The detection of two serologically distinct HLA‐A28 specificities
Identifieur interne : 000834 ( Istex/Corpus ); précédent : 000833; suivant : 000835The detection of two serologically distinct HLA‐A28 specificities
Auteurs : D. J. Holmes ; W. V. Miller ; G. E. RodeySource :
- Tissue Antigens [ 0001-2815 ] ; 1982-03.
Abstract
We describe a serum, 9045, that detects a subset of A28 bearing cells in a mixed population of American blacks, Caucasians of Jewish ancestry and non‐Jewish Caucasians. The serum reacts preferentially with A28 positive cells from American Blacks and Jewish Caucasians and does not react with non‐Jewish Caucasians. This new specificity, referred to as A28.2, is strongly associated with HLA‐B14. In contrast, A28 positive but 9045 negative cells (referred to as A28.1) do not show this association. The A28.2 determinants appears to be antigenically similar to the A28.1 determinant, but distinct from the public antigenic determinants shared by A28, A2, A9 and by A28, Aw33, Aw34 and A26. Based upon this analysis, we conclude that HLA—A28, as defined by most A28 antisera, comprises at least two populations of molecules. The A28.2 form may have arisen in the Mediterranean basin region, whereas the A28.1 form seems to be more prevalent in non‐Jewish Caucasians of Northern European ancestry.
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DOI: 10.1111/j.1399-0039.1982.tb01435.x
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Miller</name><affiliation><mods:affiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</mods:affiliation></affiliation></author><author><name sortKey="Rodey, G E" sort="Rodey, G E" uniqKey="Rodey G" first="G. E." last="Rodey">G. E. Rodey</name><affiliation><mods:affiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:5436F512DE37D1E0B593432C51A1CF5DF66EE24D</idno><date when="1982" year="1982">1982</date><idno type="doi">10.1111/j.1399-0039.1982.tb01435.x</idno><idno type="url">https://api.istex.fr/document/5436F512DE37D1E0B593432C51A1CF5DF66EE24D/fulltext/pdf</idno><idno type="wicri:Area/Istex/Corpus">000834</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">The detection of two serologically distinct HLA‐A28 specificities</title><author><name sortKey="Holmes, D J" sort="Holmes, D J" uniqKey="Holmes D" first="D. J." last="Holmes">D. J. Holmes</name><affiliation><mods:affiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</mods:affiliation></affiliation></author><author><name sortKey="Miller, W V" sort="Miller, W V" uniqKey="Miller W" first="W. V." last="Miller">W. V. Miller</name><affiliation><mods:affiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</mods:affiliation></affiliation></author><author><name sortKey="Rodey, G E" sort="Rodey, G E" uniqKey="Rodey G" first="G. E." last="Rodey">G. E. Rodey</name><affiliation><mods:affiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Tissue Antigens</title><idno type="ISSN">0001-2815</idno><idno type="eISSN">1399-0039</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1982-03">1982-03</date><biblScope unit="volume">19</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="161">161</biblScope><biblScope unit="page" to="167">167</biblScope></imprint><idno type="ISSN">0001-2815</idno></series><idno type="istex">5436F512DE37D1E0B593432C51A1CF5DF66EE24D</idno><idno type="DOI">10.1111/j.1399-0039.1982.tb01435.x</idno><idno type="ArticleID">TAN161</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0001-2815</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">We describe a serum, 9045, that detects a subset of A28 bearing cells in a mixed population of American blacks, Caucasians of Jewish ancestry and non‐Jewish Caucasians. The serum reacts preferentially with A28 positive cells from American Blacks and Jewish Caucasians and does not react with non‐Jewish Caucasians. This new specificity, referred to as A28.2, is strongly associated with HLA‐B14. In contrast, A28 positive but 9045 negative cells (referred to as A28.1) do not show this association. The A28.2 determinants appears to be antigenically similar to the A28.1 determinant, but distinct from the public antigenic determinants shared by A28, A2, A9 and by A28, Aw33, Aw34 and A26. Based upon this analysis, we conclude that HLA—A28, as defined by most A28 antisera, comprises at least two populations of molecules. The A28.2 form may have arisen in the Mediterranean basin region, whereas the A28.1 form seems to be more prevalent in non‐Jewish Caucasians of Northern European ancestry.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>D. J. Holmes</name><affiliations><json:string>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</json:string></affiliations></json:item><json:item><name>W. V. Miller</name><affiliations><json:string>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</json:string></affiliations></json:item><json:item><name>G. E. Rodey</name><affiliations><json:string>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</json:string></affiliations></json:item></author><articleId><json:string>TAN161</json:string></articleId><language><json:string>eng</json:string></language><abstract>We describe a serum, 9045, that detects a subset of A28 bearing cells in a mixed population of American blacks, Caucasians of Jewish ancestry and non‐Jewish Caucasians. The serum reacts preferentially with A28 positive cells from American Blacks and Jewish Caucasians and does not react with non‐Jewish Caucasians. This new specificity, referred to as A28.2, is strongly associated with HLA‐B14. In contrast, A28 positive but 9045 negative cells (referred to as A28.1) do not show this association. The A28.2 determinants appears to be antigenically similar to the A28.1 determinant, but distinct from the public antigenic determinants shared by A28, A2, A9 and by A28, Aw33, Aw34 and A26. Based upon this analysis, we conclude that HLA—A28, as defined by most A28 antisera, comprises at least two populations of molecules. 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E.</forename><surname>Rodey</surname></persName><affiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</affiliation></author></analytic><monogr><title level="j">Tissue Antigens</title><idno type="pISSN">0001-2815</idno><idno type="eISSN">1399-0039</idno><idno type="DOI">10.1111/(ISSN)1399-0039</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1982-03"></date><biblScope unit="volume">19</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="161">161</biblScope><biblScope unit="page" to="167">167</biblScope></imprint></monogr><idno type="istex">5436F512DE37D1E0B593432C51A1CF5DF66EE24D</idno><idno type="DOI">10.1111/j.1399-0039.1982.tb01435.x</idno><idno type="ArticleID">TAN161</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1982</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>We describe a serum, 9045, that detects a subset of A28 bearing cells in a mixed population of American blacks, Caucasians of Jewish ancestry and non‐Jewish Caucasians. The serum reacts preferentially with A28 positive cells from American Blacks and Jewish Caucasians and does not react with non‐Jewish Caucasians. This new specificity, referred to as A28.2, is strongly associated with HLA‐B14. In contrast, A28 positive but 9045 negative cells (referred to as A28.1) do not show this association. The A28.2 determinants appears to be antigenically similar to the A28.1 determinant, but distinct from the public antigenic determinants shared by A28, A2, A9 and by A28, Aw33, Aw34 and A26. Based upon this analysis, we conclude that HLA—A28, as defined by most A28 antisera, comprises at least two populations of molecules. 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J.</givenNames><familyName>Holmes</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1"><personName><givenNames>W. V.</givenNames><familyName>Miller</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>G. E.</givenNames><familyName>Rodey</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1"><unparsedAffiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</unparsedAffiliation></affiliation></affiliationGroup><abstractGroup><abstract type="main" xml:lang="en"><p>We describe a serum, 9045, that detects a subset of A28 bearing cells in a mixed population of American blacks, Caucasians of Jewish ancestry and non‐Jewish Caucasians. The serum reacts preferentially with A28 positive cells from American Blacks and Jewish Caucasians and does not react with non‐Jewish Caucasians. This new specificity, referred to as A28.2, is strongly associated with HLA‐B14. In contrast, A28 positive but 9045 negative cells (referred to as A28.1) do not show this association. The A28.2 determinants appears to be antigenically similar to the A28.1 determinant, but distinct from the public antigenic determinants shared by A28, A2, A9 and by A28, Aw33, Aw34 and A26. Based upon this analysis, we conclude that HLA—A28, as defined by most A28 antisera, comprises at least two populations of molecules. The A28.2 form may have arisen in the Mediterranean basin region, whereas the A28.1 form seems to be more prevalent in non‐Jewish Caucasians of Northern European ancestry.</p></abstract></abstractGroup></contentMeta><noteGroup><note xml:id="fn1" numbered="no"><p>Presented in part at the 7th annual meeting of the American Association for Clinical Histo‐compatibility Testing (AACHT). Orlando, Florida, U.S.A., April 1981.</p></note></noteGroup></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>The detection of two serologically distinct HLA‐A28 specificities</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>The detection of two serologically distinct HLA‐A28 specificities</title></titleInfo><name type="personal"><namePart type="given">D. J.</namePart><namePart type="family">Holmes</namePart><affiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">W. V.</namePart><namePart type="family">Miller</namePart><affiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">G. E.</namePart><namePart type="family">Rodey</namePart><affiliation>Department of Medicine and Pathology, Washington University School of Medicine, St. Louis, MO, the Missouri‐Illinois Regional Red Cross Blood Services, St. Louis, MO, and the American Red Cross Blood Services, Bethesda, MD, U.S.A.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1982-03</dateIssued><edition>Received for publication 9 October, accepted 29 October 1981</edition><copyrightDate encoding="w3cdtf">1982</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">14</extent></physicalDescription><abstract lang="en">We describe a serum, 9045, that detects a subset of A28 bearing cells in a mixed population of American blacks, Caucasians of Jewish ancestry and non‐Jewish Caucasians. The serum reacts preferentially with A28 positive cells from American Blacks and Jewish Caucasians and does not react with non‐Jewish Caucasians. This new specificity, referred to as A28.2, is strongly associated with HLA‐B14. In contrast, A28 positive but 9045 negative cells (referred to as A28.1) do not show this association. The A28.2 determinants appears to be antigenically similar to the A28.1 determinant, but distinct from the public antigenic determinants shared by A28, A2, A9 and by A28, Aw33, Aw34 and A26. Based upon this analysis, we conclude that HLA—A28, as defined by most A28 antisera, comprises at least two populations of molecules. The A28.2 form may have arisen in the Mediterranean basin region, whereas the A28.1 form seems to be more prevalent in non‐Jewish Caucasians of Northern European ancestry.</abstract><note type="content">*Presented in part at the 7th annual meeting of the American Association for Clinical Histo‐compatibility Testing (AACHT). Orlando, Florida, U.S.A., April 1981.</note><relatedItem type="host"><titleInfo><title>Tissue Antigens</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0001-2815</identifier><identifier type="eISSN">1399-0039</identifier><identifier type="DOI">10.1111/(ISSN)1399-0039</identifier><identifier type="PublisherID">TAN</identifier><part><date>1982</date><detail type="volume"><caption>vol.</caption><number>19</number></detail><detail type="issue"><caption>no.</caption><number>3</number></detail><extent unit="pages"><start>161</start><end>167</end><total>7</total></extent></part></relatedItem><identifier type="istex">5436F512DE37D1E0B593432C51A1CF5DF66EE24D</identifier><identifier type="DOI">10.1111/j.1399-0039.1982.tb01435.x</identifier><identifier type="ArticleID">TAN161</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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