Molecular biology of severe acute respiratory syndrome coronavirus
Identifieur interne : 001727 ( Pmc/Curation ); précédent : 001726; suivant : 001728Molecular biology of severe acute respiratory syndrome coronavirus
Auteurs : John ZiebuhrSource :
- Current Opinion in Microbiology [ 1369-5274 ] ; 2004.
Abstract
The worldwide epidemic of severe acute respiratory syndrome (SARS) in 2003 was caused by a novel coronavirus called SARS-CoV. Coronaviruses and their closest relatives possess extremely large plus-strand RNA genomes and employ unique mechanisms and enzymes in RNA synthesis that separate them from all other RNA viruses. The SARS epidemic prompted a variety of studies on multiple aspects of the coronavirus replication cycle, yielding both rapid identification of the entry mechanisms of SARS-CoV into host cells and valuable structural and functional information on SARS-CoV proteins. These recent advances in coronavirus research have important implications for the development of anti-SARS drugs and vaccines.
Url:
DOI: 10.1016/j.mib.2004.06.007
PubMed: 15358261
PubMed Central: 7108451
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<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Molecular biology of severe acute respiratory syndrome coronavirus</title><author><name sortKey="Ziebuhr, John" sort="Ziebuhr, John" uniqKey="Ziebuhr J" first="John" last="Ziebuhr">John Ziebuhr</name></author></titleStmt><publicationStmt><idno type="wicri:source">PMC</idno><idno type="pmid">15358261</idno><idno type="pmc">7108451</idno><idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7108451</idno><idno type="RBID">PMC:7108451</idno><idno type="doi">10.1016/j.mib.2004.06.007</idno><date when="2004">2004</date><idno type="wicri:Area/Pmc/Corpus">001727</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Corpus" wicri:corpus="PMC">001727</idno><idno type="wicri:Area/Pmc/Curation">001727</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Curation">001727</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">Molecular biology of severe acute respiratory syndrome coronavirus</title><author><name sortKey="Ziebuhr, John" sort="Ziebuhr, John" uniqKey="Ziebuhr J" first="John" last="Ziebuhr">John Ziebuhr</name></author></analytic><series><title level="j">Current Opinion in Microbiology</title><idno type="ISSN">1369-5274</idno><idno type="eISSN">1879-0364</idno><imprint><date when="2004">2004</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en"><p>The worldwide epidemic of severe acute respiratory syndrome (SARS) in 2003 was caused by a novel coronavirus called SARS-CoV. Coronaviruses and their closest relatives possess extremely large plus-strand RNA genomes and employ unique mechanisms and enzymes in RNA synthesis that separate them from all other RNA viruses. The SARS epidemic prompted a variety of studies on multiple aspects of the coronavirus replication cycle, yielding both rapid identification of the entry mechanisms of SARS-CoV into host cells and valuable structural and functional information on SARS-CoV proteins. These recent advances in coronavirus research have important implications for the development of anti-SARS drugs and vaccines.</p></div></front><back><div1 type="bibliography"><listBibl><biblStruct><analytic><author><name sortKey="Peiris, J S" uniqKey="Peiris J">J.S. Peiris</name></author><author><name sortKey="Yuen, K Y" uniqKey="Yuen K">K.Y. Yuen</name></author><author><name sortKey="Osterhaus, A D" uniqKey="Osterhaus A">A.D. Osterhaus</name></author><author><name sortKey="Stohr, K" uniqKey="Stohr K">K. 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<front><journal-meta><journal-id journal-id-type="nlm-ta">Curr Opin Microbiol</journal-id><journal-id journal-id-type="iso-abbrev">Curr. Opin. Microbiol</journal-id><journal-title-group><journal-title>Current Opinion in Microbiology</journal-title></journal-title-group><issn pub-type="ppub">1369-5274</issn><issn pub-type="epub">1879-0364</issn><publisher><publisher-name>Elsevier Ltd.</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="pmid">15358261</article-id><article-id pub-id-type="pmc">7108451</article-id><article-id pub-id-type="publisher-id">S1369-5274(04)00073-6</article-id><article-id pub-id-type="doi">10.1016/j.mib.2004.06.007</article-id><article-categories><subj-group subj-group-type="heading"><subject>Article</subject></subj-group></article-categories><title-group><article-title>Molecular biology of severe acute respiratory syndrome coronavirus</article-title></title-group><contrib-group><contrib contrib-type="author"><name><surname>Ziebuhr</surname><given-names>John</given-names></name><email>j.ziebuhr@mail.uni-wuerzburg.de</email></contrib></contrib-group><aff>Institute of Virology and Immunology, University of Würzburg, Versbacher Str. 7, 97078 Würzburg, Germany</aff><pub-date pub-type="pmc-release"><day>28</day><month>7</month><year>2004</year></pub-date><pmc-comment> PMC Release delay is 0 months and 0 days and was based on .</pmc-comment>
<pub-date pub-type="ppub"><month>8</month><year>2004</year></pub-date><pub-date pub-type="epub"><day>28</day><month>7</month><year>2004</year></pub-date><volume>7</volume><issue>4</issue><fpage>412</fpage><lpage>419</lpage><permissions><copyright-statement>Copyright © 2004 Elsevier Ltd. All rights reserved.</copyright-statement><copyright-year>2004</copyright-year><copyright-holder>Elsevier Ltd</copyright-holder><license><license-p>Since January 2020 Elsevier has created a COVID-19 resource centre with free information in English and Mandarin on the novel coronavirus COVID-19. The COVID-19 resource centre is hosted on Elsevier Connect, the company's public news and information website. Elsevier hereby grants permission to make all its COVID-19-related research that is available on the COVID-19 resource centre - including this research content - immediately available in PubMed Central and other publicly funded repositories, such as the WHO COVID database with rights for unrestricted research re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for free by Elsevier for as long as the COVID-19 resource centre remains active.</license-p></license></permissions><abstract><p>The worldwide epidemic of severe acute respiratory syndrome (SARS) in 2003 was caused by a novel coronavirus called SARS-CoV. Coronaviruses and their closest relatives possess extremely large plus-strand RNA genomes and employ unique mechanisms and enzymes in RNA synthesis that separate them from all other RNA viruses. The SARS epidemic prompted a variety of studies on multiple aspects of the coronavirus replication cycle, yielding both rapid identification of the entry mechanisms of SARS-CoV into host cells and valuable structural and functional information on SARS-CoV proteins. These recent advances in coronavirus research have important implications for the development of anti-SARS drugs and vaccines.</p></abstract><kwd-group><title>Abbreviations</title><kwd>2′-O-MT, 2′-O-ribose methyltransferase</kwd><kwd>3CL<sup>pro</sup>, 3C-like main protease</kwd><kwd>ADRP, ADP-ribose 1″-phosphatase</kwd><kwd>CPD, cyclic phosphodiesterase</kwd><kwd>ExoN, 3′-to-5′ exoribonuclease</kwd><kwd>HCoV-229E, human coronavirus 229E</kwd><kwd>HR, heptad repeat</kwd><kwd>NendoU, nidoviral uridylate-specific endoribonuclease</kwd><kwd>ORF, open reading frame</kwd><kwd>PL2<sup>pro</sup>, papain-like protease 2</kwd><kwd>RdRp, RNA-dependent RNA polymerase</kwd><kwd>SARS, severe acute respiratory syndrome</kwd><kwd>SARS-CoV, severe acute respiratory syndrome coronavirus</kwd><kwd>sg mRNA, subgenomic mRNA</kwd><kwd>TRS, transcription-regulating sequence</kwd></kwd-group></article-meta></front></pmc></record>Pour manipuler ce document sous Unix (Dilib)
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