Interaction of SARS and MERS Coronaviruses with the Antiviral Interferon Response.
Identifieur interne : 000C06 ( PubMed/Corpus ); précédent : 000C05; suivant : 000C07Interaction of SARS and MERS Coronaviruses with the Antiviral Interferon Response.
Auteurs : E. Kindler ; V. Thiel ; F. WeberSource :
- Advances in virus research [ 1557-8399 ] ; 2016.
English descriptors
- KwdEn :
- Coronavirus Infections (immunology), Coronavirus Infections (pathology), Coronavirus Infections (virology), Gene Expression Regulation, Humans, Immune Evasion, Immunity, Innate, Interferon-alpha (genetics), Interferon-alpha (immunology), Interferon-beta (genetics), Interferon-beta (immunology), Interferon-gamma (genetics), Interferon-gamma (immunology), Janus Kinase 1 (genetics), Janus Kinase 1 (immunology), Middle East Respiratory Syndrome Coronavirus (genetics), Middle East Respiratory Syndrome Coronavirus (immunology), Middle East Respiratory Syndrome Coronavirus (pathogenicity), RNA, Double-Stranded (genetics), RNA, Double-Stranded (immunology), RNA, Viral (genetics), RNA, Viral (immunology), SARS Virus (genetics), SARS Virus (immunology), SARS Virus (pathogenicity), STAT Transcription Factors (genetics), STAT Transcription Factors (immunology), Severe Acute Respiratory Syndrome (immunology), Severe Acute Respiratory Syndrome (pathology), Severe Acute Respiratory Syndrome (virology), Signal Transduction, Viral Proteins (genetics), Viral Proteins (immunology).
- MESH :
- chemical , genetics : Interferon-alpha, Interferon-beta, Interferon-gamma, Janus Kinase 1, RNA, Double-Stranded, RNA, Viral, STAT Transcription Factors, Viral Proteins.
- genetics : Middle East Respiratory Syndrome Coronavirus, SARS Virus.
- immunology : Coronavirus Infections, Interferon-alpha, Interferon-beta, Interferon-gamma, Janus Kinase 1, Middle East Respiratory Syndrome Coronavirus, RNA, Double-Stranded, RNA, Viral, SARS Virus, STAT Transcription Factors, Severe Acute Respiratory Syndrome, Viral Proteins.
- pathogenicity : Middle East Respiratory Syndrome Coronavirus, SARS Virus.
- pathology : Coronavirus Infections, Severe Acute Respiratory Syndrome.
- virology : Coronavirus Infections, Severe Acute Respiratory Syndrome.
- Gene Expression Regulation, Humans, Immune Evasion, Immunity, Innate, Signal Transduction.
Abstract
Severe Acute Respiratory Syndrome (SARS) and Middle East Respiratory Syndrome (MERS) are the most severe coronavirus (CoV)-associated diseases in humans. The causative agents, SARS-CoV and MERS-CoV, are of zoonotic origin but may be transmitted to humans, causing severe and often fatal respiratory disease in their new host. The two coronaviruses are thought to encode an unusually large number of factors that allow them to thrive and replicate in the presence of efficient host defense mechanisms, especially the antiviral interferon system. Here, we review the recent progress in our understanding of the strategies that highly pathogenic coronaviruses employ to escape, dampen, or block the antiviral interferon response in human cells.
DOI: 10.1016/bs.aivir.2016.08.006
PubMed: 27712625
Links to Exploration step
pubmed:27712625Le document en format XML
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<front><div type="abstract" xml:lang="en">Severe Acute Respiratory Syndrome (SARS) and Middle East Respiratory Syndrome (MERS) are the most severe coronavirus (CoV)-associated diseases in humans. The causative agents, SARS-CoV and MERS-CoV, are of zoonotic origin but may be transmitted to humans, causing severe and often fatal respiratory disease in their new host. The two coronaviruses are thought to encode an unusually large number of factors that allow them to thrive and replicate in the presence of efficient host defense mechanisms, especially the antiviral interferon system. Here, we review the recent progress in our understanding of the strategies that highly pathogenic coronaviruses employ to escape, dampen, or block the antiviral interferon response in human cells.</div>
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<Abstract><AbstractText>Severe Acute Respiratory Syndrome (SARS) and Middle East Respiratory Syndrome (MERS) are the most severe coronavirus (CoV)-associated diseases in humans. The causative agents, SARS-CoV and MERS-CoV, are of zoonotic origin but may be transmitted to humans, causing severe and often fatal respiratory disease in their new host. The two coronaviruses are thought to encode an unusually large number of factors that allow them to thrive and replicate in the presence of efficient host defense mechanisms, especially the antiviral interferon system. Here, we review the recent progress in our understanding of the strategies that highly pathogenic coronaviruses employ to escape, dampen, or block the antiviral interferon response in human cells.</AbstractText>
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