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La maladie de Parkinson au Canada (serveur d'exploration)

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Absence of cognitive deficits following deep brain stimulation of the subthalamic nucleus for the treatment of Parkinson's disease

Identifieur interne : 000633 ( PascalFrancis/Corpus ); précédent : 000632; suivant : 000634

Absence of cognitive deficits following deep brain stimulation of the subthalamic nucleus for the treatment of Parkinson's disease

Auteurs : Maria Fraraccio ; Alain Ptito ; Abbas Sadikot ; Michel Panisset ; Alain Dagher

Source :

RBID : Pascal:08-0378728

Descripteurs français

English descriptors

Abstract

Electrical stimulation of the subthalamic nucleus is an effective treatment for the motor symptoms of Parkinson's disease. While most patients who undergo this procedure do not appear to suffer behavioral side effects, a minority experience cognitive or emotional deficits, and longitudinal studies have reported declines; however, the measures of cognitive function used have been limited. One explanation for the possible disturbance of cognitive functions is that electrical stimulation of the subthalamic nucleus disrupts the normal flow of information within cortico-striatal loops involving prefrontal, associative, or limbic cortex. We wished to assess the effect of high frequency electrical stimulation of the subthalamic nucleus in Parkinson's disease patients while they performed a comprehensive neuropsychological test battery. We selected cognitive tasks known to test the function of different cortical areas, including tests of executive function, cognitive flexibility, attention, memory, language and visual perception. Patients were tested on two separate days, with the stimulators turned on or off. Test scores were also compared to preoperative performance. In our sample of 15 patients without dementia or major depression there was no deterioration on any cognitive test as a result of stimulation. We conclude that electrical stimulation of the motor subthalamic nucleus does not cause appreciable declines in cognitive function in well-selected patients.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

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A02 01      @0 ACNEET
A03   1    @0 Arch. clin. neuropsychol.
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A06       @2 4
A08 01  1  ENG  @1 Absence of cognitive deficits following deep brain stimulation of the subthalamic nucleus for the treatment of Parkinson's disease
A11 01  1    @1 FRARACCIO (Maria)
A11 02  1    @1 PTITO (Alain)
A11 03  1    @1 SADIKOT (Abbas)
A11 04  1    @1 PANISSET (Michel)
A11 05  1    @1 DAGHER (Alain)
A14 01      @1 Division of Cognitive Neuroscience, Montreal Neurological Institute, McGill University @2 Montreal, Quebec, H3A 2B4 @3 CAN @Z 1 aut. @Z 2 aut.
A14 02      @1 Division of Neurosurgery, Montreal Neurological Institute, McGill University @2 Montreal, Quebec, H3A 2B4 @3 CAN @Z 3 aut.
A14 03      @1 Unitd des Troubles du Movement André Barbeau, Pavillion Hôtel-Dieu du Centre, Hospitalier de l'Université de Montréal @2 Québec, H2W ITS @3 CAN @Z 4 aut.
A14 04      @1 Department of Neurology and Neurosurgery, Montreal Neurological Institute, McGill University @2 Montreal, Quebec, H3A 2B4 @3 CAN @Z 5 aut.
A20       @1 399-408
A21       @1 2008
A23 01      @0 ENG
A43 01      @1 INIST @2 21173 @5 354000161793440040
A44       @0 0000 @1 © 2008 INIST-CNRS. All rights reserved.
A45       @0 1 p.
A47 01  1    @0 08-0378728
A60       @1 P
A61       @0 A
A64 01  1    @0 Archives of clinical neuropsychology
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C01 01    ENG  @0 Electrical stimulation of the subthalamic nucleus is an effective treatment for the motor symptoms of Parkinson's disease. While most patients who undergo this procedure do not appear to suffer behavioral side effects, a minority experience cognitive or emotional deficits, and longitudinal studies have reported declines; however, the measures of cognitive function used have been limited. One explanation for the possible disturbance of cognitive functions is that electrical stimulation of the subthalamic nucleus disrupts the normal flow of information within cortico-striatal loops involving prefrontal, associative, or limbic cortex. We wished to assess the effect of high frequency electrical stimulation of the subthalamic nucleus in Parkinson's disease patients while they performed a comprehensive neuropsychological test battery. We selected cognitive tasks known to test the function of different cortical areas, including tests of executive function, cognitive flexibility, attention, memory, language and visual perception. Patients were tested on two separate days, with the stimulators turned on or off. Test scores were also compared to preoperative performance. In our sample of 15 patients without dementia or major depression there was no deterioration on any cognitive test as a result of stimulation. We conclude that electrical stimulation of the motor subthalamic nucleus does not cause appreciable declines in cognitive function in well-selected patients.
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Format Inist (serveur)

NO : PASCAL 08-0378728 INIST
ET : Absence of cognitive deficits following deep brain stimulation of the subthalamic nucleus for the treatment of Parkinson's disease
AU : FRARACCIO (Maria); PTITO (Alain); SADIKOT (Abbas); PANISSET (Michel); DAGHER (Alain)
AF : Division of Cognitive Neuroscience, Montreal Neurological Institute, McGill University/Montreal, Quebec, H3A 2B4/Canada (1 aut., 2 aut.); Division of Neurosurgery, Montreal Neurological Institute, McGill University/Montreal, Quebec, H3A 2B4/Canada (3 aut.); Unitd des Troubles du Movement André Barbeau, Pavillion Hôtel-Dieu du Centre, Hospitalier de l'Université de Montréal/Québec, H2W ITS/Canada (4 aut.); Department of Neurology and Neurosurgery, Montreal Neurological Institute, McGill University/Montreal, Quebec, H3A 2B4/Canada (5 aut.)
DT : Publication en série; Niveau analytique
SO : Archives of clinical neuropsychology; ISSN 0887-6177; Coden ACNEET; Pays-Bas; Da. 2008; Vol. 23; No. 4; Pp. 399-408; Bibl. 1 p.
LA : Anglais
EA : Electrical stimulation of the subthalamic nucleus is an effective treatment for the motor symptoms of Parkinson's disease. While most patients who undergo this procedure do not appear to suffer behavioral side effects, a minority experience cognitive or emotional deficits, and longitudinal studies have reported declines; however, the measures of cognitive function used have been limited. One explanation for the possible disturbance of cognitive functions is that electrical stimulation of the subthalamic nucleus disrupts the normal flow of information within cortico-striatal loops involving prefrontal, associative, or limbic cortex. We wished to assess the effect of high frequency electrical stimulation of the subthalamic nucleus in Parkinson's disease patients while they performed a comprehensive neuropsychological test battery. We selected cognitive tasks known to test the function of different cortical areas, including tests of executive function, cognitive flexibility, attention, memory, language and visual perception. Patients were tested on two separate days, with the stimulators turned on or off. Test scores were also compared to preoperative performance. In our sample of 15 patients without dementia or major depression there was no deterioration on any cognitive test as a result of stimulation. We conclude that electrical stimulation of the motor subthalamic nucleus does not cause appreciable declines in cognitive function in well-selected patients.
CC : 002B18C13; 002B17G; 002B17A01
FD : Trouble cognitif; Noyau sousthalamique; Traitement; Maladie de Parkinson; Haute fréquence; Neuropsychologie; Fonction exécutive; Cognition; Trouble motricité; Homme; Stimulation cérébrale profonde
FG : Encéphale; Système nerveux central; Pathologie de l'encéphale; Syndrome extrapyramidal; Maladie dégénérative; Pathologie du système nerveux central; Pathologie du système nerveux
ED : Cognitive disorder; Subthalamic nucleus; Treatment; Parkinson disease; High frequency; Neuropsychologia; Executive function; Cognition; Motility disorder; Human; Deep brain stimulation
EG : Encephalon; Central nervous system; Cerebral disorder; Extrapyramidal syndrome; Degenerative disease; Central nervous system disease; Nervous system diseases
SD : Trastorno cognitivo; Núcleo subtalámico; Tratamiento; Parkinson enfermedad; Alta frecuencia; Neurosicología; Función ejecutiva; Cognición; Trastorno motilidad; Hombre; Estimulación cerebral profunda
LO : INIST-21173.354000161793440040
ID : 08-0378728

Links to Exploration step

Pascal:08-0378728

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<s5>01</s5>
</fC03>
<fC03 i1="02" i2="X" l="FRE">
<s0>Noyau sousthalamique</s0>
<s5>02</s5>
</fC03>
<fC03 i1="02" i2="X" l="ENG">
<s0>Subthalamic nucleus</s0>
<s5>02</s5>
</fC03>
<fC03 i1="02" i2="X" l="SPA">
<s0>Núcleo subtalámico</s0>
<s5>02</s5>
</fC03>
<fC03 i1="03" i2="X" l="FRE">
<s0>Traitement</s0>
<s5>03</s5>
</fC03>
<fC03 i1="03" i2="X" l="ENG">
<s0>Treatment</s0>
<s5>03</s5>
</fC03>
<fC03 i1="03" i2="X" l="SPA">
<s0>Tratamiento</s0>
<s5>03</s5>
</fC03>
<fC03 i1="04" i2="X" l="FRE">
<s0>Maladie de Parkinson</s0>
<s2>NM</s2>
<s5>04</s5>
</fC03>
<fC03 i1="04" i2="X" l="ENG">
<s0>Parkinson disease</s0>
<s2>NM</s2>
<s5>04</s5>
</fC03>
<fC03 i1="04" i2="X" l="SPA">
<s0>Parkinson enfermedad</s0>
<s2>NM</s2>
<s5>04</s5>
</fC03>
<fC03 i1="05" i2="X" l="FRE">
<s0>Haute fréquence</s0>
<s5>05</s5>
</fC03>
<fC03 i1="05" i2="X" l="ENG">
<s0>High frequency</s0>
<s5>05</s5>
</fC03>
<fC03 i1="05" i2="X" l="SPA">
<s0>Alta frecuencia</s0>
<s5>05</s5>
</fC03>
<fC03 i1="06" i2="X" l="FRE">
<s0>Neuropsychologie</s0>
<s5>06</s5>
</fC03>
<fC03 i1="06" i2="X" l="ENG">
<s0>Neuropsychologia</s0>
<s5>06</s5>
</fC03>
<fC03 i1="06" i2="X" l="SPA">
<s0>Neurosicología</s0>
<s5>06</s5>
</fC03>
<fC03 i1="07" i2="X" l="FRE">
<s0>Fonction exécutive</s0>
<s5>07</s5>
</fC03>
<fC03 i1="07" i2="X" l="ENG">
<s0>Executive function</s0>
<s5>07</s5>
</fC03>
<fC03 i1="07" i2="X" l="SPA">
<s0>Función ejecutiva</s0>
<s5>07</s5>
</fC03>
<fC03 i1="08" i2="X" l="FRE">
<s0>Cognition</s0>
<s5>08</s5>
</fC03>
<fC03 i1="08" i2="X" l="ENG">
<s0>Cognition</s0>
<s5>08</s5>
</fC03>
<fC03 i1="08" i2="X" l="SPA">
<s0>Cognición</s0>
<s5>08</s5>
</fC03>
<fC03 i1="09" i2="X" l="FRE">
<s0>Trouble motricité</s0>
<s5>09</s5>
</fC03>
<fC03 i1="09" i2="X" l="ENG">
<s0>Motility disorder</s0>
<s5>09</s5>
</fC03>
<fC03 i1="09" i2="X" l="SPA">
<s0>Trastorno motilidad</s0>
<s5>09</s5>
</fC03>
<fC03 i1="10" i2="X" l="FRE">
<s0>Homme</s0>
<s5>18</s5>
</fC03>
<fC03 i1="10" i2="X" l="ENG">
<s0>Human</s0>
<s5>18</s5>
</fC03>
<fC03 i1="10" i2="X" l="SPA">
<s0>Hombre</s0>
<s5>18</s5>
</fC03>
<fC03 i1="11" i2="X" l="FRE">
<s0>Stimulation cérébrale profonde</s0>
<s4>CD</s4>
<s5>96</s5>
</fC03>
<fC03 i1="11" i2="X" l="ENG">
<s0>Deep brain stimulation</s0>
<s4>CD</s4>
<s5>96</s5>
</fC03>
<fC03 i1="11" i2="X" l="SPA">
<s0>Estimulación cerebral profunda</s0>
<s4>CD</s4>
<s5>96</s5>
</fC03>
<fC07 i1="01" i2="X" l="FRE">
<s0>Encéphale</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="ENG">
<s0>Encephalon</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="SPA">
<s0>Encéfalo</s0>
<s5>37</s5>
</fC07>
<fC07 i1="02" i2="X" l="FRE">
<s0>Système nerveux central</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="ENG">
<s0>Central nervous system</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="SPA">
<s0>Sistema nervioso central</s0>
<s5>38</s5>
</fC07>
<fC07 i1="03" i2="X" l="FRE">
<s0>Pathologie de l'encéphale</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="ENG">
<s0>Cerebral disorder</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="SPA">
<s0>Encéfalo patología</s0>
<s5>39</s5>
</fC07>
<fC07 i1="04" i2="X" l="FRE">
<s0>Syndrome extrapyramidal</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="ENG">
<s0>Extrapyramidal syndrome</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="SPA">
<s0>Extrapiramidal síndrome</s0>
<s5>40</s5>
</fC07>
<fC07 i1="05" i2="X" l="FRE">
<s0>Maladie dégénérative</s0>
<s5>41</s5>
</fC07>
<fC07 i1="05" i2="X" l="ENG">
<s0>Degenerative disease</s0>
<s5>41</s5>
</fC07>
<fC07 i1="05" i2="X" l="SPA">
<s0>Enfermedad degenerativa</s0>
<s5>41</s5>
</fC07>
<fC07 i1="06" i2="X" l="FRE">
<s0>Pathologie du système nerveux central</s0>
<s5>42</s5>
</fC07>
<fC07 i1="06" i2="X" l="ENG">
<s0>Central nervous system disease</s0>
<s5>42</s5>
</fC07>
<fC07 i1="06" i2="X" l="SPA">
<s0>Sistema nervosio central patología</s0>
<s5>42</s5>
</fC07>
<fC07 i1="07" i2="X" l="FRE">
<s0>Pathologie du système nerveux</s0>
<s5>43</s5>
</fC07>
<fC07 i1="07" i2="X" l="ENG">
<s0>Nervous system diseases</s0>
<s5>43</s5>
</fC07>
<fC07 i1="07" i2="X" l="SPA">
<s0>Sistema nervioso patología</s0>
<s5>43</s5>
</fC07>
<fN21>
<s1>245</s1>
</fN21>
</pA>
</standard>
<server>
<NO>PASCAL 08-0378728 INIST</NO>
<ET>Absence of cognitive deficits following deep brain stimulation of the subthalamic nucleus for the treatment of Parkinson's disease</ET>
<AU>FRARACCIO (Maria); PTITO (Alain); SADIKOT (Abbas); PANISSET (Michel); DAGHER (Alain)</AU>
<AF>Division of Cognitive Neuroscience, Montreal Neurological Institute, McGill University/Montreal, Quebec, H3A 2B4/Canada (1 aut., 2 aut.); Division of Neurosurgery, Montreal Neurological Institute, McGill University/Montreal, Quebec, H3A 2B4/Canada (3 aut.); Unitd des Troubles du Movement André Barbeau, Pavillion Hôtel-Dieu du Centre, Hospitalier de l'Université de Montréal/Québec, H2W ITS/Canada (4 aut.); Department of Neurology and Neurosurgery, Montreal Neurological Institute, McGill University/Montreal, Quebec, H3A 2B4/Canada (5 aut.)</AF>
<DT>Publication en série; Niveau analytique</DT>
<SO>Archives of clinical neuropsychology; ISSN 0887-6177; Coden ACNEET; Pays-Bas; Da. 2008; Vol. 23; No. 4; Pp. 399-408; Bibl. 1 p.</SO>
<LA>Anglais</LA>
<EA>Electrical stimulation of the subthalamic nucleus is an effective treatment for the motor symptoms of Parkinson's disease. While most patients who undergo this procedure do not appear to suffer behavioral side effects, a minority experience cognitive or emotional deficits, and longitudinal studies have reported declines; however, the measures of cognitive function used have been limited. One explanation for the possible disturbance of cognitive functions is that electrical stimulation of the subthalamic nucleus disrupts the normal flow of information within cortico-striatal loops involving prefrontal, associative, or limbic cortex. We wished to assess the effect of high frequency electrical stimulation of the subthalamic nucleus in Parkinson's disease patients while they performed a comprehensive neuropsychological test battery. We selected cognitive tasks known to test the function of different cortical areas, including tests of executive function, cognitive flexibility, attention, memory, language and visual perception. Patients were tested on two separate days, with the stimulators turned on or off. Test scores were also compared to preoperative performance. In our sample of 15 patients without dementia or major depression there was no deterioration on any cognitive test as a result of stimulation. We conclude that electrical stimulation of the motor subthalamic nucleus does not cause appreciable declines in cognitive function in well-selected patients.</EA>
<CC>002B18C13; 002B17G; 002B17A01</CC>
<FD>Trouble cognitif; Noyau sousthalamique; Traitement; Maladie de Parkinson; Haute fréquence; Neuropsychologie; Fonction exécutive; Cognition; Trouble motricité; Homme; Stimulation cérébrale profonde</FD>
<FG>Encéphale; Système nerveux central; Pathologie de l'encéphale; Syndrome extrapyramidal; Maladie dégénérative; Pathologie du système nerveux central; Pathologie du système nerveux</FG>
<ED>Cognitive disorder; Subthalamic nucleus; Treatment; Parkinson disease; High frequency; Neuropsychologia; Executive function; Cognition; Motility disorder; Human; Deep brain stimulation</ED>
<EG>Encephalon; Central nervous system; Cerebral disorder; Extrapyramidal syndrome; Degenerative disease; Central nervous system disease; Nervous system diseases</EG>
<SD>Trastorno cognitivo; Núcleo subtalámico; Tratamiento; Parkinson enfermedad; Alta frecuencia; Neurosicología; Función ejecutiva; Cognición; Trastorno motilidad; Hombre; Estimulación cerebral profunda</SD>
<LO>INIST-21173.354000161793440040</LO>
<ID>08-0378728</ID>
</server>
</inist>
</record>

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