Serveur d'exploration sur la maladie de Parkinson

Attention, ce site est en cours de développement !
Attention, site généré par des moyens informatiques à partir de corpus bruts.
Les informations ne sont donc pas validées.

Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways

Identifieur interne : 002A38 ( Main/Corpus ); précédent : 002A37; suivant : 002A39

Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways

Auteurs : Wim T. J. Aanhaanen ; Antoon F. M. Moorman ; Vincent M. Christoffels

Source :

RBID : ISTEX:52ED3228596B2916AC57AB9B12D4367C13243B7E

English descriptors

Abstract

Defects originating from the atrioventricular canal region are part of a wide spectrum of congenital cardiovascular malformations that frequently affect newborns. These defects include partial or complete atrioventricular septal defects, atrioventricular valve defects, and arrhythmias, such as atrioventricular re‐entry tachycardia, atrioventricular nodal block, and ventricular preexcitation. Insight into the cellular origin of the atrioventricular canal myocardium and the molecular mechanisms that control its development will aid in the understanding of the etiology of the atrioventricular defects. This review discusses current knowledge concerning the origin and fate of the atrioventricular canal myocardium, the molecular mechanisms that determine its specification and differentiation, and its role in the development of certain malformations such as those that underlie ventricular preexcitation. Birth Defects Research (Part A), 2011. © 2011Wiley‐Liss, Inc.

Url:
DOI: 10.1002/bdra.20826

Links to Exploration step

ISTEX:52ED3228596B2916AC57AB9B12D4367C13243B7E

Le document en format XML

<record>
<TEI wicri:istexFullTextTei="biblStruct">
<teiHeader>
<fileDesc>
<titleStmt>
<title xml:lang="en">Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways</title>
<author>
<name sortKey="Aanhaanen, Wim T J" sort="Aanhaanen, Wim T J" uniqKey="Aanhaanen W" first="Wim T. J." last="Aanhaanen">Wim T. J. Aanhaanen</name>
<affiliation>
<mods:affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</mods:affiliation>
</affiliation>
</author>
<author>
<name sortKey="Moorman, Antoon F M" sort="Moorman, Antoon F M" uniqKey="Moorman A" first="Antoon F. M." last="Moorman">Antoon F. M. Moorman</name>
<affiliation>
<mods:affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</mods:affiliation>
</affiliation>
</author>
<author>
<name sortKey="Christoffels, Vincent M" sort="Christoffels, Vincent M" uniqKey="Christoffels V" first="Vincent M." last="Christoffels">Vincent M. Christoffels</name>
<affiliation>
<mods:affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</mods:affiliation>
</affiliation>
</author>
</titleStmt>
<publicationStmt>
<idno type="wicri:source">ISTEX</idno>
<idno type="RBID">ISTEX:52ED3228596B2916AC57AB9B12D4367C13243B7E</idno>
<date when="2011" year="2011">2011</date>
<idno type="doi">10.1002/bdra.20826</idno>
<idno type="url">https://api.istex.fr/document/52ED3228596B2916AC57AB9B12D4367C13243B7E/fulltext/pdf</idno>
<idno type="wicri:Area/Main/Corpus">002A38</idno>
</publicationStmt>
<sourceDesc>
<biblStruct>
<analytic>
<title level="a" type="main" xml:lang="en">Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways</title>
<author>
<name sortKey="Aanhaanen, Wim T J" sort="Aanhaanen, Wim T J" uniqKey="Aanhaanen W" first="Wim T. J." last="Aanhaanen">Wim T. J. Aanhaanen</name>
<affiliation>
<mods:affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</mods:affiliation>
</affiliation>
</author>
<author>
<name sortKey="Moorman, Antoon F M" sort="Moorman, Antoon F M" uniqKey="Moorman A" first="Antoon F. M." last="Moorman">Antoon F. M. Moorman</name>
<affiliation>
<mods:affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</mods:affiliation>
</affiliation>
</author>
<author>
<name sortKey="Christoffels, Vincent M" sort="Christoffels, Vincent M" uniqKey="Christoffels V" first="Vincent M." last="Christoffels">Vincent M. Christoffels</name>
<affiliation>
<mods:affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</mods:affiliation>
</affiliation>
</author>
</analytic>
<monogr></monogr>
<series>
<title level="j">Birth Defects Research Part A: Clinical and Molecular Teratology</title>
<title level="j" type="abbrev">Birth Defects Research Part A: Clinical and Molecular Teratology</title>
<idno type="ISSN">1542-0752</idno>
<idno type="eISSN">1542-0760</idno>
<imprint>
<publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher>
<pubPlace>Hoboken</pubPlace>
<date type="published" when="2011-06">2011-06</date>
<biblScope unit="volume">91</biblScope>
<biblScope unit="issue">6</biblScope>
<biblScope unit="page" from="565">565</biblScope>
<biblScope unit="page" to="577">577</biblScope>
</imprint>
<idno type="ISSN">1542-0752</idno>
</series>
<idno type="istex">52ED3228596B2916AC57AB9B12D4367C13243B7E</idno>
<idno type="DOI">10.1002/bdra.20826</idno>
<idno type="ArticleID">BDRA20826</idno>
</biblStruct>
</sourceDesc>
<seriesStmt>
<idno type="ISSN">1542-0752</idno>
</seriesStmt>
</fileDesc>
<profileDesc>
<textClass>
<keywords scheme="KwdEn" xml:lang="en">
<term>T‐box transcription factors</term>
<term>WPW</term>
<term>atrioventricular canal</term>
<term>atrioventricular conduction system</term>
<term>lineage analysis</term>
<term>preexcitation</term>
</keywords>
</textClass>
<langUsage>
<language ident="en">en</language>
</langUsage>
</profileDesc>
</teiHeader>
<front>
<div type="abstract" xml:lang="en">Defects originating from the atrioventricular canal region are part of a wide spectrum of congenital cardiovascular malformations that frequently affect newborns. These defects include partial or complete atrioventricular septal defects, atrioventricular valve defects, and arrhythmias, such as atrioventricular re‐entry tachycardia, atrioventricular nodal block, and ventricular preexcitation. Insight into the cellular origin of the atrioventricular canal myocardium and the molecular mechanisms that control its development will aid in the understanding of the etiology of the atrioventricular defects. This review discusses current knowledge concerning the origin and fate of the atrioventricular canal myocardium, the molecular mechanisms that determine its specification and differentiation, and its role in the development of certain malformations such as those that underlie ventricular preexcitation. Birth Defects Research (Part A), 2011. © 2011Wiley‐Liss, Inc.</div>
</front>
</TEI>
<istex>
<corpusName>wiley</corpusName>
<author>
<json:item>
<name>Wim T. J. Aanhaanen</name>
<affiliations>
<json:string>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</json:string>
</affiliations>
</json:item>
<json:item>
<name>Antoon F. M. Moorman</name>
<affiliations>
<json:string>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</json:string>
</affiliations>
</json:item>
<json:item>
<name>Vincent M. Christoffels</name>
<affiliations>
<json:string>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</json:string>
</affiliations>
</json:item>
</author>
<subject>
<json:item>
<lang>
<json:string>eng</json:string>
</lang>
<value>atrioventricular conduction system</value>
</json:item>
<json:item>
<lang>
<json:string>eng</json:string>
</lang>
<value>atrioventricular canal</value>
</json:item>
<json:item>
<lang>
<json:string>eng</json:string>
</lang>
<value>preexcitation</value>
</json:item>
<json:item>
<lang>
<json:string>eng</json:string>
</lang>
<value>lineage analysis</value>
</json:item>
<json:item>
<lang>
<json:string>eng</json:string>
</lang>
<value>WPW</value>
</json:item>
<json:item>
<lang>
<json:string>eng</json:string>
</lang>
<value>T‐box transcription factors</value>
</json:item>
</subject>
<articleId>
<json:string>BDRA20826</json:string>
</articleId>
<language>
<json:string>eng</json:string>
</language>
<abstract>Defects originating from the atrioventricular canal region are part of a wide spectrum of congenital cardiovascular malformations that frequently affect newborns. These defects include partial or complete atrioventricular septal defects, atrioventricular valve defects, and arrhythmias, such as atrioventricular re‐entry tachycardia, atrioventricular nodal block, and ventricular preexcitation. Insight into the cellular origin of the atrioventricular canal myocardium and the molecular mechanisms that control its development will aid in the understanding of the etiology of the atrioventricular defects. This review discusses current knowledge concerning the origin and fate of the atrioventricular canal myocardium, the molecular mechanisms that determine its specification and differentiation, and its role in the development of certain malformations such as those that underlie ventricular preexcitation. Birth Defects Research (Part A), 2011. © 2011Wiley‐Liss, Inc.</abstract>
<qualityIndicators>
<score>6.5</score>
<pdfVersion>1.3</pdfVersion>
<pdfPageSize>612 x 810 pts</pdfPageSize>
<refBibsNative>true</refBibsNative>
<keywordCount>6</keywordCount>
<abstractCharCount>971</abstractCharCount>
<pdfWordCount>10627</pdfWordCount>
<pdfCharCount>68886</pdfCharCount>
<pdfPageCount>13</pdfPageCount>
<abstractWordCount>125</abstractWordCount>
</qualityIndicators>
<title>Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways</title>
<genre>
<json:string>review-article</json:string>
</genre>
<host>
<volume>91</volume>
<publisherId>
<json:string>BDRA</json:string>
</publisherId>
<pages>
<total>13</total>
<last>577</last>
<first>565</first>
</pages>
<issn>
<json:string>1542-0752</json:string>
</issn>
<issue>6</issue>
<author>
<json:item>
<name>Simon J. Conway</name>
</json:item>
<json:item>
<name>Paul R. Riley</name>
</json:item>
</author>
<subject>
<json:item>
<value>Review Article</value>
</json:item>
</subject>
<genre>
<json:string>Journal</json:string>
</genre>
<language>
<json:string>unknown</json:string>
</language>
<eissn>
<json:string>1542-0760</json:string>
</eissn>
<title>Birth Defects Research Part A: Clinical and Molecular Teratology</title>
<doi>
<json:string>10.1002/(ISSN)1542-0760</json:string>
</doi>
</host>
<publicationDate>2011</publicationDate>
<copyrightDate>2011</copyrightDate>
<doi>
<json:string>10.1002/bdra.20826</json:string>
</doi>
<id>52ED3228596B2916AC57AB9B12D4367C13243B7E</id>
<fulltext>
<json:item>
<original>true</original>
<mimetype>application/pdf</mimetype>
<extension>pdf</extension>
<uri>https://api.istex.fr/document/52ED3228596B2916AC57AB9B12D4367C13243B7E/fulltext/pdf</uri>
</json:item>
<json:item>
<original>false</original>
<mimetype>application/zip</mimetype>
<extension>zip</extension>
<uri>https://api.istex.fr/document/52ED3228596B2916AC57AB9B12D4367C13243B7E/fulltext/zip</uri>
</json:item>
<istex:fulltextTEI uri="https://api.istex.fr/document/52ED3228596B2916AC57AB9B12D4367C13243B7E/fulltext/tei">
<teiHeader>
<fileDesc>
<titleStmt>
<title level="a" type="main" xml:lang="en">Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways</title>
</titleStmt>
<publicationStmt>
<authority>ISTEX</authority>
<publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher>
<pubPlace>Hoboken</pubPlace>
<availability>
<p>WILEY</p>
</availability>
<date>2011</date>
</publicationStmt>
<sourceDesc>
<biblStruct type="inbook">
<analytic>
<title level="a" type="main" xml:lang="en">Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways</title>
<author>
<persName>
<forename type="first">Wim T. J.</forename>
<surname>Aanhaanen</surname>
</persName>
<affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</affiliation>
</author>
<author>
<persName>
<forename type="first">Antoon F. M.</forename>
<surname>Moorman</surname>
</persName>
<affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</affiliation>
</author>
<author>
<persName>
<forename type="first">Vincent M.</forename>
<surname>Christoffels</surname>
</persName>
<note type="correspondence">
<p>Correspondence: Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands===</p>
</note>
<affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</affiliation>
</author>
</analytic>
<monogr>
<title level="j">Birth Defects Research Part A: Clinical and Molecular Teratology</title>
<title level="j" type="abbrev">Birth Defects Research Part A: Clinical and Molecular Teratology</title>
<idno type="pISSN">1542-0752</idno>
<idno type="eISSN">1542-0760</idno>
<idno type="DOI">10.1002/(ISSN)1542-0760</idno>
<imprint>
<publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher>
<pubPlace>Hoboken</pubPlace>
<date type="published" when="2011-06"></date>
<biblScope unit="volume">91</biblScope>
<biblScope unit="issue">6</biblScope>
<biblScope unit="page" from="565">565</biblScope>
<biblScope unit="page" to="577">577</biblScope>
</imprint>
</monogr>
<idno type="istex">52ED3228596B2916AC57AB9B12D4367C13243B7E</idno>
<idno type="DOI">10.1002/bdra.20826</idno>
<idno type="ArticleID">BDRA20826</idno>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc>
<creation>
<date>2011</date>
</creation>
<langUsage>
<language ident="en">en</language>
</langUsage>
<abstract xml:lang="en">
<p>Defects originating from the atrioventricular canal region are part of a wide spectrum of congenital cardiovascular malformations that frequently affect newborns. These defects include partial or complete atrioventricular septal defects, atrioventricular valve defects, and arrhythmias, such as atrioventricular re‐entry tachycardia, atrioventricular nodal block, and ventricular preexcitation. Insight into the cellular origin of the atrioventricular canal myocardium and the molecular mechanisms that control its development will aid in the understanding of the etiology of the atrioventricular defects. This review discusses current knowledge concerning the origin and fate of the atrioventricular canal myocardium, the molecular mechanisms that determine its specification and differentiation, and its role in the development of certain malformations such as those that underlie ventricular preexcitation. Birth Defects Research (Part A), 2011. © 2011Wiley‐Liss, Inc.</p>
</abstract>
<textClass xml:lang="en">
<keywords scheme="keyword">
<list>
<head>Keywords</head>
<item>
<term>atrioventricular conduction system</term>
</item>
<item>
<term>atrioventricular canal</term>
</item>
<item>
<term>preexcitation</term>
</item>
<item>
<term>lineage analysis</term>
</item>
<item>
<term>WPW</term>
</item>
<item>
<term>T‐box transcription factors</term>
</item>
</list>
</keywords>
</textClass>
<textClass>
<keywords scheme="Journal Subject">
<list>
<head>article category</head>
<item>
<term>Review Article</term>
</item>
</list>
</keywords>
</textClass>
</profileDesc>
<revisionDesc>
<change when="2010-12-15">Received</change>
<change when="2011-03-14">Registration</change>
<change when="2011-06">Published</change>
</revisionDesc>
</teiHeader>
</istex:fulltextTEI>
<json:item>
<original>false</original>
<mimetype>text/plain</mimetype>
<extension>txt</extension>
<uri>https://api.istex.fr/document/52ED3228596B2916AC57AB9B12D4367C13243B7E/fulltext/txt</uri>
</json:item>
</fulltext>
<metadata>
<istex:metadataXml wicri:clean="Wiley, elements deleted: body">
<istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration>
<istex:document>
<component version="2.0" type="serialArticle" xml:lang="en">
<header>
<publicationMeta level="product">
<publisherInfo>
<publisherName>Wiley Subscription Services, Inc., A Wiley Company</publisherName>
<publisherLoc>Hoboken</publisherLoc>
</publisherInfo>
<doi registered="yes">10.1002/(ISSN)1542-0760</doi>
<issn type="print">1542-0752</issn>
<issn type="electronic">1542-0760</issn>
<idGroup>
<id type="product" value="BDRA"></id>
</idGroup>
<titleGroup>
<title type="main" xml:lang="en" sort="BIRTH DEFECTS RESEARCH PART A: CLINICAL AND MOLECULAR TERATOLOGY">Birth Defects Research Part A: Clinical and Molecular Teratology</title>
<title type="short">Birth Defects Research Part A: Clinical and Molecular Teratology</title>
</titleGroup>
<selfCitationGroup>
<citation type="ancestor" xml:id="cit1">
<journalTitle>Teratology</journalTitle>
<accessionId ref="info:x-wiley/issn/00403709">0040-3709</accessionId>
<pubYear year="2002">2002</pubYear>
<vol>66</vol>
<issue>6</issue>
</citation>
</selfCitationGroup>
</publicationMeta>
<publicationMeta level="part" position="60">
<doi origin="wiley" registered="yes">10.1002/bdra.v91.6</doi>
<titleGroup>
<title type="specialIssueTitle">Congenital Heart Defects</title>
</titleGroup>
<numberingGroup>
<numbering type="journalVolume" number="91">91</numbering>
<numbering type="journalIssue">6</numbering>
</numberingGroup>
<creators>
<creator xml:id="sped1" creatorRole="sponsoringEditor">
<personName>
<givenNames>Simon J.</givenNames>
<familyName>Conway</familyName>
</personName>
</creator>
<creator xml:id="sped2" creatorRole="sponsoringEditor">
<personName>
<givenNames>Paul R.</givenNames>
<familyName>Riley</familyName>
</personName>
</creator>
</creators>
<coverDate startDate="2011-06">June 2011</coverDate>
</publicationMeta>
<publicationMeta level="unit" type="reviewArticle" position="150" status="forIssue">
<doi origin="wiley" registered="yes">10.1002/bdra.20826</doi>
<idGroup>
<id type="unit" value="BDRA20826"></id>
</idGroup>
<countGroup>
<count type="pageTotal" number="13"></count>
</countGroup>
<titleGroup>
<title type="articleCategory">Review Article</title>
<title type="tocHeading1">Review Articles</title>
</titleGroup>
<copyright ownership="publisher">Copyright © 2011 Wiley‐Liss, Inc.</copyright>
<eventGroup>
<event type="manuscriptReceived" date="2010-12-15"></event>
<event type="manuscriptRevised" date="2011-03-11"></event>
<event type="manuscriptAccepted" date="2011-03-14"></event>
<event type="xmlConverted" agent="Converter:JWSART34_TO_WML3G version:2.5.2 mode:FullText" date="2011-06-21"></event>
<event type="publishedOnlineEarlyUnpaginated" date="2011-05-31"></event>
<event type="publishedOnlineFinalForm" date="2011-06-06"></event>
<event type="firstOnline" date="2011-05-31"></event>
<event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:4.0.1" date="2014-03-15"></event>
<event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-15"></event>
</eventGroup>
<numberingGroup>
<numbering type="pageFirst">565</numbering>
<numbering type="pageLast">577</numbering>
</numberingGroup>
<correspondenceTo>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands===</correspondenceTo>
<linkGroup>
<link type="toTypesetVersion" href="file:BDRA.BDRA20826.pdf"></link>
</linkGroup>
</publicationMeta>
<contentMeta>
<countGroup>
<count type="figureTotal" number="5"></count>
<count type="tableTotal" number="0"></count>
<count type="referenceTotal" number="148"></count>
<count type="wordTotal" number="11025"></count>
</countGroup>
<titleGroup>
<title type="main" xml:lang="en">Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways</title>
<title type="short" xml:lang="en">Origin and Development of the Atrioventricular Myocardial Lineage</title>
</titleGroup>
<creators>
<creator xml:id="au1" creatorRole="author" affiliationRef="#af1">
<personName>
<givenNames>Wim T. J.</givenNames>
<familyName>Aanhaanen</familyName>
</personName>
</creator>
<creator xml:id="au2" creatorRole="author" affiliationRef="#af1">
<personName>
<givenNames>Antoon F. M.</givenNames>
<familyName>Moorman</familyName>
</personName>
</creator>
<creator xml:id="au3" creatorRole="author" affiliationRef="#af1" corresponding="yes">
<personName>
<givenNames>Vincent M.</givenNames>
<familyName>Christoffels</familyName>
</personName>
<contactDetails>
<email>v.m.christoffels@amc.nl</email>
</contactDetails>
</creator>
</creators>
<affiliationGroup>
<affiliation xml:id="af1" countryCode="NL" type="organization">
<unparsedAffiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</unparsedAffiliation>
</affiliation>
</affiliationGroup>
<keywordGroup xml:lang="en" type="author">
<keyword xml:id="kwd1">atrioventricular conduction system</keyword>
<keyword xml:id="kwd2">atrioventricular canal</keyword>
<keyword xml:id="kwd3">preexcitation</keyword>
<keyword xml:id="kwd4">lineage analysis</keyword>
<keyword xml:id="kwd5">WPW</keyword>
<keyword xml:id="kwd6">T‐box transcription factors</keyword>
</keywordGroup>
<abstractGroup>
<abstract type="main" xml:lang="en">
<title type="main">Abstract</title>
<p>Defects originating from the atrioventricular canal region are part of a wide spectrum of congenital cardiovascular malformations that frequently affect newborns. These defects include partial or complete atrioventricular septal defects, atrioventricular valve defects, and arrhythmias, such as atrioventricular re‐entry tachycardia, atrioventricular nodal block, and ventricular preexcitation. Insight into the cellular origin of the atrioventricular canal myocardium and the molecular mechanisms that control its development will aid in the understanding of the etiology of the atrioventricular defects. This review discusses current knowledge concerning the origin and fate of the atrioventricular canal myocardium, the molecular mechanisms that determine its specification and differentiation, and its role in the development of certain malformations such as those that underlie ventricular preexcitation. Birth Defects Research (Part A), 2011. © 2011Wiley‐Liss, Inc.</p>
</abstract>
</abstractGroup>
</contentMeta>
</header>
</component>
</istex:document>
</istex:metadataXml>
<mods version="3.6">
<titleInfo lang="en">
<title>Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways</title>
</titleInfo>
<titleInfo type="abbreviated" lang="en">
<title>Origin and Development of the Atrioventricular Myocardial Lineage</title>
</titleInfo>
<titleInfo type="alternative" contentType="CDATA" lang="en">
<title>Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways</title>
</titleInfo>
<name type="personal">
<namePart type="given">Wim T. J.</namePart>
<namePart type="family">Aanhaanen</namePart>
<affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</affiliation>
<role>
<roleTerm type="text">author</roleTerm>
</role>
</name>
<name type="personal">
<namePart type="given">Antoon F. M.</namePart>
<namePart type="family">Moorman</namePart>
<affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</affiliation>
<role>
<roleTerm type="text">author</roleTerm>
</role>
</name>
<name type="personal">
<namePart type="given">Vincent M.</namePart>
<namePart type="family">Christoffels</namePart>
<affiliation>Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands</affiliation>
<description>Correspondence: Heart Failure Research Center, Academic Medical Center, Meibergdreef 15, 1105 AZ, Amsterdam, The Netherlands===</description>
<role>
<roleTerm type="text">author</roleTerm>
</role>
</name>
<typeOfResource>text</typeOfResource>
<genre type="review-article" displayLabel="reviewArticle"></genre>
<originInfo>
<publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher>
<place>
<placeTerm type="text">Hoboken</placeTerm>
</place>
<dateIssued encoding="w3cdtf">2011-06</dateIssued>
<dateCaptured encoding="w3cdtf">2010-12-15</dateCaptured>
<dateValid encoding="w3cdtf">2011-03-14</dateValid>
<copyrightDate encoding="w3cdtf">2011</copyrightDate>
</originInfo>
<language>
<languageTerm type="code" authority="rfc3066">en</languageTerm>
<languageTerm type="code" authority="iso639-2b">eng</languageTerm>
</language>
<physicalDescription>
<internetMediaType>text/html</internetMediaType>
<extent unit="figures">5</extent>
<extent unit="references">148</extent>
<extent unit="words">11025</extent>
</physicalDescription>
<abstract lang="en">Defects originating from the atrioventricular canal region are part of a wide spectrum of congenital cardiovascular malformations that frequently affect newborns. These defects include partial or complete atrioventricular septal defects, atrioventricular valve defects, and arrhythmias, such as atrioventricular re‐entry tachycardia, atrioventricular nodal block, and ventricular preexcitation. Insight into the cellular origin of the atrioventricular canal myocardium and the molecular mechanisms that control its development will aid in the understanding of the etiology of the atrioventricular defects. This review discusses current knowledge concerning the origin and fate of the atrioventricular canal myocardium, the molecular mechanisms that determine its specification and differentiation, and its role in the development of certain malformations such as those that underlie ventricular preexcitation. Birth Defects Research (Part A), 2011. © 2011Wiley‐Liss, Inc.</abstract>
<subject lang="en">
<genre>Keywords</genre>
<topic>atrioventricular conduction system</topic>
<topic>atrioventricular canal</topic>
<topic>preexcitation</topic>
<topic>lineage analysis</topic>
<topic>WPW</topic>
<topic>T‐box transcription factors</topic>
</subject>
<relatedItem type="host">
<titleInfo>
<title>Birth Defects Research Part A: Clinical and Molecular Teratology</title>
</titleInfo>
<titleInfo type="abbreviated">
<title>Birth Defects Research Part A: Clinical and Molecular Teratology</title>
</titleInfo>
<name type="personal">
<namePart type="given">Simon J.</namePart>
<namePart type="family">Conway</namePart>
</name>
<name type="personal">
<namePart type="given">Paul R.</namePart>
<namePart type="family">Riley</namePart>
</name>
<genre type="Journal">journal</genre>
<subject>
<genre>article category</genre>
<topic>Review Article</topic>
</subject>
<identifier type="ISSN">1542-0752</identifier>
<identifier type="eISSN">1542-0760</identifier>
<identifier type="DOI">10.1002/(ISSN)1542-0760</identifier>
<identifier type="PublisherID">BDRA</identifier>
<part>
<date>2011</date>
<detail type="title">
<title>Congenital Heart Defects</title>
</detail>
<detail type="volume">
<caption>vol.</caption>
<number>91</number>
</detail>
<detail type="issue">
<caption>no.</caption>
<number>6</number>
</detail>
<extent unit="pages">
<start>565</start>
<end>577</end>
<total>13</total>
</extent>
</part>
</relatedItem>
<relatedItem type="preceding">
<titleInfo>
<title>Teratology</title>
</titleInfo>
<identifier type="ISSN">0040-3709</identifier>
<part>
<date point="end">2002</date>
<detail type="volume">
<caption>last vol.</caption>
<number>66</number>
</detail>
<detail type="issue">
<caption>last no.</caption>
<number>6</number>
</detail>
</part>
</relatedItem>
<identifier type="istex">52ED3228596B2916AC57AB9B12D4367C13243B7E</identifier>
<identifier type="DOI">10.1002/bdra.20826</identifier>
<identifier type="ArticleID">BDRA20826</identifier>
<accessCondition type="use and reproduction" contentType="copyright">Copyright © 2011 Wiley‐Liss, Inc.</accessCondition>
<recordInfo>
<recordContentSource>WILEY</recordContentSource>
<recordOrigin>Wiley Subscription Services, Inc., A Wiley Company</recordOrigin>
</recordInfo>
</mods>
</metadata>
<serie></serie>
</istex>
</record>

Pour manipuler ce document sous Unix (Dilib)

EXPLOR_STEP=$WICRI_ROOT/Wicri/Sante/explor/ParkinsonV1/Data/Main/Corpus
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 002A38 | SxmlIndent | more

Ou

HfdSelect -h $EXPLOR_AREA/Data/Main/Corpus/biblio.hfd -nk 002A38 | SxmlIndent | more

Pour mettre un lien sur cette page dans le réseau Wicri

{{Explor lien
   |wiki=    Wicri/Sante
   |area=    ParkinsonV1
   |flux=    Main
   |étape=   Corpus
   |type=    RBID
   |clé=     ISTEX:52ED3228596B2916AC57AB9B12D4367C13243B7E
   |texte=   Origin and development of the atrioventricular myocardial lineage: Insight into the development of accessory pathways
}}

Wicri

This area was generated with Dilib version V0.6.23.
Data generation: Sun Jul 3 18:06:51 2016. Site generation: Wed Mar 6 18:46:03 2024