HPA‐axis activity as a predictor of future disruptive behaviors in young adolescents
Identifieur interne : 001446 ( Istex/Corpus ); précédent : 001445; suivant : 001447HPA‐axis activity as a predictor of future disruptive behaviors in young adolescents
Auteurs : Frouke E. P. L. Sondeijker ; Robert F. Ferdinand ; Albertine J. Oldehinkel ; Henning Tiemeier ; Johan Ormel ; Frank C. VerhulstSource :
- Psychophysiology [ 0048-5772 ] ; 2008-05.
English descriptors
Abstract
Low HPA‐axis activity has been proposed as a risk factor for disruptive behaviors. However longitudinal data on this topic are practically lacking. In the present study we investigated if low HPA‐axis activity predicted future disruptive behaviors. We included 1,399 boys and girls from the Dutch general population, initially aged 10–12 years. At the first assessment, basal cortisol levels were assessed. At the first assessment and at follow‐up 2 years later disruptive behaviors were assessed with parent and self‐report questionnaires. The results suggest that the association between low cortisol levels at 8.00 p.m. and future disruptive behaviors according to the parents was only present for boys. More importantly however, the results suggest that low HPA‐axis activity is not a good predictor for disruptive behaviors, but could be valuable to identify those with a poor prognosis, once disruptive behaviors are present in preadolescence.
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DOI: 10.1111/j.1469-8986.2008.00639.x
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ISTEX:1D359CB6C8C4B8B67DEC2D084CA954C1DB1240E8Le document en format XML
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Ferdinand</name><affiliation><mods:affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</mods:affiliation></affiliation></author><author><name sortKey="Oldehinkel, Albertine J" sort="Oldehinkel, Albertine J" uniqKey="Oldehinkel A" first="Albertine J." last="Oldehinkel">Albertine J. Oldehinkel</name><affiliation><mods:affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</mods:affiliation></affiliation></author><author><name sortKey="Tiemeier, Henning" sort="Tiemeier, Henning" uniqKey="Tiemeier H" first="Henning" last="Tiemeier">Henning Tiemeier</name><affiliation><mods:affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Department of Epidemiology and Biostatistics, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation></author><author><name sortKey="Ormel, Johan" sort="Ormel, Johan" uniqKey="Ormel J" first="Johan" last="Ormel">Johan Ormel</name><affiliation><mods:affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</mods:affiliation></affiliation></author><author><name sortKey="Verhulst, Frank C" sort="Verhulst, Frank C" uniqKey="Verhulst F" first="Frank C." last="Verhulst">Frank C. 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L." last="Sondeijker">Frouke E. P. L. Sondeijker</name><affiliation><mods:affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation></author><author><name sortKey="Ferdinand, Robert F" sort="Ferdinand, Robert F" uniqKey="Ferdinand R" first="Robert F." last="Ferdinand">Robert F. Ferdinand</name><affiliation><mods:affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</mods:affiliation></affiliation></author><author><name sortKey="Oldehinkel, Albertine J" sort="Oldehinkel, Albertine J" uniqKey="Oldehinkel A" first="Albertine J." last="Oldehinkel">Albertine J. Oldehinkel</name><affiliation><mods:affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</mods:affiliation></affiliation></author><author><name sortKey="Tiemeier, Henning" sort="Tiemeier, Henning" uniqKey="Tiemeier H" first="Henning" last="Tiemeier">Henning Tiemeier</name><affiliation><mods:affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Department of Epidemiology and Biostatistics, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation></author><author><name sortKey="Ormel, Johan" sort="Ormel, Johan" uniqKey="Ormel J" first="Johan" last="Ormel">Johan Ormel</name><affiliation><mods:affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</mods:affiliation></affiliation><affiliation><mods:affiliation>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</mods:affiliation></affiliation></author><author><name sortKey="Verhulst, Frank C" sort="Verhulst, Frank C" uniqKey="Verhulst F" first="Frank C." last="Verhulst">Frank C. Verhulst</name><affiliation><mods:affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Psychophysiology</title><idno type="ISSN">0048-5772</idno><idno type="eISSN">1469-8986</idno><imprint><publisher>Blackwell Publishing Inc</publisher><pubPlace>Malden, USA</pubPlace><date type="published" when="2008-05">2008-05</date><biblScope unit="volume">45</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="398">398</biblScope><biblScope unit="page" to="404">404</biblScope></imprint><idno type="ISSN">0048-5772</idno></series><idno type="istex">1D359CB6C8C4B8B67DEC2D084CA954C1DB1240E8</idno><idno type="DOI">10.1111/j.1469-8986.2008.00639.x</idno><idno type="ArticleID">PSYP639</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0048-5772</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Adolescents</term><term>Cortisol</term><term>Disruptive behaviors</term><term>General population</term><term>HPA‐axis</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Low HPA‐axis activity has been proposed as a risk factor for disruptive behaviors. However longitudinal data on this topic are practically lacking. In the present study we investigated if low HPA‐axis activity predicted future disruptive behaviors. We included 1,399 boys and girls from the Dutch general population, initially aged 10–12 years. At the first assessment, basal cortisol levels were assessed. At the first assessment and at follow‐up 2 years later disruptive behaviors were assessed with parent and self‐report questionnaires. The results suggest that the association between low cortisol levels at 8.00 p.m. and future disruptive behaviors according to the parents was only present for boys. More importantly however, the results suggest that low HPA‐axis activity is not a good predictor for disruptive behaviors, but could be valuable to identify those with a poor prognosis, once disruptive behaviors are present in preadolescence.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Frouke E.P.L. Sondeijker</name><affiliations><json:string>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</json:string></affiliations></json:item><json:item><name>Robert F. Ferdinand</name><affiliations><json:string>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</json:string><json:string>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</json:string></affiliations></json:item><json:item><name>Albertine J. Oldehinkel</name><affiliations><json:string>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</json:string><json:string>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</json:string><json:string>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</json:string></affiliations></json:item><json:item><name>Henning Tiemeier</name><affiliations><json:string>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</json:string><json:string>Department of Epidemiology and Biostatistics, Erasmus Medical Center, Rotterdam, The Netherlands</json:string></affiliations></json:item><json:item><name>Johan Ormel</name><affiliations><json:string>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</json:string><json:string>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</json:string></affiliations></json:item><json:item><name>Frank C. Verhulst</name><affiliations><json:string>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>HPA‐axis</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Disruptive behaviors</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Cortisol</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Adolescents</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>General population</value></json:item></subject><articleId><json:string>PSYP639</json:string></articleId><language><json:string>eng</json:string></language><originalGenre><json:string>article</json:string></originalGenre><abstract>Low HPA‐axis activity has been proposed as a risk factor for disruptive behaviors. However longitudinal data on this topic are practically lacking. In the present study we investigated if low HPA‐axis activity predicted future disruptive behaviors. We included 1,399 boys and girls from the Dutch general population, initially aged 10–12 years. At the first assessment, basal cortisol levels were assessed. At the first assessment and at follow‐up 2 years later disruptive behaviors were assessed with parent and self‐report questionnaires. The results suggest that the association between low cortisol levels at 8.00 p.m. and future disruptive behaviors according to the parents was only present for boys. 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adolescents</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>Blackwell Publishing Inc</publisher><pubPlace>Malden, USA</pubPlace><availability><p>Copyright © 2008 Society for Psychophysiological Research</p></availability><date>2008</date></publicationStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">HPA‐axis activity as a predictor of future disruptive behaviors in young adolescents</title><author xml:id="author-1"><persName><forename type="first">Frouke E.P.L.</forename><surname>Sondeijker</surname></persName><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation></author><author xml:id="author-2"><persName><forename type="first">Robert F.</forename><surname>Ferdinand</surname></persName><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation><affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</affiliation></author><author xml:id="author-3"><persName><forename type="first">Albertine J.</forename><surname>Oldehinkel</surname></persName><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation><affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</affiliation><affiliation>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</affiliation></author><author xml:id="author-4"><persName><forename type="first">Henning</forename><surname>Tiemeier</surname></persName><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation><affiliation>Department of Epidemiology and Biostatistics, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation></author><author xml:id="author-5"><persName><forename type="first">Johan</forename><surname>Ormel</surname></persName><affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</affiliation><affiliation>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</affiliation></author><author xml:id="author-6"><persName><forename type="first">Frank C.</forename><surname>Verhulst</surname></persName><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation></author></analytic><monogr><title level="j">Psychophysiology</title><idno type="pISSN">0048-5772</idno><idno type="eISSN">1469-8986</idno><idno type="DOI">10.1111/(ISSN)1469-8986</idno><imprint><publisher>Blackwell Publishing Inc</publisher><pubPlace>Malden, USA</pubPlace><date type="published" when="2008-05"></date><biblScope unit="volume">45</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="398">398</biblScope><biblScope unit="page" to="404">404</biblScope></imprint></monogr><idno type="istex">1D359CB6C8C4B8B67DEC2D084CA954C1DB1240E8</idno><idno type="DOI">10.1111/j.1469-8986.2008.00639.x</idno><idno type="ArticleID">PSYP639</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>2008</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Low HPA‐axis activity has been proposed as a risk factor for disruptive behaviors. However longitudinal data on this topic are practically lacking. In the present study we investigated if low HPA‐axis activity predicted future disruptive behaviors. We included 1,399 boys and girls from the Dutch general population, initially aged 10–12 years. At the first assessment, basal cortisol levels were assessed. At the first assessment and at follow‐up 2 years later disruptive behaviors were assessed with parent and self‐report questionnaires. The results suggest that the association between low cortisol levels at 8.00 p.m. and future disruptive behaviors according to the parents was only present for boys. More importantly however, the results suggest that low HPA‐axis activity is not a good predictor for disruptive behaviors, but could be valuable to identify those with a poor prognosis, once disruptive behaviors are present in preadolescence.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>keywords</head><item><term>HPA‐axis</term></item><item><term>Disruptive behaviors</term></item><item><term>Cortisol</term></item><item><term>Adolescents</term></item><item><term>General population</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="2008-05">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/1D359CB6C8C4B8B67DEC2D084CA954C1DB1240E8/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Inc</publisherName><publisherLoc>Malden, USA</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1469-8986</doi><issn type="print">0048-5772</issn><issn type="electronic">1469-8986</issn><idGroup><id type="product" value="PSYP"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PSYCHOPHYSIOLOGY">Psychophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="05003"><doi origin="wiley">10.1111/psyp.2008.45.issue-3</doi><numberingGroup><numbering type="journalVolume" number="45">45</numbering><numbering type="journalIssue" number="3">3</numbering></numberingGroup><coverDate startDate="2008-05">May 2008</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="8" status="forIssue"><doi origin="wiley">10.1111/j.1469-8986.2008.00639.x</doi><idGroup><id type="unit" value="PSYP639"></id><id type="supplier" value="639"></id></idGroup><countGroup><count type="pageTotal" number="7"></count></countGroup><titleGroup><title type="tocHeading1">Original Articles</title></titleGroup><copyright>Copyright © 2008 Society for Psychophysiological Research</copyright><eventGroup><event type="firstOnline" date="2008-02-28"></event><event type="publishedOnlineFinalForm" date="2008-02-28"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:FullText result:FullText" date="2010-03-03"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-07"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-11-03"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="398">398</numbering><numbering type="pageLast" number="404">404</numbering></numberingGroup><correspondenceTo>Address reprint requests to: Robert F. Ferdinand, Erasmus Medical Center Rotterdam/Sophia Children's Hospital, Department of Child & Adolescent Psychiatry, Dr. Molewaterplein 60, 3015 GJ Rotterdam, The Netherlands. E‐mail: <email normalForm="r.f.ferdinand@erasmusmc.nl">r.f.ferdinand@erasmusmc.nl</email></correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:PSYP.PSYP639.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>(Received September 6, 2006; Accepted September 30, 2007)</unparsedEditorialHistory><countGroup><count type="figureTotal" number="0"></count><count type="tableTotal" number="0"></count><count type="formulaTotal" number="1"></count><count type="referenceTotal" number="52"></count><count type="wordTotal" number="7393"></count><count type="linksCrossRef" number="56"></count></countGroup><titleGroup><title type="main">HPA‐axis activity as a predictor of future disruptive behaviors in young adolescents</title><title type="shortAuthors">F.E.P.L. Sondeijker et al.</title><title type="short">HPA‐axis activity and future disruptive behaviors</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1"><personName><givenNames>Frouke E.P.L.</givenNames><familyName>Sondeijker</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1 #a2"><personName><givenNames>Robert F.</givenNames><familyName>Ferdinand</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1 #a2 #a3"><personName><givenNames>Albertine J.</givenNames><familyName>Oldehinkel</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a1 #a4"><personName><givenNames>Henning</givenNames><familyName>Tiemeier</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a2 #a3"><personName><givenNames>Johan</givenNames><familyName>Ormel</familyName></personName></creator><creator creatorRole="author" xml:id="cr6" affiliationRef="#a1"><personName><givenNames>Frank C.</givenNames><familyName>Verhulst</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="NL"><unparsedAffiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</unparsedAffiliation></affiliation><affiliation xml:id="a2" countryCode="NL"><unparsedAffiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</unparsedAffiliation></affiliation><affiliation xml:id="a3" countryCode="NL"><unparsedAffiliation>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</unparsedAffiliation></affiliation><affiliation xml:id="a4" countryCode="NL"><unparsedAffiliation>Department of Epidemiology and Biostatistics, Erasmus Medical Center, Rotterdam, The Netherlands</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">HPA‐axis</keyword><keyword xml:id="k2">Disruptive behaviors</keyword><keyword xml:id="k3">Cortisol</keyword><keyword xml:id="k4">Adolescents</keyword><keyword xml:id="k5">General population</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><title type="main">Abstract</title><p>Low HPA‐axis activity has been proposed as a risk factor for disruptive behaviors. However longitudinal data on this topic are practically lacking. In the present study we investigated if low HPA‐axis activity predicted future disruptive behaviors. We included 1,399 boys and girls from the Dutch general population, initially aged 10–12 years. At the first assessment, basal cortisol levels were assessed. At the first assessment and at follow‐up 2 years later disruptive behaviors were assessed with parent and self‐report questionnaires. The results suggest that the association between low cortisol levels at 8.00 p.m. and future disruptive behaviors according to the parents was only present for boys. More importantly however, the results suggest that low HPA‐axis activity is not a good predictor for disruptive behaviors, but could be valuable to identify those with a poor prognosis, once disruptive behaviors are present in preadolescence.</p></abstract></abstractGroup></contentMeta><noteGroup><note xml:id="fn1" numbered="no"><p>This research is part of the TRacking Adolescents' Individual Lives Survey (TRAILS). Participating centers of TRAILS include various Departments of the University of Groningen, the Erasmus Medical Center of Rotterdam, the University of Nijmegen, University of Utrecht, and the Trimbos Institute, The Netherlands. TRAILS is financially supported by grants from the Netherlands Organization for Scientific Research (GB‐MW 940‐38‐011, GB‐MAGW 480‐01‐006, GB‐MAGW 457‐03‐018, GB‐MAGW 175.010.2003.005, ZonMw 100‐001‐001 “Geestkracht” Program, ZonMw 60‐60600‐98‐018), the Sophia Foundation for Medical Research (projects 301 and 393), the Dutch Ministry of Justice, and by the participating centers.</p></note></noteGroup></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>HPA‐axis activity as a predictor of future disruptive behaviors in young adolescents</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>HPA‐axis activity and future disruptive behaviors</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>HPA‐axis activity as a predictor of future disruptive behaviors in young adolescents</title></titleInfo><name type="personal"><namePart type="given">Frouke E.P.L.</namePart><namePart type="family">Sondeijker</namePart><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Robert F.</namePart><namePart type="family">Ferdinand</namePart><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation><affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Albertine J.</namePart><namePart type="family">Oldehinkel</namePart><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation><affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</affiliation><affiliation>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Henning</namePart><namePart type="family">Tiemeier</namePart><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation><affiliation>Department of Epidemiology and Biostatistics, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Johan</namePart><namePart type="family">Ormel</namePart><affiliation>Department of Psychiatry and Graduate School of Behavioral and Cognitive Neurosciences, Groningen University Medical Center, Groningen, The Netherlands</affiliation><affiliation>Graduate School for Experimental Psychopathology, Groningen, The Netherlands</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Frank C.</namePart><namePart type="family">Verhulst</namePart><affiliation>Department of Child and Adolescent Psychiatry, Erasmus Medical Center, Rotterdam, The Netherlands</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Inc</publisher><place><placeTerm type="text">Malden, USA</placeTerm></place><dateIssued encoding="w3cdtf">2008-05</dateIssued><edition>(Received September 6, 2006; Accepted September 30, 2007)</edition><copyrightDate encoding="w3cdtf">2008</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="formulas">1</extent><extent unit="references">52</extent><extent unit="words">7393</extent></physicalDescription><abstract lang="en">Low HPA‐axis activity has been proposed as a risk factor for disruptive behaviors. However longitudinal data on this topic are practically lacking. In the present study we investigated if low HPA‐axis activity predicted future disruptive behaviors. We included 1,399 boys and girls from the Dutch general population, initially aged 10–12 years. At the first assessment, basal cortisol levels were assessed. At the first assessment and at follow‐up 2 years later disruptive behaviors were assessed with parent and self‐report questionnaires. The results suggest that the association between low cortisol levels at 8.00 p.m. and future disruptive behaviors according to the parents was only present for boys. More importantly however, the results suggest that low HPA‐axis activity is not a good predictor for disruptive behaviors, but could be valuable to identify those with a poor prognosis, once disruptive behaviors are present in preadolescence.</abstract><subject lang="en"><genre>keywords</genre><topic>HPA‐axis</topic><topic>Disruptive behaviors</topic><topic>Cortisol</topic><topic>Adolescents</topic><topic>General population</topic></subject><relatedItem type="host"><titleInfo><title>Psychophysiology</title></titleInfo><genre type="journal">journal</genre><identifier type="ISSN">0048-5772</identifier><identifier type="eISSN">1469-8986</identifier><identifier type="DOI">10.1111/(ISSN)1469-8986</identifier><identifier type="PublisherID">PSYP</identifier><part><date>2008</date><detail type="volume"><caption>vol.</caption><number>45</number></detail><detail type="issue"><caption>no.</caption><number>3</number></detail><extent unit="pages"><start>398</start><end>404</end><total>7</total></extent></part></relatedItem><identifier type="istex">1D359CB6C8C4B8B67DEC2D084CA954C1DB1240E8</identifier><identifier type="DOI">10.1111/j.1469-8986.2008.00639.x</identifier><identifier type="ArticleID">PSYP639</identifier><accessCondition type="use and reproduction" contentType="copyright">Copyright © 2008 Society for Psychophysiological Research</accessCondition><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Inc</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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