Association of human-leukocyte-antigen class I (B*0703) and class II (DRB1*0301) genotypes with susceptibility and resistance to the development of severe acute respiratory syndrome.
Identifieur interne : 000942 ( Ncbi/Merge ); précédent : 000941; suivant : 000943Association of human-leukocyte-antigen class I (B*0703) and class II (DRB1*0301) genotypes with susceptibility and resistance to the development of severe acute respiratory syndrome.
Auteurs : Margaret H L. Ng [République populaire de Chine] ; Kin-Mang Lau ; Libby Li ; Suk-Hang Cheng ; Wing Y. Chan ; Pak K. Hui ; Benny Zee ; Chi-Bon Leung ; Joseph J Y. SungSource :
- The Journal of infectious diseases [ 0022-1899 ] ; 2004.
Descripteurs français
- KwdFr :
- Adulte, Adulte d'âge moyen, Antigènes HLA-B (génétique), Antigènes HLA-DR (génétique), Chaines HLA-DRB1, Dépistage génétique, Femelle, Fréquence d'allèle, Génotype, Humains, Indice de gravité médicale, Mâle, Prédisposition génétique à une maladie, Sujet âgé, Susceptibilité à une maladie, Syndrome respiratoire aigu sévère (génétique), Syndrome respiratoire aigu sévère (immunologie), Syndrome respiratoire aigu sévère (physiopathologie), Virus du SRAS (pathogénicité).
- MESH :
- génétique : Antigènes HLA-B, Antigènes HLA-DR, Syndrome respiratoire aigu sévère.
- immunologie : Syndrome respiratoire aigu sévère.
- pathogénicité : Virus du SRAS.
- physiopathologie : Syndrome respiratoire aigu sévère.
- Adulte, Adulte d'âge moyen, Chaines HLA-DRB1, Dépistage génétique, Femelle, Fréquence d'allèle, Génotype, Humains, Indice de gravité médicale, Mâle, Prédisposition génétique à une maladie, Sujet âgé, Susceptibilité à une maladie.
English descriptors
- KwdEn :
- Adult, Aged, Disease Susceptibility, Female, Gene Frequency, Genetic Predisposition to Disease, Genetic Testing, Genotype, HLA-B Antigens (genetics), HLA-DR Antigens (genetics), HLA-DRB1 Chains, Humans, Male, Middle Aged, SARS Virus (pathogenicity), Severe Acute Respiratory Syndrome (genetics), Severe Acute Respiratory Syndrome (immunology), Severe Acute Respiratory Syndrome (physiopathology), Severity of Illness Index.
- MESH :
- chemical , genetics : HLA-B Antigens, HLA-DR Antigens.
- genetics : Severe Acute Respiratory Syndrome.
- immunology : Severe Acute Respiratory Syndrome.
- pathogenicity : SARS Virus.
- physiopathology : Severe Acute Respiratory Syndrome.
- Adult, Aged, Disease Susceptibility, Female, Gene Frequency, Genetic Predisposition to Disease, Genetic Testing, Genotype, HLA-DRB1 Chains, Humans, Male, Middle Aged, Severity of Illness Index.
Abstract
Severe acute respiratory syndrome (SARS) is a public health concern worldwide. By studying the human leukocyte antigen (HLA) types A, B, DR, and DQ alleles in 90 Chinese patients with serologically confirmed SARS infections, we identified a strong association between HLA-B*0703 (OR, 4.08; 95% CI, 2.03-8.18; P=.00072 [Bonferroni-corrected P value, P(c) <.0022]) and -DRB1*0301 (OR, 0.06; 95%, 0.01-0.47; P=.00008 [after Bonferroni correction, P<.0042]) and the development of SARS. Moreover, the frequency of B*0703 and B60 coinheritance (9.6%; 95% CI, 4.6%-19.0%) in our SARS group was significantly higher (P=3x10(-9)) than that expected in the general population (0.4%). These genetic data will critically affect both the study of the pathogenesis of SARS and the design of vaccination programs.
DOI: 10.1086/421523
PubMed: 15243926
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Ng</name><affiliation wicri:level="4"><nlm:affiliation>Department of Anatomical and Cellular Pathology, The Chinese University of Hong Kong, Prince of Wales Hospital, Hong Kong SAR, China. margaretng@cuhk.edu.hk</nlm:affiliation><country xml:lang="fr">République populaire de Chine</country><wicri:regionArea>Department of Anatomical and Cellular Pathology, The Chinese University of Hong Kong, Prince of Wales Hospital, Hong Kong SAR</wicri:regionArea><orgName type="university">Université chinoise de Hong Kong</orgName><placeName><settlement type="city">Sha Tin</settlement></placeName></affiliation></author><author><name sortKey="Lau, Kin Mang" sort="Lau, Kin Mang" uniqKey="Lau K" first="Kin-Mang" last="Lau">Kin-Mang Lau</name></author><author><name sortKey="Li, Libby" sort="Li, Libby" uniqKey="Li L" first="Libby" last="Li">Libby Li</name></author><author><name sortKey="Cheng, Suk Hang" sort="Cheng, Suk Hang" uniqKey="Cheng S" first="Suk-Hang" last="Cheng">Suk-Hang Cheng</name></author><author><name sortKey="Chan, Wing Y" sort="Chan, Wing Y" uniqKey="Chan W" first="Wing Y" last="Chan">Wing Y. 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Ng</name><affiliation wicri:level="4"><nlm:affiliation>Department of Anatomical and Cellular Pathology, The Chinese University of Hong Kong, Prince of Wales Hospital, Hong Kong SAR, China. margaretng@cuhk.edu.hk</nlm:affiliation><country xml:lang="fr">République populaire de Chine</country><wicri:regionArea>Department of Anatomical and Cellular Pathology, The Chinese University of Hong Kong, Prince of Wales Hospital, Hong Kong SAR</wicri:regionArea><orgName type="university">Université chinoise de Hong Kong</orgName><placeName><settlement type="city">Sha Tin</settlement></placeName></affiliation></author><author><name sortKey="Lau, Kin Mang" sort="Lau, Kin Mang" uniqKey="Lau K" first="Kin-Mang" last="Lau">Kin-Mang Lau</name></author><author><name sortKey="Li, Libby" sort="Li, Libby" uniqKey="Li L" first="Libby" last="Li">Libby Li</name></author><author><name sortKey="Cheng, Suk Hang" sort="Cheng, Suk Hang" uniqKey="Cheng S" first="Suk-Hang" last="Cheng">Suk-Hang Cheng</name></author><author><name sortKey="Chan, Wing Y" sort="Chan, Wing Y" uniqKey="Chan W" first="Wing Y" last="Chan">Wing Y. Chan</name></author><author><name sortKey="Hui, Pak K" sort="Hui, Pak K" uniqKey="Hui P" first="Pak K" last="Hui">Pak K. Hui</name></author><author><name sortKey="Zee, Benny" sort="Zee, Benny" uniqKey="Zee B" first="Benny" last="Zee">Benny Zee</name></author><author><name sortKey="Leung, Chi Bon" sort="Leung, Chi Bon" uniqKey="Leung C" first="Chi-Bon" last="Leung">Chi-Bon Leung</name></author><author><name sortKey="Sung, Joseph J Y" sort="Sung, Joseph J Y" uniqKey="Sung J" first="Joseph J Y" last="Sung">Joseph J Y. 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By studying the human leukocyte antigen (HLA) types A, B, DR, and DQ alleles in 90 Chinese patients with serologically confirmed SARS infections, we identified a strong association between HLA-B*0703 (OR, 4.08; 95% CI, 2.03-8.18; P=.00072 [Bonferroni-corrected P value, P(c) <.0022]) and -DRB1*0301 (OR, 0.06; 95%, 0.01-0.47; P=.00008 [after Bonferroni correction, P<.0042]) and the development of SARS. Moreover, the frequency of B*0703 and B60 coinheritance (9.6%; 95% CI, 4.6%-19.0%) in our SARS group was significantly higher (P=3x10(-9)) than that expected in the general population (0.4%). These genetic data will critically affect both the study of the pathogenesis of SARS and the design of vaccination programs.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">15243926</PMID><DateCompleted><Year>2004</Year><Month>08</Month><Day>24</Day></DateCompleted><DateRevised><Year>2020</Year><Month>04</Month><Day>03</Day></DateRevised><Article PubModel="Print-Electronic"><Journal><ISSN IssnType="Print">0022-1899</ISSN><JournalIssue CitedMedium="Print"><Volume>190</Volume><Issue>3</Issue><PubDate><Year>2004</Year><Month>Aug</Month><Day>01</Day></PubDate></JournalIssue><Title>The Journal of infectious diseases</Title><ISOAbbreviation>J. Infect. Dis.</ISOAbbreviation></Journal><ArticleTitle>Association of human-leukocyte-antigen class I (B*0703) and class II (DRB1*0301) genotypes with susceptibility and resistance to the development of severe acute respiratory syndrome.</ArticleTitle><Pagination><MedlinePgn>515-8</MedlinePgn></Pagination><Abstract><AbstractText>Severe acute respiratory syndrome (SARS) is a public health concern worldwide. By studying the human leukocyte antigen (HLA) types A, B, DR, and DQ alleles in 90 Chinese patients with serologically confirmed SARS infections, we identified a strong association between HLA-B*0703 (OR, 4.08; 95% CI, 2.03-8.18; P=.00072 [Bonferroni-corrected P value, P(c) <.0022]) and -DRB1*0301 (OR, 0.06; 95%, 0.01-0.47; P=.00008 [after Bonferroni correction, P<.0042]) and the development of SARS. Moreover, the frequency of B*0703 and B60 coinheritance (9.6%; 95% CI, 4.6%-19.0%) in our SARS group was significantly higher (P=3x10(-9)) than that expected in the general population (0.4%). These genetic data will critically affect both the study of the pathogenesis of SARS and the design of vaccination programs.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Ng</LastName><ForeName>Margaret H L</ForeName><Initials>MH</Initials><AffiliationInfo><Affiliation>Department of Anatomical and Cellular Pathology, The Chinese University of Hong Kong, Prince of Wales Hospital, Hong Kong SAR, China. margaretng@cuhk.edu.hk</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Lau</LastName><ForeName>Kin-Mang</ForeName><Initials>KM</Initials></Author><Author ValidYN="Y"><LastName>Li</LastName><ForeName>Libby</ForeName><Initials>L</Initials></Author><Author ValidYN="Y"><LastName>Cheng</LastName><ForeName>Suk-Hang</ForeName><Initials>SH</Initials></Author><Author ValidYN="Y"><LastName>Chan</LastName><ForeName>Wing Y</ForeName><Initials>WY</Initials></Author><Author ValidYN="Y"><LastName>Hui</LastName><ForeName>Pak K</ForeName><Initials>PK</Initials></Author><Author ValidYN="Y"><LastName>Zee</LastName><ForeName>Benny</ForeName><Initials>B</Initials></Author><Author ValidYN="Y"><LastName>Leung</LastName><ForeName>Chi-Bon</ForeName><Initials>CB</Initials></Author><Author ValidYN="Y"><LastName>Sung</LastName><ForeName>Joseph J Y</ForeName><Initials>JJ</Initials></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D003160">Comparative Study</PublicationType><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType></PublicationTypeList><ArticleDate DateType="Electronic"><Year>2004</Year><Month>07</Month><Day>07</Day></ArticleDate></Article><MedlineJournalInfo><Country>United States</Country><MedlineTA>J Infect Dis</MedlineTA><NlmUniqueID>0413675</NlmUniqueID><ISSNLinking>0022-1899</ISSNLinking></MedlineJournalInfo><ChemicalList><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D015235">HLA-B Antigens</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D006684">HLA-DR Antigens</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D059811">HLA-DRB1 Chains</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="C553502">HLA-DRB1*03:01 antigen</NameOfSubstance></Chemical></ChemicalList><CitationSubset>AIM</CitationSubset><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D000328" MajorTopicYN="N">Adult</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D000368" MajorTopicYN="N">Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D004198" MajorTopicYN="N">Disease Susceptibility</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005787" MajorTopicYN="N">Gene Frequency</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D020022" MajorTopicYN="Y">Genetic Predisposition to Disease</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005820" MajorTopicYN="N">Genetic Testing</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005838" MajorTopicYN="N">Genotype</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D015235" MajorTopicYN="N">HLA-B Antigens</DescriptorName><QualifierName UI="Q000235" MajorTopicYN="Y">genetics</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D006684" MajorTopicYN="N">HLA-DR Antigens</DescriptorName><QualifierName UI="Q000235" MajorTopicYN="Y">genetics</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D059811" MajorTopicYN="N">HLA-DRB1 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21;326(7403):1358-62</Citation><ArticleIdList><ArticleId IdType="pubmed">12816821</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>Tissue Antigens. 1995 Nov;46(5):355-67</Citation><ArticleIdList><ArticleId IdType="pubmed">8838344</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>Hum Immunol. 1997 Aug-Sep;56(1-2):125-35</Citation><ArticleIdList><ArticleId IdType="pubmed">9455501</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>BMJ. 1995 Jan 21;310(6973):170</Citation><ArticleIdList><ArticleId IdType="pubmed">7833759</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>Transplant Proc. 2000 Nov;32(7):1859-61</Citation><ArticleIdList><ArticleId IdType="pubmed">11119972</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>N Engl J Med. 2003 May 15;348(20):1986-94</Citation><ArticleIdList><ArticleId IdType="pubmed">12682352</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>J Infect Dis. 2001 Jul 1;184(1):16-21</Citation><ArticleIdList><ArticleId IdType="pubmed">11398104</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>J Virol. 2002 May;76(10):4792-7</Citation><ArticleIdList><ArticleId IdType="pubmed">11967296</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>J Viral Hepat. 2001 May;8(3):174-9</Citation><ArticleIdList><ArticleId IdType="pubmed">11380794</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>Lancet. 2003 Apr 19;361(9366):1319-25</Citation><ArticleIdList><ArticleId IdType="pubmed">12711465</ArticleId></ArticleIdList></Reference></ReferenceList><ReferenceList><Reference><Citation>BMC Med Genet. 2003 Sep 12;4:9</Citation><ArticleIdList><ArticleId IdType="pubmed">12969506</ArticleId></ArticleIdList></Reference></ReferenceList></PubmedData></pubmed><affiliations><list><country><li>République populaire de Chine</li></country><settlement><li>Sha Tin</li></settlement><orgName><li>Université chinoise de Hong Kong</li></orgName></list><tree><noCountry><name sortKey="Chan, Wing Y" sort="Chan, Wing Y" uniqKey="Chan W" first="Wing Y" last="Chan">Wing Y. Chan</name><name sortKey="Cheng, Suk Hang" sort="Cheng, Suk Hang" uniqKey="Cheng S" first="Suk-Hang" last="Cheng">Suk-Hang Cheng</name><name sortKey="Hui, Pak K" sort="Hui, Pak K" uniqKey="Hui P" first="Pak K" last="Hui">Pak K. Hui</name><name sortKey="Lau, Kin Mang" sort="Lau, Kin Mang" uniqKey="Lau K" first="Kin-Mang" last="Lau">Kin-Mang Lau</name><name sortKey="Leung, Chi Bon" sort="Leung, Chi Bon" uniqKey="Leung C" first="Chi-Bon" last="Leung">Chi-Bon Leung</name><name sortKey="Li, Libby" sort="Li, Libby" uniqKey="Li L" first="Libby" last="Li">Libby Li</name><name sortKey="Sung, Joseph J Y" sort="Sung, Joseph J Y" uniqKey="Sung J" first="Joseph J Y" last="Sung">Joseph J Y. Sung</name><name sortKey="Zee, Benny" sort="Zee, Benny" uniqKey="Zee B" first="Benny" last="Zee">Benny Zee</name></noCountry><country name="République populaire de Chine"><noRegion><name sortKey="Ng, Margaret H L" sort="Ng, Margaret H L" uniqKey="Ng M" first="Margaret H L" last="Ng">Margaret H L. 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