HBV/HIV co-infection and APOBEC3G polymorphisms in a population from Burkina Faso.
Identifieur interne : 000194 ( Main/Curation ); précédent : 000193; suivant : 000195HBV/HIV co-infection and APOBEC3G polymorphisms in a population from Burkina Faso.
Auteurs : Tegwinde Rebeca Compaore [Burkina Faso] ; Birama Diarra [Burkina Faso] ; Maleki Assih [Burkina Faso] ; Dorcas Obiri-Yeboah [Ghana] ; Serge Theophile Soubeiga [Burkina Faso] ; Abdoul Karim Ouattara [Burkina Faso] ; Damehan Tchelougou [Burkina Faso] ; Cyrille Bisseye [Burkina Faso] ; Didier Romuald Bakouan [Burkina Faso] ; Issaka Pierre Compaore [Burkina Faso] ; Augustine Dembele [Burkina Faso] ; Wendkuuni Florencia Djigma [Burkina Faso] ; Jacques Simpore [Burkina Faso]Source :
- BMC infectious diseases [ 1471-2334 ] ; 2016.
Descripteurs français
- KwdFr :
- APOBEC-3G Deaminase (génétique), Adulte, Allèles, Burkina (épidémiologie), Co-infection (génétique), Co-infection (épidémiologie), Femelle, Fréquence d'allèle, Génotype, Haplotypes, Humains, Hépatite B (génétique), Hépatite B (épidémiologie), Infections à VIH (génétique), Infections à VIH (épidémiologie), Mâle, Polymorphisme de nucléotide simple, Prédisposition génétique à une maladie, Prévalence, VIH-1 (Virus de l'Immunodéficience Humaine de type 1) (pathogénicité), Virus de l'hépatite B (pathogénicité), Études cas-témoins, Études d'associations génétiques.
- MESH :
- génétique : APOBEC-3G Deaminase, Co-infection, Hépatite B, Infections à VIH.
- pathogénicité : VIH-1 (Virus de l'Immunodéficience Humaine de type 1), Virus de l'hépatite B.
- épidémiologie : Burkina, Co-infection, Hépatite B, Infections à VIH.
- Adulte, Allèles, Femelle, Fréquence d'allèle, Génotype, Haplotypes, Humains, Mâle, Polymorphisme de nucléotide simple, Prédisposition génétique à une maladie, Prévalence, Études cas-témoins, Études d'associations génétiques.
English descriptors
- KwdEn :
- APOBEC-3G Deaminase (genetics), Adult, Alleles, Burkina Faso (epidemiology), Case-Control Studies, Coinfection (epidemiology), Coinfection (genetics), Female, Gene Frequency, Genetic Association Studies, Genetic Predisposition to Disease, Genotype, HIV Infections (epidemiology), HIV Infections (genetics), HIV-1 (pathogenicity), Haplotypes, Hepatitis B (epidemiology), Hepatitis B (genetics), Hepatitis B virus (pathogenicity), Humans, Male, Polymorphism, Single Nucleotide, Prevalence.
- MESH :
- chemical , genetics : APOBEC-3G Deaminase.
- epidemiology : Burkina Faso, Coinfection, HIV Infections, Hepatitis B.
- genetics : Coinfection, HIV Infections, Hepatitis B.
- pathogenicity : HIV-1, Hepatitis B virus.
- Adult, Alleles, Case-Control Studies, Female, Gene Frequency, Genetic Association Studies, Genetic Predisposition to Disease, Genotype, Haplotypes, Humans, Male, Polymorphism, Single Nucleotide, Prevalence.
Abstract
Apolipoprotein B mRNA editing enzyme catalytic polypeptide-like 3G (APOBEC3G) is a potent host defense factor, which interferes with HIV-1 and HBV. Our study had three objectives, to screen a population of HIV-1 infected and uninfected patients in Burkina Faso for HBV, to screen the population for APOBEC3G variants rs6001417, rs8177832, and rs35228531 previously described, and to analyze the effect of these three variants and their haplotypes on HIV-1/HBV co-infection in Burkina Faso.
DOI: 10.1186/s12879-016-1672-2
PubMed: 27449138
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pubmed:27449138Le document en format XML
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<author><name sortKey="Ouattara, Abdoul Karim" sort="Ouattara, Abdoul Karim" uniqKey="Ouattara A" first="Abdoul Karim" last="Ouattara">Abdoul Karim Ouattara</name>
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<author><name sortKey="Tchelougou, Damehan" sort="Tchelougou, Damehan" uniqKey="Tchelougou D" first="Damehan" last="Tchelougou">Damehan Tchelougou</name>
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<country xml:lang="fr">Burkina Faso</country>
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<author><name sortKey="Bisseye, Cyrille" sort="Bisseye, Cyrille" uniqKey="Bisseye C" first="Cyrille" last="Bisseye">Cyrille Bisseye</name>
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<country xml:lang="fr">Burkina Faso</country>
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<author><name sortKey="Bakouan, Didier Romuald" sort="Bakouan, Didier Romuald" uniqKey="Bakouan D" first="Didier Romuald" last="Bakouan">Didier Romuald Bakouan</name>
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<author><name sortKey="Compaore, Issaka Pierre" sort="Compaore, Issaka Pierre" uniqKey="Compaore I" first="Issaka Pierre" last="Compaore">Issaka Pierre Compaore</name>
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<country xml:lang="fr">Burkina Faso</country>
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<author><name sortKey="Dembele, Augustine" sort="Dembele, Augustine" uniqKey="Dembele A" first="Augustine" last="Dembele">Augustine Dembele</name>
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<author><name sortKey="Djigma, Wendkuuni Florencia" sort="Djigma, Wendkuuni Florencia" uniqKey="Djigma W" first="Wendkuuni Florencia" last="Djigma">Wendkuuni Florencia Djigma</name>
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<country xml:lang="fr">Burkina Faso</country>
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<author><name sortKey="Simpore, Jacques" sort="Simpore, Jacques" uniqKey="Simpore J" first="Jacques" last="Simpore">Jacques Simpore</name>
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<series><title level="j">BMC infectious diseases</title>
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<imprint><date when="2016" type="published">2016</date>
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>APOBEC-3G Deaminase (genetics)</term>
<term>Adult</term>
<term>Alleles</term>
<term>Burkina Faso (epidemiology)</term>
<term>Case-Control Studies</term>
<term>Coinfection (epidemiology)</term>
<term>Coinfection (genetics)</term>
<term>Female</term>
<term>Gene Frequency</term>
<term>Genetic Association Studies</term>
<term>Genetic Predisposition to Disease</term>
<term>Genotype</term>
<term>HIV Infections (epidemiology)</term>
<term>HIV Infections (genetics)</term>
<term>HIV-1 (pathogenicity)</term>
<term>Haplotypes</term>
<term>Hepatitis B (epidemiology)</term>
<term>Hepatitis B (genetics)</term>
<term>Hepatitis B virus (pathogenicity)</term>
<term>Humans</term>
<term>Male</term>
<term>Polymorphism, Single Nucleotide</term>
<term>Prevalence</term>
</keywords>
<keywords scheme="KwdFr" xml:lang="fr"><term>APOBEC-3G Deaminase (génétique)</term>
<term>Adulte</term>
<term>Allèles</term>
<term>Burkina (épidémiologie)</term>
<term>Co-infection (génétique)</term>
<term>Co-infection (épidémiologie)</term>
<term>Femelle</term>
<term>Fréquence d'allèle</term>
<term>Génotype</term>
<term>Haplotypes</term>
<term>Humains</term>
<term>Hépatite B (génétique)</term>
<term>Hépatite B (épidémiologie)</term>
<term>Infections à VIH (génétique)</term>
<term>Infections à VIH (épidémiologie)</term>
<term>Mâle</term>
<term>Polymorphisme de nucléotide simple</term>
<term>Prédisposition génétique à une maladie</term>
<term>Prévalence</term>
<term>VIH-1 (Virus de l'Immunodéficience Humaine de type 1) (pathogénicité)</term>
<term>Virus de l'hépatite B (pathogénicité)</term>
<term>Études cas-témoins</term>
<term>Études d'associations génétiques</term>
</keywords>
<keywords scheme="MESH" type="chemical" qualifier="genetics" xml:lang="en"><term>APOBEC-3G Deaminase</term>
</keywords>
<keywords scheme="MESH" qualifier="epidemiology" xml:lang="en"><term>Burkina Faso</term>
<term>Coinfection</term>
<term>HIV Infections</term>
<term>Hepatitis B</term>
</keywords>
<keywords scheme="MESH" qualifier="genetics" xml:lang="en"><term>Coinfection</term>
<term>HIV Infections</term>
<term>Hepatitis B</term>
</keywords>
<keywords scheme="MESH" qualifier="génétique" xml:lang="fr"><term>APOBEC-3G Deaminase</term>
<term>Co-infection</term>
<term>Hépatite B</term>
<term>Infections à VIH</term>
</keywords>
<keywords scheme="MESH" qualifier="pathogenicity" xml:lang="en"><term>HIV-1</term>
<term>Hepatitis B virus</term>
</keywords>
<keywords scheme="MESH" qualifier="pathogénicité" xml:lang="fr"><term>VIH-1 (Virus de l'Immunodéficience Humaine de type 1)</term>
<term>Virus de l'hépatite B</term>
</keywords>
<keywords scheme="MESH" qualifier="épidémiologie" xml:lang="fr"><term>Burkina</term>
<term>Co-infection</term>
<term>Hépatite B</term>
<term>Infections à VIH</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Adult</term>
<term>Alleles</term>
<term>Case-Control Studies</term>
<term>Female</term>
<term>Gene Frequency</term>
<term>Genetic Association Studies</term>
<term>Genetic Predisposition to Disease</term>
<term>Genotype</term>
<term>Haplotypes</term>
<term>Humans</term>
<term>Male</term>
<term>Polymorphism, Single Nucleotide</term>
<term>Prevalence</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Adulte</term>
<term>Allèles</term>
<term>Femelle</term>
<term>Fréquence d'allèle</term>
<term>Génotype</term>
<term>Haplotypes</term>
<term>Humains</term>
<term>Mâle</term>
<term>Polymorphisme de nucléotide simple</term>
<term>Prédisposition génétique à une maladie</term>
<term>Prévalence</term>
<term>Études cas-témoins</term>
<term>Études d'associations génétiques</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">Apolipoprotein B mRNA editing enzyme catalytic polypeptide-like 3G (APOBEC3G) is a potent host defense factor, which interferes with HIV-1 and HBV. Our study had three objectives, to screen a population of HIV-1 infected and uninfected patients in Burkina Faso for HBV, to screen the population for APOBEC3G variants rs6001417, rs8177832, and rs35228531 previously described, and to analyze the effect of these three variants and their haplotypes on HIV-1/HBV co-infection in Burkina Faso.</div>
</front>
</TEI>
</record>
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