StressCovidV1, PascalFrancis, Corpus, bibRecord, 000314

Stress-associated immune modulation : Relevance to viral infections and chronic fatigue syndrome

Identifieur interne : 000314 ( PascalFrancis/Corpus ); précédent : 000313; suivant : 000315

Stress-associated immune modulation : Relevance to viral infections and chronic fatigue syndrome

Auteurs : R. Glaser ; J. K. Kiecolt-Glaser

Source :

The American journal of medicine [ 0002-9343 ] ; 1998.

RBID : Pascal:99-0039814

Descripteurs français

Pascal (Inist)
- Fatigue chronique syndrome, Prévalence, Association, Virose, Stress, Facteur risque, Réponse immune, Modulation, Etude comparative, Homme.

English descriptors

KwdEn :
- Association, Chronic fatigue syndrome, Comparative study, Human, Immune response, Modulation, Prevalence, Risk factor, Stress, Viral disease.

Abstract

The frequent association of an active viral infection with the symptoms of CFS led researchers to hypothesize that chronic fatigue syndrome (CFS) is induced by a virus. Results of these studies indicated that despite clinical support for this hypothesis, there were no clear data linking viruses to CFS. In this overview, we will explore the interrelation of the immune, endocrine, and central nervous systems, and the possibility that stress and/or the reactivation/ replication of a latent virus (such as Epstein Barr virus) could modulate the immune system to induce CFS. Relevant research conducted in the developing field of psychoneuroimmunology will be reviewed, with a particular focus on cytokine synthesis, natural killer (NK) cell activity, and T-lymphocyte function, as they relate to CFS.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

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A02	`01`			`@0 AJMEAZ`
A03		`1`		`@0 Am. j. med.`
A05				`@2 105`
A06				`@2 3A`
A08	`01`	`1`	`ENG`	`@1 Stress-associated immune modulation : Relevance to viral infections and chronic fatigue syndrome`
A09	`01`	`1`	`ENG`	`@1 Recent developments in chronic fatigue syndrome`
A11	`01`	`1`		`@1 GLASER (R.)`
A11	`02`	`1`		`@1 KIECOLT-GLASER (J. K.)`
A12	`01`	`1`		`@1 LEVINE (Paul H.) @9 ed.`
A14	`01`			`@1 Department of Medical Microbiology and Immunology and the Department of Psychiatry, Institute for Behavioral Medicine Research, Comprehensive Cancer Center, The Ohio State University College of Medicine @2 Columbus, Ohio @3 USA`
A15	`01`			`@1 George Washington University Medical Center @2 Washington DC @3 USA @Z 1 aut.`
A15	`02`			`@1 Viral Epidemiology Branch, National Cancer Institute, National Institutes of Health @2 Bethesda MD @3 USA @Z 1 aut.`
A20				`@2 35S-42S`
A21				`@1 1998`
A23	`01`			`@0 ENG`
A43	`01`			`@1 INIST @2 4562 @5 354000071365760070`
A44				`@0 0000 @1 © 1999 INIST-CNRS. All rights reserved.`
A45				`@0 81 ref.`
A47	`01`	`1`		`@0 99-0039814`
A60				`@1 P`
A61				`@0 A`
A64		`1`		`@0 The American journal of medicine`
A66	`01`			`@0 USA`
C01	`01`		`ENG`	@0 The frequent association of an active viral infection with the symptoms of CFS led researchers to hypothesize that chronic fatigue syndrome (CFS) is induced by a virus. Results of these studies indicated that despite clinical support for this hypothesis, there were no clear data linking viruses to CFS. In this overview, we will explore the interrelation of the immune, endocrine, and central nervous systems, and the possibility that stress and/or the reactivation/ replication of a latent virus (such as Epstein Barr virus) could modulate the immune system to induce CFS. Relevant research conducted in the developing field of psychoneuroimmunology will be reviewed, with a particular focus on cytokine synthesis, natural killer (NK) cell activity, and T-lymphocyte function, as they relate to CFS.
C02	`01`	`X`		`@0 002B17I`
C03	`01`	`X`	`FRE`	`@0 Fatigue chronique syndrome @2 NM @5 01`
C03	`01`	`X`	`ENG`	`@0 Chronic fatigue syndrome @2 NM @5 01`
C03	`01`	`X`	`SPA`	`@0 Fatiga crónica síndrome @2 NM @5 01`
C03	`02`	`X`	`FRE`	`@0 Prévalence @5 02`
C03	`02`	`X`	`ENG`	`@0 Prevalence @5 02`
C03	`02`	`X`	`SPA`	`@0 Prevalencia @5 02`
C03	`03`	`X`	`FRE`	`@0 Association @5 03`
C03	`03`	`X`	`ENG`	`@0 Association @5 03`
C03	`03`	`X`	`SPA`	`@0 Asociación @5 03`
C03	`04`	`X`	`FRE`	`@0 Virose @5 04`
C03	`04`	`X`	`ENG`	`@0 Viral disease @5 04`
C03	`04`	`X`	`SPA`	`@0 Virosis @5 04`
C03	`05`	`X`	`FRE`	`@0 Stress @5 05`
C03	`05`	`X`	`ENG`	`@0 Stress @5 05`
C03	`05`	`X`	`SPA`	`@0 Stress @5 05`
C03	`06`	`X`	`FRE`	`@0 Facteur risque @5 06`
C03	`06`	`X`	`ENG`	`@0 Risk factor @5 06`
C03	`06`	`X`	`SPA`	`@0 Factor riesgo @5 06`
C03	`07`	`X`	`FRE`	`@0 Réponse immune @5 07`
C03	`07`	`X`	`ENG`	`@0 Immune response @5 07`
C03	`07`	`X`	`SPA`	`@0 Respuesta inmune @5 07`
C03	`08`	`X`	`FRE`	`@0 Modulation @5 08`
C03	`08`	`X`	`ENG`	`@0 Modulation @5 08`
C03	`08`	`X`	`SPA`	`@0 Modulación @5 08`
C03	`09`	`X`	`FRE`	`@0 Etude comparative @5 09`
C03	`09`	`X`	`ENG`	`@0 Comparative study @5 09`
C03	`09`	`X`	`GER`	`@0 Vergleich @5 09`
C03	`09`	`X`	`SPA`	`@0 Estudio comparativo @5 09`
C03	`10`	`X`	`FRE`	`@0 Homme @5 10`
C03	`10`	`X`	`ENG`	`@0 Human @5 10`
C03	`10`	`X`	`SPA`	`@0 Hombre @5 10`
C07	`01`	`X`	`FRE`	`@0 Infection`
C07	`01`	`X`	`ENG`	`@0 Infection`
C07	`01`	`X`	`SPA`	`@0 Infección`
C07	`02`	`X`	`FRE`	`@0 Système nerveux pathologie @5 37`
C07	`02`	`X`	`ENG`	`@0 Nervous system diseases @5 37`
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C07	`03`	`X`	`FRE`	`@0 Immunopathologie @5 53`
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N21				`@1 018`

Format Inist (serveur)

NO :	PASCAL 99-0039814 INIST
ET :	Stress-associated immune modulation : Relevance to viral infections and chronic fatigue syndrome
AU :	GLASER (R.); KIECOLT-GLASER (J. K.); LEVINE (Paul H.)
AF :	Department of Medical Microbiology and Immunology and the Department of Psychiatry, Institute for Behavioral Medicine Research, Comprehensive Cancer Center, The Ohio State University College of Medicine/Columbus, Ohio/Etats-Unis; George Washington University Medical Center/Washington DC/Etats-Unis (1 aut.); Viral Epidemiology Branch, National Cancer Institute, National Institutes of Health/Bethesda MD/Etats-Unis (1 aut.)
DT :	Publication en série; Niveau analytique
SO :	The American journal of medicine; ISSN 0002-9343; Coden AJMEAZ; Etats-Unis; Da. 1998; Vol. 105; No. 3A; 35S-42S; Bibl. 81 ref.
LA :	Anglais
EA :	The frequent association of an active viral infection with the symptoms of CFS led researchers to hypothesize that chronic fatigue syndrome (CFS) is induced by a virus. Results of these studies indicated that despite clinical support for this hypothesis, there were no clear data linking viruses to CFS. In this overview, we will explore the interrelation of the immune, endocrine, and central nervous systems, and the possibility that stress and/or the reactivation/ replication of a latent virus (such as Epstein Barr virus) could modulate the immune system to induce CFS. Relevant research conducted in the developing field of psychoneuroimmunology will be reviewed, with a particular focus on cytokine synthesis, natural killer (NK) cell activity, and T-lymphocyte function, as they relate to CFS.
CC :	002B17I
FD :	Fatigue chronique syndrome; Prévalence; Association; Virose; Stress; Facteur risque; Réponse immune; Modulation; Etude comparative; Homme
FG :	Infection; Système nerveux pathologie; Immunopathologie
ED :	Chronic fatigue syndrome; Prevalence; Association; Viral disease; Stress; Risk factor; Immune response; Modulation; Comparative study; Human
EG :	Infection; Nervous system diseases; Immunopathology
GD :	Vergleich
SD :	Fatiga crónica síndrome; Prevalencia; Asociación; Virosis; Stress; Factor riesgo; Respuesta inmune; Modulación; Estudio comparativo; Hombre
LO :	INIST-4562.354000071365760070
ID :	99-0039814

Links to Exploration step

Pascal:99-0039814

Le document en format XML

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<front><div type="abstract" xml:lang="en">The frequent association of an active viral infection with the symptoms of CFS led researchers to hypothesize that chronic fatigue syndrome (CFS) is induced by a virus. Results of these studies indicated that despite clinical support for this hypothesis, there were no clear data linking viruses to CFS. In this overview, we will explore the interrelation of the immune, endocrine, and central nervous systems, and the possibility that stress and/or the reactivation/ replication of a latent virus (such as Epstein Barr virus) could modulate the immune system to induce CFS. Relevant research conducted in the developing field of psychoneuroimmunology will be reviewed, with a particular focus on cytokine synthesis, natural killer (NK) cell activity, and T-lymphocyte function, as they relate to CFS.</div>
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   |texte=   Stress-associated immune modulation : Relevance to viral infections and chronic fatigue syndrome
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Stress-associated immune modulation : Relevance to viral infections and chronic fatigue syndrome

Stress-associated immune modulation : Relevance to viral infections and chronic fatigue syndrome

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