The association of lung shadowing with hypersensitivity responses in patients with allergic broncho‐pulmonary aspergillosis
Identifieur interne : 000079 ( Istex/Corpus ); précédent : 000078; suivant : 000080The association of lung shadowing with hypersensitivity responses in patients with allergic broncho‐pulmonary aspergillosis
Auteurs : K. J. Turner ; Janet L. Elder ; Janice O'Mahony ; S. G. O. JohanssonSource :
- Clinical & Experimental Allergy [ 0954-7894 ] ; 1974-06.
Abstract
The levels of total serum IgE and IgE antibodies to Aspergillus fumigatus allergens were highest during an episode of symptoms associated with fresh pulmonary shadowing and a rise in the direct cosinophil count in patients with allergic bronchopulmonary aspergillosis. Agglutinin titres were greater than those found in the sera of control subjects hut did not always correlate in magnitude with total serum IgE and IgE antibody levels. The highest agglutinin titre was found in the serum of a patient with an aspergilloma in association with allergic aspergillosis. The maximum in vitro histamine release from leucocytes exposed to A. fumigatus extracts or anti‐human IgE was independent of the concentration of total IgE or IgL antibodies. The leucocytes appeared to he relatively insensitive to both allergic and reverse allergic histamine release. This was considered to result from either continuous in vivo desensitization of the leucocytes by antigens shed from the fungal hyphae or, to the effect of drugs used in the treatment of (he disease, It was concluded that serial serum IgE measurements may be of value in monitoring the onset of the active phase of this disease.
Url:
DOI: 10.1111/j.1365-2222.1974.tb01372.x
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Shenton Park, Western Australia</mods:affiliation></affiliation></author><author><name sortKey="O Mahony, Janice" sort="O Mahony, Janice" uniqKey="O Mahony J" first="Janice" last="O'Mahony">Janice O'Mahony</name><affiliation><mods:affiliation>Clinical Immunology Unit, Department of Microbiology, University of Western Australia</mods:affiliation></affiliation></author><author><name sortKey="Johansson, S G O" sort="Johansson, S G O" uniqKey="Johansson S" first="S. G. O." last="Johansson">S. G. O. 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This was considered to result from either continuous in vivo desensitization of the leucocytes by antigens shed from the fungal hyphae or, to the effect of drugs used in the treatment of (he disease, It was concluded that serial serum IgE measurements may be of value in monitoring the onset of the active phase of this disease.</p></abstract></profileDesc><revisionDesc><change when="1974-06">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/DC84DB43874140C263DB25D33ABCBF699D162C85/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1365-2222</doi><issn type="print">0954-7894</issn><issn type="electronic">1365-2222</issn><idGroup><id type="product" value="CEA"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="CLINICAL EXPERIMENTAL ALLERGY">Clinical & Experimental Allergy</title></titleGroup></publicationMeta><publicationMeta level="part" position="06002"><doi origin="wiley">10.1111/cea.1974.4.issue-2</doi><numberingGroup><numbering type="journalVolume" number="4">4</numbering><numbering type="journalIssue" number="2">2</numbering></numberingGroup><coverDate startDate="1974-06">June 1974</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0014900" status="forIssue"><doi origin="wiley">10.1111/j.1365-2222.1974.tb01372.x</doi><idGroup><id type="unit" value="CEA149"></id></idGroup><countGroup><count type="pageTotal" number="12"></count></countGroup><eventGroup><event type="firstOnline" date="2006-04-27"></event><event type="publishedOnlineFinalForm" date="2006-04-27"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:HeaderRef result:HeaderRef" date="2010-03-09"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-01-09"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-16"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="149">149</numbering><numbering type="pageLast" number="160">160</numbering></numberingGroup><correspondenceTo>Dr K. 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J.</givenNames><familyName>TURNER</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a2"><personName><givenNames>JANET L.</givenNames><familyName>ELDER</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>JANICE</givenNames><familyName>O'MAHONY</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a3"><personName><givenNames>S. G. O.</givenNames><familyName>JOHANSSON</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="AU"><unparsedAffiliation>Clinical Immunology Unit, Department of Microbiology, University of Western Australia</unparsedAffiliation></affiliation><affiliation xml:id="a2" countryCode="AU"><unparsedAffiliation>Thoracic Unit, Sir Charles Gairdner Hospital. Shenton Park, Western Australia</unparsedAffiliation></affiliation><affiliation xml:id="a3" countryCode="SE"><unparsedAffiliation>The Blood Center, Unirersity Hospital, Uppsala, Sweden</unparsedAffiliation></affiliation></affiliationGroup><abstractGroup><abstract type="main" xml:lang="en"><title type="main">Summary</title><p>The levels of total serum IgE and IgE antibodies to <i>Aspergillus fumigatus</i> allergens were highest during an episode of symptoms associated with fresh pulmonary shadowing and a rise in the direct cosinophil count in patients with allergic bronchopulmonary aspergillosis. Agglutinin titres were greater than those found in the sera of control subjects hut did not always correlate in magnitude with total serum IgE and IgE antibody levels. The highest agglutinin titre was found in the serum of a patient with an aspergilloma in association with allergic aspergillosis. The maximum <i>in vitro</i> histamine release from leucocytes exposed to <i>A. fumigatus</i> extracts or anti‐human IgE was independent of the concentration of total IgE or IgL antibodies. The leucocytes appeared to he relatively insensitive to both allergic and reverse allergic histamine release. This was considered to result from either continuous <i>in vivo</i> desensitization of the leucocytes by antigens shed from the fungal hyphae or, to the effect of drugs used in the treatment of (he disease, It was concluded that serial serum IgE measurements may be of value in monitoring the onset of the active phase of this disease.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>The association of lung shadowing with hypersensitivity responses in patients with allergic broncho‐pulmonary aspergillosis</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>The association of lung shadowing with hypersensitivity responses in patients with allergic broncho‐pulmonary aspergillosis</title></titleInfo><name type="personal"><namePart type="given">K. 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O.</namePart><namePart type="family">JOHANSSON</namePart><affiliation>The Blood Center, Unirersity Hospital, Uppsala, Sweden</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1974-06</dateIssued><copyrightDate encoding="w3cdtf">1974</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">35</extent></physicalDescription><abstract lang="en">The levels of total serum IgE and IgE antibodies to Aspergillus fumigatus allergens were highest during an episode of symptoms associated with fresh pulmonary shadowing and a rise in the direct cosinophil count in patients with allergic bronchopulmonary aspergillosis. Agglutinin titres were greater than those found in the sera of control subjects hut did not always correlate in magnitude with total serum IgE and IgE antibody levels. The highest agglutinin titre was found in the serum of a patient with an aspergilloma in association with allergic aspergillosis. The maximum in vitro histamine release from leucocytes exposed to A. fumigatus extracts or anti‐human IgE was independent of the concentration of total IgE or IgL antibodies. The leucocytes appeared to he relatively insensitive to both allergic and reverse allergic histamine release. This was considered to result from either continuous in vivo desensitization of the leucocytes by antigens shed from the fungal hyphae or, to the effect of drugs used in the treatment of (he disease, It was concluded that serial serum IgE measurements may be of value in monitoring the onset of the active phase of this disease.</abstract><relatedItem type="host"><titleInfo><title>Clinical & Experimental Allergy</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0954-7894</identifier><identifier type="eISSN">1365-2222</identifier><identifier type="DOI">10.1111/(ISSN)1365-2222</identifier><identifier type="PublisherID">CEA</identifier><part><date>1974</date><detail type="volume"><caption>vol.</caption><number>4</number></detail><detail type="issue"><caption>no.</caption><number>2</number></detail><extent unit="pages"><start>149</start><end>160</end><total>12</total></extent></part></relatedItem><identifier type="istex">DC84DB43874140C263DB25D33ABCBF699D162C85</identifier><identifier type="DOI">10.1111/j.1365-2222.1974.tb01372.x</identifier><identifier type="ArticleID">CEA149</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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