Amebiasis.
Identifieur interne : 000469 ( PubMed/Checkpoint ); précédent : 000468; suivant : 000470Amebiasis.
Auteurs : Badalamenti [États-Unis] ; Jameson ; ReddySource :
- Current treatment options in gastroenterology [ 1092-8472 ] ; 1999.
Abstract
More than 80% of cases of amebic liver abscess can be managed with a 14-day course of intravenous or oral metronidazole. In cases of suspected amebic liver abscess, treatment should be started before diagnostic confirmation. If no clinical improvement is evident by 72 to 96 hours, treatment should be changed to dehydroemetine and chloroquine. Invasive treatment is necessary only in patients in whom medical treatment fails within 5 days or in whom signs of clinically severe disease are present. A 10-day course with a luminal agent such as paromomycin to eliminate intestinal cysts, which are resistant to imidazoles, should always follow treatment of the liver abscess. Percutaneous catheter drainage is indicated in patients with impending rupture, with a lesion 6 cm or more in diameter, with an abscess located in the left lobe or high in the dome of the right lobe, or in whom medical treatment fails. Although sympathetic pleural effusion is not an indication for drainage, direct pulmonary involvement or spread to pleural or lung tissues requires drainage. Intraperitoneal rupture and peritonitis necessitate open surgical drainage. Only a small minority of amebic liver abscesses are secondarily infected by other organisms. Because relapses are possible, feces should be checked for cysts monthly for several months after therapy.
DOI: 10.1007/s11938-999-0036-z
PubMed: 11096579
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In cases of suspected amebic liver abscess, treatment should be started before diagnostic confirmation. If no clinical improvement is evident by 72 to 96 hours, treatment should be changed to dehydroemetine and chloroquine. Invasive treatment is necessary only in patients in whom medical treatment fails within 5 days or in whom signs of clinically severe disease are present. A 10-day course with a luminal agent such as paromomycin to eliminate intestinal cysts, which are resistant to imidazoles, should always follow treatment of the liver abscess. Percutaneous catheter drainage is indicated in patients with impending rupture, with a lesion 6 cm or more in diameter, with an abscess located in the left lobe or high in the dome of the right lobe, or in whom medical treatment fails. Although sympathetic pleural effusion is not an indication for drainage, direct pulmonary involvement or spread to pleural or lung tissues requires drainage. Intraperitoneal rupture and peritonitis necessitate open surgical drainage. Only a small minority of amebic liver abscesses are secondarily infected by other organisms. Because relapses are possible, feces should be checked for cysts monthly for several months after therapy.</div></front></TEI><pubmed><MedlineCitation Status="PubMed-not-MEDLINE" Owner="NLM"><PMID Version="1">11096579</PMID><DateRevised><Year>2019</Year><Month>11</Month><Day>20</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">1092-8472</ISSN><JournalIssue CitedMedium="Print"><Volume>2</Volume><Issue>2</Issue><PubDate><Year>1999</Year><Month>Apr</Month></PubDate></JournalIssue><Title>Current treatment options in gastroenterology</Title><ISOAbbreviation>Curr Treat Options Gastroenterol</ISOAbbreviation></Journal><ArticleTitle>Amebiasis.</ArticleTitle><Pagination><MedlinePgn>97-103</MedlinePgn></Pagination><Abstract><AbstractText>More than 80% of cases of amebic liver abscess can be managed with a 14-day course of intravenous or oral metronidazole. In cases of suspected amebic liver abscess, treatment should be started before diagnostic confirmation. If no clinical improvement is evident by 72 to 96 hours, treatment should be changed to dehydroemetine and chloroquine. Invasive treatment is necessary only in patients in whom medical treatment fails within 5 days or in whom signs of clinically severe disease are present. A 10-day course with a luminal agent such as paromomycin to eliminate intestinal cysts, which are resistant to imidazoles, should always follow treatment of the liver abscess. Percutaneous catheter drainage is indicated in patients with impending rupture, with a lesion 6 cm or more in diameter, with an abscess located in the left lobe or high in the dome of the right lobe, or in whom medical treatment fails. Although sympathetic pleural effusion is not an indication for drainage, direct pulmonary involvement or spread to pleural or lung tissues requires drainage. Intraperitoneal rupture and peritonitis necessitate open surgical drainage. Only a small minority of amebic liver abscesses are secondarily infected by other organisms. Because relapses are possible, feces should be checked for cysts monthly for several months after therapy.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Badalamenti</LastName><Initials>S</Initials><AffiliationInfo><Affiliation>University of Miami School of Medicine, Center for Liver Diseases, 1500 NW 12th Avenue, Suite 1101, Miami, FL 33136.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Jameson</LastName><Initials>JE</Initials></Author><Author ValidYN="Y"><LastName>Reddy</LastName><Initials>KR</Initials></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>United States</Country><MedlineTA>Curr Treat Options Gastroenterol</MedlineTA><NlmUniqueID>9815941</NlmUniqueID><ISSNLinking>1092-8472</ISSNLinking></MedlineJournalInfo></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>2000</Year><Month>11</Month><Day>30</Day><Hour>0</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2000</Year><Month>11</Month><Day>30</Day><Hour>0</Hour><Minute>1</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>2000</Year><Month>11</Month><Day>30</Day><Hour>0</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">11096579</ArticleId><ArticleId IdType="doi">10.1007/s11938-999-0036-z</ArticleId></ArticleIdList><ReferenceList><Reference><Citation>Rev Infect Dis. 1985 Mar-Apr;7(2):171-9</Citation><ArticleIdList><ArticleId IdType="pubmed">4001714</ArticleId></ArticleIdList></Reference><Reference><Citation>Gastroenterol Clin North Am. 1996 Sep;25(3):471-92</Citation><ArticleIdList><ArticleId IdType="pubmed">8863036</ArticleId></ArticleIdList></Reference><Reference><Citation>Am J Surg. 1989 Jul;158(1):58-62</Citation><ArticleIdList><ArticleId IdType="pubmed">2662790</ArticleId></ArticleIdList></Reference><Reference><Citation>Am J Gastroenterol. 1996 Dec;91(12):2584-8</Citation><ArticleIdList><ArticleId IdType="pubmed">8946991</ArticleId></ArticleIdList></Reference><Reference><Citation>JAMA. 1964 May 11;188:519-21</Citation><ArticleIdList><ArticleId IdType="pubmed">14159068</ArticleId></ArticleIdList></Reference><Reference><Citation>AJR Am J Roentgenol. 1998 Apr;170(4):1035-9</Citation><ArticleIdList><ArticleId IdType="pubmed">9530055</ArticleId></ArticleIdList></Reference><Reference><Citation>South Med J. 1994 Oct;87(10):985-90</Citation><ArticleIdList><ArticleId IdType="pubmed">7939926</ArticleId></ArticleIdList></Reference><Reference><Citation>Dig Dis Sci. 1992 Feb;37(2):240-7</Citation><ArticleIdList><ArticleId IdType="pubmed">1735342</ArticleId></ArticleIdList></Reference><Reference><Citation>Postgrad Med. 1988 Jun;83(8):65-8, 72</Citation><ArticleIdList><ArticleId IdType="pubmed">2897682</ArticleId></ArticleIdList></Reference><Reference><Citation>Infect Immun. 1994 Apr;62(4):1166-70</Citation><ArticleIdList><ArticleId IdType="pubmed">8132322</ArticleId></ArticleIdList></Reference><Reference><Citation>Radiology. 1987 Dec;165(3):805-7</Citation><ArticleIdList><ArticleId IdType="pubmed">3317505</ArticleId></ArticleIdList></Reference><Reference><Citation>World J Surg. 1999 Jan;23(1):102-6</Citation><ArticleIdList><ArticleId IdType="pubmed">9841772</ArticleId></ArticleIdList></Reference><Reference><Citation>Surg Clin North Am. 1989 Apr;69(2):259-71</Citation><ArticleIdList><ArticleId IdType="pubmed">2648615</ArticleId></ArticleIdList></Reference><Reference><Citation>South Med J. 1998 Sep;91(9):829-37</Citation><ArticleIdList><ArticleId IdType="pubmed">9743053</ArticleId></ArticleIdList></Reference><Reference><Citation>Clin Infect Dis. 1995 Jun;20(6):1453-64; quiz 1465-6</Citation><ArticleIdList><ArticleId IdType="pubmed">7548493</ArticleId></ArticleIdList></Reference></ReferenceList></PubmedData></pubmed><affiliations><list><country><li>États-Unis</li></country><region><li>Floride</li></region></list><tree><noCountry><name sortKey="Jameson" sort="Jameson" uniqKey="Jameson" last="Jameson">Jameson</name><name sortKey="Reddy" sort="Reddy" uniqKey="Reddy" last="Reddy">Reddy</name></noCountry><country name="États-Unis"><region name="Floride"><name sortKey="Badalamenti" sort="Badalamenti" uniqKey="Badalamenti" last="Badalamenti">Badalamenti</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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