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Serveur d'exploration sur le cobalt au Maghreb - Curation (Istex)

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List of bibliographic references

Number of relevant bibliographic references: 8.
Ident.Authors (with country if any)Title
000578 (1992) Stephen M. Oppenheimer [Canada] ; Tarek Saleh [Canada] ; David F. Cechetto [Canada]Lateral hypothalamic area neurotransmission and neuromodulation of the specific cardiac effects of insular cortex stimulation
000594 (1986) N. Riveros [Chili] ; F. Orrego [Chili]N -methylaspartate-activated calcium channels in rat brain cortex slices. Effect of calcium channel blockers and of inhibitory and depressant substances
000814 (1996) Nikolaj K. Isaev [Russie] ; Dmitry B. Zorov [Russie] ; Elena V. Stelmashook [Russie] ; Rustem E. Uzbekov [Russie] ; Maxim B. Kozhemyakin [Russie] ; Ilya V. Victorov [Russie]Neurotoxic glutamate treatment of cultured cerebellar granule cells induces Ca2+-dependent collapse of mitochondrial membrane potential and ultrastructural alterations of mitochondria
000818 (1990) P. Calabresi [Italie] ; N. B. Mercuri [Italie] ; M. De Murtas [Italie] ; G. Bernardi [Italie]Endogenous GABA mediates presynaptic inhibition of spontaneous and evoked excitatory synaptic potentials in the rat neostriatum
000820 (1998) Nikolaj K. Isaev [Russie] ; Elena V. Stelmashook [Russie] ; Olga P. Alexandrova [Russie] ; Nadezhda A. Andreeva [Russie] ; Irina A. Polyakova [Russie] ; Ilya V. Victorov [Russie] ; Dmitry B. Zorov [Russie]The lack of extracellular Na+ exacerbates Ca2+-dependent damage of cultured cerebellar granule cells
000B84 (2004) Victoria L. Harvey [Royaume-Uni] ; Gary J. Stephens [Royaume-Uni]Mechanism of GABAB receptor‐mediated inhibition of spontaneous GABA release onto cerebellar Purkinje cells
000C92 (1995) Andras Papp [Allemagne] ; Manfred R. Klee [Allemagne]Alterations in the action potential of Aplysia neurons evoked by a phorbolester are mediated by protein kinase C
000E98 (1987) O. W Witte [Allemagne] ; E.-J Speckmann [Allemagne] ; J. Walden [Allemagne]Motor cortical epileptic foci in vivo: actions of a calcium channel blocker on paroxysmal neuronal depolarizations

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