Active and passive smoking: assessment issues in periodontal research
Identifieur interne : 000D20 ( Istex/Corpus ); précédent : 000D19; suivant : 000D21Active and passive smoking: assessment issues in periodontal research
Auteurs : Clemens Walter ; Elizabeth K. Kaye ; Thomas DietrichSource :
- Periodontology 2000 [ 0906-6713 ] ; 2012-02.
English descriptors
- KwdEn :
- Abstinence, Active smoking, Actual smoker, Addict behav, Arch environ health, Attachment loss, Biochemical, Biochemical measures, Biochemical validation, Biomarkers, Bogus pipeline, Carbon monoxide, Cessation, Cessation studies, Cessation trial, Chronic periodontitis, Cigarette smoking, Clin, Clin epidemiol, Clin periodontol, Clinical research, Clinical studies, Cognitive impairment, Comprehensive smoking index, Coronary heart disease, Cotinine, Cotinine levels, Current smoker, Current smokers, Dental practices, Different approaches, Different smoking characteristics, Disease associations, Disease risk, Environmental tobacco smoke, Epidemiol, Epidemiological studies, Exclusive categories, Former smokers, Fresh sidestream smoke, General practice, Health outcomes, Human services, Intensive intervention studies, Intervention studies, Involuntary smokers, Involuntary smoking, Logistic regression analysis, Lung cancer, Mainstream smoke, Mass index, Melanin pigmentation, Monoxide, Nicotine, Odds ratio, Passive smoking, Periodontal, Periodontal disease, Periodontal disease prevalence, Periodontal diseases, Periodontal outcomes, Periodontal parameters, Periodontal pathogenesis, Periodontal research, Periodontal therapy, Periodontitis, Periodontol, Pigmentation, Potential confounders, Progression, Public health, Regression model, Relative risk, Risk factor, Robust evidence, Salivary, Salivary cotinine, Salivary cotinine levels, Salivary markers, Same effect, Same population, Secondhand smoke, Several issues, Sidestream, Sidestream smoke, Smoker, Smoking, Smoking assessment, Smoking cessation, Smoking cessation program, Smoking cessation programme, Smoking effect, Smoking exposure, Smoking history, Smoking intensity, Smoking period, Smoking status, Study subjects, Systemic disease, Tobacco cessation, Tooth loss.
- Teeft :
- Abstinence, Active smoking, Actual smoker, Addict behav, Arch environ health, Attachment loss, Biochemical, Biochemical measures, Biochemical validation, Biomarkers, Bogus pipeline, Carbon monoxide, Cessation, Cessation studies, Cessation trial, Chronic periodontitis, Cigarette smoking, Clin, Clin epidemiol, Clin periodontol, Clinical research, Clinical studies, Cognitive impairment, Comprehensive smoking index, Coronary heart disease, Cotinine, Cotinine levels, Current smoker, Current smokers, Dental practices, Different approaches, Different smoking characteristics, Disease associations, Disease risk, Environmental tobacco smoke, Epidemiol, Epidemiological studies, Exclusive categories, Former smokers, Fresh sidestream smoke, General practice, Health outcomes, Human services, Intensive intervention studies, Intervention studies, Involuntary smokers, Involuntary smoking, Logistic regression analysis, Lung cancer, Mainstream smoke, Mass index, Melanin pigmentation, Monoxide, Nicotine, Odds ratio, Passive smoking, Periodontal, Periodontal disease, Periodontal disease prevalence, Periodontal diseases, Periodontal outcomes, Periodontal parameters, Periodontal pathogenesis, Periodontal research, Periodontal therapy, Periodontitis, Periodontol, Pigmentation, Potential confounders, Progression, Public health, Regression model, Relative risk, Risk factor, Robust evidence, Salivary, Salivary cotinine, Salivary cotinine levels, Salivary markers, Same effect, Same population, Secondhand smoke, Several issues, Sidestream, Sidestream smoke, Smoker, Smoking, Smoking assessment, Smoking cessation, Smoking cessation program, Smoking cessation programme, Smoking effect, Smoking exposure, Smoking history, Smoking intensity, Smoking period, Smoking status, Study subjects, Systemic disease, Tobacco cessation, Tooth loss.
Abstract
Tobacco use, particularly active cigarette smoking, is a strong modifiable risk factor for periodontitis. Our primary aim is to provide a systematic review of the impact of environmental tobacco smoke on periodontal diseases. Nine publications were identified by electronic literature search up to September 2010. Epidemiological data for a US population sample and a cohort of Japanese men suggest that environmental tobacco smoke exposure may be associated with increase periodontitis prevalence and increased risk of periodontitis progression. Secondly, several methodological issues related to cigarette smoking in clinical and epidemiological research studies are discussed. Tobacco use history is a multi‐dimensional phenomenon, with characteristics such as intensity, duration and time since cessation. Understanding these complexities of smoking history and accurate measurement of the exposure, using biomarkers and/or self‐reported data as appropriate, are of central importance in clinical and epidemiological studies on the effects of active and passive smoking. The most appropriate approach to assess smoking exposure will vary according to the purpose and design of a particular study.
Url:
DOI: 10.1111/j.1600-0757.2011.00417.x
Links to Exploration step
ISTEX:1AC77B6A22460AB6C5F364B9FDF6433C7BE6FF5CLe document en format XML
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research</term><term>Periodontal therapy</term><term>Periodontitis</term><term>Periodontol</term><term>Pigmentation</term><term>Potential confounders</term><term>Progression</term><term>Public health</term><term>Regression model</term><term>Relative risk</term><term>Risk factor</term><term>Robust evidence</term><term>Salivary</term><term>Salivary cotinine</term><term>Salivary cotinine levels</term><term>Salivary markers</term><term>Same effect</term><term>Same population</term><term>Secondhand smoke</term><term>Several issues</term><term>Sidestream</term><term>Sidestream smoke</term><term>Smoker</term><term>Smoking</term><term>Smoking assessment</term><term>Smoking cessation</term><term>Smoking cessation program</term><term>Smoking cessation programme</term><term>Smoking effect</term><term>Smoking exposure</term><term>Smoking history</term><term>Smoking intensity</term><term>Smoking period</term><term>Smoking status</term><term>Study subjects</term><term>Systemic 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Our primary aim is to provide a systematic review of the impact of environmental tobacco smoke on periodontal diseases. Nine publications were identified by electronic literature search up to September 2010. Epidemiological data for a US population sample and a cohort of Japanese men suggest that environmental tobacco smoke exposure may be associated with increase periodontitis prevalence and increased risk of periodontitis progression. Secondly, several methodological issues related to cigarette smoking in clinical and epidemiological research studies are discussed. Tobacco use history is a multi‐dimensional phenomenon, with characteristics such as intensity, duration and time since cessation. Understanding these complexities of smoking history and accurate measurement of the exposure, using biomarkers and/or self‐reported data as appropriate, are of central importance in clinical and epidemiological studies on the effects of active and passive smoking. The most appropriate approach to assess smoking exposure will vary according to the purpose and design of a particular study.</div></front></TEI><istex><corpusName>wiley</corpusName><keywords><teeft><json:string>periodontal</json:string><json:string>smoker</json:string><json:string>periodontitis</json:string><json:string>cessation</json:string><json:string>cotinine</json:string><json:string>periodontal disease</json:string><json:string>periodontol</json:string><json:string>passive smoking</json:string><json:string>clin</json:string><json:string>nicotine</json:string><json:string>smoking cessation</json:string><json:string>monoxide</json:string><json:string>sidestream</json:string><json:string>smoking</json:string><json:string>environmental tobacco smoke</json:string><json:string>biochemical measures</json:string><json:string>biomarkers</json:string><json:string>smoking intensity</json:string><json:string>smoking history</json:string><json:string>active smoking</json:string><json:string>pigmentation</json:string><json:string>epidemiol</json:string><json:string>tooth loss</json:string><json:string>carbon monoxide</json:string><json:string>sidestream smoke</json:string><json:string>cessation studies</json:string><json:string>clin periodontol</json:string><json:string>salivary</json:string><json:string>melanin pigmentation</json:string><json:string>smoking assessment</json:string><json:string>smoking status</json:string><json:string>periodontal research</json:string><json:string>current smokers</json:string><json:string>former smokers</json:string><json:string>mainstream smoke</json:string><json:string>involuntary smoking</json:string><json:string>salivary cotinine levels</json:string><json:string>clinical research</json:string><json:string>bogus pipeline</json:string><json:string>public health</json:string><json:string>cigarette smoking</json:string><json:string>abstinence</json:string><json:string>smoking exposure</json:string><json:string>involuntary smokers</json:string><json:string>biochemical validation</json:string><json:string>clin epidemiol</json:string><json:string>chronic periodontitis</json:string><json:string>same population</json:string><json:string>epidemiological studies</json:string><json:string>exclusive categories</json:string><json:string>lung cancer</json:string><json:string>regression model</json:string><json:string>smoking effect</json:string><json:string>salivary cotinine</json:string><json:string>risk factor</json:string><json:string>current smoker</json:string><json:string>periodontal parameters</json:string><json:string>biochemical</json:string><json:string>progression</json:string><json:string>actual smoker</json:string><json:string>human services</json:string><json:string>robust evidence</json:string><json:string>periodontal therapy</json:string><json:string>cessation trial</json:string><json:string>fresh sidestream smoke</json:string><json:string>study subjects</json:string><json:string>smoking cessation program</json:string><json:string>dental practices</json:string><json:string>odds ratio</json:string><json:string>several issues</json:string><json:string>clinical studies</json:string><json:string>cotinine levels</json:string><json:string>intensive intervention studies</json:string><json:string>intervention studies</json:string><json:string>tobacco cessation</json:string><json:string>different approaches</json:string><json:string>cognitive impairment</json:string><json:string>different smoking characteristics</json:string><json:string>secondhand smoke</json:string><json:string>mass index</json:string><json:string>same effect</json:string><json:string>relative risk</json:string><json:string>comprehensive smoking index</json:string><json:string>disease risk</json:string><json:string>periodontal diseases</json:string><json:string>smoking period</json:string><json:string>coronary heart disease</json:string><json:string>arch environ health</json:string><json:string>periodontal outcomes</json:string><json:string>general practice</json:string><json:string>smoking cessation programme</json:string><json:string>systemic disease</json:string><json:string>periodontal disease prevalence</json:string><json:string>periodontal pathogenesis</json:string><json:string>health outcomes</json:string><json:string>potential confounders</json:string><json:string>disease associations</json:string><json:string>logistic regression analysis</json:string><json:string>addict behav</json:string><json:string>salivary markers</json:string><json:string>attachment loss</json:string></teeft></keywords><author><json:item><name>Clemens Walter</name></json:item><json:item><name>Elizabeth K. Kaye</name></json:item><json:item><name>Thomas Dietrich</name></json:item></author><articleId><json:string>PRD417</json:string></articleId><arkIstex>ark:/67375/WNG-S73L1Z7V-P</arkIstex><language><json:string>eng</json:string></language><originalGenre><json:string>article</json:string></originalGenre><abstract>Tobacco use, particularly active cigarette smoking, is a strong modifiable risk factor for periodontitis. Our primary aim is to provide a systematic review of the impact of environmental tobacco smoke on periodontal diseases. Nine publications were identified by electronic literature search up to September 2010. Epidemiological data for a US population sample and a cohort of Japanese men suggest that environmental tobacco smoke exposure may be associated with increase periodontitis prevalence and increased risk of periodontitis progression. Secondly, several methodological issues related to cigarette smoking in clinical and epidemiological research studies are discussed. Tobacco use history is a multi‐dimensional phenomenon, with characteristics such as intensity, duration and time since cessation. Understanding these complexities of smoking history and accurate measurement of the exposure, using biomarkers and/or self‐reported data as appropriate, are of central importance in clinical and epidemiological studies on the effects of active and passive smoking. The most appropriate approach to assess smoking exposure will vary according to the purpose and design of a particular study.</abstract><qualityIndicators><score>8.992</score><pdfWordCount>5844</pdfWordCount><pdfCharCount>37386</pdfCharCount><pdfVersion>1.3</pdfVersion><pdfPageCount>9</pdfPageCount><pdfPageSize>595.276 x 782.362 pts</pdfPageSize><refBibsNative>true</refBibsNative><abstractWordCount>166</abstractWordCount><abstractCharCount>1200</abstractCharCount><keywordCount>0</keywordCount></qualityIndicators><title>Active and passive smoking: assessment issues in periodontal research</title><pmid><json:string>22133368</json:string></pmid><genre><json:string>article</json:string></genre><host><title>Periodontology 2000</title><language><json:string>unknown</json:string></language><doi><json:string>10.1111/(ISSN)1600-0757</json:string></doi><issn><json:string>0906-6713</json:string></issn><eissn><json:string>1600-0757</json:string></eissn><publisherId><json:string>PRD</json:string></publisherId><volume>58</volume><issue>1</issue><pages><first>84</first><last>92</last><total>9</total></pages><genre><json:string>journal</json:string></genre></host><namedEntities><unitex><date><json:string>2012</json:string><json:string>2010</json:string><json:string>1980s</json:string></date><geogName></geogName><orgName><json:string>US Department of Health and Human Services</json:string></orgName><orgName_funder></orgName_funder><orgName_provider></orgName_provider><persName><json:string>Self</json:string></persName><placeName><json:string>UK</json:string></placeName><ref_url></ref_url><ref_bibl><json:string>Arbes et al.</json:string><json:string>Walter et al.</json:string><json:string>Yamamoto et al.</json:string><json:string>Erdemir et al.</json:string><json:string>Nishida et al.</json:string></ref_bibl><bibl></bibl></unitex></namedEntities><ark><json:string>ark:/67375/WNG-S73L1Z7V-P</json:string></ark><categories><wos><json:string>1 - 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Our primary aim is to provide a systematic review of the impact of environmental tobacco smoke on periodontal diseases. Nine publications were identified by electronic literature search up to September 2010. Epidemiological data for a US population sample and a cohort of Japanese men suggest that environmental tobacco smoke exposure may be associated with increase periodontitis prevalence and increased risk of periodontitis progression. Secondly, several methodological issues related to cigarette smoking in clinical and epidemiological research studies are discussed. Tobacco use history is a multi‐dimensional phenomenon, with characteristics such as intensity, duration and time since cessation. Understanding these complexities of smoking history and accurate measurement of the exposure, using biomarkers and/or self‐reported data as appropriate, are of central importance in clinical and epidemiological studies on the effects of active and passive smoking. The most appropriate approach to assess smoking exposure will vary according to the purpose and design of a particular study.</p></abstract><textClass><keywords rend="tocHeading1"><term>Original Articles</term></keywords></textClass><langUsage><language ident="en"></language></langUsage></profileDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/1AC77B6A22460AB6C5F364B9FDF6433C7BE6FF5C/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1600-0757</doi><issn type="print">0906-6713</issn><issn type="electronic">1600-0757</issn><idGroup><id type="product" value="PRD"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PERIODONTOLOGY 2000">Periodontology 2000</title></titleGroup></publicationMeta><publicationMeta level="part" position="02101"><doi origin="wiley">10.1111/prd.2011.58.issue-1</doi><numberingGroup><numbering type="journalVolume" number="58">58</numbering><numbering type="journalIssue" number="1">1</numbering></numberingGroup><coverDate startDate="2012-02">February 2012</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="6" status="forIssue"><doi origin="wiley">10.1111/j.1600-0757.2011.00417.x</doi><idGroup><id type="unit" value="PRD417"></id></idGroup><countGroup><count type="pageTotal" number="9"></count></countGroup><titleGroup><title type="tocHeading1">Original Articles</title></titleGroup><copyright>© 2012 John Wiley & Sons A/S</copyright><eventGroup><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:3.0.1 mode:FullText" date="2011-12-01"></event><event type="firstOnline" date="2011-12-01"></event><event type="publishedOnlineFinalForm" date="2011-12-01"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-07"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.3.4 mode:FullText" date="2015-02-25"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="84">84</numbering><numbering type="pageLast" number="92">92</numbering></numberingGroup><linkGroup><link type="toTypesetVersion" href="file:PRD.PRD417.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Accepted: 20 March 2011</unparsedEditorialHistory><countGroup><count type="figureTotal" number="0"></count><count type="tableTotal" number="1"></count></countGroup><titleGroup><title type="main">Active and passive smoking: assessment issues in periodontal research</title><title type="shortAuthors"><i>Walter et al.</i></title><title type="short"><i>Smoking assessment in periodontal research</i></title></titleGroup><creators><creator creatorRole="author" xml:id="cr1"><personName><givenNames>Clemens</givenNames><familyName>Walter</familyName></personName></creator><creator creatorRole="author" xml:id="cr2"><personName><givenNames>Elizabeth K. </givenNames><familyName>Kaye</familyName></personName></creator><creator creatorRole="author" xml:id="cr3"><personName><givenNames>Thomas</givenNames><familyName>Dietrich</familyName></personName></creator></creators><abstractGroup><abstract type="main" xml:lang="en"><title type="main">Abstract</title><p>Tobacco use, particularly active cigarette smoking, is a strong modifiable risk factor for periodontitis. 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