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Index « Auteurs » - entrée « Makoto Ujike »
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Makoto Takeda < Makoto Ujike < Malathy Satkunarajah  Facettes :

List of bibliographic references

Number of relevant bibliographic references: 7.
Ident.Authors (with country if any)Title
001128 (2005) Shutoku Matsuyama [Japon] ; Makoto Ujike ; Shigeru Morikawa ; Masato Tashiro ; Fumihiro TaguchiProtease-mediated enhancement of severe acute respiratory syndrome coronavirus infection.
001703 (2006) Shutoku Matsuyama [Japon] ; Makoto Ujike ; Koji Ishii ; Shuetsu Fukushi ; Shigeru Morikawa ; Masato Tashiro ; Fumihiro TaguchiEnhancement of SARS-CoV infection by proteases.
001A89 (2008) Makoto Ujike [Japon] ; Hiroki Nishikawa ; Akira Otaka ; Naoki Yamamoto ; Norio Yamamoto ; Masao Matsuoka ; Eiichi Kodama ; Nobutaka Fujii ; Fumihiro TaguchiHeptad repeat-derived peptides block protease-mediated direct entry from the cell surface of severe acute respiratory syndrome coronavirus but not entry via the endosomal pathway.
002375 (2011) Kazuya Shirato [Japon] ; Madoka Maejima ; Shutoku Matsuyama ; Makoto Ujike ; Ayako Miyazaki ; Natsumi Takeyama ; Hidetoshi Ikeda ; Fumihiro TaguchiMutation in the cytoplasmic retrieval signal of porcine epidemic diarrhea virus spike (S) protein is responsible for enhanced fusion activity.
002438 (2012) Makoto Ujike [Japon] ; Cheng Huang [États-Unis] ; Kazuya Shirato [Japon] ; Shutoku Matsuyama [Japon] ; Shinji Makino [États-Unis] ; Fumihiro Taguchi [Japon]Two palmitylated cysteine residues of the severe acute respiratory syndrome coronavirus spike (S) protein are critical for S incorporation into virus-like particles, but not for M-S co-localization.
002621 (2013) Kozue Shimabukuro [Japon] ; Makoto Ujike ; Toshihiro Ito ; Hiroshi Tsunemitsu ; Hitoshi Oshitani ; Fumihiro TaguchiHemagglutination mediated by the spike protein of cell-adapted bovine torovirus.
002C53 (2016) Makoto Ujike ; Cheng Huang ; Kazuya Shirato ; Shinji Makino ; Fumihiro TaguchiThe contribution of the cytoplasmic retrieval signal of severe acute respiratory syndrome coronavirus to intracellular accumulation of S proteins and incorporation of S protein into virus-like particles

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