Cell Regeneration in Lung Injury
Identifieur interne : 000F92 ( Pmc/Checkpoint ); précédent : 000F91; suivant : 000F93Cell Regeneration in Lung Injury
Auteurs :Source :
- Intensive Care Medicine ; 2007.
Abstract
The acute respiratory distress syndrome (ARDS) is a lethal inflammatory disorder of the lung. Its incidence is estimated at 75 cases per 100,000 population and appears to be increasing [
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DOI: 10.1007/978-0-387-49518-7_28
PubMed: NONE
PubMed Central: 7122047
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<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Cell Regeneration in Lung Injury</title></titleStmt><publicationStmt><idno type="wicri:source">PMC</idno><idno type="pmc">7122047</idno><idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7122047</idno><idno type="RBID">PMC:7122047</idno><idno type="doi">10.1007/978-0-387-49518-7_28</idno><idno type="pmid">NONE</idno><date when="2007">2007</date><idno type="wicri:Area/Pmc/Corpus">000078</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Corpus" wicri:corpus="PMC">000078</idno><idno type="wicri:Area/Pmc/Curation">000078</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Curation">000078</idno><idno type="wicri:Area/Pmc/Checkpoint">000F92</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Checkpoint">000F92</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">Cell Regeneration in Lung Injury</title></analytic><series><title level="j">Intensive Care Medicine</title><imprint><date when="2007">2007</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en"><p>The acute respiratory distress syndrome (ARDS) is a lethal inflammatory disorder of the lung. Its incidence is estimated at 75 cases per 100,000 population and appears to be increasing [<xref ref-type="bibr" rid="CR1">1</xref>]. Even with optimal treatment, mortality is about 30% [<xref ref-type="bibr" rid="CR1">1</xref>–<xref ref-type="bibr" rid="CR3">3</xref>]. As such, ARDS represents a major public health problem. The effects of two recent crises created by unusual viral infections of the respiratory tract — the severe acute respiratory syndrome (SARS) epidemic caused by the novel SARS coronavirus [<xref ref-type="bibr" rid="CR4">4</xref>, <xref ref-type="bibr" rid="CR5">5</xref>] and the bird flu [<xref ref-type="bibr" rid="CR6">6</xref>] highlight the importance of research into ARDS. Both viruses cause an ARDS-like picture. Because lung repair and regeneration contribute substantially to the pathophysiology of ARDS, understanding these processes is essential [<xref ref-type="bibr" rid="CR7">7</xref>]. This chapter focuses on specific cell populations and markers involved in cell division and regeneration. In addition, a brief review of two pathways intimately associated with cell division is provided because of their potential for pharmacologic manipulation.</p></div></front><back><div1 type="bibliography"><listBibl><biblStruct><analytic><author><name sortKey="Ware, L B" uniqKey="Ware L">L.B. Ware</name></author><author><name sortKey="Matthay, M A" uniqKey="Matthay M">M.A. Matthay</name></author></analytic></biblStruct><biblStruct></biblStruct><biblStruct><analytic><author><name sortKey="Brower, R G" uniqKey="Brower R">R.G. Brower</name></author><author><name sortKey="Lanken, P N" uniqKey="Lanken P">P.N. Lanken</name></author><author><name sortKey="Macintyre, N" uniqKey="Macintyre N">N. MacIntyre</name></author></analytic></biblStruct><biblStruct><analytic><author><name sortKey="Ksiazek, T G" uniqKey="Ksiazek T">T.G. 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<front><journal-meta><journal-id journal-id-type="publisher-id">978-0-387-49518-7</journal-id><journal-id journal-id-type="doi">10.1007/978-0-387-49518-7</journal-id><journal-id journal-id-type="nlm-ta">Intensive Care Medicine</journal-id><journal-title-group><journal-title>Intensive Care Medicine</journal-title><journal-subtitle>Annual Update 2007</journal-subtitle></journal-title-group><isbn publication-format="print">978-0-387-49517-0</isbn><isbn publication-format="electronic">978-0-387-49518-7</isbn></journal-meta><article-meta><article-id pub-id-type="pmc">7122047</article-id><article-id pub-id-type="publisher-id">28</article-id><article-id pub-id-type="doi">10.1007/978-0-387-49518-7_28</article-id><article-categories><subj-group subj-group-type="heading"><subject>Article</subject></subj-group></article-categories><title-group><article-title>Cell Regeneration in Lung Injury</article-title></title-group><contrib-group content-type="book editors"><contrib contrib-type="editor"><name><surname>Vincent</surname><given-names>Jean-Louis</given-names></name><degrees>MD, PhD, FCCM, FCCP</degrees><xref ref-type="aff" rid="Aff1"></xref></contrib><aff id="Aff1"><institution-wrap><institution-id institution-id-type="GRID">grid.8767.e</institution-id><institution-id institution-id-type="ISNI">0000 0001 2290 8069</institution-id><institution>Department of Intensive Care Erasme Hospital,</institution><institution>Free University of Brussels,</institution></institution-wrap>Route de Lennik 808, 1070 Brussels, Belgium</aff></contrib-group><contrib-group><contrib contrib-type="author"><name><surname>Bromberg</surname><given-names>Z.</given-names></name><xref ref-type="aff" rid="Aff2">2</xref></contrib><contrib contrib-type="author"><name><surname>Deutschman</surname><given-names>C. S.</given-names></name><xref ref-type="aff" rid="Aff3">3</xref></contrib><contrib contrib-type="author"><name><surname>Weiss</surname><given-names>Y. G.</given-names></name><xref ref-type="aff" rid="Aff2">2</xref></contrib><aff id="Aff2"><label>2</label><institution-wrap><institution-id institution-id-type="GRID">grid.17788.31</institution-id><institution-id institution-id-type="ISNI">0000000122212926</institution-id><institution>Department of Anesthesiology and Critical Care Medicine,</institution><institution>Hadassah Medical Organization,</institution></institution-wrap>Kiryat Hadassah, P.O Box 12000, Jerusalem, 91120 Israel</aff><aff id="Aff3"><label>3</label><institution-wrap><institution-id institution-id-type="GRID">grid.25879.31</institution-id><institution-id institution-id-type="ISNI">0000000419368972</institution-id><institution>Department of Anesthesiology and Critical Care,</institution><institution>University of Pennsylvania School of Medicine,</institution></institution-wrap>3400 Spruce Street, Philadelphia, PA 19104-4283 USA</aff></contrib-group><pub-date pub-type="ppub"><year>2007</year></pub-date><fpage>310</fpage><lpage>319</lpage><permissions><copyright-statement>© Springer Science + Business Media Inc. 2007</copyright-statement><license><license-p>This article is made available via the PMC Open Access Subset for unrestricted research re-use and secondary analysis in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the World Health Organization (WHO) declaration of COVID-19 as a global pandemic.</license-p></license></permissions><abstract id="Abs1"><p>The acute respiratory distress syndrome (ARDS) is a lethal inflammatory disorder of the lung. Its incidence is estimated at 75 cases per 100,000 population and appears to be increasing [<xref ref-type="bibr" rid="CR1">1</xref>]. Even with optimal treatment, mortality is about 30% [<xref ref-type="bibr" rid="CR1">1</xref>–<xref ref-type="bibr" rid="CR3">3</xref>]. As such, ARDS represents a major public health problem. The effects of two recent crises created by unusual viral infections of the respiratory tract — the severe acute respiratory syndrome (SARS) epidemic caused by the novel SARS coronavirus [<xref ref-type="bibr" rid="CR4">4</xref>, <xref ref-type="bibr" rid="CR5">5</xref>] and the bird flu [<xref ref-type="bibr" rid="CR6">6</xref>] highlight the importance of research into ARDS. Both viruses cause an ARDS-like picture. Because lung repair and regeneration contribute substantially to the pathophysiology of ARDS, understanding these processes is essential [<xref ref-type="bibr" rid="CR7">7</xref>]. This chapter focuses on specific cell populations and markers involved in cell division and regeneration. In addition, a brief review of two pathways intimately associated with cell division is provided because of their potential for pharmacologic manipulation.</p></abstract><kwd-group xml:lang="en"><title>Keywords</title><kwd>Lung Injury</kwd><kwd>Hepatocyte Growth Factor</kwd><kwd>Acute Lung Injury</kwd><kwd>Idiopathic Pulmonary Fibrosis</kwd><kwd>Acute Respiratory Distress Syndrome</kwd></kwd-group><custom-meta-group><custom-meta><meta-name>issue-copyright-statement</meta-name><meta-value>© Springer Science + Business Media Inc. 2007</meta-value></custom-meta></custom-meta-group></article-meta></front></pmc><affiliations><list></list><tree></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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