Management of Tachyarrhythmias with Dual‐Chamber Pacemakers
Identifieur interne : 003100 ( Main/Corpus ); précédent : 003099; suivant : 003101Management of Tachyarrhythmias with Dual‐Chamber Pacemakers
Auteurs : Victor Medina-Ravell ; Agustin Castellanos ; Bolivar Portillo-Acosta ; Castor Maduro-Maytin ; Luis Rodriguez-Salas ; Manuel Hernandez-Arenas ; Raul La Salle-Toro ; Ivan Mendoza-Mujica ; Manuel Ortega-Maldonado ; Barouh V. BerkovitsSource :
- Pacing and Clinical Electrophysiology [ 0147-8389 ] ; 1983-03.
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Abstract
Des stimulaleurs multiprogrammables AV séquentiels de type “double‐demande” (DVI, MN) ont été implantés chez 23 patienls porteurs ?anomalies variées de tachycardies supraventriculaires. De plus, des stimuloteurs AV séquenfiels à paire “nonobligée” (DVI, MN e(DDD, M) ont été utilisés pour le traitement de lachyarhythmies ventriculaires. Nous avons observé que ľexpérience avec ee type de stimulateur multiprogrammable est favorable chez les patients sans fibrillation auriculaire chronique, qui ont besoin ?un stimutafeur et qui nécéssitent un froilement anti‐tachycardique. Ľévolution des stimulateurs du type DDD contribuera sans doute à un traitement encore plus éfficace. Multi‐programmable dual‐demand AV sequential (DVI, MN) pacemakers were implanted in twenty‐three potients (in one of them a DVI, MN unit was used as a VVI, MN with the aid of an atrial plug) with suproventricular tachycardias after electrophysiological studies revealed a great voriety of AV reentry circuits. The latter included tachycardios involving accessory pathways of the Kent type, manifest or concealed Wolff‐Parkinson‐White syndromes, nodo‐ventricular (Mahaim) tracts, “enhanced” AV node for extra AV nodal) pothwaysand dual AV pathways. In addition, multiprogrammable “non‐committed” AV sequential (DVI, MN and DDD, M) pacemakers were permanently implonted to treat different forms of ventricular tachyarrhythmias that included: torsode de pointes in the Romano‐Ward syndrome and Chagas' cardiomyopathy, ventricular tachycordia which is bradycardia‐dependent (in Chagas' cardiomyopathy) and reciprocal beats indueed by, and producing severe hemodynamic derangements in a patient with a conventional VVI unit. With smallsize multiprogrammable units, arrhythmias may be treated by changing parameters non‐invasively. By temporary inhibition, one may analyze the underlying rhythm and pacemaker dependency. In potients without chronic atrial flutterJfibrillation who require pacing and possibly tachyarrhythmia control, our experience with multiprogrammable “non‐committed” AV sequential pacing has been very satisfactory. The evolution toward newer pacing modes which provide atrial sensing and trackmg (DDD), and thus preserve AV synchrony over a wider range of atrial rates, may contribute even further to successful patient management. This may be applicable to pediatric patients as well.
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DOI: 10.1111/j.1540-8159.1983.tb04370.x
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<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Management of Tachyarrhythmias with Dual‐Chamber Pacemakers</title><author><name sortKey="Medina Avell, Victor" sort="Medina Avell, Victor" uniqKey="Medina Avell V" first="Victor" last="Medina-Ravell">Victor Medina-Ravell</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Castellanos, Agustin" sort="Castellanos, Agustin" uniqKey="Castellanos A" first="Agustin" last="Castellanos">Agustin Castellanos</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Portillo Costa, Bolivar" sort="Portillo Costa, Bolivar" uniqKey="Portillo Costa B" first="Bolivar" last="Portillo-Acosta">Bolivar Portillo-Acosta</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Maduro Aytin, Castor" sort="Maduro Aytin, Castor" uniqKey="Maduro Aytin C" first="Castor" last="Maduro-Maytin">Castor Maduro-Maytin</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. 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Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Ortega Aldonado, Manuel" sort="Ortega Aldonado, Manuel" uniqKey="Ortega Aldonado M" first="Manuel" last="Ortega-Maldonado">Manuel Ortega-Maldonado</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Berkovits, Barouh V" sort="Berkovits, Barouh V" uniqKey="Berkovits B" first="Barouh V." last="Berkovits">Barouh V. Berkovits</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:AACFFEDD8E44364702A7E58A9B5774404E99670F</idno><date when="1983" year="1983">1983</date><idno type="doi">10.1111/j.1540-8159.1983.tb04370.x</idno><idno type="url">https://api.istex.fr/document/AACFFEDD8E44364702A7E58A9B5774404E99670F/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">003100</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Management of Tachyarrhythmias with Dual‐Chamber Pacemakers</title><author><name sortKey="Medina Avell, Victor" sort="Medina Avell, Victor" uniqKey="Medina Avell V" first="Victor" last="Medina-Ravell">Victor Medina-Ravell</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Castellanos, Agustin" sort="Castellanos, Agustin" uniqKey="Castellanos A" first="Agustin" last="Castellanos">Agustin Castellanos</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Portillo Costa, Bolivar" sort="Portillo Costa, Bolivar" uniqKey="Portillo Costa B" first="Bolivar" last="Portillo-Acosta">Bolivar Portillo-Acosta</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Maduro Aytin, Castor" sort="Maduro Aytin, Castor" uniqKey="Maduro Aytin C" first="Castor" last="Maduro-Maytin">Castor Maduro-Maytin</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Rodriguez Alas, Luis" sort="Rodriguez Alas, Luis" uniqKey="Rodriguez Alas L" first="Luis" last="Rodriguez-Salas">Luis Rodriguez-Salas</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Hernandez Renas, Manuel" sort="Hernandez Renas, Manuel" uniqKey="Hernandez Renas M" first="Manuel" last="Hernandez-Arenas">Manuel Hernandez-Arenas</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="La Salle Oro, Raul" sort="La Salle Oro, Raul" uniqKey="La Salle Oro R" first="Raul" last="La Salle-Toro">Raul La Salle-Toro</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Mendoza Ujica, Ivan" sort="Mendoza Ujica, Ivan" uniqKey="Mendoza Ujica I" first="Ivan" last="Mendoza-Mujica">Ivan Mendoza-Mujica</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Ortega Aldonado, Manuel" sort="Ortega Aldonado, Manuel" uniqKey="Ortega Aldonado M" first="Manuel" last="Ortega-Maldonado">Manuel Ortega-Maldonado</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author><author><name sortKey="Berkovits, Barouh V" sort="Berkovits, Barouh V" uniqKey="Berkovits B" first="Barouh V." last="Berkovits">Barouh V. Berkovits</name><affiliation><mods:affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Pacing and Clinical Electrophysiology</title><idno type="ISSN">0147-8389</idno><idno type="eISSN">1540-8159</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1983-03">1983-03</date><biblScope unit="volume">6</biblScope><biblScope unit="issue">2</biblScope><biblScope unit="page" from="333">333</biblScope><biblScope unit="page" to="345">345</biblScope></imprint><idno type="ISSN">0147-8389</idno></series><idno type="istex">AACFFEDD8E44364702A7E58A9B5774404E99670F</idno><idno type="DOI">10.1111/j.1540-8159.1983.tb04370.x</idno><idno type="ArticleID">PACE333</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0147-8389</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>dualchamber pacemakers</term><term>multiprogrammable pacemakers</term><term>supraventriculor tachycardias</term><term>ventricular tachyarrhythmias</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Des stimulaleurs multiprogrammables AV séquentiels de type “double‐demande” (DVI, MN) ont été implantés chez 23 patienls porteurs ?anomalies variées de tachycardies supraventriculaires. De plus, des stimuloteurs AV séquenfiels à paire “nonobligée” (DVI, MN e(DDD, M) ont été utilisés pour le traitement de lachyarhythmies ventriculaires. Nous avons observé que ľexpérience avec ee type de stimulateur multiprogrammable est favorable chez les patients sans fibrillation auriculaire chronique, qui ont besoin ?un stimutafeur et qui nécéssitent un froilement anti‐tachycardique. Ľévolution des stimulateurs du type DDD contribuera sans doute à un traitement encore plus éfficace. Multi‐programmable dual‐demand AV sequential (DVI, MN) pacemakers were implanted in twenty‐three potients (in one of them a DVI, MN unit was used as a VVI, MN with the aid of an atrial plug) with suproventricular tachycardias after electrophysiological studies revealed a great voriety of AV reentry circuits. The latter included tachycardios involving accessory pathways of the Kent type, manifest or concealed Wolff‐Parkinson‐White syndromes, nodo‐ventricular (Mahaim) tracts, “enhanced” AV node for extra AV nodal) pothwaysand dual AV pathways. In addition, multiprogrammable “non‐committed” AV sequential (DVI, MN and DDD, M) pacemakers were permanently implonted to treat different forms of ventricular tachyarrhythmias that included: torsode de pointes in the Romano‐Ward syndrome and Chagas' cardiomyopathy, ventricular tachycordia which is bradycardia‐dependent (in Chagas' cardiomyopathy) and reciprocal beats indueed by, and producing severe hemodynamic derangements in a patient with a conventional VVI unit. With smallsize multiprogrammable units, arrhythmias may be treated by changing parameters non‐invasively. By temporary inhibition, one may analyze the underlying rhythm and pacemaker dependency. In potients without chronic atrial flutterJfibrillation who require pacing and possibly tachyarrhythmia control, our experience with multiprogrammable “non‐committed” AV sequential pacing has been very satisfactory. The evolution toward newer pacing modes which provide atrial sensing and trackmg (DDD), and thus preserve AV synchrony over a wider range of atrial rates, may contribute even further to successful patient management. This may be applicable to pediatric patients as well.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>VICTOR MEDINA‐RAVELL</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item><json:item><name>AGUSTIN CASTELLANOS</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item><json:item><name>BOLIVAR PORTILLO‐ACOSTA</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item><json:item><name>CASTOR MADURO‐MAYTIN</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item><json:item><name>LUIS RODRIGUEZ‐SALAS</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item><json:item><name>MANUEL HERNANDEZ‐ARENAS</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item><json:item><name>RAUL LA SALLE‐TORO</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item><json:item><name>IVAN MENDOZA‐MUJICA</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item><json:item><name>MANUEL ORTEGA‐MALDONADO</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item><json:item><name>BAROUH V. BERKOVITS</name><affiliations><json:string>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>supraventriculor tachycardias</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>multiprogrammable pacemakers</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>ventricular tachyarrhythmias</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>dualchamber pacemakers</value></json:item></subject><articleId><json:string>PACE333</json:string></articleId><language><json:string>eng</json:string></language><abstract>Des stimulaleurs multiprogrammables AV séquentiels de type “double‐demande” (DVI, MN) ont été implantés chez 23 patienls porteurs ?anomalies variées de tachycardies supraventriculaires. De plus, des stimuloteurs AV séquenfiels à paire “nonobligée” (DVI, MN e(DDD, M) ont été utilisés pour le traitement de lachyarhythmies ventriculaires. Nous avons observé que ľexpérience avec ee type de stimulateur multiprogrammable est favorable chez les patients sans fibrillation auriculaire chronique, qui ont besoin ?un stimutafeur et qui nécéssitent un froilement anti‐tachycardique. Ľévolution des stimulateurs du type DDD contribuera sans doute à un traitement encore plus éfficace. Multi‐programmable dual‐demand AV sequential (DVI, MN) pacemakers were implanted in twenty‐three potients (in one of them a DVI, MN unit was used as a VVI, MN with the aid of an atrial plug) with suproventricular tachycardias after electrophysiological studies revealed a great voriety of AV reentry circuits. The latter included tachycardios involving accessory pathways of the Kent type, manifest or concealed Wolff‐Parkinson‐White syndromes, nodo‐ventricular (Mahaim) tracts, “enhanced” AV node for extra AV nodal) pothwaysand dual AV pathways. In addition, multiprogrammable “non‐committed” AV sequential (DVI, MN and DDD, M) pacemakers were permanently implonted to treat different forms of ventricular tachyarrhythmias that included: torsode de pointes in the Romano‐Ward syndrome and Chagas' cardiomyopathy, ventricular tachycordia which is bradycardia‐dependent (in Chagas' cardiomyopathy) and reciprocal beats indueed by, and producing severe hemodynamic derangements in a patient with a conventional VVI unit. With smallsize multiprogrammable units, arrhythmias may be treated by changing parameters non‐invasively. By temporary inhibition, one may analyze the underlying rhythm and pacemaker dependency. In potients without chronic atrial flutterJfibrillation who require pacing and possibly tachyarrhythmia control, our experience with multiprogrammable “non‐committed” AV sequential pacing has been very satisfactory. The evolution toward newer pacing modes which provide atrial sensing and trackmg (DDD), and thus preserve AV synchrony over a wider range of atrial rates, may contribute even further to successful patient management. 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Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author><author><persName><forename type="first">AGUSTIN</forename><surname>CASTELLANOS</surname></persName><note type="correspondence"><p>Correspondence: Address for reprinls: Agustin Caslellanos, M.D., Division of Cardioiogy (D‐39), University of Miami, School of Medicine. P.O. Box 016960. Miami, Florida 33101.</p></note><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author><author><persName><forename type="first">BOLIVAR</forename><surname>PORTILLO‐ACOSTA</surname></persName><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author><author><persName><forename type="first">CASTOR</forename><surname>MADURO‐MAYTIN</surname></persName><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author><author><persName><forename type="first">LUIS</forename><surname>RODRIGUEZ‐SALAS</surname></persName><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author><author><persName><forename type="first">MANUEL</forename><surname>HERNANDEZ‐ARENAS</surname></persName><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author><author><persName><forename type="first">RAUL</forename><surname>LA SALLE‐TORO</surname></persName><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author><author><persName><forename type="first">IVAN</forename><surname>MENDOZA‐MUJICA</surname></persName><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author><author><persName><forename type="first">MANUEL</forename><surname>ORTEGA‐MALDONADO</surname></persName><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author><author><persName><forename type="first">BAROUH V.</forename><surname>BERKOVITS</surname></persName><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation></author></analytic><monogr><title level="j">Pacing and Clinical Electrophysiology</title><idno type="pISSN">0147-8389</idno><idno type="eISSN">1540-8159</idno><idno type="DOI">10.1111/(ISSN)1540-8159</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1983-03"></date><biblScope unit="volume">6</biblScope><biblScope unit="issue">2</biblScope><biblScope unit="page" from="333">333</biblScope><biblScope unit="page" to="345">345</biblScope></imprint></monogr><idno type="istex">AACFFEDD8E44364702A7E58A9B5774404E99670F</idno><idno type="DOI">10.1111/j.1540-8159.1983.tb04370.x</idno><idno type="ArticleID">PACE333</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1983</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Des stimulaleurs multiprogrammables AV séquentiels de type “double‐demande” (DVI, MN) ont été implantés chez 23 patienls porteurs ?anomalies variées de tachycardies supraventriculaires. De plus, des stimuloteurs AV séquenfiels à paire “nonobligée” (DVI, MN e(DDD, M) ont été utilisés pour le traitement de lachyarhythmies ventriculaires. Nous avons observé que ľexpérience avec ee type de stimulateur multiprogrammable est favorable chez les patients sans fibrillation auriculaire chronique, qui ont besoin ?un stimutafeur et qui nécéssitent un froilement anti‐tachycardique. Ľévolution des stimulateurs du type DDD contribuera sans doute à un traitement encore plus éfficace. Multi‐programmable dual‐demand AV sequential (DVI, MN) pacemakers were implanted in twenty‐three potients (in one of them a DVI, MN unit was used as a VVI, MN with the aid of an atrial plug) with suproventricular tachycardias after electrophysiological studies revealed a great voriety of AV reentry circuits. The latter included tachycardios involving accessory pathways of the Kent type, manifest or concealed Wolff‐Parkinson‐White syndromes, nodo‐ventricular (Mahaim) tracts, “enhanced” AV node for extra AV nodal) pothwaysand dual AV pathways. In addition, multiprogrammable “non‐committed” AV sequential (DVI, MN and DDD, M) pacemakers were permanently implonted to treat different forms of ventricular tachyarrhythmias that included: torsode de pointes in the Romano‐Ward syndrome and Chagas' cardiomyopathy, ventricular tachycordia which is bradycardia‐dependent (in Chagas' cardiomyopathy) and reciprocal beats indueed by, and producing severe hemodynamic derangements in a patient with a conventional VVI unit. With smallsize multiprogrammable units, arrhythmias may be treated by changing parameters non‐invasively. By temporary inhibition, one may analyze the underlying rhythm and pacemaker dependency. In potients without chronic atrial flutterJfibrillation who require pacing and possibly tachyarrhythmia control, our experience with multiprogrammable “non‐committed” AV sequential pacing has been very satisfactory. The evolution toward newer pacing modes which provide atrial sensing and trackmg (DDD), and thus preserve AV synchrony over a wider range of atrial rates, may contribute even further to successful patient management. This may be applicable to pediatric patients as well.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>supraventriculor tachycardias</term></item><item><term>multiprogrammable pacemakers</term></item><item><term>ventricular tachyarrhythmias</term></item><item><term>dualchamber pacemakers</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1983-03">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/AACFFEDD8E44364702A7E58A9B5774404E99670F/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1540-8159</doi><issn type="print">0147-8389</issn><issn type="electronic">1540-8159</issn><idGroup><id type="product" value="PACE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PACING CLINICAL ELECTROPHYSIOLOGY">Pacing and Clinical Electrophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="03002"><doi origin="wiley">10.1111/pace.1983.6.issue-2</doi><numberingGroup><numbering type="journalVolume" number="6">6</numbering><numbering type="journalIssue" number="2">2</numbering></numberingGroup><coverDate startDate="1983-03">March 1983</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0033300" status="forIssue"><doi origin="wiley">10.1111/j.1540-8159.1983.tb04370.x</doi><idGroup><id type="unit" value="PACE333"></id></idGroup><countGroup><count type="pageTotal" number="13"></count></countGroup><titleGroup><title type="tocHeading1">PHYSIOLOGY OF DUAL‐CHAMBER PACING</title></titleGroup><eventGroup><event type="firstOnline" date="2006-06-30"></event><event type="publishedOnlineFinalForm" date="2006-06-30"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:HeaderRef result:HeaderRef" date="2010-03-02"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-06"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-11-03"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="333">333</numbering><numbering type="pageLast" number="345">345</numbering></numberingGroup><correspondenceTo>Address for reprinls: Agustin Caslellanos, M.D., Division of Cardioiogy (D‐39), University of Miami, School of Medicine. P.O. Box 016960. Miami, Florida 33101.</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:PACE.PACE333.pdf"></link></linkGroup></publicationMeta><contentMeta><countGroup><count type="referenceTotal" number="25"></count><count type="linksCrossRef" number="11"></count></countGroup><titleGroup><title type="main">Management of Tachyarrhythmias with Dual‐Chamber Pacemakers</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1"><personName><givenNames>VICTOR</givenNames><familyName>MEDINA‐RAVELL</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1" corresponding="yes"><personName><givenNames>AGUSTIN</givenNames><familyName>CASTELLANOS</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>BOLIVAR</givenNames><familyName>PORTILLO‐ACOSTA</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a1"><personName><givenNames>CASTOR</givenNames><familyName>MADURO‐MAYTIN</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a1"><personName><givenNames>LUIS</givenNames><familyName>RODRIGUEZ‐SALAS</familyName></personName></creator><creator creatorRole="author" xml:id="cr6" affiliationRef="#a1"><personName><givenNames>MANUEL</givenNames><familyName>HERNANDEZ‐ARENAS</familyName></personName></creator><creator creatorRole="author" xml:id="cr7" affiliationRef="#a1"><personName><givenNames>RAUL</givenNames><familyNamePrefix>LA</familyNamePrefix><familyName>SALLE‐TORO</familyName></personName></creator><creator creatorRole="author" xml:id="cr8" affiliationRef="#a1"><personName><givenNames>IVAN</givenNames><familyName>MENDOZA‐MUJICA</familyName></personName></creator><creator creatorRole="author" xml:id="cr9" affiliationRef="#a1"><personName><givenNames>MANUEL</givenNames><familyName>ORTEGA‐MALDONADO</familyName></personName></creator><creator creatorRole="author" xml:id="cr10" affiliationRef="#a1"><personName><givenNames>BAROUH V.</givenNames><familyName>BERKOVITS</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="US"><unparsedAffiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">supraventriculor tachycardias</keyword><keyword xml:id="k2">multiprogrammable pacemakers</keyword><keyword xml:id="k3">ventricular tachyarrhythmias</keyword><keyword xml:id="k4">dualchamber pacemakers</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>Des stimulaleurs multiprogrammables AV séquentiels de type “double‐demande” (DVI, MN) ont été implantés chez 23 patienls porteurs ?anomalies variées de tachycardies supraventriculaires. De plus, des stimuloteurs AV séquenfiels à paire “nonobligée” (DVI, MN e(DDD, M) ont été utilisés pour le traitement de lachyarhythmies ventriculaires. Nous avons observé que ľexpérience avec ee type de stimulateur multiprogrammable est favorable chez les patients sans fibrillation auriculaire chronique, qui ont besoin ?un stimutafeur et qui nécéssitent un froilement anti‐tachycardique. Ľévolution des stimulateurs du type DDD contribuera sans doute à un traitement encore plus éfficace.</p><p>Multi‐programmable dual‐demand AV sequential (DVI, MN) pacemakers were implanted in twenty‐three potients (in one of them a DVI, MN unit was used as a VVI, MN with the aid of an atrial plug) with suproventricular tachycardias after electrophysiological studies revealed a great voriety of AV reentry circuits. The latter included tachycardios involving accessory pathways of the Kent type, manifest or concealed Wolff‐Parkinson‐White syndromes, nodo‐ventricular (Mahaim) tracts, “enhanced” AV node for extra AV nodal) pothwaysand dual AV pathways. In addition, multiprogrammable “non‐committed” AV sequential (DVI, MN and DDD, M) pacemakers were permanently implonted to treat different forms of ventricular tachyarrhythmias that included: torsode de pointes in the Romano‐Ward syndrome and Chagas' cardiomyopathy, ventricular tachycordia which is bradycardia‐dependent (in Chagas' cardiomyopathy) and reciprocal beats indueed by, and producing severe hemodynamic derangements in a patient with a conventional VVI unit. With smallsize multiprogrammable units, arrhythmias may be treated by changing parameters non‐invasively. By temporary inhibition, one may analyze the underlying rhythm and pacemaker dependency. In potients without chronic atrial flutterJfibrillation who require pacing and possibly tachyarrhythmia control, our experience with multiprogrammable “non‐committed” AV sequential pacing has been very satisfactory. The evolution toward newer pacing modes which provide atrial sensing and trackmg (DDD), and thus preserve AV synchrony over a wider range of atrial rates, may contribute even further to successful patient management. This may be applicable to pediatric patients as well.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Management of Tachyarrhythmias with Dual‐Chamber Pacemakers</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Management of Tachyarrhythmias with Dual‐Chamber Pacemakers</title></titleInfo><name type="personal"><namePart type="given">VICTOR</namePart><namePart type="family">MEDINA‐RAVELL</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">AGUSTIN</namePart><namePart type="family">CASTELLANOS</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><description>Correspondence: Address for reprinls: Agustin Caslellanos, M.D., Division of Cardioiogy (D‐39), University of Miami, School of Medicine. P.O. Box 016960. Miami, Florida 33101.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">BOLIVAR</namePart><namePart type="family">PORTILLO‐ACOSTA</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">CASTOR</namePart><namePart type="family">MADURO‐MAYTIN</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">LUIS</namePart><namePart type="family">RODRIGUEZ‐SALAS</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">MANUEL</namePart><namePart type="family">HERNANDEZ‐ARENAS</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">RAUL</namePart><namePart type="family">LA SALLE‐TORO</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">IVAN</namePart><namePart type="family">MENDOZA‐MUJICA</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">MANUEL</namePart><namePart type="family">ORTEGA‐MALDONADO</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">BAROUH V.</namePart><namePart type="family">BERKOVITS</namePart><affiliation>From Fundacion COR de Valencia and Hospital Central de Valencia. Valencia Venezuela and the Division of Cardiology, University of Miami School of Medicine, Miami, Florida</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1983-03</dateIssued><copyrightDate encoding="w3cdtf">1983</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">25</extent></physicalDescription><abstract lang="en">Des stimulaleurs multiprogrammables AV séquentiels de type “double‐demande” (DVI, MN) ont été implantés chez 23 patienls porteurs ?anomalies variées de tachycardies supraventriculaires. De plus, des stimuloteurs AV séquenfiels à paire “nonobligée” (DVI, MN e(DDD, M) ont été utilisés pour le traitement de lachyarhythmies ventriculaires. Nous avons observé que ľexpérience avec ee type de stimulateur multiprogrammable est favorable chez les patients sans fibrillation auriculaire chronique, qui ont besoin ?un stimutafeur et qui nécéssitent un froilement anti‐tachycardique. Ľévolution des stimulateurs du type DDD contribuera sans doute à un traitement encore plus éfficace. Multi‐programmable dual‐demand AV sequential (DVI, MN) pacemakers were implanted in twenty‐three potients (in one of them a DVI, MN unit was used as a VVI, MN with the aid of an atrial plug) with suproventricular tachycardias after electrophysiological studies revealed a great voriety of AV reentry circuits. The latter included tachycardios involving accessory pathways of the Kent type, manifest or concealed Wolff‐Parkinson‐White syndromes, nodo‐ventricular (Mahaim) tracts, “enhanced” AV node for extra AV nodal) pothwaysand dual AV pathways. In addition, multiprogrammable “non‐committed” AV sequential (DVI, MN and DDD, M) pacemakers were permanently implonted to treat different forms of ventricular tachyarrhythmias that included: torsode de pointes in the Romano‐Ward syndrome and Chagas' cardiomyopathy, ventricular tachycordia which is bradycardia‐dependent (in Chagas' cardiomyopathy) and reciprocal beats indueed by, and producing severe hemodynamic derangements in a patient with a conventional VVI unit. With smallsize multiprogrammable units, arrhythmias may be treated by changing parameters non‐invasively. By temporary inhibition, one may analyze the underlying rhythm and pacemaker dependency. In potients without chronic atrial flutterJfibrillation who require pacing and possibly tachyarrhythmia control, our experience with multiprogrammable “non‐committed” AV sequential pacing has been very satisfactory. The evolution toward newer pacing modes which provide atrial sensing and trackmg (DDD), and thus preserve AV synchrony over a wider range of atrial rates, may contribute even further to successful patient management. This may be applicable to pediatric patients as well.</abstract><subject lang="en"><genre>Keywords</genre><topic>supraventriculor tachycardias</topic><topic>multiprogrammable pacemakers</topic><topic>ventricular tachyarrhythmias</topic><topic>dualchamber pacemakers</topic></subject><relatedItem type="host"><titleInfo><title>Pacing and Clinical Electrophysiology</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0147-8389</identifier><identifier type="eISSN">1540-8159</identifier><identifier type="DOI">10.1111/(ISSN)1540-8159</identifier><identifier type="PublisherID">PACE</identifier><part><date>1983</date><detail type="volume"><caption>vol.</caption><number>6</number></detail><detail type="issue"><caption>no.</caption><number>2</number></detail><extent unit="pages"><start>333</start><end>345</end><total>13</total></extent></part></relatedItem><identifier type="istex">AACFFEDD8E44364702A7E58A9B5774404E99670F</identifier><identifier type="DOI">10.1111/j.1540-8159.1983.tb04370.x</identifier><identifier type="ArticleID">PACE333</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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