C-reactive protein, but not low-density lipoprotein cholesterol levels, associate with coronary atheroma regression and cardiovascular events after maximally intensive statin therapy.
Identifieur interne : 001466 ( Ncbi/Curation ); précédent : 001465; suivant : 001467C-reactive protein, but not low-density lipoprotein cholesterol levels, associate with coronary atheroma regression and cardiovascular events after maximally intensive statin therapy.
Auteurs : Rishi Puri ; Steven E. Nissen ; Peter Libby ; Mingyuan Shao ; Christie M. Ballantyne ; Phillip J. Barter ; M John Chapman ; Raimund Erbel ; Joel S. Raichlen ; Kiyoko Uno ; Yu Kataoka ; Stephen J. NichollsSource :
- Circulation [ 1524-4539 ] ; 2013.
Descripteurs français
- KwdFr :
- Acides heptanoïques (administration et posologie), Adulte d'âge moyen, Analyse multivariée, Atorvastatine de calcium, Cholestérol LDL (métabolisme), Estimation de Kaplan-Meier, Femelle, Fluorobenzènes (administration et posologie), Humains, Inhibiteurs de l'hydroxyméthylglutaryl-CoA réductase (administration et posologie), Lipoprotéines LDL (métabolisme), Maladie des artères coronaires (mortalité), Maladie des artères coronaires (métabolisme), Maladie des artères coronaires (traitement médicamenteux), Modèles de hasards proportionnels, Mâle, Protéine C-réactive (métabolisme), Pyrimidines (administration et posologie), Pyrroles (administration et posologie), Rosuvastatine de calcium, Sujet âgé, Sulfonamides (administration et posologie), Études de suivi.
- MESH :
- administration et posologie : Acides heptanoïques, Fluorobenzènes, Inhibiteurs de l'hydroxyméthylglutaryl-CoA réductase, Pyrimidines, Pyrroles, Sulfonamides.
- mortalité : Maladie des artères coronaires.
- métabolisme : Cholestérol LDL, Lipoprotéines LDL, Maladie des artères coronaires, Protéine C-réactive.
- traitement médicamenteux : Maladie des artères coronaires.
- Adulte d'âge moyen, Analyse multivariée, Atorvastatine de calcium, Estimation de Kaplan-Meier, Femelle, Humains, Modèles de hasards proportionnels, Mâle, Rosuvastatine de calcium, Sujet âgé, Études de suivi.
English descriptors
- KwdEn :
- Aged, Atorvastatin Calcium, C-Reactive Protein (metabolism), Cholesterol, LDL (metabolism), Coronary Artery Disease (drug therapy), Coronary Artery Disease (metabolism), Coronary Artery Disease (mortality), Female, Fluorobenzenes (administration & dosage), Follow-Up Studies, Heptanoic Acids (administration & dosage), Humans, Hydroxymethylglutaryl-CoA Reductase Inhibitors (administration & dosage), Kaplan-Meier Estimate, Lipoproteins, LDL (metabolism), Male, Middle Aged, Multivariate Analysis, Proportional Hazards Models, Pyrimidines (administration & dosage), Pyrroles (administration & dosage), Rosuvastatin Calcium, Sulfonamides (administration & dosage).
- MESH :
- chemical , administration & dosage : Fluorobenzenes, Heptanoic Acids, Hydroxymethylglutaryl-CoA Reductase Inhibitors, Pyrimidines, Pyrroles, Sulfonamides.
- chemical , metabolism : C-Reactive Protein, Cholesterol, LDL, Lipoproteins, LDL.
- chemical : Atorvastatin Calcium, Rosuvastatin Calcium.
- drug therapy : Coronary Artery Disease.
- metabolism : Coronary Artery Disease.
- mortality : Coronary Artery Disease.
- Aged, Female, Follow-Up Studies, Humans, Kaplan-Meier Estimate, Male, Middle Aged, Multivariate Analysis, Proportional Hazards Models.
Abstract
Baseline C-reactive protein (CRP) levels predict major adverse cardiovascular events (MACE: death, myocardial infarction, stroke, coronary revascularization, and hospitalization for unstable angina). The association between changes in CRP levels with plaque progression and MACE in the setting of maximally intensive statin therapy is unknown.
DOI: 10.1161/CIRCULATIONAHA.113.004243
PubMed: 24043299
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Rishi Puri<affiliation><nlm:affiliation>Department of Cardiovascular Medicine, Cleveland Clinic, Cleveland, OH (R.P., S.E.N., K.U., Y.K., S.J.N.); Cardiovascular Division, Brigham and Women's Hospital, Boston, MA (P.L.); C5Research, Cleveland Clinic, Cleveland, OH (M.S.); Section of Cardiovascular Research, Baylor College of Medicine, and the Methodist DeBakey Heart and Vascular Center, Houston, TX (C.M.B.); Centre for Vascular Research, University of New South Wales, Sydney, Australia (P.J.B.); INSERM Dyslipidaemia and Atherosclerosis Research Unit, Hôpital de la Pitié, Paris, France (M.J.C.); West German Heart Center, Essen, Germany (R.E.); AstraZeneca, Wilmington, DE (J.S.R.); and South Australian Health and Medical Research Institute, University of Adelaide, Adelaide, Australia (S.J.N.).</nlm:affiliation><wicri:noCountry code="subField">Australia (S.J.N.).</wicri:noCountry></affiliation>Le document en format XML
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Nissen</name></author><author><name sortKey="Libby, Peter" sort="Libby, Peter" uniqKey="Libby P" first="Peter" last="Libby">Peter Libby</name></author><author><name sortKey="Shao, Mingyuan" sort="Shao, Mingyuan" uniqKey="Shao M" first="Mingyuan" last="Shao">Mingyuan Shao</name></author><author><name sortKey="Ballantyne, Christie M" sort="Ballantyne, Christie M" uniqKey="Ballantyne C" first="Christie M" last="Ballantyne">Christie M. Ballantyne</name></author><author><name sortKey="Barter, Phillip J" sort="Barter, Phillip J" uniqKey="Barter P" first="Phillip J" last="Barter">Phillip J. Barter</name></author><author><name sortKey="Chapman, M John" sort="Chapman, M John" uniqKey="Chapman M" first="M John" last="Chapman">M John Chapman</name></author><author><name sortKey="Erbel, Raimund" sort="Erbel, Raimund" uniqKey="Erbel R" first="Raimund" last="Erbel">Raimund Erbel</name></author><author><name sortKey="Raichlen, Joel S" sort="Raichlen, Joel S" uniqKey="Raichlen J" first="Joel S" last="Raichlen">Joel S. Raichlen</name></author><author><name sortKey="Uno, Kiyoko" sort="Uno, Kiyoko" uniqKey="Uno K" first="Kiyoko" last="Uno">Kiyoko Uno</name></author><author><name sortKey="Kataoka, Yu" sort="Kataoka, Yu" uniqKey="Kataoka Y" first="Yu" last="Kataoka">Yu Kataoka</name></author><author><name sortKey="Nicholls, Stephen J" sort="Nicholls, Stephen J" uniqKey="Nicholls S" first="Stephen J" last="Nicholls">Stephen J. Nicholls</name></author></analytic><series><title level="j">Circulation</title><idno type="eISSN">1524-4539</idno><imprint><date when="2013" type="published">2013</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Atorvastatin Calcium</term><term>C-Reactive Protein (metabolism)</term><term>Cholesterol, LDL (metabolism)</term><term>Coronary Artery Disease (drug therapy)</term><term>Coronary Artery Disease (metabolism)</term><term>Coronary Artery Disease (mortality)</term><term>Female</term><term>Fluorobenzenes (administration & dosage)</term><term>Follow-Up Studies</term><term>Heptanoic Acids (administration & dosage)</term><term>Humans</term><term>Hydroxymethylglutaryl-CoA Reductase Inhibitors (administration & dosage)</term><term>Kaplan-Meier Estimate</term><term>Lipoproteins, LDL (metabolism)</term><term>Male</term><term>Middle Aged</term><term>Multivariate Analysis</term><term>Proportional Hazards Models</term><term>Pyrimidines (administration & dosage)</term><term>Pyrroles (administration & dosage)</term><term>Rosuvastatin Calcium</term><term>Sulfonamides (administration & dosage)</term></keywords><keywords scheme="KwdFr" xml:lang="fr"><term>Acides heptanoïques (administration et posologie)</term><term>Adulte d'âge moyen</term><term>Analyse multivariée</term><term>Atorvastatine de calcium</term><term>Cholestérol LDL (métabolisme)</term><term>Estimation de Kaplan-Meier</term><term>Femelle</term><term>Fluorobenzènes (administration et posologie)</term><term>Humains</term><term>Inhibiteurs de l'hydroxyméthylglutaryl-CoA réductase (administration et posologie)</term><term>Lipoprotéines LDL (métabolisme)</term><term>Maladie des artères coronaires (mortalité)</term><term>Maladie des artères coronaires (métabolisme)</term><term>Maladie des artères coronaires (traitement médicamenteux)</term><term>Modèles de hasards proportionnels</term><term>Mâle</term><term>Protéine C-réactive (métabolisme)</term><term>Pyrimidines (administration et posologie)</term><term>Pyrroles (administration et posologie)</term><term>Rosuvastatine de calcium</term><term>Sujet âgé</term><term>Sulfonamides (administration et posologie)</term><term>Études de suivi</term></keywords><keywords scheme="MESH" type="chemical" qualifier="administration & dosage" xml:lang="en"><term>Fluorobenzenes</term><term>Heptanoic Acids</term><term>Hydroxymethylglutaryl-CoA Reductase Inhibitors</term><term>Pyrimidines</term><term>Pyrroles</term><term>Sulfonamides</term></keywords><keywords scheme="MESH" type="chemical" qualifier="metabolism" xml:lang="en"><term>C-Reactive Protein</term><term>Cholesterol, LDL</term><term>Lipoproteins, LDL</term></keywords><keywords scheme="MESH" type="chemical" xml:lang="en"><term>Atorvastatin Calcium</term><term>Rosuvastatin Calcium</term></keywords><keywords scheme="MESH" qualifier="administration et posologie" xml:lang="fr"><term>Acides heptanoïques</term><term>Fluorobenzènes</term><term>Inhibiteurs de l'hydroxyméthylglutaryl-CoA réductase</term><term>Pyrimidines</term><term>Pyrroles</term><term>Sulfonamides</term></keywords><keywords scheme="MESH" qualifier="drug therapy" xml:lang="en"><term>Coronary Artery Disease</term></keywords><keywords scheme="MESH" qualifier="metabolism" xml:lang="en"><term>Coronary Artery Disease</term></keywords><keywords scheme="MESH" qualifier="mortality" xml:lang="en"><term>Coronary Artery Disease</term></keywords><keywords scheme="MESH" qualifier="mortalité" xml:lang="fr"><term>Maladie des artères coronaires</term></keywords><keywords scheme="MESH" qualifier="métabolisme" xml:lang="fr"><term>Cholestérol LDL</term><term>Lipoprotéines LDL</term><term>Maladie des artères coronaires</term><term>Protéine C-réactive</term></keywords><keywords scheme="MESH" qualifier="traitement médicamenteux" xml:lang="fr"><term>Maladie des artères coronaires</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Female</term><term>Follow-Up Studies</term><term>Humans</term><term>Kaplan-Meier Estimate</term><term>Male</term><term>Middle Aged</term><term>Multivariate Analysis</term><term>Proportional Hazards Models</term></keywords><keywords scheme="MESH" xml:lang="fr"><term>Adulte d'âge moyen</term><term>Analyse multivariée</term><term>Atorvastatine de calcium</term><term>Estimation de Kaplan-Meier</term><term>Femelle</term><term>Humains</term><term>Modèles de hasards proportionnels</term><term>Mâle</term><term>Rosuvastatine de calcium</term><term>Sujet âgé</term><term>Études de suivi</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Baseline C-reactive protein (CRP) levels predict major adverse cardiovascular events (MACE: death, myocardial infarction, stroke, coronary revascularization, and hospitalization for unstable angina). The association between changes in CRP levels with plaque progression and MACE in the setting of maximally intensive statin therapy is unknown.</div></front></TEI></record>Pour manipuler ce document sous Unix (Dilib)
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