StressCovidV1, Main, Exploration, bibRecord, 000897

Diversity of Th1/Th2 immunity in mice with acute lung injury induced by the H1N1 influenza virus and lipopolysaccharides.

Identifieur interne : 000897 ( Main/Exploration ); précédent : 000896; suivant : 000898

Diversity of Th1/Th2 immunity in mice with acute lung injury induced by the H1N1 influenza virus and lipopolysaccharides.

Auteurs : Jian-Xing Liu [République populaire de Chine] ; Ying Zhang [République populaire de Chine] ; Miao An [République populaire de Chine] ; Qing-Guang Wu [République populaire de Chine] ; Ya Zhao [République populaire de Chine] ; Xiong Li [République populaire de Chine] ; Geng Li [République populaire de Chine]

Source :

Journal of infection in developing countries [ 1972-2680 ] ; 2019.

RBID : pubmed:32058989

Abstract

The polarization of T helper (Th) cells plays an important role in the inflammatory response, pathogen removal, and tissue damage processes of infectious acute lung injury (ALI). However, Th cell polarization in viral- or bacterial-mediated ALI is not well defined. Herein, an influenza virus (A/FM/1/47, H1N1) and lipopolysaccharide (LPS) were chosen to induce ALI in mice, and the resultant diversity of Th-cell polarization was explored.

DOI: 10.3855/jidc.10338
PubMed: 32058989

Affiliations:

Le document en format XML

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<front><div type="abstract" xml:lang="en">The polarization of T helper (Th) cells plays an important role in the inflammatory response, pathogen removal, and tissue damage processes of infectious acute lung injury (ALI). However, Th cell polarization in viral- or bacterial-mediated ALI is not well defined. Herein, an influenza virus (A/FM/1/47, H1N1) and lipopolysaccharide (LPS) were chosen to induce ALI in mice, and the resultant diversity of Th-cell polarization was explored.</div>
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Diversity of Th1/Th2 immunity in mice with acute lung injury induced by the H1N1 influenza virus and lipopolysaccharides.

Diversity of Th1/Th2 immunity in mice with acute lung injury induced by the H1N1 influenza virus and lipopolysaccharides.

Source :

Abstract

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Le document en format XML

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