EsturgeonV1, PascalFrancis, Corpus, bibRecord, 000065

Histopathological alterations of juvenile green (Acipenser medirostris) and white sturgeon (Acipenser transmontanus) exposed to graded levels of dietary methylmercury

Identifieur interne : 000065 ( PascalFrancis/Corpus ); précédent : 000064; suivant : 000066

Histopathological alterations of juvenile green (Acipenser medirostris) and white sturgeon (Acipenser transmontanus) exposed to graded levels of dietary methylmercury

Auteurs : Jang-Won Lee ; Jae-Won Kim ; NICOLA DE RIU ; Giuseppe Moniello ; Silas S. O. Hung

Source :

Aquatic toxicology [ 0166-445X ] ; 2012.

RBID : Pascal:12-0172963

Descripteurs français

Pascal (Inist)
- Altération, Animal jeune, Régime alimentaire, Histopathologie, Milieu aquatique, Ecotoxicologie, Toxicité, Acipenser transmontanus, Mercure(méthyl).

English descriptors

KwdEn :
- Acipenser transmontanus, Alteration, Aquatic environment, Diet, Ecotoxicology, Histopathology, Toxicity, Young animal.

Abstract

Triplicate groups of juvenile green and white sturgeon (average weight of 30±2g) were exposed to one of four concentrations of dietary methylmercury (MeHg; 0, 25, 50, and 100 mg MeHg/kg diet) for 8 weeks to determine and compare the sensitivity of the two sturgeon species from a histopathological perspective. After 4- and 8-week exposure, histological changes were examined in the kidney, liver, gill, skeletal muscle, and heart muscle of both species using light microscopy. Marked abnormalities were observed in the kidney and liver of both sturgeon species after each exposure period; the abnormalities showed progressive histological alterations in severity with increasing doses and duration of exposure. Renal lesions included tubular epithelium degeneration and necrosis, renal corpuscular disintegration, and interstitial tissue degeneration. The changes observed in the livers of both sturgeon species were glycogen depletion and vacuolar degeneration. In the gill and skeletal and heart muscle of green and white sturgeon fed MeHg-added diets, mild histological changes were observed but did not show pronounced difference between the two species. Although the lowest observed effect concentration in both species was the 25 mg MeHg/kg diet, the histological changes in the kidney and liver were more pronounced at all treatments groups of green sturgeon than those of white sturgeon. The current results on structural changes of kidney and liver (i.e., more severe glycogen depletion and tubular epithelium degeneration in green sturgeon) confirmed our previous results, in that green sturgeon exhibited a higher mortality, lower growth rate, and lower protein, lipid, and energy contents in their whole body than white sturgeon under the same MeHg exposures.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

A01	`01`	`1`		`@0 0166-445X`
A02	`01`			`@0 AQTODG`
A03		`1`		`@0 Aquat. toxicol.`
A05				`@2 109`
A08	`01`	`1`	`ENG`	`@1 Histopathological alterations of juvenile green (Acipenser medirostris) and white sturgeon (Acipenser transmontanus) exposed to graded levels of dietary methylmercury`
A11	`01`	`1`		`@1 LEE (Jang-Won)`
A11	`02`	`1`		`@1 KIM (Jae-Won)`
A11	`03`	`1`		`@1 NICOLA DE RIU`
A11	`04`	`1`		`@1 MONIELLO (Giuseppe)`
A11	`05`	`1`		`@1 HUNG (Silas S. O.)`
A14	`01`			`@1 Department of Animal Science, University of California, One Shields Avenue @2 Davis, CA 95616-8521 @3 USA @Z 1 aut. @Z 5 aut.`
A14	`02`			`@1 Department of Marine Life-Science, Gangwon Provincial College, 115 Gyohang st., Jumunjin-eup Gangneung @2 Gangwon 210-804 @3 KOR @Z 2 aut.`
A14	`03`			`@1 Department of Animal Biology, University of Sassari, Via Vienna 2 @2 07100 Sassari @3 ITA @Z 3 aut. @Z 4 aut.`
A20				`@1 90-99`
A21				`@1 2012`
A23	`01`			`@0 ENG`
A43	`01`			`@1 INIST @2 18841 @5 354000509616490100`
A44				`@0 0000 @1 © 2012 INIST-CNRS. All rights reserved.`
A45				`@0 3/4 p.`
A47	`01`	`1`		`@0 12-0172963`
A60				`@1 P`
A61				`@0 A`
A64	`01`	`1`		`@0 Aquatic toxicology`
A66	`01`			`@0 NLD`
C01	`01`		`ENG`	@0 Triplicate groups of juvenile green and white sturgeon (average weight of 30±2g) were exposed to one of four concentrations of dietary methylmercury (MeHg; 0, 25, 50, and 100 mg MeHg/kg diet) for 8 weeks to determine and compare the sensitivity of the two sturgeon species from a histopathological perspective. After 4- and 8-week exposure, histological changes were examined in the kidney, liver, gill, skeletal muscle, and heart muscle of both species using light microscopy. Marked abnormalities were observed in the kidney and liver of both sturgeon species after each exposure period; the abnormalities showed progressive histological alterations in severity with increasing doses and duration of exposure. Renal lesions included tubular epithelium degeneration and necrosis, renal corpuscular disintegration, and interstitial tissue degeneration. The changes observed in the livers of both sturgeon species were glycogen depletion and vacuolar degeneration. In the gill and skeletal and heart muscle of green and white sturgeon fed MeHg-added diets, mild histological changes were observed but did not show pronounced difference between the two species. Although the lowest observed effect concentration in both species was the 25 mg MeHg/kg diet, the histological changes in the kidney and liver were more pronounced at all treatments groups of green sturgeon than those of white sturgeon. The current results on structural changes of kidney and liver (i.e., more severe glycogen depletion and tubular epithelium degeneration in green sturgeon) confirmed our previous results, in that green sturgeon exhibited a higher mortality, lower growth rate, and lower protein, lipid, and energy contents in their whole body than white sturgeon under the same MeHg exposures.
C02	`01`	`X`		`@0 002A14D05A`
C02	`02`	`X`		`@0 002A14B02A`
C03	`01`	`X`	`FRE`	`@0 Altération @5 01`
C03	`01`	`X`	`ENG`	`@0 Alteration @5 01`
C03	`01`	`X`	`SPA`	`@0 Alteración @5 01`
C03	`02`	`X`	`FRE`	`@0 Animal jeune @5 02`
C03	`02`	`X`	`ENG`	`@0 Young animal @5 02`
C03	`02`	`X`	`SPA`	`@0 Animal joven @5 02`
C03	`03`	`X`	`FRE`	`@0 Régime alimentaire @5 03`
C03	`03`	`X`	`ENG`	`@0 Diet @5 03`
C03	`03`	`X`	`SPA`	`@0 Régimen alimentario @5 03`
C03	`04`	`X`	`FRE`	`@0 Histopathologie @5 04`
C03	`04`	`X`	`ENG`	`@0 Histopathology @5 04`
C03	`04`	`X`	`SPA`	`@0 Histopatología @5 04`
C03	`05`	`X`	`FRE`	`@0 Milieu aquatique @5 05`
C03	`05`	`X`	`ENG`	`@0 Aquatic environment @5 05`
C03	`05`	`X`	`SPA`	`@0 Medio acuático @5 05`
C03	`06`	`X`	`FRE`	`@0 Ecotoxicologie @5 06`
C03	`06`	`X`	`ENG`	`@0 Ecotoxicology @5 06`
C03	`06`	`X`	`SPA`	`@0 Ecotoxicología @5 06`
C03	`07`	`X`	`FRE`	`@0 Toxicité @5 23`
C03	`07`	`X`	`ENG`	`@0 Toxicity @5 23`
C03	`07`	`X`	`SPA`	`@0 Toxicidad @5 23`
C03	`08`	`X`	`FRE`	`@0 Acipenser transmontanus @2 NS @5 49`
C03	`08`	`X`	`ENG`	`@0 Acipenser transmontanus @2 NS @5 49`
C03	`08`	`X`	`SPA`	`@0 Acipenser transmontanus @2 NS @5 49`
C03	`09`	`X`	`FRE`	`@0 Mercure(méthyl) @4 INC @5 87`
C07	`01`	`X`	`FRE`	`@0 Alimentation @5 17`
C07	`01`	`X`	`ENG`	`@0 Feeding @5 17`
C07	`01`	`X`	`SPA`	`@0 Alimentación @5 17`
C07	`02`	`X`	`FRE`	`@0 Composé organique @2 NA @5 18`
C07	`02`	`X`	`ENG`	`@0 Organic compounds @2 NA @5 18`
C07	`02`	`X`	`SPA`	`@0 Compuesto orgánico @2 NA @5 18`
C07	`03`	`X`	`FRE`	`@0 Pisces @2 NS @5 29`
C07	`03`	`X`	`ENG`	`@0 Pisces @2 NS @5 29`
C07	`03`	`X`	`SPA`	`@0 Pisces @2 NS @5 29`
C07	`04`	`X`	`FRE`	`@0 Vertebrata @2 NS`
C07	`04`	`X`	`ENG`	`@0 Vertebrata @2 NS`
C07	`04`	`X`	`SPA`	`@0 Vertebrata @2 NS`
C07	`05`	`X`	`FRE`	`@0 Acipenseridae @4 INC @5 70`
N21				`@1 129`
N44	`01`			`@1 OTO`
N82				`@1 OTO`

Format Inist (serveur)

NO :	PASCAL 12-0172963 INIST
ET :	Histopathological alterations of juvenile green (Acipenser medirostris) and white sturgeon (Acipenser transmontanus) exposed to graded levels of dietary methylmercury
AU :	LEE (Jang-Won); KIM (Jae-Won); NICOLA DE RIU; MONIELLO (Giuseppe); HUNG (Silas S. O.)
AF :	Department of Animal Science, University of California, One Shields Avenue/Davis, CA 95616-8521/Etats-Unis (1 aut., 5 aut.); Department of Marine Life-Science, Gangwon Provincial College, 115 Gyohang st., Jumunjin-eup Gangneung/Gangwon 210-804/Corée, République de (2 aut.); Department of Animal Biology, University of Sassari, Via Vienna 2/07100 Sassari/Italie (3 aut., 4 aut.)
DT :	Publication en série; Niveau analytique
SO :	Aquatic toxicology; ISSN 0166-445X; Coden AQTODG; Pays-Bas; Da. 2012; Vol. 109; Pp. 90-99; Bibl. 3/4 p.
LA :	Anglais
EA :	Triplicate groups of juvenile green and white sturgeon (average weight of 30±2g) were exposed to one of four concentrations of dietary methylmercury (MeHg; 0, 25, 50, and 100 mg MeHg/kg diet) for 8 weeks to determine and compare the sensitivity of the two sturgeon species from a histopathological perspective. After 4- and 8-week exposure, histological changes were examined in the kidney, liver, gill, skeletal muscle, and heart muscle of both species using light microscopy. Marked abnormalities were observed in the kidney and liver of both sturgeon species after each exposure period; the abnormalities showed progressive histological alterations in severity with increasing doses and duration of exposure. Renal lesions included tubular epithelium degeneration and necrosis, renal corpuscular disintegration, and interstitial tissue degeneration. The changes observed in the livers of both sturgeon species were glycogen depletion and vacuolar degeneration. In the gill and skeletal and heart muscle of green and white sturgeon fed MeHg-added diets, mild histological changes were observed but did not show pronounced difference between the two species. Although the lowest observed effect concentration in both species was the 25 mg MeHg/kg diet, the histological changes in the kidney and liver were more pronounced at all treatments groups of green sturgeon than those of white sturgeon. The current results on structural changes of kidney and liver (i.e., more severe glycogen depletion and tubular epithelium degeneration in green sturgeon) confirmed our previous results, in that green sturgeon exhibited a higher mortality, lower growth rate, and lower protein, lipid, and energy contents in their whole body than white sturgeon under the same MeHg exposures.
CC :	002A14D05A; 002A14B02A
FD :	Altération; Animal jeune; Régime alimentaire; Histopathologie; Milieu aquatique; Ecotoxicologie; Toxicité; Acipenser transmontanus; Mercure(méthyl)
FG :	Alimentation; Composé organique; Pisces; Vertebrata; Acipenseridae
ED :	Alteration; Young animal; Diet; Histopathology; Aquatic environment; Ecotoxicology; Toxicity; Acipenser transmontanus
EG :	Feeding; Organic compounds; Pisces; Vertebrata
SD :	Alteración; Animal joven; Régimen alimentario; Histopatología; Medio acuático; Ecotoxicología; Toxicidad; Acipenser transmontanus
LO :	INIST-18841.354000509616490100
ID :	12-0172963

Links to Exploration step

Pascal:12-0172963

Le document en format XML

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<front><div type="abstract" xml:lang="en">Triplicate groups of juvenile green and white sturgeon (average weight of 30±2g) were exposed to one of four concentrations of dietary methylmercury (MeHg; 0, 25, 50, and 100 mg MeHg/kg diet) for 8 weeks to determine and compare the sensitivity of the two sturgeon species from a histopathological perspective. After 4- and 8-week exposure, histological changes were examined in the kidney, liver, gill, skeletal muscle, and heart muscle of both species using light microscopy. Marked abnormalities were observed in the kidney and liver of both sturgeon species after each exposure period; the abnormalities showed progressive histological alterations in severity with increasing doses and duration of exposure. Renal lesions included tubular epithelium degeneration and necrosis, renal corpuscular disintegration, and interstitial tissue degeneration. The changes observed in the livers of both sturgeon species were glycogen depletion and vacuolar degeneration. In the gill and skeletal and heart muscle of green and white sturgeon fed MeHg-added diets, mild histological changes were observed but did not show pronounced difference between the two species. Although the lowest observed effect concentration in both species was the 25 mg MeHg/kg diet, the histological changes in the kidney and liver were more pronounced at all treatments groups of green sturgeon than those of white sturgeon. The current results on structural changes of kidney and liver (i.e., more severe glycogen depletion and tubular epithelium degeneration in green sturgeon) confirmed our previous results, in that green sturgeon exhibited a higher mortality, lower growth rate, and lower protein, lipid, and energy contents in their whole body than white sturgeon under the same MeHg exposures.</div>
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<fA08 i1="01" i2="1" l="ENG"><s1>Histopathological alterations of juvenile green (Acipenser medirostris) and white sturgeon (Acipenser transmontanus) exposed to graded levels of dietary methylmercury</s1>
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<fA11 i1="01" i2="1"><s1>LEE (Jang-Won)</s1>
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<fA14 i1="02"><s1>Department of Marine Life-Science, Gangwon Provincial College, 115 Gyohang st., Jumunjin-eup Gangneung</s1>
<s2>Gangwon 210-804</s2>
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<sZ>2 aut.</sZ>
</fA14>
<fA14 i1="03"><s1>Department of Animal Biology, University of Sassari, Via Vienna 2</s1>
<s2>07100 Sassari</s2>
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<fC01 i1="01" l="ENG"><s0>Triplicate groups of juvenile green and white sturgeon (average weight of 30±2g) were exposed to one of four concentrations of dietary methylmercury (MeHg; 0, 25, 50, and 100 mg MeHg/kg diet) for 8 weeks to determine and compare the sensitivity of the two sturgeon species from a histopathological perspective. After 4- and 8-week exposure, histological changes were examined in the kidney, liver, gill, skeletal muscle, and heart muscle of both species using light microscopy. Marked abnormalities were observed in the kidney and liver of both sturgeon species after each exposure period; the abnormalities showed progressive histological alterations in severity with increasing doses and duration of exposure. Renal lesions included tubular epithelium degeneration and necrosis, renal corpuscular disintegration, and interstitial tissue degeneration. The changes observed in the livers of both sturgeon species were glycogen depletion and vacuolar degeneration. In the gill and skeletal and heart muscle of green and white sturgeon fed MeHg-added diets, mild histological changes were observed but did not show pronounced difference between the two species. Although the lowest observed effect concentration in both species was the 25 mg MeHg/kg diet, the histological changes in the kidney and liver were more pronounced at all treatments groups of green sturgeon than those of white sturgeon. The current results on structural changes of kidney and liver (i.e., more severe glycogen depletion and tubular epithelium degeneration in green sturgeon) confirmed our previous results, in that green sturgeon exhibited a higher mortality, lower growth rate, and lower protein, lipid, and energy contents in their whole body than white sturgeon under the same MeHg exposures.</s0>
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</fC03>
<fC03 i1="02" i2="X" l="FRE"><s0>Animal jeune</s0>
<s5>02</s5>
</fC03>
<fC03 i1="02" i2="X" l="ENG"><s0>Young animal</s0>
<s5>02</s5>
</fC03>
<fC03 i1="02" i2="X" l="SPA"><s0>Animal joven</s0>
<s5>02</s5>
</fC03>
<fC03 i1="03" i2="X" l="FRE"><s0>Régime alimentaire</s0>
<s5>03</s5>
</fC03>
<fC03 i1="03" i2="X" l="ENG"><s0>Diet</s0>
<s5>03</s5>
</fC03>
<fC03 i1="03" i2="X" l="SPA"><s0>Régimen alimentario</s0>
<s5>03</s5>
</fC03>
<fC03 i1="04" i2="X" l="FRE"><s0>Histopathologie</s0>
<s5>04</s5>
</fC03>
<fC03 i1="04" i2="X" l="ENG"><s0>Histopathology</s0>
<s5>04</s5>
</fC03>
<fC03 i1="04" i2="X" l="SPA"><s0>Histopatología</s0>
<s5>04</s5>
</fC03>
<fC03 i1="05" i2="X" l="FRE"><s0>Milieu aquatique</s0>
<s5>05</s5>
</fC03>
<fC03 i1="05" i2="X" l="ENG"><s0>Aquatic environment</s0>
<s5>05</s5>
</fC03>
<fC03 i1="05" i2="X" l="SPA"><s0>Medio acuático</s0>
<s5>05</s5>
</fC03>
<fC03 i1="06" i2="X" l="FRE"><s0>Ecotoxicologie</s0>
<s5>06</s5>
</fC03>
<fC03 i1="06" i2="X" l="ENG"><s0>Ecotoxicology</s0>
<s5>06</s5>
</fC03>
<fC03 i1="06" i2="X" l="SPA"><s0>Ecotoxicología</s0>
<s5>06</s5>
</fC03>
<fC03 i1="07" i2="X" l="FRE"><s0>Toxicité</s0>
<s5>23</s5>
</fC03>
<fC03 i1="07" i2="X" l="ENG"><s0>Toxicity</s0>
<s5>23</s5>
</fC03>
<fC03 i1="07" i2="X" l="SPA"><s0>Toxicidad</s0>
<s5>23</s5>
</fC03>
<fC03 i1="08" i2="X" l="FRE"><s0>Acipenser transmontanus</s0>
<s2>NS</s2>
<s5>49</s5>
</fC03>
<fC03 i1="08" i2="X" l="ENG"><s0>Acipenser transmontanus</s0>
<s2>NS</s2>
<s5>49</s5>
</fC03>
<fC03 i1="08" i2="X" l="SPA"><s0>Acipenser transmontanus</s0>
<s2>NS</s2>
<s5>49</s5>
</fC03>
<fC03 i1="09" i2="X" l="FRE"><s0>Mercure(méthyl)</s0>
<s4>INC</s4>
<s5>87</s5>
</fC03>
<fC07 i1="01" i2="X" l="FRE"><s0>Alimentation</s0>
<s5>17</s5>
</fC07>
<fC07 i1="01" i2="X" l="ENG"><s0>Feeding</s0>
<s5>17</s5>
</fC07>
<fC07 i1="01" i2="X" l="SPA"><s0>Alimentación</s0>
<s5>17</s5>
</fC07>
<fC07 i1="02" i2="X" l="FRE"><s0>Composé organique</s0>
<s2>NA</s2>
<s5>18</s5>
</fC07>
<fC07 i1="02" i2="X" l="ENG"><s0>Organic compounds</s0>
<s2>NA</s2>
<s5>18</s5>
</fC07>
<fC07 i1="02" i2="X" l="SPA"><s0>Compuesto orgánico</s0>
<s2>NA</s2>
<s5>18</s5>
</fC07>
<fC07 i1="03" i2="X" l="FRE"><s0>Pisces</s0>
<s2>NS</s2>
<s5>29</s5>
</fC07>
<fC07 i1="03" i2="X" l="ENG"><s0>Pisces</s0>
<s2>NS</s2>
<s5>29</s5>
</fC07>
<fC07 i1="03" i2="X" l="SPA"><s0>Pisces</s0>
<s2>NS</s2>
<s5>29</s5>
</fC07>
<fC07 i1="04" i2="X" l="FRE"><s0>Vertebrata</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="04" i2="X" l="ENG"><s0>Vertebrata</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="04" i2="X" l="SPA"><s0>Vertebrata</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="05" i2="X" l="FRE"><s0>Acipenseridae</s0>
<s4>INC</s4>
<s5>70</s5>
</fC07>
<fN21><s1>129</s1>
</fN21>
<fN44 i1="01"><s1>OTO</s1>
</fN44>
<fN82><s1>OTO</s1>
</fN82>
</pA>
</standard>
<server><NO>PASCAL 12-0172963 INIST</NO>
<ET>Histopathological alterations of juvenile green (Acipenser medirostris) and white sturgeon (Acipenser transmontanus) exposed to graded levels of dietary methylmercury</ET>
<AU>LEE (Jang-Won); KIM (Jae-Won); NICOLA DE RIU; MONIELLO (Giuseppe); HUNG (Silas S. O.)</AU>
<AF>Department of Animal Science, University of California, One Shields Avenue/Davis, CA 95616-8521/Etats-Unis (1 aut., 5 aut.); Department of Marine Life-Science, Gangwon Provincial College, 115 Gyohang st., Jumunjin-eup Gangneung/Gangwon 210-804/Corée, République de (2 aut.); Department of Animal Biology, University of Sassari, Via Vienna 2/07100 Sassari/Italie (3 aut., 4 aut.)</AF>
<DT>Publication en série; Niveau analytique</DT>
<SO>Aquatic toxicology; ISSN 0166-445X; Coden AQTODG; Pays-Bas; Da. 2012; Vol. 109; Pp. 90-99; Bibl. 3/4 p.</SO>
<LA>Anglais</LA>
<EA>Triplicate groups of juvenile green and white sturgeon (average weight of 30±2g) were exposed to one of four concentrations of dietary methylmercury (MeHg; 0, 25, 50, and 100 mg MeHg/kg diet) for 8 weeks to determine and compare the sensitivity of the two sturgeon species from a histopathological perspective. After 4- and 8-week exposure, histological changes were examined in the kidney, liver, gill, skeletal muscle, and heart muscle of both species using light microscopy. Marked abnormalities were observed in the kidney and liver of both sturgeon species after each exposure period; the abnormalities showed progressive histological alterations in severity with increasing doses and duration of exposure. Renal lesions included tubular epithelium degeneration and necrosis, renal corpuscular disintegration, and interstitial tissue degeneration. The changes observed in the livers of both sturgeon species were glycogen depletion and vacuolar degeneration. In the gill and skeletal and heart muscle of green and white sturgeon fed MeHg-added diets, mild histological changes were observed but did not show pronounced difference between the two species. Although the lowest observed effect concentration in both species was the 25 mg MeHg/kg diet, the histological changes in the kidney and liver were more pronounced at all treatments groups of green sturgeon than those of white sturgeon. The current results on structural changes of kidney and liver (i.e., more severe glycogen depletion and tubular epithelium degeneration in green sturgeon) confirmed our previous results, in that green sturgeon exhibited a higher mortality, lower growth rate, and lower protein, lipid, and energy contents in their whole body than white sturgeon under the same MeHg exposures.</EA>
<CC>002A14D05A; 002A14B02A</CC>
<FD>Altération; Animal jeune; Régime alimentaire; Histopathologie; Milieu aquatique; Ecotoxicologie; Toxicité; Acipenser transmontanus; Mercure(méthyl)</FD>
<FG>Alimentation; Composé organique; Pisces; Vertebrata; Acipenseridae</FG>
<ED>Alteration; Young animal; Diet; Histopathology; Aquatic environment; Ecotoxicology; Toxicity; Acipenser transmontanus</ED>
<EG>Feeding; Organic compounds; Pisces; Vertebrata</EG>
<SD>Alteración; Animal joven; Régimen alimentario; Histopatología; Medio acuático; Ecotoxicología; Toxicidad; Acipenser transmontanus</SD>
<LO>INIST-18841.354000509616490100</LO>
<ID>12-0172963</ID>
</server>
</inist>
</record>

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Histopathological alterations of juvenile green (Acipenser medirostris) and white sturgeon (Acipenser transmontanus) exposed to graded levels of dietary methylmercury

Histopathological alterations of juvenile green (Acipenser medirostris) and white sturgeon (Acipenser transmontanus) exposed to graded levels of dietary methylmercury

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