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Serveur d'exploration SRAS - Curation (Ncbi)

Index « Keywords » - entrée « ADAM17 Protein »
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ADAM Proteins (metabolism) < ADAM17 Protein < ADAM17 Protein (antagonists & inhibitors)  Facettes :

List of bibliographic references

Number of relevant bibliographic references: 4.
Ident.Authors (with country if any)Title
001028 (2005) Daniel W. Lambert [Royaume-Uni] ; Mike Yarski ; Fiona J. Warner ; Paul Thornhill ; Edward T. Parkin ; A Ian Smith ; Nigel M. Hooper ; Anthony J. TurnerTumor necrosis factor-alpha convertase (ADAM17) mediates regulated ectodomain shedding of the severe-acute respiratory syndrome-coronavirus (SARS-CoV) receptor, angiotensin-converting enzyme-2 (ACE2).
001C64 (2008) Shiori Haga [Japon] ; Norio Yamamoto ; Chikako Nakai-Murakami ; Yoshiaki Osawa ; Kenzo Tokunaga ; Tetsutaro Sata ; Naoki Yamamoto ; Takehiko Sasazuki ; Yukihito IshizakaModulation of TNF-alpha-converting enzyme by the spike protein of SARS-CoV and ACE2 induces TNF-alpha production and facilitates viral entry.
002043 (2010) Shiori Haga [Japon] ; Noriyo Nagata ; Tadashi Okamura ; Norio Yamamoto ; Tetsutaro Sata ; Naoki Yamamoto ; Takehiko Sasazuki ; Yukihito IshizakaTACE antagonists blocking ACE2 shedding caused by the spike protein of SARS-CoV are candidate antiviral compounds.
002780 (2014) Adeline Heurich [Allemagne] ; Heike Hofmann-Winkler ; Stefanie Gierer ; Thomas Liepold ; Olaf Jahn ; Stefan PöhlmannTMPRSS2 and ADAM17 cleave ACE2 differentially and only proteolysis by TMPRSS2 augments entry driven by the severe acute respiratory syndrome coronavirus spike protein.

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   |flux=    Ncbi
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