TRANSVENOUS ABLATION OF ATRIOVENTRICULAR CONDUCTION FOR REFRACTORY OR MALIGNANT SUPRAVENTRICULAR ARRHYTHMIAS
Identifieur interne : 002E80 ( Main/Corpus ); précédent : 002E79; suivant : 002E81TRANSVENOUS ABLATION OF ATRIOVENTRICULAR CONDUCTION FOR REFRACTORY OR MALIGNANT SUPRAVENTRICULAR ARRHYTHMIAS
Auteurs : M. J. E. Davis ; G. C. Mews ; G. D. CopeSource :
- Australian and New Zealand Journal of Medicine [ 0004-8291 ] ; 1984-08.
English descriptors
Abstract
In nine patients with recurrent disabling supraventricular arrhythmia refractory to, or intolerant of, multiple drug combinations, and two patients with Wolff‐Parkinson‐White (WPW) syndrome and documented malignant atrial fibrillation, transvenous ablation of atrioventricular (AV) conduction, utilising synchronised unipolar DC shocks delivered by catheter to the AV node‐His bundle or to the accessory AV pathway, was attempted. One to two 200–300 J discharges produced complete heart block in all of the nine patients, with markedly improved symptomatic status at one to ten month follow‐up including the one patient with recovery of modified AV conduction. Single 100–150 J shocks ablated pre‐excitation for five to fifteen minutes in the two WPW patients without subsequent modification of accessory pathway conduction. There were no complications. This simple technique has great potential and may supplant some open‐heart procedures. With refinement it may be possible to slow rather than to ablate AV‐His conduction, to ablate conduction via accessory AV pathways permanently and to interrupt ventricular re‐entrant circuits.
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DOI: 10.1111/j.1445-5994.1984.tb03620.x
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Davis</name><affiliation><mods:affiliation>Cardiology Registrar, Royal Perth Hospital, WA</mods:affiliation></affiliation></author><author><name sortKey="Mews, G C" sort="Mews, G C" uniqKey="Mews G" first="G. C." last="Mews">G. C. Mews</name><affiliation><mods:affiliation>Cardiologist, Royal Perth Hospital, WA</mods:affiliation></affiliation></author><author><name sortKey="Cope, G D" sort="Cope, G D" uniqKey="Cope G" first="G. D." last="Cope">G. D. Cope</name><affiliation><mods:affiliation>Cardiologist, Royal Perth Hospital, WA</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Australian and New Zealand Journal of Medicine</title><idno type="ISSN">0004-8291</idno><idno type="eISSN">1445-5994</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1984-08">1984-08</date><biblScope unit="volume">14</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="479">479</biblScope><biblScope unit="page" to="486">486</biblScope></imprint><idno type="ISSN">0004-8291</idno></series><idno type="istex">A44A92106D13F35B6343AC3017E347B56E8C7B45</idno><idno type="DOI">10.1111/j.1445-5994.1984.tb03620.x</idno><idno type="ArticleID">IMJ479</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0004-8291</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Heart conduction system</term><term>Wolff‐Parkinson‐White syndrome</term><term>arrhythmia</term><term>therapy</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">In nine patients with recurrent disabling supraventricular arrhythmia refractory to, or intolerant of, multiple drug combinations, and two patients with Wolff‐Parkinson‐White (WPW) syndrome and documented malignant atrial fibrillation, transvenous ablation of atrioventricular (AV) conduction, utilising synchronised unipolar DC shocks delivered by catheter to the AV node‐His bundle or to the accessory AV pathway, was attempted. One to two 200–300 J discharges produced complete heart block in all of the nine patients, with markedly improved symptomatic status at one to ten month follow‐up including the one patient with recovery of modified AV conduction. Single 100–150 J shocks ablated pre‐excitation for five to fifteen minutes in the two WPW patients without subsequent modification of accessory pathway conduction. There were no complications. This simple technique has great potential and may supplant some open‐heart procedures. With refinement it may be possible to slow rather than to ablate AV‐His conduction, to ablate conduction via accessory AV pathways permanently and to interrupt ventricular re‐entrant circuits.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>M. J. E. DAVIS</name><affiliations><json:string>Cardiology Registrar, Royal Perth Hospital, WA</json:string></affiliations></json:item><json:item><name>G. C. 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COPE</name><affiliations><json:string>Cardiologist, Royal Perth Hospital, WA</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Heart conduction system</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>arrhythmia</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Wolff‐Parkinson‐White syndrome</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>therapy</value></json:item></subject><articleId><json:string>IMJ479</json:string></articleId><language><json:string>eng</json:string></language><abstract>In nine patients with recurrent disabling supraventricular arrhythmia refractory to, or intolerant of, multiple drug combinations, and two patients with Wolff‐Parkinson‐White (WPW) syndrome and documented malignant atrial fibrillation, transvenous ablation of atrioventricular (AV) conduction, utilising synchronised unipolar DC shocks delivered by catheter to the AV node‐His bundle or to the accessory AV pathway, was attempted. 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J. E.</forename><surname>DAVIS</surname></persName><affiliation>Cardiology Registrar, Royal Perth Hospital, WA</affiliation></author><author><persName><forename type="first">G. C.</forename><surname>MEWS</surname></persName><affiliation>Cardiologist, Royal Perth Hospital, WA</affiliation></author><author><persName><forename type="first">G. D.</forename><surname>COPE</surname></persName><note type="correspondence"><p>Correspondence: Department of Cardiology, Royal Perth Hospital, Box X. 2213, GPO, Perth, Western Australia, 6001.</p></note><affiliation>Cardiologist, Royal Perth Hospital, WA</affiliation></author></analytic><monogr><title level="j">Australian and New Zealand Journal of Medicine</title><idno type="pISSN">0004-8291</idno><idno type="eISSN">1445-5994</idno><idno type="DOI">10.1111/(ISSN)1445-5994a</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1984-08"></date><biblScope unit="volume">14</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="479">479</biblScope><biblScope unit="page" to="486">486</biblScope></imprint></monogr><idno type="istex">A44A92106D13F35B6343AC3017E347B56E8C7B45</idno><idno type="DOI">10.1111/j.1445-5994.1984.tb03620.x</idno><idno type="ArticleID">IMJ479</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1984</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>In nine patients with recurrent disabling supraventricular arrhythmia refractory to, or intolerant of, multiple drug combinations, and two patients with Wolff‐Parkinson‐White (WPW) syndrome and documented malignant atrial fibrillation, transvenous ablation of atrioventricular (AV) conduction, utilising synchronised unipolar DC shocks delivered by catheter to the AV node‐His bundle or to the accessory AV pathway, was attempted. One to two 200–300 J discharges produced complete heart block in all of the nine patients, with markedly improved symptomatic status at one to ten month follow‐up including the one patient with recovery of modified AV conduction. Single 100–150 J shocks ablated pre‐excitation for five to fifteen minutes in the two WPW patients without subsequent modification of accessory pathway conduction. There were no complications. This simple technique has great potential and may supplant some open‐heart procedures. 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D.</givenNames><familyName>COPE</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1"><unparsedAffiliation>Cardiology Registrar, Royal Perth Hospital, WA</unparsedAffiliation></affiliation><affiliation xml:id="a2"><unparsedAffiliation>Cardiologist, Royal Perth Hospital, WA</unparsedAffiliation></affiliation><affiliation xml:id="a3"><unparsedAffiliation>Cardiologist, Royal Perth Hospital, WA</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">Heart conduction system</keyword><keyword xml:id="k2">arrhythmia</keyword><keyword xml:id="k3">Wolff‐Parkinson‐White syndrome</keyword><keyword xml:id="k4">therapy</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><title type="main">Abstract:</title><p>In nine patients with recurrent disabling supraventricular arrhythmia refractory to, or intolerant of, multiple drug combinations, and two patients with Wolff‐Parkinson‐White (WPW) syndrome and documented malignant atrial fibrillation, transvenous ablation of atrioventricular (AV) conduction, utilising synchronised unipolar DC shocks delivered by catheter to the AV node‐His bundle or to the accessory AV pathway, was attempted. One to two 200–300 J discharges produced complete heart block in all of the nine patients, with markedly improved symptomatic status at one to ten month follow‐up including the one patient with recovery of modified AV conduction. Single 100–150 J shocks ablated pre‐excitation for five to fifteen minutes in the two WPW patients without subsequent modification of accessory pathway conduction. There were no complications. This simple technique has great potential and may supplant some open‐heart procedures. With refinement it may be possible to slow rather than to ablate AV‐His conduction, to ablate conduction <i>via</i> accessory AV pathways permanently and to interrupt ventricular re‐entrant circuits.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>TRANSVENOUS ABLATION OF ATRIOVENTRICULAR CONDUCTION FOR REFRACTORY OR MALIGNANT SUPRAVENTRICULAR ARRHYTHMIAS</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>TRANSVENOUS ABLATION OF ATRIOVENTRICULAR CONDUCTION FOR REFRACTORY OR MALIGNANT SUPRAVENTRICULAR ARRHYTHMIAS</title></titleInfo><name type="personal"><namePart type="given">M. J. E.</namePart><namePart type="family">DAVIS</namePart><affiliation>Cardiology Registrar, Royal Perth Hospital, WA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">G. C.</namePart><namePart type="family">MEWS</namePart><affiliation>Cardiologist, Royal Perth Hospital, WA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">G. D.</namePart><namePart type="family">COPE</namePart><affiliation>Cardiologist, Royal Perth Hospital, WA</affiliation><description>Correspondence: Department of Cardiology, Royal Perth Hospital, Box X. 2213, GPO, Perth, Western Australia, 6001.</description><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1984-08</dateIssued><copyrightDate encoding="w3cdtf">1984</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">17</extent></physicalDescription><abstract lang="en">In nine patients with recurrent disabling supraventricular arrhythmia refractory to, or intolerant of, multiple drug combinations, and two patients with Wolff‐Parkinson‐White (WPW) syndrome and documented malignant atrial fibrillation, transvenous ablation of atrioventricular (AV) conduction, utilising synchronised unipolar DC shocks delivered by catheter to the AV node‐His bundle or to the accessory AV pathway, was attempted. One to two 200–300 J discharges produced complete heart block in all of the nine patients, with markedly improved symptomatic status at one to ten month follow‐up including the one patient with recovery of modified AV conduction. Single 100–150 J shocks ablated pre‐excitation for five to fifteen minutes in the two WPW patients without subsequent modification of accessory pathway conduction. There were no complications. This simple technique has great potential and may supplant some open‐heart procedures. With refinement it may be possible to slow rather than to ablate AV‐His conduction, to ablate conduction via accessory AV pathways permanently and to interrupt ventricular re‐entrant circuits.</abstract><subject lang="en"><genre>Keywords</genre><topic>Heart conduction system</topic><topic>arrhythmia</topic><topic>Wolff‐Parkinson‐White syndrome</topic><topic>therapy</topic></subject><relatedItem type="host"><titleInfo><title>Australian and New Zealand Journal of Medicine</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0004-8291</identifier><identifier type="eISSN">1445-5994</identifier><identifier type="DOI">10.1111/(ISSN)1445-5994a</identifier><identifier type="PublisherID">IMJ</identifier><part><date>1984</date><detail type="volume"><caption>vol.</caption><number>14</number></detail><detail type="issue"><caption>no.</caption><number>4</number></detail><extent unit="pages"><start>479</start><end>486</end><total>8</total></extent></part></relatedItem><identifier type="istex">A44A92106D13F35B6343AC3017E347B56E8C7B45</identifier><identifier type="DOI">10.1111/j.1445-5994.1984.tb03620.x</identifier><identifier type="ArticleID">IMJ479</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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