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Index « Auteurs » - entrée « Katia Aquilano »
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Kati M. Donner < Katia Aquilano < Katia M C. Verhamme  Facettes :

List of bibliographic references

Number of relevant bibliographic references: 8.
Ident.Authors (with country if any)Title
000957 (2013) Katia Aquilano ; Sara Baldelli ; Maria R. CirioloNuclear Recruitment of Neuronal Nitric-oxide Synthase by α-Syntrophin Is Crucial for the Induction of Mitochondrial Biogenesis*
000A11 (2014) Daniele Lettieri Barbato [Italie] ; Giuseppe Tatulli [Italie] ; Katia Aquilano [Italie] ; Maria R. Ciriolo [Italie]Inhibition of Age-Related Cytokines Production by ATGL: A Mechanism Linked to the Anti-Inflammatory Effect of Resveratrol
000A66 (2014) Katia Aquilano [Italie] ; Sara Baldelli [Italie] ; Maria R. Ciriolo [Italie]Glutathione: new roles in redox signaling for an old antioxidant
000A68 (2014) Sara Baldelli [Italie] ; Daniele Lettieri Barbato [Italie] ; Giuseppe Tatulli [Italie] ; Katia Aquilano [Italie] ; Maria Rosa Ciriolo [Italie]The role of nNOS and PGC-1α in skeletal muscle cells.
000B87 (2015) Daniele Lettieri Barbato [Italie] ; Giuseppe Tatulli [Italie] ; Stefano Maria Cannata [Italie] ; Sergio Bernardini [Italie] ; Katia Aquilano [Italie] ; Maria R. Ciriolo [Italie]Glutathione Decrement Drives Thermogenic Program In Adipose Cells
000C17 (2015) Daniele Lettieri Barbato [Italie] ; Giuseppe Tatulli [Italie] ; Rolando Vegliante [Italie] ; Stefano M. Cannata [Italie] ; Sergio Bernardini [Italie] ; Maria R. Ciriolo [Italie] ; Katia Aquilano [Italie]Dietary fat overload reprograms brown fat mitochondria
000C20 (2015) Daniele Lettieri Barbato [Italie] ; Giuseppe Tatulli [Italie] ; Katia Aquilano [Italie] ; Maria R. Ciriolo [Italie]Mitochondrial Hormesis links nutrient restriction to improved metabolism in fat cell
000C83 (2016) Katia Aquilano [Italie] ; Sara Baldelli [Italie] ; Livia La Barbera [Italie] ; Daniele Lettieri Barbato [Italie] ; Giuseppe Tatulli [Italie] ; Maria Rosa Ciriolo [Italie]Adipose triglycerides lipase decrement affects skeletal muscle homeostasis during aging through FAs-PPARα-PGC-1α antioxidant response.

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