Factors affecting amylase output by the isolated perfused liver
Identifieur interne : 003240 ( Istex/Corpus ); précédent : 003239; suivant : 003241Factors affecting amylase output by the isolated perfused liver
Auteurs : Robert L. Mcgeachin ; Betty Ann Potter ; Agamemnon DespopoulosSource :
- Archives of Biochemistry and Biophysics [ 0003-9861 ] ; 1962.
Abstract
During the perfusion of isolated rat livers with either diluted whole rat blood or washed red blood cells in a reconstituted medium, the amylase content of the plasma increased two- to threefold, and it was determined that there was a net gain in the amylase of the total system. Amylase was found in the bile excreted from these livers during the perfusion. Administration of N-nitrosodimethylamine to rats produced damage to their livers. Amylase output and bile formation by these damaged livers were below normal as were the amylase contents of the livers themselves. Lack of oxygenation of the perfusing blood resulted in lowered amylase output and bile formation by the perfused livers. No significant effects of the hormones, glucagon and insulin, on amylase production were noted. Epinephrine in large amounts inhibited amylase output probably because of its effect on blood flow through the liver. Amylase was found to be uniformly distributed throughout all lobes of the rat liver.
Url:
DOI: 10.1016/0003-9861(62)90149-2
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Mcgeachin</name><affiliation><mods:affiliation>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</mods:affiliation></affiliation></author><author><name sortKey="Potter, Betty Ann" sort="Potter, Betty Ann" uniqKey="Potter B" first="Betty Ann" last="Potter">Betty Ann Potter</name><affiliation><mods:affiliation>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</mods:affiliation></affiliation></author><author><name sortKey="Despopoulos, Agamemnon" sort="Despopoulos, Agamemnon" uniqKey="Despopoulos A" first="Agamemnon" last="Despopoulos">Agamemnon Despopoulos</name><affiliation><mods:affiliation>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Archives of Biochemistry and Biophysics</title><title level="j" type="abbrev">YABBI</title><idno type="ISSN">0003-9861</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1962">1962</date><biblScope unit="volume">98</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="89">89</biblScope><biblScope unit="page" to="94">94</biblScope></imprint><idno type="ISSN">0003-9861</idno></series><idno type="istex">8D9B1A7A22E1A9D4897351739CE46AB37A6636E7</idno><idno type="DOI">10.1016/0003-9861(62)90149-2</idno><idno type="PII">0003-9861(62)90149-2</idno><idno type="ArticleID">62901492</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0003-9861</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">During the perfusion of isolated rat livers with either diluted whole rat blood or washed red blood cells in a reconstituted medium, the amylase content of the plasma increased two- to threefold, and it was determined that there was a net gain in the amylase of the total system. Amylase was found in the bile excreted from these livers during the perfusion. Administration of N-nitrosodimethylamine to rats produced damage to their livers. Amylase output and bile formation by these damaged livers were below normal as were the amylase contents of the livers themselves. Lack of oxygenation of the perfusing blood resulted in lowered amylase output and bile formation by the perfused livers. No significant effects of the hormones, glucagon and insulin, on amylase production were noted. Epinephrine in large amounts inhibited amylase output probably because of its effect on blood flow through the liver. Amylase was found to be uniformly distributed throughout all lobes of the rat liver.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>Robert L. McGeachin</name><affiliations><json:string>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</json:string></affiliations></json:item><json:item><name>Betty Ann Potter</name><affiliations><json:string>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</json:string></affiliations></json:item><json:item><name>Agamemnon Despopoulos</name><affiliations><json:string>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</json:string></affiliations></json:item></author><articleId><json:string>62901492</json:string></articleId><language><json:string>eng</json:string></language><abstract>During the perfusion of isolated rat livers with either diluted whole rat blood or washed red blood cells in a reconstituted medium, the amylase content of the plasma increased two- to threefold, and it was determined that there was a net gain in the amylase of the total system. Amylase was found in the bile excreted from these livers during the perfusion. Administration of N-nitrosodimethylamine to rats produced damage to their livers. Amylase output and bile formation by these damaged livers were below normal as were the amylase contents of the livers themselves. Lack of oxygenation of the perfusing blood resulted in lowered amylase output and bile formation by the perfused livers. No significant effects of the hormones, glucagon and insulin, on amylase production were noted. Epinephrine in large amounts inhibited amylase output probably because of its effect on blood flow through the liver. 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Please direct requests for reprints to the first author.</note></notesStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Factors affecting amylase output by the isolated perfused liver</title><author><persName><forename type="first">Robert L.</forename><surname>McGeachin</surname></persName><affiliation>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</affiliation></author><author><persName><forename type="first">Betty Ann</forename><surname>Potter</surname></persName><affiliation>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</affiliation></author><author><persName><forename type="first">Agamemnon</forename><surname>Despopoulos</surname></persName><affiliation>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</affiliation></author></analytic><monogr><title level="j">Archives of Biochemistry and Biophysics</title><title level="j" type="abbrev">YABBI</title><idno type="pISSN">0003-9861</idno><idno type="PII">S0003-9861(00)X0745-X</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1962"></date><biblScope unit="volume">98</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="89">89</biblScope><biblScope unit="page" to="94">94</biblScope></imprint></monogr><idno type="istex">8D9B1A7A22E1A9D4897351739CE46AB37A6636E7</idno><idno type="DOI">10.1016/0003-9861(62)90149-2</idno><idno type="PII">0003-9861(62)90149-2</idno><idno type="ArticleID">62901492</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1962</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>During the perfusion of isolated rat livers with either diluted whole rat blood or washed red blood cells in a reconstituted medium, the amylase content of the plasma increased two- to threefold, and it was determined that there was a net gain in the amylase of the total system. Amylase was found in the bile excreted from these livers during the perfusion. Administration of N-nitrosodimethylamine to rats produced damage to their livers. Amylase output and bile formation by these damaged livers were below normal as were the amylase contents of the livers themselves. Lack of oxygenation of the perfusing blood resulted in lowered amylase output and bile formation by the perfused livers. No significant effects of the hormones, glucagon and insulin, on amylase production were noted. Epinephrine in large amounts inhibited amylase output probably because of its effect on blood flow through the liver. Amylase was found to be uniformly distributed throughout all lobes of the rat liver.</p></abstract></profileDesc><revisionDesc><change when="1962-02-08">Received</change><change when="1962">Published</change></revisionDesc></teiHeader></istex:fulltextTEI></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier, elements deleted: tail"><istex:xmlDeclaration>version="1.0" encoding="UTF-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 4.5.2//EN//XML" URI="art452.dtd" name="istex:docType"></istex:docType><istex:document><converted-article version="4.5.2" docsubtype="fla" xml:lang="en"><item-info><jid>YABBI</jid><aid>62901492</aid><ce:pii>0003-9861(62)90149-2</ce:pii><ce:doi>10.1016/0003-9861(62)90149-2</ce:doi><ce:copyright type="unknown" year="1962"></ce:copyright></item-info><head><ce:article-footnote><ce:label>☆</ce:label><ce:note-para>This work was partially supported by grants (A-2610 C1, 2, 3) from the National Institute for Arthritis and Metabolic Diseases of the National Institutes of Health and from the Owensboro-Daviess County Heart Association. Please direct requests for reprints to the first author.</ce:note-para></ce:article-footnote><ce:title>Factors affecting amylase output by the isolated perfused liver</ce:title><ce:author-group><ce:author><ce:given-name>Robert L.</ce:given-name><ce:surname>McGeachin</ce:surname></ce:author><ce:author><ce:given-name>Betty Ann</ce:given-name><ce:surname>Potter</ce:surname></ce:author><ce:author><ce:given-name>Agamemnon</ce:given-name><ce:surname>Despopoulos</ce:surname></ce:author><ce:affiliation><ce:textfn>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</ce:textfn></ce:affiliation></ce:author-group><ce:date-received day="8" month="2" year="1962"></ce:date-received><ce:abstract class="author"><ce:section-title>Abstract</ce:section-title><ce:abstract-sec><ce:simple-para view="all" id="simple-para.0010">During the perfusion of isolated rat livers with either diluted whole rat blood or washed red blood cells in a reconstituted medium, the amylase content of the plasma increased two- to threefold, and it was determined that there was a net gain in the amylase of the total system. Amylase was found in the bile excreted from these livers during the perfusion. Administration of <ce:italic>N</ce:italic>-nitrosodimethylamine to rats produced damage to their livers. Amylase output and bile formation by these damaged livers were below normal as were the amylase contents of the livers themselves. Lack of oxygenation of the perfusing blood resulted in lowered amylase output and bile formation by the perfused livers. No significant effects of the hormones, glucagon and insulin, on amylase production were noted. Epinephrine in large amounts inhibited amylase output probably because of its effect on blood flow through the liver. Amylase was found to be uniformly distributed throughout all lobes of the rat liver.</ce:simple-para></ce:abstract-sec></ce:abstract></head></converted-article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Factors affecting amylase output by the isolated perfused liver</title></titleInfo><titleInfo type="alternative" lang="en" contentType="CDATA"><title>Factors affecting amylase output by the isolated perfused liver</title></titleInfo><name type="personal"><namePart type="given">Robert L.</namePart><namePart type="family">McGeachin</namePart><affiliation>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Betty Ann</namePart><namePart type="family">Potter</namePart><affiliation>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Agamemnon</namePart><namePart type="family">Despopoulos</namePart><affiliation>From the Departments of Biochemistry and Pharmacology, University of Louisville School of Medicine, Louisville, Kentucky, USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="Full-length article"></genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1962</dateIssued><dateCaptured encoding="w3cdtf">1962-02-08</dateCaptured><copyrightDate encoding="w3cdtf">1962</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">During the perfusion of isolated rat livers with either diluted whole rat blood or washed red blood cells in a reconstituted medium, the amylase content of the plasma increased two- to threefold, and it was determined that there was a net gain in the amylase of the total system. Amylase was found in the bile excreted from these livers during the perfusion. Administration of N-nitrosodimethylamine to rats produced damage to their livers. Amylase output and bile formation by these damaged livers were below normal as were the amylase contents of the livers themselves. Lack of oxygenation of the perfusing blood resulted in lowered amylase output and bile formation by the perfused livers. No significant effects of the hormones, glucagon and insulin, on amylase production were noted. Epinephrine in large amounts inhibited amylase output probably because of its effect on blood flow through the liver. Amylase was found to be uniformly distributed throughout all lobes of the rat liver.</abstract><note>This work was partially supported by grants (A-2610 C1, 2, 3) from the National Institute for Arthritis and Metabolic Diseases of the National Institutes of Health and from the Owensboro-Daviess County Heart Association. 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J</title><editor>e ,:,m, 1'. S., f~~~r/~</editor><imprint><biblScope unit="volume">76</biblScope><biblScope unit="page">109</biblScope><date type="published" when="1960"></date></imprint></monogr></biblStruct><biblStruct><monogr><author><persName><forename type="first">Ilorc~s</forename><forename type="middle">I</forename><surname>Fl</surname></persName></author><imprint><date type="published" when="1951"></date></imprint></monogr></biblStruct><biblStruct><analytic></analytic><monogr><title level="j">IIorau</title></monogr></biblStruct></listBibl></back></text></istex:refBibTEI></enrichments><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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