Serveur d'exploration sur la maladie de Parkinson

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Dopaminergic deficiency and delayed visual evoked potentials in humans

Identifieur interne : 002B21 ( Main/Exploration ); précédent : 002B20; suivant : 002B22

Dopaminergic deficiency and delayed visual evoked potentials in humans

Auteurs : Bodis‐Wollner [États-Unis] ; Melvin D. Yahr [États-Unis] ; Leland Mylin [États-Unis] ; John Thornton [États-Unis]

Source :

RBID : ISTEX:DFFF8973BD334A0FDE6E6F3F8A3EAA7402E26834

Abstract

Abnormal delay in visual evoked potential (VEP) could be normalized in 9 of 14 previously untreated parkinsonian patients under the influence of levodopa/carbidopa therapy. In 6 of 11 previously untreated schizophrenic patients the VEP latency became abnormal following therapy with agents known to cause dopaminergic blockade. Besides defects in retrobulbar conduction secondary to demyelination, synaptic dysfunction due to neurotransmitter deficiency or receptor blockade must also be considered as a mechanism underlying VEP delays in humans.

Url:
DOI: 10.1002/ana.410110507


Affiliations:


Links toward previous steps (curation, corpus...)


Le document en format XML

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