Inhibition of cytokine gene expression and induction of chemokine genes in non-lymphatic cells infected with SARS coronavirus.
Identifieur interne : 002296 ( PubMed/Curation ); précédent : 002295; suivant : 002297Inhibition of cytokine gene expression and induction of chemokine genes in non-lymphatic cells infected with SARS coronavirus.
Auteurs : Martin Spiegel [Allemagne] ; Friedemann WeberSource :
- Virology journal [ 1743-422X ] ; 2006.
Descripteurs français
- KwdFr :
- ARN messager (génétique), ARN messager (métabolisme), Chimiokines (génétique), Cytokines (génétique), Extinction de l'expression des gènes, Humains, Lignée cellulaire, Rein (métabolisme), Rein (virologie), Régulation négative (génétique), Régulation positive (génétique), Transcription génétique, Tumeurs du côlon (génétique), Tumeurs du côlon (virologie), Virus du SRAS (physiologie).
- MESH :
- génétique : ARN messager, Chimiokines, Cytokines, Régulation négative, Régulation positive, Tumeurs du côlon.
- métabolisme : ARN messager, Rein.
- physiologie : Virus du SRAS.
- virologie : Rein, Tumeurs du côlon.
- Extinction de l'expression des gènes, Humains, Lignée cellulaire, Transcription génétique.
English descriptors
- KwdEn :
- Cell Line, Chemokines (genetics), Colonic Neoplasms (genetics), Colonic Neoplasms (virology), Cytokines (genetics), Down-Regulation (genetics), Gene Silencing, Humans, Kidney (metabolism), Kidney (virology), RNA, Messenger (genetics), RNA, Messenger (metabolism), SARS Virus (physiology), Transcription, Genetic, Up-Regulation (genetics).
- MESH :
- chemical , genetics : Chemokines, Cytokines, RNA, Messenger.
- genetics : Colonic Neoplasms, Down-Regulation, Up-Regulation.
- metabolism : Kidney, RNA, Messenger.
- physiology : SARS Virus.
- virology : Colonic Neoplasms, Kidney.
- Cell Line, Gene Silencing, Humans, Transcription, Genetic.
Abstract
SARS coronavirus (SARS-CoV) is the etiologic agent of the severe acute respiratory syndrome. SARS-CoV mainly infects tissues of non-lymphatic origin, and the cytokine profile of those cells can determine the course of disease. Here, we investigated the cytokine response of two human non-lymphatic cell lines, Caco-2 and HEK 293, which are fully permissive for SARS-CoV.
DOI: 10.1186/1743-422X-3-17
PubMed: 16571117
Links toward previous steps (curation, corpus...)
- to stream PubMed, to step Corpus: Pour aller vers cette notice dans l'étape Curation :002296
Links to Exploration step
pubmed:16571117Le document en format XML
<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Inhibition of cytokine gene expression and induction of chemokine genes in non-lymphatic cells infected with SARS coronavirus.</title>
<author><name sortKey="Spiegel, Martin" sort="Spiegel, Martin" uniqKey="Spiegel M" first="Martin" last="Spiegel">Martin Spiegel</name>
<affiliation wicri:level="1"><nlm:affiliation>Abteilung Virologie, Institut für Medizinische Mikrobiologie und Hygiene, Universität, Freiburg, D-79008 Freiburg, Germany. martin.spiegel@uniklinik-freiburg.de</nlm:affiliation>
<country xml:lang="fr">Allemagne</country>
<wicri:regionArea>Abteilung Virologie, Institut für Medizinische Mikrobiologie und Hygiene, Universität, Freiburg, D-79008 Freiburg</wicri:regionArea>
</affiliation>
</author>
<author><name sortKey="Weber, Friedemann" sort="Weber, Friedemann" uniqKey="Weber F" first="Friedemann" last="Weber">Friedemann Weber</name>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">PubMed</idno>
<date when="2006">2006</date>
<idno type="RBID">pubmed:16571117</idno>
<idno type="pmid">16571117</idno>
<idno type="doi">10.1186/1743-422X-3-17</idno>
<idno type="wicri:Area/PubMed/Corpus">002296</idno>
<idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Corpus" wicri:corpus="PubMed">002296</idno>
<idno type="wicri:Area/PubMed/Curation">002296</idno>
<idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Curation">002296</idno>
</publicationStmt>
<sourceDesc><biblStruct><analytic><title xml:lang="en">Inhibition of cytokine gene expression and induction of chemokine genes in non-lymphatic cells infected with SARS coronavirus.</title>
<author><name sortKey="Spiegel, Martin" sort="Spiegel, Martin" uniqKey="Spiegel M" first="Martin" last="Spiegel">Martin Spiegel</name>
<affiliation wicri:level="1"><nlm:affiliation>Abteilung Virologie, Institut für Medizinische Mikrobiologie und Hygiene, Universität, Freiburg, D-79008 Freiburg, Germany. martin.spiegel@uniklinik-freiburg.de</nlm:affiliation>
<country xml:lang="fr">Allemagne</country>
<wicri:regionArea>Abteilung Virologie, Institut für Medizinische Mikrobiologie und Hygiene, Universität, Freiburg, D-79008 Freiburg</wicri:regionArea>
</affiliation>
</author>
<author><name sortKey="Weber, Friedemann" sort="Weber, Friedemann" uniqKey="Weber F" first="Friedemann" last="Weber">Friedemann Weber</name>
</author>
</analytic>
<series><title level="j">Virology journal</title>
<idno type="eISSN">1743-422X</idno>
<imprint><date when="2006" type="published">2006</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Cell Line</term>
<term>Chemokines (genetics)</term>
<term>Colonic Neoplasms (genetics)</term>
<term>Colonic Neoplasms (virology)</term>
<term>Cytokines (genetics)</term>
<term>Down-Regulation (genetics)</term>
<term>Gene Silencing</term>
<term>Humans</term>
<term>Kidney (metabolism)</term>
<term>Kidney (virology)</term>
<term>RNA, Messenger (genetics)</term>
<term>RNA, Messenger (metabolism)</term>
<term>SARS Virus (physiology)</term>
<term>Transcription, Genetic</term>
<term>Up-Regulation (genetics)</term>
</keywords>
<keywords scheme="KwdFr" xml:lang="fr"><term>ARN messager (génétique)</term>
<term>ARN messager (métabolisme)</term>
<term>Chimiokines (génétique)</term>
<term>Cytokines (génétique)</term>
<term>Extinction de l'expression des gènes</term>
<term>Humains</term>
<term>Lignée cellulaire</term>
<term>Rein (métabolisme)</term>
<term>Rein (virologie)</term>
<term>Régulation négative (génétique)</term>
<term>Régulation positive (génétique)</term>
<term>Transcription génétique</term>
<term>Tumeurs du côlon (génétique)</term>
<term>Tumeurs du côlon (virologie)</term>
<term>Virus du SRAS (physiologie)</term>
</keywords>
<keywords scheme="MESH" type="chemical" qualifier="genetics" xml:lang="en"><term>Chemokines</term>
<term>Cytokines</term>
<term>RNA, Messenger</term>
</keywords>
<keywords scheme="MESH" qualifier="genetics" xml:lang="en"><term>Colonic Neoplasms</term>
<term>Down-Regulation</term>
<term>Up-Regulation</term>
</keywords>
<keywords scheme="MESH" qualifier="génétique" xml:lang="fr"><term>ARN messager</term>
<term>Chimiokines</term>
<term>Cytokines</term>
<term>Régulation négative</term>
<term>Régulation positive</term>
<term>Tumeurs du côlon</term>
</keywords>
<keywords scheme="MESH" qualifier="metabolism" xml:lang="en"><term>Kidney</term>
<term>RNA, Messenger</term>
</keywords>
<keywords scheme="MESH" qualifier="métabolisme" xml:lang="fr"><term>ARN messager</term>
<term>Rein</term>
</keywords>
<keywords scheme="MESH" qualifier="physiologie" xml:lang="fr"><term>Virus du SRAS</term>
</keywords>
<keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>SARS Virus</term>
</keywords>
<keywords scheme="MESH" qualifier="virologie" xml:lang="fr"><term>Rein</term>
<term>Tumeurs du côlon</term>
</keywords>
<keywords scheme="MESH" qualifier="virology" xml:lang="en"><term>Colonic Neoplasms</term>
<term>Kidney</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Cell Line</term>
<term>Gene Silencing</term>
<term>Humans</term>
<term>Transcription, Genetic</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Extinction de l'expression des gènes</term>
<term>Humains</term>
<term>Lignée cellulaire</term>
<term>Transcription génétique</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">SARS coronavirus (SARS-CoV) is the etiologic agent of the severe acute respiratory syndrome. SARS-CoV mainly infects tissues of non-lymphatic origin, and the cytokine profile of those cells can determine the course of disease. Here, we investigated the cytokine response of two human non-lymphatic cell lines, Caco-2 and HEK 293, which are fully permissive for SARS-CoV.</div>
</front>
</TEI>
<pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">16571117</PMID>
<DateCompleted><Year>2006</Year>
<Month>07</Month>
<Day>20</Day>
</DateCompleted>
<DateRevised><Year>2018</Year>
<Month>11</Month>
<Day>13</Day>
</DateRevised>
<Article PubModel="Electronic"><Journal><ISSN IssnType="Electronic">1743-422X</ISSN>
<JournalIssue CitedMedium="Internet"><Volume>3</Volume>
<PubDate><Year>2006</Year>
<Month>Mar</Month>
<Day>29</Day>
</PubDate>
</JournalIssue>
<Title>Virology journal</Title>
<ISOAbbreviation>Virol. J.</ISOAbbreviation>
</Journal>
<ArticleTitle>Inhibition of cytokine gene expression and induction of chemokine genes in non-lymphatic cells infected with SARS coronavirus.</ArticleTitle>
<Pagination><MedlinePgn>17</MedlinePgn>
</Pagination>
<Abstract><AbstractText Label="BACKGROUND" NlmCategory="BACKGROUND">SARS coronavirus (SARS-CoV) is the etiologic agent of the severe acute respiratory syndrome. SARS-CoV mainly infects tissues of non-lymphatic origin, and the cytokine profile of those cells can determine the course of disease. Here, we investigated the cytokine response of two human non-lymphatic cell lines, Caco-2 and HEK 293, which are fully permissive for SARS-CoV.</AbstractText>
<AbstractText Label="RESULTS" NlmCategory="RESULTS">A comparison with established cytokine-inducing viruses revealed that SARS-CoV only weakly triggered a cytokine response. In particular, SARS-CoV did not activate significant transcription of the interferons IFN-alpha, IFN-beta, IFN-lambda1, IFN-lambda2/3, as well as of the interferon-induced antiviral genes ISG56 and MxA, the chemokine RANTES and the interleukine IL-6. Interestingly, however, SARS-CoV strongly induced the chemokines IP-10 and IL-8 in the colon carcinoma cell line Caco-2, but not in the embryonic kidney cell line 293.</AbstractText>
<AbstractText Label="CONCLUSION" NlmCategory="CONCLUSIONS">Our data indicate that SARS-CoV suppresses the antiviral cytokine system of non-immune cells to a large extent, thus buying time for dissemination in the host. However, synthesis of IP-10 and IL-8, which are established markers for acute-stage SARS, escapes the virus-induced silencing at least in some cell types. Therefore, the progressive infiltration of immune cells into the infected lungs observed in SARS patients could be due to the production of these chemokines by the infected tissue cells.</AbstractText>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Spiegel</LastName>
<ForeName>Martin</ForeName>
<Initials>M</Initials>
<AffiliationInfo><Affiliation>Abteilung Virologie, Institut für Medizinische Mikrobiologie und Hygiene, Universität, Freiburg, D-79008 Freiburg, Germany. martin.spiegel@uniklinik-freiburg.de</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y"><LastName>Weber</LastName>
<ForeName>Friedemann</ForeName>
<Initials>F</Initials>
</Author>
</AuthorList>
<Language>eng</Language>
<PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType>
<PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType>
</PublicationTypeList>
<ArticleDate DateType="Electronic"><Year>2006</Year>
<Month>03</Month>
<Day>29</Day>
</ArticleDate>
</Article>
<MedlineJournalInfo><Country>England</Country>
<MedlineTA>Virol J</MedlineTA>
<NlmUniqueID>101231645</NlmUniqueID>
<ISSNLinking>1743-422X</ISSNLinking>
</MedlineJournalInfo>
<ChemicalList><Chemical><RegistryNumber>0</RegistryNumber>
<NameOfSubstance UI="D018925">Chemokines</NameOfSubstance>
</Chemical>
<Chemical><RegistryNumber>0</RegistryNumber>
<NameOfSubstance UI="D016207">Cytokines</NameOfSubstance>
</Chemical>
<Chemical><RegistryNumber>0</RegistryNumber>
<NameOfSubstance UI="D012333">RNA, Messenger</NameOfSubstance>
</Chemical>
</ChemicalList>
<CitationSubset>IM</CitationSubset>
<MeshHeadingList><MeshHeading><DescriptorName UI="D002460" MajorTopicYN="N">Cell Line</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D018925" MajorTopicYN="N">Chemokines</DescriptorName>
<QualifierName UI="Q000235" MajorTopicYN="Y">genetics</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D003110" MajorTopicYN="N">Colonic Neoplasms</DescriptorName>
<QualifierName UI="Q000235" MajorTopicYN="N">genetics</QualifierName>
<QualifierName UI="Q000821" MajorTopicYN="N">virology</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D016207" MajorTopicYN="N">Cytokines</DescriptorName>
<QualifierName UI="Q000235" MajorTopicYN="Y">genetics</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D015536" MajorTopicYN="N">Down-Regulation</DescriptorName>
<QualifierName UI="Q000235" MajorTopicYN="Y">genetics</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D020868" MajorTopicYN="N">Gene Silencing</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D007668" MajorTopicYN="N">Kidney</DescriptorName>
<QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName>
<QualifierName UI="Q000821" MajorTopicYN="N">virology</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D012333" MajorTopicYN="N">RNA, Messenger</DescriptorName>
<QualifierName UI="Q000235" MajorTopicYN="N">genetics</QualifierName>
<QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D045473" MajorTopicYN="N">SARS Virus</DescriptorName>
<QualifierName UI="Q000502" MajorTopicYN="Y">physiology</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D014158" MajorTopicYN="N">Transcription, Genetic</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D015854" MajorTopicYN="N">Up-Regulation</DescriptorName>
<QualifierName UI="Q000235" MajorTopicYN="Y">genetics</QualifierName>
</MeshHeading>
</MeshHeadingList>
</MedlineCitation>
<PubmedData><History><PubMedPubDate PubStatus="received"><Year>2005</Year>
<Month>10</Month>
<Day>28</Day>
</PubMedPubDate>
<PubMedPubDate PubStatus="accepted"><Year>2006</Year>
<Month>03</Month>
<Day>29</Day>
</PubMedPubDate>
<PubMedPubDate PubStatus="pubmed"><Year>2006</Year>
<Month>3</Month>
<Day>31</Day>
<Hour>9</Hour>
<Minute>0</Minute>
</PubMedPubDate>
<PubMedPubDate PubStatus="medline"><Year>2006</Year>
<Month>7</Month>
<Day>21</Day>
<Hour>9</Hour>
<Minute>0</Minute>
</PubMedPubDate>
<PubMedPubDate PubStatus="entrez"><Year>2006</Year>
<Month>3</Month>
<Day>31</Day>
<Hour>9</Hour>
<Minute>0</Minute>
</PubMedPubDate>
</History>
<PublicationStatus>epublish</PublicationStatus>
<ArticleIdList><ArticleId IdType="pubmed">16571117</ArticleId>
<ArticleId IdType="pii">1743-422X-3-17</ArticleId>
<ArticleId IdType="doi">10.1186/1743-422X-3-17</ArticleId>
<ArticleId IdType="pmc">PMC1444920</ArticleId>
</ArticleIdList>
<ReferenceList><Reference><Citation>Proc Natl Acad Sci U S A. 1988 Jul;85(14):5259-63</Citation>
<ArticleIdList><ArticleId IdType="pubmed">3134658</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Blood. 1997 Aug 1;90(3):909-28</Citation>
<ArticleIdList><ArticleId IdType="pubmed">9242519</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Virol. 1999 Aug;73(8):6559-65</Citation>
<ArticleIdList><ArticleId IdType="pubmed">10400752</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Virus Res. 2005 Jan;107(1):93-101</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15567038</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Nat Med. 2004 Dec;10(12 Suppl):S88-97</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15577937</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Med Virol. 2005 Feb;75(2):185-94</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15602737</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Viral Immunol. 2004;17(4):498-515</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15671747</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Viral Immunol. 2004;17(4):535-44</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15671750</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>BMC Immunol. 2005;6:2</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15655079</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Virol. 2005 Feb;79(4):2079-86</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15681410</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Virol. 2005 Mar;79(6):3846-50</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15731278</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Arch Virol. 2005 May;150(5):1023-31</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15645376</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Virol. 2005 May;79(10):6180-93</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15858003</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Virol. 2005 Jun;79(12):7819-26</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15919935</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Exp Med. 2005 Aug 1;202(3):415-24</Citation>
<ArticleIdList><ArticleId IdType="pubmed">16043521</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Blood. 2005 Oct 1;106(7):2366-74</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15860669</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>EMBO J. 2000 Dec 15;19(24):6891-9</Citation>
<ArticleIdList><ArticleId IdType="pubmed">11118224</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Clin Microbiol Rev. 2001 Oct;14(4):778-809, table of contents</Citation>
<ArticleIdList><ArticleId IdType="pubmed">11585785</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Virol. 2002 Aug;76(16):7949-55</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12133999</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Traffic. 2002 Oct;3(10):710-7</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12230469</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Nat Immunol. 2003 Jan;4(1):69-77</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12483210</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Virol. 2003 Jan;77(2):1092-104</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12502825</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Lancet. 2003 Apr 19;361(9366):1319-25</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12711465</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>N Engl J Med. 2003 May 15;348(20):1967-76</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12690091</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>N Engl J Med. 2003 May 15;348(20):1953-66</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12690092</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Lancet. 2003 May 24;361(9371):1767-72</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12781535</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Lancet. 2003 May 24;361(9371):1773-8</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12781536</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Pathol. 2003 Jul;200(3):282-9</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12845623</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Lancet. 2003 Jul 26;362(9380):263-70</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12892955</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Lancet. 2003 Jul 26;362(9380):293-4</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12892961</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Zhonghua Gan Zang Bing Za Zhi. 2003 Aug;11(8):493-6</Citation>
<ArticleIdList><ArticleId IdType="pubmed">12939186</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Chin Med J (Engl). 2003 Sep;116(9):1283-7</Citation>
<ArticleIdList><ArticleId IdType="pubmed">14527349</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Gen Virol. 2003 Dec;84(Pt 12):3291-303</Citation>
<ArticleIdList><ArticleId IdType="pubmed">14645910</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Pathology. 2003 Dec;35(6):526-31</Citation>
<ArticleIdList><ArticleId IdType="pubmed">14660106</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Clin Microbiol. 2004 Jan;42(1):347-50</Citation>
<ArticleIdList><ArticleId IdType="pubmed">14715775</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Virol. 2004 Feb;78(4):1706-17</Citation>
<ArticleIdList><ArticleId IdType="pubmed">14747536</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Nat Med. 2004 Mar;10(3):290-3</Citation>
<ArticleIdList><ArticleId IdType="pubmed">14981511</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Clin Exp Immunol. 2004 Apr;136(1):95-103</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15030519</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Emerg Infect Dis. 2004 Feb;10(2):317-9</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15030704</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Infect Dis. 2004 Apr 1;189(7):1164-7</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15031783</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Emerg Infect Dis. 2004 Mar;10(3):489-93</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15109419</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Clin Virol. 2004 Jul;30(3):211-3</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15135736</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Expert Opin Biol Ther. 2004 Jun;4(6):827-36</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15174965</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Infect Immun. 2004 Aug;72(8):4410-5</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15271897</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Cell Mol Life Sci. 2004 Aug;61(16):2100-12</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15316659</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>BMC Infect Dis. 2004 Sep 9;4:34</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15357874</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Clin Infect Dis. 2004 Oct 1;39(7):1071-5</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15472864</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>Emerg Infect Dis. 2004 Sep;10(9):1550-7</Citation>
<ArticleIdList><ArticleId IdType="pubmed">15498155</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Gen Virol. 1977 Jul;36(1):59-74</Citation>
<ArticleIdList><ArticleId IdType="pubmed">886304</ArticleId>
</ArticleIdList>
</Reference>
<Reference><Citation>J Gen Virol. 1979 Apr;43(1):247-52</Citation>
<ArticleIdList><ArticleId IdType="pubmed">113494</ArticleId>
</ArticleIdList>
</Reference>
</ReferenceList>
</PubmedData>
</pubmed>
</record>
Pour manipuler ce document sous Unix (Dilib)
EXPLOR_STEP=$WICRI_ROOT/Sante/explor/SrasV1/Data/PubMed/Curation
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 002296 | SxmlIndent | more
Ou
HfdSelect -h $EXPLOR_AREA/Data/PubMed/Curation/biblio.hfd -nk 002296 | SxmlIndent | more
Pour mettre un lien sur cette page dans le réseau Wicri
{{Explor lien |wiki= Sante |area= SrasV1 |flux= PubMed |étape= Curation |type= RBID |clé= pubmed:16571117 |texte= Inhibition of cytokine gene expression and induction of chemokine genes in non-lymphatic cells infected with SARS coronavirus. }}
Pour générer des pages wiki
HfdIndexSelect -h $EXPLOR_AREA/Data/PubMed/Curation/RBID.i -Sk "pubmed:16571117" \ | HfdSelect -Kh $EXPLOR_AREA/Data/PubMed/Curation/biblio.hfd \ | NlmPubMed2Wicri -a SrasV1
![]() | This area was generated with Dilib version V0.6.33. | ![]() |