Normalization of nutritional status by various levels of vitamin E supplementation during murine AIDS
Identifieur interne : 003501 ( Istex/Curation ); précédent : 003500; suivant : 003502Normalization of nutritional status by various levels of vitamin E supplementation during murine AIDS
Auteurs : Yuejian Wang [États-Unis] ; Bailin Liang [États-Unis] ; Ronald R. Watson [États-Unis]Source :
- Nutrition Research [ 0271-5317 ] ; 1994.
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Abstract
Female C57BL/6 mice were infected with LP-BM5 retrovirus, causing murine AIDS which is functionally similar to human AIDS. Dietary supplementation, with a 15-, 150- and 450-fold increase of vitamin E in a liquid diet (National Council Research), significantly restored serum and haptic vitamin A and E which had been reduced by retrovirus infection. They also significantly restored hepatic copper, which had been reduced by retrovirus infection, whereas only 150- and 450-fold vitamin E improved hepatic zinc level. Vitamin E supplementation at all levels had no effects on hepatic zinc and copper levels in normal mice, whereas they significantly increased serum and hepatic vitamin A and E concentrations. Vitamin E supplementation at all levels significantly increased intestinal vitamin A and E levels during murine AIDS, whereas only intestinal vitamin E levels was altered by various levels of vitamin E supplementation. Interestingly, vitamin E supplementation had no effect on intestinal copper level, whereas they significantly increased intestinal zinc level in the normal mice and only 450-fold vitamin E supplementation significantly elevated intestinal level of zinc. These data indicate that dietary vitamin E supplementation at extremely high levels was not toxic, can improve undernutrition initiated by retrovirus infection during progression to murine AIDS, which should favorably affect immune response.
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DOI: 10.1016/S0271-5317(05)80297-0
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Ronald R. Watson<affiliation><mods:affiliation>To whom correspondence should be sent: Ronald R. Watson, Ph.D, Department of Family and Community Medicine, University of Arizona, Tucson, AZ 85724, USA. (602) 626-6001; Fax (602)626-2030</mods:affiliation>
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<front><div type="abstract" xml:lang="en">Female C57BL/6 mice were infected with LP-BM5 retrovirus, causing murine AIDS which is functionally similar to human AIDS. Dietary supplementation, with a 15-, 150- and 450-fold increase of vitamin E in a liquid diet (National Council Research), significantly restored serum and haptic vitamin A and E which had been reduced by retrovirus infection. They also significantly restored hepatic copper, which had been reduced by retrovirus infection, whereas only 150- and 450-fold vitamin E improved hepatic zinc level. Vitamin E supplementation at all levels had no effects on hepatic zinc and copper levels in normal mice, whereas they significantly increased serum and hepatic vitamin A and E concentrations. Vitamin E supplementation at all levels significantly increased intestinal vitamin A and E levels during murine AIDS, whereas only intestinal vitamin E levels was altered by various levels of vitamin E supplementation. Interestingly, vitamin E supplementation had no effect on intestinal copper level, whereas they significantly increased intestinal zinc level in the normal mice and only 450-fold vitamin E supplementation significantly elevated intestinal level of zinc. These data indicate that dietary vitamin E supplementation at extremely high levels was not toxic, can improve undernutrition initiated by retrovirus infection during progression to murine AIDS, which should favorably affect immune response.</div>
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