Myxomatous mitral valve disease and related entities: The role of matrix in valvular heart disease
Identifieur interne : 003F58 ( Main/Exploration ); précédent : 003F57; suivant : 003F59Myxomatous mitral valve disease and related entities: The role of matrix in valvular heart disease
Auteurs : Wanda M. Lester [Canada]Source :
- Cardiovascular Pathology [ 1054-8807 ] ; 1995.
Abstract
Myxomatous mitral valve disease is a cause of prolapse of mitral valve leaflet(s) into the left atrium. In this condition there is a loss of collagen from the leaflet and an accumulation of glycosaminoglycan. Complications of myxomatous mitral valve disease include infective endocarditis, mitral regurgitation, sudden death, and stroke. Mitral valve prolapse has been reported to occur in association with many syndromes. In some of these, such as Marfan syndrome, the pathology of the mitral valve has been well described and is that of a myxomatous valve. However, in some of the other disorders associated with mitral valve prolapse, there is no information yet available on the valve pathology, and this would be useful in order to better understand the pathogenesis of myxomatous mitral valve disease.
Url:
DOI: 10.1016/1054-8807(95)00052-7
Affiliations:
Links toward previous steps (curation, corpus...)
- to stream Istex, to step Corpus: 002409
- to stream Istex, to step Curation: 002409
- to stream Istex, to step Checkpoint: 001932
- to stream Main, to step Merge: 004680
- to stream Main, to step Curation: 003F58
Le document en format XML
<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title>Myxomatous mitral valve disease and related entities: The role of matrix in valvular heart disease</title>
<author><name sortKey="Lester, Wanda M" sort="Lester, Wanda M" uniqKey="Lester W" first="Wanda M." last="Lester">Wanda M. Lester</name>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">ISTEX</idno>
<idno type="RBID">ISTEX:EB7485855C0DBA7004352E7362ABD014CC89FF3D</idno>
<date when="1995" year="1995">1995</date>
<idno type="doi">10.1016/1054-8807(95)00052-7</idno>
<idno type="url">https://api-v5.istex.fr/document/EB7485855C0DBA7004352E7362ABD014CC89FF3D/fulltext/pdf</idno>
<idno type="wicri:Area/Istex/Corpus">002409</idno>
<idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Corpus" wicri:corpus="ISTEX">002409</idno>
<idno type="wicri:Area/Istex/Curation">002409</idno>
<idno type="wicri:Area/Istex/Checkpoint">001932</idno>
<idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Checkpoint">001932</idno>
<idno type="wicri:doubleKey">1054-8807:1995:Lester W:myxomatous:mitral:valve</idno>
<idno type="wicri:Area/Main/Merge">004680</idno>
<idno type="wicri:Area/Main/Curation">003F58</idno>
<idno type="wicri:Area/Main/Exploration">003F58</idno>
</publicationStmt>
<sourceDesc><biblStruct><analytic><title level="a">Myxomatous mitral valve disease and related entities: The role of matrix in valvular heart disease</title>
<author><name sortKey="Lester, Wanda M" sort="Lester, Wanda M" uniqKey="Lester W" first="Wanda M." last="Lester">Wanda M. Lester</name>
<affiliation wicri:level="4"><country xml:lang="fr">Canada</country>
<wicri:regionArea>Department of Pathology, Foothills Hospital and the University of Calgary, Alberta</wicri:regionArea>
<orgName type="university">Université de Calgary</orgName>
<placeName><settlement type="city">Calgary</settlement>
<region type="state">Alberta</region>
</placeName>
</affiliation>
</author>
</analytic>
<monogr></monogr>
<series><title level="j">Cardiovascular Pathology</title>
<title level="j" type="abbrev">CVP</title>
<idno type="ISSN">1054-8807</idno>
<imprint><publisher>ELSEVIER</publisher>
<date type="published" when="1995">1995</date>
<biblScope unit="volume">4</biblScope>
<biblScope unit="issue">4</biblScope>
<biblScope unit="page" from="257">257</biblScope>
<biblScope unit="page" to="264">264</biblScope>
</imprint>
<idno type="ISSN">1054-8807</idno>
</series>
</biblStruct>
</sourceDesc>
<seriesStmt><idno type="ISSN">1054-8807</idno>
</seriesStmt>
</fileDesc>
<profileDesc><textClass></textClass>
<langUsage><language ident="en">en</language>
</langUsage>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">Myxomatous mitral valve disease is a cause of prolapse of mitral valve leaflet(s) into the left atrium. In this condition there is a loss of collagen from the leaflet and an accumulation of glycosaminoglycan. Complications of myxomatous mitral valve disease include infective endocarditis, mitral regurgitation, sudden death, and stroke. Mitral valve prolapse has been reported to occur in association with many syndromes. In some of these, such as Marfan syndrome, the pathology of the mitral valve has been well described and is that of a myxomatous valve. However, in some of the other disorders associated with mitral valve prolapse, there is no information yet available on the valve pathology, and this would be useful in order to better understand the pathogenesis of myxomatous mitral valve disease.</div>
</front>
</TEI>
<affiliations><list><country><li>Canada</li>
</country>
<region><li>Alberta</li>
</region>
<settlement><li>Calgary</li>
</settlement>
<orgName><li>Université de Calgary</li>
</orgName>
</list>
<tree><country name="Canada"><region name="Alberta"><name sortKey="Lester, Wanda M" sort="Lester, Wanda M" uniqKey="Lester W" first="Wanda M." last="Lester">Wanda M. Lester</name>
</region>
</country>
</tree>
</affiliations>
</record>
Pour manipuler ce document sous Unix (Dilib)
EXPLOR_STEP=$WICRI_ROOT/Wicri/Canada/explor/ParkinsonCanadaV1/Data/Main/Exploration
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 003F58 | SxmlIndent | more
Ou
HfdSelect -h $EXPLOR_AREA/Data/Main/Exploration/biblio.hfd -nk 003F58 | SxmlIndent | more
Pour mettre un lien sur cette page dans le réseau Wicri
{{Explor lien |wiki= Wicri/Canada |area= ParkinsonCanadaV1 |flux= Main |étape= Exploration |type= RBID |clé= ISTEX:EB7485855C0DBA7004352E7362ABD014CC89FF3D |texte= Myxomatous mitral valve disease and related entities: The role of matrix in valvular heart disease }}
This area was generated with Dilib version V0.6.29. |