Antimicrobial resistance with focus on oral beta-lactamases.
Identifieur interne : 000534 ( Main/Corpus ); précédent : 000533; suivant : 000535Antimicrobial resistance with focus on oral beta-lactamases.
Auteurs : T. Handal ; I. OlsenSource :
- European journal of oral sciences [ 0909-8836 ] ; 2000.
English descriptors
- KwdEn :
- MESH :
- chemical , chemistry : Anti-Bacterial Agents.
- chemical , metabolism : beta-Lactamases.
- chemical , pharmacology : Anti-Bacterial Agents.
- drug effects : Cell Wall.
- enzymology : Bacteria.
- microbiology : Mouth.
- physiology : beta-Lactam Resistance.
- Humans, beta-Lactamase Inhibitors, beta-Lactams.
Abstract
Over the last 10-15 yr antibiotic resistance has increased in the oral microflora. The beta-lactam antibiotics, i.e., penicillins and cephalosporins, are the most frequently used antimicrobial agents. Unfortunately, the efficiency of these drugs is increasingly being challenged by the emergence of resistant bacteria, which is mainly due to their production of beta-lactamases. In this paper, mechanisms of antibiotic resistance are reviewed, with emphasis on beta-lactamases. This review also discusses how the presence of beta-lactamases in oral microorganisms may affect the treatment of oral diseases. Dentists can influence the emerging global crisis of antibiotic resistance by carefully evaluating the indications for antibiotic treatment. General guidelines for when and how to use antibiotics in dentistry are reviewed.
DOI: 10.1034/j.1600-0722.2000.108003163.x
PubMed: 10872985
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pubmed:10872985Le document en format XML
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Olsen</name></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="2000">2000</date><idno type="RBID">pubmed:10872985</idno><idno type="pmid">10872985</idno><idno type="doi">10.1034/j.1600-0722.2000.108003163.x</idno><idno type="wicri:Area/Main/Corpus">000534</idno><idno type="wicri:explorRef" wicri:stream="Main" wicri:step="Corpus" wicri:corpus="PubMed">000534</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">Antimicrobial resistance with focus on oral beta-lactamases.</title><author><name sortKey="Handal, T" sort="Handal, T" uniqKey="Handal T" first="T" last="Handal">T. Handal</name><affiliation><nlm:affiliation>Department of Oral Biology, Dental Faculty, University of Oslo, Norway. thandal@odont.uio.no</nlm:affiliation></affiliation></author><author><name sortKey="Olsen, I" sort="Olsen, I" uniqKey="Olsen I" first="I" last="Olsen">I. Olsen</name></author></analytic><series><title level="j">European journal of oral sciences</title><idno type="ISSN">0909-8836</idno><imprint><date when="2000" type="published">2000</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Anti-Bacterial Agents (chemistry)</term><term>Anti-Bacterial Agents (pharmacology)</term><term>Bacteria (enzymology)</term><term>Cell Wall (drug effects)</term><term>Humans (MeSH)</term><term>Mouth (microbiology)</term><term>beta-Lactam Resistance (physiology)</term><term>beta-Lactamase Inhibitors (MeSH)</term><term>beta-Lactamases (metabolism)</term><term>beta-Lactams (MeSH)</term></keywords><keywords scheme="MESH" type="chemical" qualifier="chemistry" xml:lang="en"><term>Anti-Bacterial Agents</term></keywords><keywords scheme="MESH" type="chemical" qualifier="metabolism" xml:lang="en"><term>beta-Lactamases</term></keywords><keywords scheme="MESH" type="chemical" qualifier="pharmacology" xml:lang="en"><term>Anti-Bacterial Agents</term></keywords><keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Cell Wall</term></keywords><keywords scheme="MESH" qualifier="enzymology" xml:lang="en"><term>Bacteria</term></keywords><keywords scheme="MESH" qualifier="microbiology" xml:lang="en"><term>Mouth</term></keywords><keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>beta-Lactam Resistance</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Humans</term><term>beta-Lactamase Inhibitors</term><term>beta-Lactams</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Over the last 10-15 yr antibiotic resistance has increased in the oral microflora. The beta-lactam antibiotics, i.e., penicillins and cephalosporins, are the most frequently used antimicrobial agents. Unfortunately, the efficiency of these drugs is increasingly being challenged by the emergence of resistant bacteria, which is mainly due to their production of beta-lactamases. In this paper, mechanisms of antibiotic resistance are reviewed, with emphasis on beta-lactamases. This review also discusses how the presence of beta-lactamases in oral microorganisms may affect the treatment of oral diseases. Dentists can influence the emerging global crisis of antibiotic resistance by carefully evaluating the indications for antibiotic treatment. General guidelines for when and how to use antibiotics in dentistry are reviewed.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">10872985</PMID><DateCompleted><Year>2000</Year><Month>10</Month><Day>05</Day></DateCompleted><DateRevised><Year>2019</Year><Month>11</Month><Day>04</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0909-8836</ISSN><JournalIssue CitedMedium="Print"><Volume>108</Volume><Issue>3</Issue><PubDate><Year>2000</Year><Month>Jun</Month></PubDate></JournalIssue><Title>European journal of oral sciences</Title><ISOAbbreviation>Eur J Oral Sci</ISOAbbreviation></Journal><ArticleTitle>Antimicrobial resistance with focus on oral beta-lactamases.</ArticleTitle><Pagination><MedlinePgn>163-74</MedlinePgn></Pagination><Abstract><AbstractText>Over the last 10-15 yr antibiotic resistance has increased in the oral microflora. The beta-lactam antibiotics, i.e., penicillins and cephalosporins, are the most frequently used antimicrobial agents. Unfortunately, the efficiency of these drugs is increasingly being challenged by the emergence of resistant bacteria, which is mainly due to their production of beta-lactamases. In this paper, mechanisms of antibiotic resistance are reviewed, with emphasis on beta-lactamases. This review also discusses how the presence of beta-lactamases in oral microorganisms may affect the treatment of oral diseases. Dentists can influence the emerging global crisis of antibiotic resistance by carefully evaluating the indications for antibiotic treatment. General guidelines for when and how to use antibiotics in dentistry are reviewed.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Handal</LastName><ForeName>T</ForeName><Initials>T</Initials><AffiliationInfo><Affiliation>Department of Oral Biology, Dental Faculty, University of Oslo, Norway. thandal@odont.uio.no</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Olsen</LastName><ForeName>I</ForeName><Initials>I</Initials></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D016454">Review</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>England</Country><MedlineTA>Eur J Oral Sci</MedlineTA><NlmUniqueID>9504563</NlmUniqueID><ISSNLinking>0909-8836</ISSNLinking></MedlineJournalInfo><ChemicalList><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D000900">Anti-Bacterial Agents</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D065093">beta-Lactamase Inhibitors</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D047090">beta-Lactams</NameOfSubstance></Chemical><Chemical><RegistryNumber>EC 3.5.2.6</RegistryNumber><NameOfSubstance UI="D001618">beta-Lactamases</NameOfSubstance></Chemical></ChemicalList><CitationSubset>D</CitationSubset><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D000900" MajorTopicYN="N">Anti-Bacterial Agents</DescriptorName><QualifierName UI="Q000737" MajorTopicYN="N">chemistry</QualifierName><QualifierName UI="Q000494" MajorTopicYN="N">pharmacology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D001419" MajorTopicYN="N">Bacteria</DescriptorName><QualifierName UI="Q000201" MajorTopicYN="N">enzymology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D002473" MajorTopicYN="N">Cell Wall</DescriptorName><QualifierName UI="Q000187" MajorTopicYN="N">drug effects</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D009055" MajorTopicYN="N">Mouth</DescriptorName><QualifierName UI="Q000382" MajorTopicYN="Y">microbiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D018440" MajorTopicYN="N">beta-Lactam Resistance</DescriptorName><QualifierName UI="Q000502" MajorTopicYN="Y">physiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D065093" MajorTopicYN="N">beta-Lactamase Inhibitors</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D001618" MajorTopicYN="N">beta-Lactamases</DescriptorName><QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D047090" MajorTopicYN="N">beta-Lactams</DescriptorName></MeshHeading></MeshHeadingList><NumberOfReferences>54</NumberOfReferences></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>2000</Year><Month>6</Month><Day>29</Day><Hour>11</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2000</Year><Month>10</Month><Day>7</Day><Hour>11</Hour><Minute>1</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>2000</Year><Month>6</Month><Day>29</Day><Hour>11</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">10872985</ArticleId><ArticleId IdType="doi">10.1034/j.1600-0722.2000.108003163.x</ArticleId></ArticleIdList></PubmedData></pubmed></record>Pour manipuler ce document sous Unix (Dilib)
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