SidaGhanaV1, PubMed, Checkpoint, bibRecord, 000536

Detection of highly prevalent hepatitis B virus coinfection among HIV-seropositive persons in Ghana.

Identifieur interne : 000536 ( PubMed/Checkpoint ); précédent : 000535; suivant : 000537

Detection of highly prevalent hepatitis B virus coinfection among HIV-seropositive persons in Ghana.

Auteurs : Anna Maria Geretti [Royaume-Uni] ; Mauli Patel ; Fred Stephen Sarfo ; David Chadwick ; Jens Verheyen ; Maria Fraune ; Ana Garcia ; Richard Odame Phillips

Source :

Journal of clinical microbiology [ 1098-660X ] ; 2010.

RBID : pubmed:20631103

Descripteurs français

KwdFr :
- ADN viral (génétique), ADN viral (sang), Analyse de séquence d'ADN, Antigènes de surface du virus de l'hépatite B (génétique), Antigènes de surface du virus de l'hépatite B (sang), Charge virale, DNA-directed DNA polymerase (génétique), Ghana, Génotype, Humains, Hépatite B (diagnostic), Hépatite B (épidémiologie), Infections à VIH (), Mutation faux-sens, Protéines virales (génétique), Réaction de polymérisation en chaîne, Sensibilité et spécificité, Virus de l'hépatite B (), Virus de l'hépatite B (génétique), Virus de l'hépatite B (immunologie), Virus de l'hépatite B (isolement et purification).
MESH :
- diagnostic : Hépatite B.
- génétique : ADN viral, Antigènes de surface du virus de l'hépatite B, DNA-directed DNA polymerase, Protéines virales, Virus de l'hépatite B.
- immunologie : Virus de l'hépatite B.
- isolement et purification : Virus de l'hépatite B.
- sang : ADN viral, Antigènes de surface du virus de l'hépatite B.
- épidémiologie : Hépatite B.
- Analyse de séquence d'ADN, Charge virale, Ghana, Génotype, Humains, Infections à VIH, Mutation faux-sens, Réaction de polymérisation en chaîne, Sensibilité et spécificité, Virus de l'hépatite B.
Wicri :
- geographic : Ghana.

English descriptors

KwdEn :
- DNA, Viral (blood), DNA, Viral (genetics), DNA-Directed DNA Polymerase (genetics), Genotype, Ghana, HIV Infections (complications), Hepatitis B (diagnosis), Hepatitis B (epidemiology), Hepatitis B Surface Antigens (blood), Hepatitis B Surface Antigens (genetics), Hepatitis B virus (classification), Hepatitis B virus (genetics), Hepatitis B virus (immunology), Hepatitis B virus (isolation & purification), Humans, Mutation, Missense, Polymerase Chain Reaction, Sensitivity and Specificity, Sequence Analysis, DNA, Viral Load, Viral Proteins (genetics).
MESH :
- chemical , blood : DNA, Viral, Hepatitis B Surface Antigens.
- chemical , genetics : DNA, Viral, DNA-Directed DNA Polymerase, Hepatitis B Surface Antigens, Viral Proteins.
- geographic : Ghana.
- classification : Hepatitis B virus.
- complications : HIV Infections.
- diagnosis : Hepatitis B.
- epidemiology : Hepatitis B.
- genetics : Hepatitis B virus.
- immunology : Hepatitis B virus.
- isolation & purification : Hepatitis B virus.
- Genotype, Humans, Mutation, Missense, Polymerase Chain Reaction, Sensitivity and Specificity, Sequence Analysis, DNA, Viral Load.

Abstract

Simple hepatitis B surface antigen (HBsAg) tests may facilitate ascertainment of hepatitis B virus (HBV) infection in settings with high endemicity but limited infrastructure. We evaluated two rapid HBsAg tests and characterized HBV coinfection in a Ghanaian HIV-positive cohort. Samples from 838 patients were tested by the rapid assays Determine and Vikia and the reference assays Architect, Murex version 3, and Liaison Ultra. The assays were also evaluated using the 2nd International Standard, a seroconversion panel, and two mutant panels. HBsAg-positive samples underwent HBV DNA quantification by real-time PCR and surface and polymerase gene population sequencing. Overall, 140/838 patients (16.7%; 95% confidence interval, 14.2 to 19.2%) were HBsAg positive, and of these, 103/140 (73.6%) were e-antigen negative and 118/140 (84.3%) showed an HBV DNA level of >14 IU/ml (median, 8,279 IU/ml). Assay sensitivities and specificities were as follows: Architect, 97.9 and 99.6%; Liaison, 97.1 and 99.4%; Murex, 98.6 and 99.3%; Determine, 69.3 and 100%; and Vikia, 70.7 and 100%. With Determine, the limit of detection was >1.5 to 3.4 HBsAg IU/ml, and the median HBV DNA loads were 598 and 10,905 IU/ml in Determine-negative and -positive samples, respectively (P = 0.0005). Results were similar with the Vikia assay. HBV DNA sequencing indicated infection with genotype E in 82/86 (95.3%) patients. HBsAg mutations affected assay performance, including a T123A mutant that escaped detection by Architect. Major drug resistance mutations were observed in 4/86 patients (4.6%). The prevalence of HBV coinfection was high in this HIV-positive Ghanaian cohort. The two rapid assays identified HBsAg-positive patients at risk for liver disease with high specificity, albeit with only moderate sensitivity.

DOI: 10.1128/JCM.02231-09
PubMed: 20631103

Affiliations:

Royaume-Uni

Links toward previous steps (curation, corpus...)

to stream PubMed, to step Corpus: 000237
to stream PubMed, to step Curation: 000237

Links to Exploration step

pubmed:20631103

Le document en format XML

<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Detection of highly prevalent hepatitis B virus coinfection among HIV-seropositive persons in Ghana.</title>
<author><name sortKey="Geretti, Anna Maria" sort="Geretti, Anna Maria" uniqKey="Geretti A" first="Anna Maria" last="Geretti">Anna Maria Geretti</name>
<affiliation wicri:level="1"><nlm:affiliation>Department of Virology, Royal Free Hampstead NHS Trust & UCL Medical School, Pond Street, London NW3 2QG, United Kingdom. a.geretti@medsch.ucl.ac.uk</nlm:affiliation>
<country xml:lang="fr">Royaume-Uni</country>
<wicri:regionArea>Department of Virology, Royal Free Hampstead NHS Trust & UCL Medical School, Pond Street, London NW3 2QG</wicri:regionArea>
<wicri:noRegion>London NW3 2QG</wicri:noRegion>
</affiliation>
</author>
<author><name sortKey="Patel, Mauli" sort="Patel, Mauli" uniqKey="Patel M" first="Mauli" last="Patel">Mauli Patel</name>
</author>
<author><name sortKey="Sarfo, Fred Stephen" sort="Sarfo, Fred Stephen" uniqKey="Sarfo F" first="Fred Stephen" last="Sarfo">Fred Stephen Sarfo</name>
</author>
<author><name sortKey="Chadwick, David" sort="Chadwick, David" uniqKey="Chadwick D" first="David" last="Chadwick">David Chadwick</name>
</author>
<author><name sortKey="Verheyen, Jens" sort="Verheyen, Jens" uniqKey="Verheyen J" first="Jens" last="Verheyen">Jens Verheyen</name>
</author>
<author><name sortKey="Fraune, Maria" sort="Fraune, Maria" uniqKey="Fraune M" first="Maria" last="Fraune">Maria Fraune</name>
</author>
<author><name sortKey="Garcia, Ana" sort="Garcia, Ana" uniqKey="Garcia A" first="Ana" last="Garcia">Ana Garcia</name>
</author>
<author><name sortKey="Phillips, Richard Odame" sort="Phillips, Richard Odame" uniqKey="Phillips R" first="Richard Odame" last="Phillips">Richard Odame Phillips</name>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">PubMed</idno>
<date when="2010">2010</date>
<idno type="RBID">pubmed:20631103</idno>
<idno type="pmid">20631103</idno>
<idno type="doi">10.1128/JCM.02231-09</idno>
<idno type="wicri:Area/PubMed/Corpus">000237</idno>
<idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Corpus" wicri:corpus="PubMed">000237</idno>
<idno type="wicri:Area/PubMed/Curation">000237</idno>
<idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Curation">000237</idno>
<idno type="wicri:Area/PubMed/Checkpoint">000237</idno>
<idno type="wicri:explorRef" wicri:stream="Checkpoint" wicri:step="PubMed">000237</idno>
</publicationStmt>
<sourceDesc><biblStruct><analytic><title xml:lang="en">Detection of highly prevalent hepatitis B virus coinfection among HIV-seropositive persons in Ghana.</title>
<author><name sortKey="Geretti, Anna Maria" sort="Geretti, Anna Maria" uniqKey="Geretti A" first="Anna Maria" last="Geretti">Anna Maria Geretti</name>
<affiliation wicri:level="1"><nlm:affiliation>Department of Virology, Royal Free Hampstead NHS Trust & UCL Medical School, Pond Street, London NW3 2QG, United Kingdom. a.geretti@medsch.ucl.ac.uk</nlm:affiliation>
<country xml:lang="fr">Royaume-Uni</country>
<wicri:regionArea>Department of Virology, Royal Free Hampstead NHS Trust & UCL Medical School, Pond Street, London NW3 2QG</wicri:regionArea>
<wicri:noRegion>London NW3 2QG</wicri:noRegion>
</affiliation>
</author>
<author><name sortKey="Patel, Mauli" sort="Patel, Mauli" uniqKey="Patel M" first="Mauli" last="Patel">Mauli Patel</name>
</author>
<author><name sortKey="Sarfo, Fred Stephen" sort="Sarfo, Fred Stephen" uniqKey="Sarfo F" first="Fred Stephen" last="Sarfo">Fred Stephen Sarfo</name>
</author>
<author><name sortKey="Chadwick, David" sort="Chadwick, David" uniqKey="Chadwick D" first="David" last="Chadwick">David Chadwick</name>
</author>
<author><name sortKey="Verheyen, Jens" sort="Verheyen, Jens" uniqKey="Verheyen J" first="Jens" last="Verheyen">Jens Verheyen</name>
</author>
<author><name sortKey="Fraune, Maria" sort="Fraune, Maria" uniqKey="Fraune M" first="Maria" last="Fraune">Maria Fraune</name>
</author>
<author><name sortKey="Garcia, Ana" sort="Garcia, Ana" uniqKey="Garcia A" first="Ana" last="Garcia">Ana Garcia</name>
</author>
<author><name sortKey="Phillips, Richard Odame" sort="Phillips, Richard Odame" uniqKey="Phillips R" first="Richard Odame" last="Phillips">Richard Odame Phillips</name>
</author>
</analytic>
<series><title level="j">Journal of clinical microbiology</title>
<idno type="eISSN">1098-660X</idno>
<imprint><date when="2010" type="published">2010</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>DNA, Viral (blood)</term>
<term>DNA, Viral (genetics)</term>
<term>DNA-Directed DNA Polymerase (genetics)</term>
<term>Genotype</term>
<term>Ghana</term>
<term>HIV Infections (complications)</term>
<term>Hepatitis B (diagnosis)</term>
<term>Hepatitis B (epidemiology)</term>
<term>Hepatitis B Surface Antigens (blood)</term>
<term>Hepatitis B Surface Antigens (genetics)</term>
<term>Hepatitis B virus (classification)</term>
<term>Hepatitis B virus (genetics)</term>
<term>Hepatitis B virus (immunology)</term>
<term>Hepatitis B virus (isolation & purification)</term>
<term>Humans</term>
<term>Mutation, Missense</term>
<term>Polymerase Chain Reaction</term>
<term>Sensitivity and Specificity</term>
<term>Sequence Analysis, DNA</term>
<term>Viral Load</term>
<term>Viral Proteins (genetics)</term>
</keywords>
<keywords scheme="KwdFr" xml:lang="fr"><term>ADN viral (génétique)</term>
<term>ADN viral (sang)</term>
<term>Analyse de séquence d'ADN</term>
<term>Antigènes de surface du virus de l'hépatite B (génétique)</term>
<term>Antigènes de surface du virus de l'hépatite B (sang)</term>
<term>Charge virale</term>
<term>DNA-directed DNA polymerase (génétique)</term>
<term>Ghana</term>
<term>Génotype</term>
<term>Humains</term>
<term>Hépatite B (diagnostic)</term>
<term>Hépatite B (épidémiologie)</term>
<term>Infections à VIH ()</term>
<term>Mutation faux-sens</term>
<term>Protéines virales (génétique)</term>
<term>Réaction de polymérisation en chaîne</term>
<term>Sensibilité et spécificité</term>
<term>Virus de l'hépatite B ()</term>
<term>Virus de l'hépatite B (génétique)</term>
<term>Virus de l'hépatite B (immunologie)</term>
<term>Virus de l'hépatite B (isolement et purification)</term>
</keywords>
<keywords scheme="MESH" type="chemical" qualifier="blood" xml:lang="en"><term>DNA, Viral</term>
<term>Hepatitis B Surface Antigens</term>
</keywords>
<keywords scheme="MESH" type="chemical" qualifier="genetics" xml:lang="en"><term>DNA, Viral</term>
<term>DNA-Directed DNA Polymerase</term>
<term>Hepatitis B Surface Antigens</term>
<term>Viral Proteins</term>
</keywords>
<keywords scheme="MESH" type="geographic" xml:lang="en"><term>Ghana</term>
</keywords>
<keywords scheme="MESH" qualifier="classification" xml:lang="en"><term>Hepatitis B virus</term>
</keywords>
<keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>HIV Infections</term>
</keywords>
<keywords scheme="MESH" qualifier="diagnosis" xml:lang="en"><term>Hepatitis B</term>
</keywords>
<keywords scheme="MESH" qualifier="diagnostic" xml:lang="fr"><term>Hépatite B</term>
</keywords>
<keywords scheme="MESH" qualifier="epidemiology" xml:lang="en"><term>Hepatitis B</term>
</keywords>
<keywords scheme="MESH" qualifier="genetics" xml:lang="en"><term>Hepatitis B virus</term>
</keywords>
<keywords scheme="MESH" qualifier="génétique" xml:lang="fr"><term>ADN viral</term>
<term>Antigènes de surface du virus de l'hépatite B</term>
<term>DNA-directed DNA polymerase</term>
<term>Protéines virales</term>
<term>Virus de l'hépatite B</term>
</keywords>
<keywords scheme="MESH" qualifier="immunologie" xml:lang="fr"><term>Virus de l'hépatite B</term>
</keywords>
<keywords scheme="MESH" qualifier="immunology" xml:lang="en"><term>Hepatitis B virus</term>
</keywords>
<keywords scheme="MESH" qualifier="isolation & purification" xml:lang="en"><term>Hepatitis B virus</term>
</keywords>
<keywords scheme="MESH" qualifier="isolement et purification" xml:lang="fr"><term>Virus de l'hépatite B</term>
</keywords>
<keywords scheme="MESH" qualifier="sang" xml:lang="fr"><term>ADN viral</term>
<term>Antigènes de surface du virus de l'hépatite B</term>
</keywords>
<keywords scheme="MESH" qualifier="épidémiologie" xml:lang="fr"><term>Hépatite B</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Genotype</term>
<term>Humans</term>
<term>Mutation, Missense</term>
<term>Polymerase Chain Reaction</term>
<term>Sensitivity and Specificity</term>
<term>Sequence Analysis, DNA</term>
<term>Viral Load</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Analyse de séquence d'ADN</term>
<term>Charge virale</term>
<term>Ghana</term>
<term>Génotype</term>
<term>Humains</term>
<term>Infections à VIH</term>
<term>Mutation faux-sens</term>
<term>Réaction de polymérisation en chaîne</term>
<term>Sensibilité et spécificité</term>
<term>Virus de l'hépatite B</term>
</keywords>
<keywords scheme="Wicri" type="geographic" xml:lang="fr"><term>Ghana</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">Simple hepatitis B surface antigen (HBsAg) tests may facilitate ascertainment of hepatitis B virus (HBV) infection in settings with high endemicity but limited infrastructure. We evaluated two rapid HBsAg tests and characterized HBV coinfection in a Ghanaian HIV-positive cohort. Samples from 838 patients were tested by the rapid assays Determine and Vikia and the reference assays Architect, Murex version 3, and Liaison Ultra. The assays were also evaluated using the 2nd International Standard, a seroconversion panel, and two mutant panels. HBsAg-positive samples underwent HBV DNA quantification by real-time PCR and surface and polymerase gene population sequencing. Overall, 140/838 patients (16.7%; 95% confidence interval, 14.2 to 19.2%) were HBsAg positive, and of these, 103/140 (73.6%) were e-antigen negative and 118/140 (84.3%) showed an HBV DNA level of >14 IU/ml (median, 8,279 IU/ml). Assay sensitivities and specificities were as follows: Architect, 97.9 and 99.6%; Liaison, 97.1 and 99.4%; Murex, 98.6 and 99.3%; Determine, 69.3 and 100%; and Vikia, 70.7 and 100%. With Determine, the limit of detection was >1.5 to 3.4 HBsAg IU/ml, and the median HBV DNA loads were 598 and 10,905 IU/ml in Determine-negative and -positive samples, respectively (P = 0.0005). Results were similar with the Vikia assay. HBV DNA sequencing indicated infection with genotype E in 82/86 (95.3%) patients. HBsAg mutations affected assay performance, including a T123A mutant that escaped detection by Architect. Major drug resistance mutations were observed in 4/86 patients (4.6%). The prevalence of HBV coinfection was high in this HIV-positive Ghanaian cohort. The two rapid assays identified HBsAg-positive patients at risk for liver disease with high specificity, albeit with only moderate sensitivity.</div>
</front>
</TEI>
<pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">20631103</PMID>
<DateCreated><Year>2010</Year>
<Month>08</Month>
<Day>31</Day>
</DateCreated>
<DateCompleted><Year>2010</Year>
<Month>12</Month>
<Day>08</Day>
</DateCompleted>
<DateRevised><Year>2014</Year>
<Month>12</Month>
<Day>03</Day>
</DateRevised>
<Article PubModel="Print-Electronic"><Journal><ISSN IssnType="Electronic">1098-660X</ISSN>
<JournalIssue CitedMedium="Internet"><Volume>48</Volume>
<Issue>9</Issue>
<PubDate><Year>2010</Year>
<Month>Sep</Month>
</PubDate>
</JournalIssue>
<Title>Journal of clinical microbiology</Title>
<ISOAbbreviation>J. Clin. Microbiol.</ISOAbbreviation>
</Journal>
<ArticleTitle>Detection of highly prevalent hepatitis B virus coinfection among HIV-seropositive persons in Ghana.</ArticleTitle>
<Pagination><MedlinePgn>3223-30</MedlinePgn>
</Pagination>
<ELocationID EIdType="doi" ValidYN="Y">10.1128/JCM.02231-09</ELocationID>
<Abstract><AbstractText>Simple hepatitis B surface antigen (HBsAg) tests may facilitate ascertainment of hepatitis B virus (HBV) infection in settings with high endemicity but limited infrastructure. We evaluated two rapid HBsAg tests and characterized HBV coinfection in a Ghanaian HIV-positive cohort. Samples from 838 patients were tested by the rapid assays Determine and Vikia and the reference assays Architect, Murex version 3, and Liaison Ultra. The assays were also evaluated using the 2nd International Standard, a seroconversion panel, and two mutant panels. HBsAg-positive samples underwent HBV DNA quantification by real-time PCR and surface and polymerase gene population sequencing. Overall, 140/838 patients (16.7%; 95% confidence interval, 14.2 to 19.2%) were HBsAg positive, and of these, 103/140 (73.6%) were e-antigen negative and 118/140 (84.3%) showed an HBV DNA level of >14 IU/ml (median, 8,279 IU/ml). Assay sensitivities and specificities were as follows: Architect, 97.9 and 99.6%; Liaison, 97.1 and 99.4%; Murex, 98.6 and 99.3%; Determine, 69.3 and 100%; and Vikia, 70.7 and 100%. With Determine, the limit of detection was >1.5 to 3.4 HBsAg IU/ml, and the median HBV DNA loads were 598 and 10,905 IU/ml in Determine-negative and -positive samples, respectively (P = 0.0005). Results were similar with the Vikia assay. HBV DNA sequencing indicated infection with genotype E in 82/86 (95.3%) patients. HBsAg mutations affected assay performance, including a T123A mutant that escaped detection by Architect. Major drug resistance mutations were observed in 4/86 patients (4.6%). The prevalence of HBV coinfection was high in this HIV-positive Ghanaian cohort. The two rapid assays identified HBsAg-positive patients at risk for liver disease with high specificity, albeit with only moderate sensitivity.</AbstractText>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Geretti</LastName>
<ForeName>Anna Maria</ForeName>
<Initials>AM</Initials>
<AffiliationInfo><Affiliation>Department of Virology, Royal Free Hampstead NHS Trust & UCL Medical School, Pond Street, London NW3 2QG, United Kingdom. a.geretti@medsch.ucl.ac.uk</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y"><LastName>Patel</LastName>
<ForeName>Mauli</ForeName>
<Initials>M</Initials>
</Author>
<Author ValidYN="Y"><LastName>Sarfo</LastName>
<ForeName>Fred Stephen</ForeName>
<Initials>FS</Initials>
</Author>
<Author ValidYN="Y"><LastName>Chadwick</LastName>
<ForeName>David</ForeName>
<Initials>D</Initials>
</Author>
<Author ValidYN="Y"><LastName>Verheyen</LastName>
<ForeName>Jens</ForeName>
<Initials>J</Initials>
</Author>
<Author ValidYN="Y"><LastName>Fraune</LastName>
<ForeName>Maria</ForeName>
<Initials>M</Initials>
</Author>
<Author ValidYN="Y"><LastName>Garcia</LastName>
<ForeName>Ana</ForeName>
<Initials>A</Initials>
</Author>
<Author ValidYN="Y"><LastName>Phillips</LastName>
<ForeName>Richard Odame</ForeName>
<Initials>RO</Initials>
</Author>
</AuthorList>
<Language>eng</Language>
<PublicationTypeList><PublicationType UI="D003160">Comparative Study</PublicationType>
<PublicationType UI="D023362">Evaluation Studies</PublicationType>
<PublicationType UI="D016428">Journal Article</PublicationType>
</PublicationTypeList>
<ArticleDate DateType="Electronic"><Year>2010</Year>
<Month>07</Month>
<Day>14</Day>
</ArticleDate>
</Article>
<MedlineJournalInfo><Country>United States</Country>
<MedlineTA>J Clin Microbiol</MedlineTA>
<NlmUniqueID>7505564</NlmUniqueID>
<ISSNLinking>0095-1137</ISSNLinking>
</MedlineJournalInfo>
<ChemicalList><Chemical><RegistryNumber>0</RegistryNumber>
<NameOfSubstance UI="D004279">DNA, Viral</NameOfSubstance>
</Chemical>
<Chemical><RegistryNumber>0</RegistryNumber>
<NameOfSubstance UI="D006514">Hepatitis B Surface Antigens</NameOfSubstance>
</Chemical>
<Chemical><RegistryNumber>0</RegistryNumber>
<NameOfSubstance UI="D014764">Viral Proteins</NameOfSubstance>
</Chemical>
<Chemical><RegistryNumber>EC 2.7.7.7</RegistryNumber>
<NameOfSubstance UI="D004259">DNA-Directed DNA Polymerase</NameOfSubstance>
</Chemical>
</ChemicalList>
<CitationSubset>IM</CitationSubset>
<CommentsCorrectionsList><CommentsCorrections RefType="Cites"><RefSource>Rev Med Virol. 1999 Jul;9(3):183-209</RefSource>
<PMID Version="1">10479779</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Med Virol. 1999 Sep;59(1):19-24</RefSource>
<PMID Version="1">10440803</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Hepatology. 1999 Nov;30(5):1302-6</RefSource>
<PMID Version="1">10534354</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Med Virol. 2000 Feb;60(2):113-21</RefSource>
<PMID Version="1">10596008</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Gen Virol. 2000 May;81(Pt 5):1165-74</RefSource>
<PMID Version="1">10769057</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Hepatology. 2000 May;31(5):1176-82</RefSource>
<PMID Version="1">10796895</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Am J Trop Med Hyg. 2000 Feb;62(2):301-9</RefSource>
<PMID Version="1">10813489</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Hepatology. 2001 Nov;34(5):1027-34</RefSource>
<PMID Version="1">11679975</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Clin Virol. 2002 Aug;25(2):97-106</RefSource>
<PMID Version="1">12367644</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Lancet. 2002 Dec 14;360(9349):1921-6</RefSource>
<PMID Version="1">12493258</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Blood. 2003 Mar 15;101(6):2419-25</RefSource>
<PMID Version="1">12393691</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>AIDS. 2005 Mar 24;19(6):593-601</RefSource>
<PMID Version="1">15802978</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Clin Infect Dis. 2005 Nov 15;41(10):1525-8</RefSource>
<PMID Version="1">16231268</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Emerg Infect Dis. 2006 Feb;12(2):198-203</RefSource>
<PMID Version="1">16494742</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Med Microbiol. 2006 May;55(Pt 5):593-7</RefSource>
<PMID Version="1">16585647</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Med Virol. 2006;78 Suppl 1:S66-70</RefSource>
<PMID Version="1">16622881</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Med Virol. 2006 Jun;78(6):730-3</RefSource>
<PMID Version="1">16628587</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Clin Microbiol. 2006 Jul;44(7):2321-6</RefSource>
<PMID Version="1">16825343</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Virol Methods. 2006 Sep;136(1-2):254-6</RefSource>
<PMID Version="1">16707169</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Arch Intern Med. 2006 Aug 14-28;166(15):1632-41</RefSource>
<PMID Version="1">16908797</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>N Engl J Med. 2006 Sep 14;355(11):1141-53</RefSource>
<PMID Version="1">16971720</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Viral Hepat. 2006 Nov;13(11):715-24</RefSource>
<PMID Version="1">17052270</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Hepatology. 2006 Nov;44(5):1110-6</RefSource>
<PMID Version="1">17058225</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Antivir Ther. 2007;12(1):119-22</RefSource>
<PMID Version="1">17503756</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Clin Infect Dis. 2007 Sep 1;45(5):624-32</RefSource>
<PMID Version="1">17682999</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Med Microbiol Immunol. 2007 Dec;196(4):247-52</RefSource>
<PMID Version="1">17503077</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Clin Microbiol. 2007 Sep;45(9):2971-8</RefSource>
<PMID Version="1">17609325</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Gen Virol. 2007 Oct;88(Pt 10):2686-95</RefSource>
<PMID Version="1">17872520</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Med Virol. 2007 Dec;79(12):1797-801</RefSource>
<PMID Version="1">17935164</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Viral Hepat. 2007 Nov;14 Suppl 1:11-5</RefSource>
<PMID Version="1">17958637</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Virol. 2007 Dec;81(23):13057-66</RefSource>
<PMID Version="1">17898062</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Med Microbiol Immunol. 2008 Mar;197(1):55-64</RefSource>
<PMID Version="1">17899185</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Clin Infect Dis. 2008 Feb 1;46(3):361-6</RefSource>
<PMID Version="1">18171303</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>AIDS. 2008 Jun 19;22(10):1221-2</RefSource>
<PMID Version="1">18525268</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Med Virol. 2008 Aug;80(8):1332-6</RefSource>
<PMID Version="1">18551596</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Infect. 2008 Jul;57(1):72-7</RefSource>
<PMID Version="1">18555534</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>AIDS. 2008 Jul 31;22(12):1399-410</RefSource>
<PMID Version="1">18614862</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Virol Methods. 2008 Aug;151(2):294-7</RefSource>
<PMID Version="1">18462816</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Ann Hepatol. 2008 Apr-Jun;7(2):152-6</RefSource>
<PMID Version="1">18626434</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Clin Microbiol. 2008 Oct;46(10):3319-24</RefSource>
<PMID Version="1">18701669</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Clin Infect Dis. 2008 Dec 1;47(11):1479-85</RefSource>
<PMID Version="1">18937580</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Clin Infect Dis. 2009 Apr 1;48(7):988-91</RefSource>
<PMID Version="1">19231976</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Hepatology. 2009 Apr;49(4):1158-65</RefSource>
<PMID Version="1">19263474</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Clin Infect Dis. 2009 Jun 15;48(12):1763-71</RefSource>
<PMID Version="1">19435436</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Antiviral Res. 2003 Oct;60(2):75-8</RefSource>
<PMID Version="1">14638401</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Methods Mol Med. 2004;95:125-49</RefSource>
<PMID Version="1">14762302</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Gut. 2004 Oct;53(10):1499-503</RefSource>
<PMID Version="1">15361503</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Lancet. 1990 Aug 11;336(8711):325-9</RefSource>
<PMID Version="1">1697396</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>J Virol. 1994 Apr;68(4):2671-6</RefSource>
<PMID Version="1">8139044</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Virology. 1995 Aug 20;211(2):535-43</RefSource>
<PMID Version="1">7645257</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Hepatology. 1999 Apr;29(4):1306-10</RefSource>
<PMID Version="1">10094979</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites"><RefSource>Intervirology. 2004;47(6):289-309</RefSource>
<PMID Version="1">15564741</PMID>
</CommentsCorrections>
</CommentsCorrectionsList>
<MeshHeadingList><MeshHeading><DescriptorName UI="D004279" MajorTopicYN="N">DNA, Viral</DescriptorName>
<QualifierName UI="Q000097" MajorTopicYN="N">blood</QualifierName>
<QualifierName UI="Q000235" MajorTopicYN="N">genetics</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D004259" MajorTopicYN="N">DNA-Directed DNA Polymerase</DescriptorName>
<QualifierName UI="Q000235" MajorTopicYN="N">genetics</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D005838" MajorTopicYN="N">Genotype</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D005869" MajorTopicYN="N" Type="Geographic">Ghana</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D015658" MajorTopicYN="N">HIV Infections</DescriptorName>
<QualifierName UI="Q000150" MajorTopicYN="Y">complications</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D006509" MajorTopicYN="N">Hepatitis B</DescriptorName>
<QualifierName UI="Q000175" MajorTopicYN="Y">diagnosis</QualifierName>
<QualifierName UI="Q000453" MajorTopicYN="Y">epidemiology</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D006514" MajorTopicYN="N">Hepatitis B Surface Antigens</DescriptorName>
<QualifierName UI="Q000097" MajorTopicYN="Y">blood</QualifierName>
<QualifierName UI="Q000235" MajorTopicYN="N">genetics</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D006515" MajorTopicYN="N">Hepatitis B virus</DescriptorName>
<QualifierName UI="Q000145" MajorTopicYN="N">classification</QualifierName>
<QualifierName UI="Q000235" MajorTopicYN="N">genetics</QualifierName>
<QualifierName UI="Q000276" MajorTopicYN="N">immunology</QualifierName>
<QualifierName UI="Q000302" MajorTopicYN="Y">isolation & purification</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D020125" MajorTopicYN="N">Mutation, Missense</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D016133" MajorTopicYN="N">Polymerase Chain Reaction</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D012680" MajorTopicYN="N">Sensitivity and Specificity</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D017422" MajorTopicYN="N">Sequence Analysis, DNA</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D019562" MajorTopicYN="N">Viral Load</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D014764" MajorTopicYN="N">Viral Proteins</DescriptorName>
<QualifierName UI="Q000235" MajorTopicYN="N">genetics</QualifierName>
</MeshHeading>
</MeshHeadingList>
<OtherID Source="NLM">PMC2937686</OtherID>
</MedlineCitation>
<PubmedData><History><PubMedPubDate PubStatus="entrez"><Year>2010</Year>
<Month>7</Month>
<Day>16</Day>
<Hour>6</Hour>
<Minute>0</Minute>
</PubMedPubDate>
<PubMedPubDate PubStatus="pubmed"><Year>2010</Year>
<Month>7</Month>
<Day>16</Day>
<Hour>6</Hour>
<Minute>0</Minute>
</PubMedPubDate>
<PubMedPubDate PubStatus="medline"><Year>2010</Year>
<Month>12</Month>
<Day>14</Day>
<Hour>6</Hour>
<Minute>0</Minute>
</PubMedPubDate>
</History>
<PublicationStatus>ppublish</PublicationStatus>
<ArticleIdList><ArticleId IdType="pubmed">20631103</ArticleId>
<ArticleId IdType="pii">JCM.02231-09</ArticleId>
<ArticleId IdType="doi">10.1128/JCM.02231-09</ArticleId>
<ArticleId IdType="pmc">PMC2937686</ArticleId>
</ArticleIdList>
</PubmedData>
</pubmed>
<affiliations><list><country><li>Royaume-Uni</li>
</country>
</list>
<tree><noCountry><name sortKey="Chadwick, David" sort="Chadwick, David" uniqKey="Chadwick D" first="David" last="Chadwick">David Chadwick</name>
<name sortKey="Fraune, Maria" sort="Fraune, Maria" uniqKey="Fraune M" first="Maria" last="Fraune">Maria Fraune</name>
<name sortKey="Garcia, Ana" sort="Garcia, Ana" uniqKey="Garcia A" first="Ana" last="Garcia">Ana Garcia</name>
<name sortKey="Patel, Mauli" sort="Patel, Mauli" uniqKey="Patel M" first="Mauli" last="Patel">Mauli Patel</name>
<name sortKey="Phillips, Richard Odame" sort="Phillips, Richard Odame" uniqKey="Phillips R" first="Richard Odame" last="Phillips">Richard Odame Phillips</name>
<name sortKey="Sarfo, Fred Stephen" sort="Sarfo, Fred Stephen" uniqKey="Sarfo F" first="Fred Stephen" last="Sarfo">Fred Stephen Sarfo</name>
<name sortKey="Verheyen, Jens" sort="Verheyen, Jens" uniqKey="Verheyen J" first="Jens" last="Verheyen">Jens Verheyen</name>
</noCountry>
<country name="Royaume-Uni"><noRegion><name sortKey="Geretti, Anna Maria" sort="Geretti, Anna Maria" uniqKey="Geretti A" first="Anna Maria" last="Geretti">Anna Maria Geretti</name>
</noRegion>
</country>
</tree>
</affiliations>
</record>

Pour manipuler ce document sous Unix (Dilib)

EXPLOR_STEP=$WICRI_ROOT/Wicri/Sante/explor/SidaGhanaV1/Data/PubMed/Checkpoint

HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 000536 | SxmlIndent | more

HfdSelect -h $EXPLOR_AREA/Data/PubMed/Checkpoint/biblio.hfd -nk 000536 | SxmlIndent | more

Pour mettre un lien sur cette page dans le réseau Wicri

{{Explor lien
   |wiki=    Wicri/Sante
   |area=    SidaGhanaV1
   |flux=    PubMed
   |étape=   Checkpoint
   |type=    RBID
   |clé=     pubmed:20631103
   |texte=   Detection of highly prevalent hepatitis B virus coinfection among HIV-seropositive persons in Ghana.
}}

Pour générer des pages wiki

HfdIndexSelect -h $EXPLOR_AREA/Data/PubMed/Checkpoint/RBID.i   -Sk "pubmed:20631103" \
       | HfdSelect -Kh $EXPLOR_AREA/Data/PubMed/Checkpoint/biblio.hfd   \
       | NlmPubMed2Wicri -a SidaGhanaV1

This area was generated with Dilib version V0.6.31.
Data generation: Tue Nov 7 18:07:38 2017. Site generation: Tue Mar 5 15:01:57 2024

	Le SIDA au Ghana (serveur d'exploration)
	Attention, ce site est en cours de développement ! Attention, site généré par des moyens informatiques à partir de corpus bruts. Les informations ne sont donc pas validées.

Le SIDA au Ghana (serveur d'exploration)

Detection of highly prevalent hepatitis B virus coinfection among HIV-seropositive persons in Ghana.

Detection of highly prevalent hepatitis B virus coinfection among HIV-seropositive persons in Ghana.

Source :

Descripteurs français

English descriptors

Abstract

Links toward previous steps (curation, corpus...)

Links to Exploration step

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri

Pour générer des pages wiki