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Epidemic dynamics at the human-animal interface

Identifieur interne : 000D29 ( Pmc/Curation ); précédent : 000D28; suivant : 000D30

Epidemic dynamics at the human-animal interface

Auteurs : James O. Lloyd-Smith [États-Unis] ; Dylan George [États-Unis] ; Kim M. Pepin [États-Unis] ; Virginia E. Pitzer [États-Unis] ; Juliet R. C. Pulliam [États-Unis] ; Andrew P. Dobson [États-Unis] ; Peter J. Hudson [États-Unis] ; Bryan T. Grenfell [États-Unis]

Source :

RBID : PMC:3891603

Abstract

Few infectious diseases are entirely human-specific: most human pathogens also circulate in animals, or else originated in non-human hosts. Influenza, plague, and trypanosomiasis are classic examples of zoonoses, or infections that transmit from animals to humans. The multi-host ecology of zoonoses leads to complex dynamics, and analytical tools such as mathematical modeling are vital to the development of effective control policies and research agendas. Much attention has focused on modeling pathogens with simpler life cycles and immediate global urgency, such as influenza and SARS, but vector-transmitted, chronic, and protozoan infections have been neglected, as have crucial processes such as cross-species transmission. Progress in understanding and combating zoonoses requires a new generation of models that addresses a broader set of pathogen life histories and integrates across host species and scientific disciplines.


Url:
DOI: 10.1126/science.1177345
PubMed: 19965751
PubMed Central: 3891603

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PMC:3891603

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<p id="P1">Few infectious diseases are entirely human-specific: most human pathogens also circulate in animals, or else originated in non-human hosts. Influenza, plague, and trypanosomiasis are classic examples of zoonoses, or infections that transmit from animals to humans. The multi-host ecology of zoonoses leads to complex dynamics, and analytical tools such as mathematical modeling are vital to the development of effective control policies and research agendas. Much attention has focused on modeling pathogens with simpler life cycles and immediate global urgency, such as influenza and SARS, but vector-transmitted, chronic, and protozoan infections have been neglected, as have crucial processes such as cross-species transmission. Progress in understanding and combating zoonoses requires a new generation of models that addresses a broader set of pathogen life histories and integrates across host species and scientific disciplines.</p>
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Department of Ecology & Evolutionary Biology, UCLA, Los Angeles CA 90095, USA</aff>
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Fogarty International Center, National Institutes of Health, Bethesda MD 20892, USA</aff>
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Department of Biology, Colorado State University, Fort Collins, CO, 80523, USA</aff>
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Center for Infectious Disease Dynamics, Pennsylvania State University, University Park 16802, USA</aff>
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Department of Ecology & Evolutionary Biology, Princeton University, Princeton NJ 08544, USA</aff>
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To whom correspondence should be addressed.
<email>jlloydsmith@ucla.edu</email>
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<p id="P1">Few infectious diseases are entirely human-specific: most human pathogens also circulate in animals, or else originated in non-human hosts. Influenza, plague, and trypanosomiasis are classic examples of zoonoses, or infections that transmit from animals to humans. The multi-host ecology of zoonoses leads to complex dynamics, and analytical tools such as mathematical modeling are vital to the development of effective control policies and research agendas. Much attention has focused on modeling pathogens with simpler life cycles and immediate global urgency, such as influenza and SARS, but vector-transmitted, chronic, and protozoan infections have been neglected, as have crucial processes such as cross-species transmission. Progress in understanding and combating zoonoses requires a new generation of models that addresses a broader set of pathogen life histories and integrates across host species and scientific disciplines.</p>
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