Serveur d'exploration SRAS - Exploration (Accueil)

Index « ISSN » - entrée « 0006-3002 »
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0006-2960 < 0006-3002 < 0006-3207  Facettes :

List of bibliographic references indexed by 0006-3002

Number of relevant bibliographic references: 9.
Ident.Authors (with country if any)Title
001276 (2015) Mukesh Mahajan [Singapour] ; Surajit Bhattacharjya [Singapour]NMR structures and localization of the potential fusion peptides and the pre-transmembrane region of SARS-CoV: Implications in membrane fusion.
001994 (2013) Carmina Verdiá-Báguena [Espagne] ; Jose L. Nieto-Torres ; Antonio Alcaraz ; Marta L. Dediego ; Luis Enjuanes ; Vicente M. AguilellaAnalysis of SARS-CoV E protein ion channel activity by tuning the protein and lipid charge.
002073 (2011) Cheng-Chang Chen [Taïwan] ; Jens Krüger ; Issara Sramala ; Hao-Jen Hsu ; Peter Henklein ; Yi-Ming Arthur Chen ; Wolfgang B. FischerORF8a of SARS-CoV forms an ion channel: experiments and molecular dynamics simulations.
003076 (2008) Jaime Guillén [Espagne] ; Paavo K J. Kinnunen ; José VillalaínMembrane insertion of the three main membranotropic sequences from SARS-CoV S2 glycoprotein.
003178 (2008) Zhongde Ye [République populaire de Chine] ; Chung Kai Wong ; Peng Li ; Yong XieA SARS-CoV protein, ORF-6, induces caspase-3 mediated, ER stress and JNK-dependent apoptosis.
003D83 (2006) John S. Mills [États-Unis]Peptides derived from HIV-1, HIV-2, Ebola virus, SARS coronavirus and coronavirus 229E exhibit high affinity binding to the formyl peptide receptor.
004761 (2005) J L Guy [Royaume-Uni] ; D W Lambert ; F J Warner ; N M Hooper ; A J TurnerMembrane-associated zinc peptidase families: comparing ACE and ACE2.
004777 (2005) Tetsuya Mizutani [Japon] ; Shuetsu Fukushi ; Masayuki Saijo ; Ichiro Kurane ; Shigeru MorikawaJNK and PI3k/Akt signaling pathways are required for establishing persistent SARS-CoV infection in Vero E6 cells.
006738 (????) Xiao-Nan Zhang [République populaire de Chine] ; Jiang-Xia Liu ; Yun-Wen Hu ; Hui Chen ; Zheng-Hong YuanHyper-activated IRF-1 and STAT1 contribute to enhanced interferon stimulated gene (ISG) expression by interferon alpha and gamma co-treatment in human hepatoma cells.

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