Toll‐like receptors, endogenous ligands, and systemic autoimmune disease
Identifieur interne : 001E25 ( Main/Exploration ); précédent : 001E24; suivant : 001E26Toll‐like receptors, endogenous ligands, and systemic autoimmune disease
Auteurs : Ian R. Rifkin [États-Unis] ; Elizabeth A. Leadbetter [États-Unis] ; Liliana Busconi [États-Unis] ; Gregory Viglianti [États-Unis] ; Ann Marshak-Rothstein [États-Unis]Source :
- Immunological Reviews [ 0105-2896 ] ; 2005-04.
English descriptors
- Teeft :
- Acad, Activation, Adaptive, Apoptosis, Apoptotic, Apoptotic cells, Atherosclerosis, Autoantibody, Autoantigen, Autoantigens, Autoimmune, Autoimmune disease, Autoimmune diseases, Autoimmune mice, Autoimmunity, Autoreactive, Biol, Biol chem, Cell death, Cell debris, Cell types, Chem, Chromatin, Clin, Culture fluids, Dendritic, Dendritic cells, Disease table, Dnase, Endogenous, Endogenous ligands, Erythematosus, Fcgr, Fragment, High affinity, Igg2a, Igg2a mabs, Immune, Immune complexes, Immune response, Immune system, Immunoglobulin, Immunol, Immunological, Immunological reviews, Inhibitor, Innate, Kinase, Ligand, Lipid, Lipoprotein, Lupus, Lymphocyte, Mabs, Macrophage, Mouse, Murine, Myeloid differentiation factor, Natl, Other tlrs, Oxidized, Oxldl, Pathway, Proc, Proc natl acad, Reactive oxygen species, Receptor, Relevant receptor, Rheumatoid, Rheumatoid factor, Rifkin, Scavenger, Scavenger receptors, Shlomchik, Stimulatory, Stimulatory capacity, Systemic, Systemic lupus erythematosus, Tlr2, Tlr4, Tlr9, Tlrs, Tolerance induction, Transgenic, Transgenic mice.
Url:
DOI: 10.1111/j.0105-2896.2005.00239.x
Affiliations:
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Le document en format XML
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<profileDesc><textClass><keywords scheme="Teeft" xml:lang="en"><term>Acad</term>
<term>Activation</term>
<term>Adaptive</term>
<term>Apoptosis</term>
<term>Apoptotic</term>
<term>Apoptotic cells</term>
<term>Atherosclerosis</term>
<term>Autoantibody</term>
<term>Autoantigen</term>
<term>Autoantigens</term>
<term>Autoimmune</term>
<term>Autoimmune disease</term>
<term>Autoimmune diseases</term>
<term>Autoimmune mice</term>
<term>Autoimmunity</term>
<term>Autoreactive</term>
<term>Biol</term>
<term>Biol chem</term>
<term>Cell death</term>
<term>Cell debris</term>
<term>Cell types</term>
<term>Chem</term>
<term>Chromatin</term>
<term>Clin</term>
<term>Culture fluids</term>
<term>Dendritic</term>
<term>Dendritic cells</term>
<term>Disease table</term>
<term>Dnase</term>
<term>Endogenous</term>
<term>Endogenous ligands</term>
<term>Erythematosus</term>
<term>Fcgr</term>
<term>Fragment</term>
<term>High affinity</term>
<term>Igg2a</term>
<term>Igg2a mabs</term>
<term>Immune</term>
<term>Immune complexes</term>
<term>Immune response</term>
<term>Immune system</term>
<term>Immunoglobulin</term>
<term>Immunol</term>
<term>Immunological</term>
<term>Immunological reviews</term>
<term>Inhibitor</term>
<term>Innate</term>
<term>Kinase</term>
<term>Ligand</term>
<term>Lipid</term>
<term>Lipoprotein</term>
<term>Lupus</term>
<term>Lymphocyte</term>
<term>Mabs</term>
<term>Macrophage</term>
<term>Mouse</term>
<term>Murine</term>
<term>Myeloid differentiation factor</term>
<term>Natl</term>
<term>Other tlrs</term>
<term>Oxidized</term>
<term>Oxldl</term>
<term>Pathway</term>
<term>Proc</term>
<term>Proc natl acad</term>
<term>Reactive oxygen species</term>
<term>Receptor</term>
<term>Relevant receptor</term>
<term>Rheumatoid</term>
<term>Rheumatoid factor</term>
<term>Rifkin</term>
<term>Scavenger</term>
<term>Scavenger receptors</term>
<term>Shlomchik</term>
<term>Stimulatory</term>
<term>Stimulatory capacity</term>
<term>Systemic</term>
<term>Systemic lupus erythematosus</term>
<term>Tlr2</term>
<term>Tlr4</term>
<term>Tlr9</term>
<term>Tlrs</term>
<term>Tolerance induction</term>
<term>Transgenic</term>
<term>Transgenic mice</term>
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