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Serveur d'exploration sur la rapamycine et les champignons - Curation (Accueil)

Index « MedMesh.i » - entrée « Peptide Initiation Factors »
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List of bibliographic references

Number of relevant bibliographic references: 9.
Ident.Authors (with country if any)Title
001842 (2005) Ryu Matsuo [Japon] ; Hiroyuki Kubota ; Tohru Obata ; Keiji Kito ; Kazuhisa Ota ; Takanari Kitazono ; Setsuro Ibayashi ; Takuma Sasaki ; Mitsuo Iida ; Takashi ItoThe yeast eIF4E-associated protein Eap1p attenuates GCN4 translation upon TOR-inactivation.
001964 (2002) John H. Connor [États-Unis] ; Douglas S. LylesVesicular stomatitis virus infection alters the eIF4F translation initiation complex and causes dephosphorylation of the eIF4E binding protein 4E-BP1.
001A18 (2001) A C Gingras [Canada] ; B. Raught ; N. SonenbergRegulation of translation initiation by FRAP/mTOR.
001A30 (2000) S. Oldham [Suisse] ; J. Montagne ; T. Radimerski ; G. Thomas ; E. HafenGenetic and biochemical characterization of dTOR, the Drosophila homolog of the target of rapamycin.
001A41 (2000) G P Cosentino [Canada] ; T. Schmelzle ; A. Haghighat ; S B Helliwell ; M N Hall ; N. SonenbergEap1p, a novel eukaryotic translation initiation factor 4E-associated protein in Saccharomyces cerevisiae.
001A65 (1998) M. Nanahoshi [Japon] ; T. Nishiuma ; Y. Tsujishita ; K. Hara ; S. Inui ; N. Sakaguchi ; K. YonezawaRegulation of protein phosphatase 2A catalytic activity by alpha4 protein and its yeast homolog Tap42.
001A73 (1998) C. Berset [Suisse] ; H. Trachsel ; M. AltmannThe TOR (target of rapamycin) signal transduction pathway regulates the stability of translation initiation factor eIF4G in the yeast Saccharomyces cerevisiae.
001A84 (1997) M. Polymenis [États-Unis] ; E V SchmidtCoupling of cell division to cell growth by translational control of the G1 cyclin CLN3 in yeast.
001A88 (1997) S M Aronica [États-Unis] ; A C Gingras ; N. Sonenberg ; S. Cooper ; N. Hague ; H E BroxmeyerMacrophage inflammatory protein-1alpha and interferon-inducible protein 10 inhibit synergistically induced growth factor stimulation of MAP kinase activity and suppress phosphorylation of eukaryotic initiation factor 4E and 4E binding protein 1.

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