Extreme prematurity: The aetiology of preterm delivery
Identifieur interne : 000D78 ( Istex/Corpus ); précédent : 000D77; suivant : 000D79Extreme prematurity: The aetiology of preterm delivery
Auteurs : Philip R. Bennett ; Murdoch G. ElderSource :
- British Medical Bulletin [ 0007-1420 ] ; 1988.
Abstract
Preterm delivery will follow either a decision to terminate the pregnancy before term for fetral or maternal reasons, or spontaneous preterm labout. A number of previously more important causes of prematurity have declined in incidence due either to improvements in prevention or treatment of the problem. Rhesus haemolytic disease of the newborn is now relatively rare and all severe cases should be managed in specialist centres. The introduction of prophylactic measures, such as the administration of anti-D gloubulin, and the success of intraperitoneal or intravascular fetal transfusion techniques ahs greatly reduced the frequency of the disease as a cause of extreme prematurity. Severe pre-existing maternal diseases—for example hypertencsion, chronic renal disease or systemic lupus—still account for a small number of preterm deliveries. With improvements in the management of pregnancy in diabetics and those with gestational diabetes, including the introduction of specialist clinics and home monitoring of blood glucose concentrations, there is rarely a need for very preterm delivery in this group. Most infants in neonatal units today will have been delivered following: severe pre-eclampsia; intrauterine growth retardation; antepartum haemorrhage; or spontaneous preterm labour.
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DOI: 10.1093/oxfordjournals.bmb.a072297
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Elder</name><affiliation><mods:affiliation>Institute of Obstetrics and Gynaecology, Hammersmith Hospital London</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">British Medical Bulletin</title><idno type="ISSN">0007-1420</idno><idno type="eISSN">1471-8391</idno><imprint><publisher>Oxford University Press</publisher><date type="published" when="1988">1988</date><biblScope unit="volume">44</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="850">850</biblScope><biblScope unit="page" to="860">860</biblScope></imprint><idno type="ISSN">0007-1420</idno></series><idno type="istex">F8A43232DD2C8A2A64F9230C75B0652CB2B23AB2</idno><idno type="DOI">10.1093/oxfordjournals.bmb.a072297</idno><idno type="ArticleID">44.4.850</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0007-1420</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract">Preterm delivery will follow either a decision to terminate the pregnancy before term for fetral or maternal reasons, or spontaneous preterm labout. A number of previously more important causes of prematurity have declined in incidence due either to improvements in prevention or treatment of the problem. Rhesus haemolytic disease of the newborn is now relatively rare and all severe cases should be managed in specialist centres. The introduction of prophylactic measures, such as the administration of anti-D gloubulin, and the success of intraperitoneal or intravascular fetal transfusion techniques ahs greatly reduced the frequency of the disease as a cause of extreme prematurity. Severe pre-existing maternal diseases—for example hypertencsion, chronic renal disease or systemic lupus—still account for a small number of preterm deliveries. With improvements in the management of pregnancy in diabetics and those with gestational diabetes, including the introduction of specialist clinics and home monitoring of blood glucose concentrations, there is rarely a need for very preterm delivery in this group. Most infants in neonatal units today will have been delivered following: severe pre-eclampsia; intrauterine growth retardation; antepartum haemorrhage; or spontaneous preterm labour.</div></front></TEI><istex><corpusName>oup</corpusName><author><json:item><name>Philip R Bennett</name><affiliations><json:string>Queen Charlotte's Maternity Hospital London</json:string></affiliations></json:item><json:item><name>Murdoch G Elder</name><affiliations><json:string>Institute of Obstetrics and Gynaecology, Hammersmith Hospital London</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Articles</value></json:item></subject><articleId><json:string>44.4.850</json:string></articleId><language><json:string>eng</json:string></language><abstract>Preterm delivery will follow either a decision to terminate the pregnancy before term for fetral or maternal reasons, or spontaneous preterm labout. A number of previously more important causes of prematurity have declined in incidence due either to improvements in prevention or treatment of the problem. Rhesus haemolytic disease of the newborn is now relatively rare and all severe cases should be managed in specialist centres. The introduction of prophylactic measures, such as the administration of anti-D gloubulin, and the success of intraperitoneal or intravascular fetal transfusion techniques ahs greatly reduced the frequency of the disease as a cause of extreme prematurity. Severe pre-existing maternal diseases—for example hypertencsion, chronic renal disease or systemic lupus—still account for a small number of preterm deliveries. With improvements in the management of pregnancy in diabetics and those with gestational diabetes, including the introduction of specialist clinics and home monitoring of blood glucose concentrations, there is rarely a need for very preterm delivery in this group. 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