Active and passive smoking: assessment issues in periodontal research
Identifieur interne : 000D20 ( Istex/Corpus ); précédent : 000D19; suivant : 000D21Active and passive smoking: assessment issues in periodontal research
Auteurs : Clemens Walter ; Elizabeth K. Kaye ; Thomas DietrichSource :
- Periodontology 2000 [ 0906-6713 ] ; 2012-02.
English descriptors
- KwdEn :
- Abstinence, Active smoking, Actual smoker, Addict behav, Arch environ health, Attachment loss, Biochemical, Biochemical measures, Biochemical validation, Biomarkers, Bogus pipeline, Carbon monoxide, Cessation, Cessation studies, Cessation trial, Chronic periodontitis, Cigarette smoking, Clin, Clin epidemiol, Clin periodontol, Clinical research, Clinical studies, Cognitive impairment, Comprehensive smoking index, Coronary heart disease, Cotinine, Cotinine levels, Current smoker, Current smokers, Dental practices, Different approaches, Different smoking characteristics, Disease associations, Disease risk, Environmental tobacco smoke, Epidemiol, Epidemiological studies, Exclusive categories, Former smokers, Fresh sidestream smoke, General practice, Health outcomes, Human services, Intensive intervention studies, Intervention studies, Involuntary smokers, Involuntary smoking, Logistic regression analysis, Lung cancer, Mainstream smoke, Mass index, Melanin pigmentation, Monoxide, Nicotine, Odds ratio, Passive smoking, Periodontal, Periodontal disease, Periodontal disease prevalence, Periodontal diseases, Periodontal outcomes, Periodontal parameters, Periodontal pathogenesis, Periodontal research, Periodontal therapy, Periodontitis, Periodontol, Pigmentation, Potential confounders, Progression, Public health, Regression model, Relative risk, Risk factor, Robust evidence, Salivary, Salivary cotinine, Salivary cotinine levels, Salivary markers, Same effect, Same population, Secondhand smoke, Several issues, Sidestream, Sidestream smoke, Smoker, Smoking, Smoking assessment, Smoking cessation, Smoking cessation program, Smoking cessation programme, Smoking effect, Smoking exposure, Smoking history, Smoking intensity, Smoking period, Smoking status, Study subjects, Systemic disease, Tobacco cessation, Tooth loss.
- Teeft :
- Abstinence, Active smoking, Actual smoker, Addict behav, Arch environ health, Attachment loss, Biochemical, Biochemical measures, Biochemical validation, Biomarkers, Bogus pipeline, Carbon monoxide, Cessation, Cessation studies, Cessation trial, Chronic periodontitis, Cigarette smoking, Clin, Clin epidemiol, Clin periodontol, Clinical research, Clinical studies, Cognitive impairment, Comprehensive smoking index, Coronary heart disease, Cotinine, Cotinine levels, Current smoker, Current smokers, Dental practices, Different approaches, Different smoking characteristics, Disease associations, Disease risk, Environmental tobacco smoke, Epidemiol, Epidemiological studies, Exclusive categories, Former smokers, Fresh sidestream smoke, General practice, Health outcomes, Human services, Intensive intervention studies, Intervention studies, Involuntary smokers, Involuntary smoking, Logistic regression analysis, Lung cancer, Mainstream smoke, Mass index, Melanin pigmentation, Monoxide, Nicotine, Odds ratio, Passive smoking, Periodontal, Periodontal disease, Periodontal disease prevalence, Periodontal diseases, Periodontal outcomes, Periodontal parameters, Periodontal pathogenesis, Periodontal research, Periodontal therapy, Periodontitis, Periodontol, Pigmentation, Potential confounders, Progression, Public health, Regression model, Relative risk, Risk factor, Robust evidence, Salivary, Salivary cotinine, Salivary cotinine levels, Salivary markers, Same effect, Same population, Secondhand smoke, Several issues, Sidestream, Sidestream smoke, Smoker, Smoking, Smoking assessment, Smoking cessation, Smoking cessation program, Smoking cessation programme, Smoking effect, Smoking exposure, Smoking history, Smoking intensity, Smoking period, Smoking status, Study subjects, Systemic disease, Tobacco cessation, Tooth loss.
Abstract
Tobacco use, particularly active cigarette smoking, is a strong modifiable risk factor for periodontitis. Our primary aim is to provide a systematic review of the impact of environmental tobacco smoke on periodontal diseases. Nine publications were identified by electronic literature search up to September 2010. Epidemiological data for a US population sample and a cohort of Japanese men suggest that environmental tobacco smoke exposure may be associated with increase periodontitis prevalence and increased risk of periodontitis progression. Secondly, several methodological issues related to cigarette smoking in clinical and epidemiological research studies are discussed. Tobacco use history is a multi‐dimensional phenomenon, with characteristics such as intensity, duration and time since cessation. Understanding these complexities of smoking history and accurate measurement of the exposure, using biomarkers and/or self‐reported data as appropriate, are of central importance in clinical and epidemiological studies on the effects of active and passive smoking. The most appropriate approach to assess smoking exposure will vary according to the purpose and design of a particular study.
Url:
DOI: 10.1111/j.1600-0757.2011.00417.x
Links to Exploration step
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<front><div type="abstract" xml:lang="en">Tobacco use, particularly active cigarette smoking, is a strong modifiable risk factor for periodontitis. Our primary aim is to provide a systematic review of the impact of environmental tobacco smoke on periodontal diseases. Nine publications were identified by electronic literature search up to September 2010. Epidemiological data for a US population sample and a cohort of Japanese men suggest that environmental tobacco smoke exposure may be associated with increase periodontitis prevalence and increased risk of periodontitis progression. Secondly, several methodological issues related to cigarette smoking in clinical and epidemiological research studies are discussed. Tobacco use history is a multi‐dimensional phenomenon, with characteristics such as intensity, duration and time since cessation. Understanding these complexities of smoking history and accurate measurement of the exposure, using biomarkers and/or self‐reported data as appropriate, are of central importance in clinical and epidemiological studies on the effects of active and passive smoking. The most appropriate approach to assess smoking exposure will vary according to the purpose and design of a particular study.</div>
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<abstract>Tobacco use, particularly active cigarette smoking, is a strong modifiable risk factor for periodontitis. Our primary aim is to provide a systematic review of the impact of environmental tobacco smoke on periodontal diseases. Nine publications were identified by electronic literature search up to September 2010. Epidemiological data for a US population sample and a cohort of Japanese men suggest that environmental tobacco smoke exposure may be associated with increase periodontitis prevalence and increased risk of periodontitis progression. Secondly, several methodological issues related to cigarette smoking in clinical and epidemiological research studies are discussed. Tobacco use history is a multi‐dimensional phenomenon, with characteristics such as intensity, duration and time since cessation. Understanding these complexities of smoking history and accurate measurement of the exposure, using biomarkers and/or self‐reported data as appropriate, are of central importance in clinical and epidemiological studies on the effects of active and passive smoking. The most appropriate approach to assess smoking exposure will vary according to the purpose and design of a particular study.</abstract>
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