Features of brain atrophy in Parkinson's disease
Identifieur interne : 002674 ( Main/Corpus ); précédent : 002673; suivant : 002675Features of brain atrophy in Parkinson's disease
Auteurs : I. Steiner ; M. Gomori ; E. MelamedSource :
- Neuroradiology [ 0028-3940 ] ; 1985-03-01.
Abstract
Summary: Multiple parameters for brain volume and mass were studied in 85 parkinsonian patients and in 149 normal controls aged 24 to 84 using CT scanning. In controls there was reduction in brain substance with advancing age. Increased brain atrophy in patients with Parkinson's disease (PD) was mainly observed in the younger age group of 24 to 49. This included parameters evaluating the size of the lateral and third ventricles (P<0.05 and P<0.01 respectively) and the size of the subarachnoid space in the frontal interhemispheric and Sylvian fissures (P<0.005 and P<0.01). With computed canonical correlation analysis a formula was obtained which expressed the tendency of the atrophic process in PD to involve the areas surrounding the third ventricle and the mesial aspect of the frontal lobes more than during normal aging.
Url:
DOI: 10.1007/BF00343788
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In controls there was reduction in brain substance with advancing age. Increased brain atrophy in patients with Parkinson's disease (PD) was mainly observed in the younger age group of 24 to 49. This included parameters evaluating the size of the lateral and third ventricles (P<0.05 and P<0.01 respectively) and the size of the subarachnoid space in the frontal interhemispheric and Sylvian fissures (P<0.005 and P<0.01). 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Radiology</OrgDivision><OrgName>Hadassah University Hospital</OrgName><OrgAddress><City>Jerusalem</City><Country>Israel</Country></OrgAddress></Affiliation></AuthorGroup><Abstract ID="Abs1" Language="En"><Heading>Summary</Heading><Para>Multiple parameters for brain volume and mass were studied in 85 parkinsonian patients and in 149 normal controls aged 24 to 84 using CT scanning. In controls there was reduction in brain substance with advancing age. Increased brain atrophy in patients with Parkinson's disease (PD) was mainly observed in the younger age group of 24 to 49. This included parameters evaluating the size of the lateral and third ventricles (P<0.05 and P<0.01 respectively) and the size of the subarachnoid space in the frontal interhemispheric and Sylvian fissures (P<0.005 and P<0.01). With computed canonical correlation analysis a formula was obtained which expressed the tendency of the atrophic process in PD to involve the areas surrounding the third ventricle and the mesial aspect of the frontal lobes more than during normal aging.</Para></Abstract><KeywordGroup Language="En"><Heading>Key words</Heading><Keyword>Brain atrophy</Keyword><Keyword>Parkinson disease</Keyword><Keyword>CT scan</Keyword></KeywordGroup></ArticleHeader><NoBody></NoBody></Article></Issue></Volume></Journal></Publisher></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Features of brain atrophy in Parkinson's disease</title><subTitle>A CT scan study</subTitle></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Features of brain atrophy in Parkinson's disease</title><subTitle>A CT scan study</subTitle></titleInfo><name type="personal"><namePart type="given">I.</namePart><namePart type="family">Steiner</namePart><affiliation>Department of Neurology, Hadassah University Hospital, Jerusalem, Israel</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">J.</namePart><namePart type="given">M.</namePart><namePart type="family">Gomori</namePart><affiliation>Department of Radiology, Hadassah University Hospital, Jerusalem, Israel</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">E.</namePart><namePart type="family">Melamed</namePart><affiliation>Department of Neurology, Hadassah University Hospital, Jerusalem, Israel</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="OriginalPaper"></genre><originInfo><publisher>Springer-Verlag</publisher><place><placeTerm type="text">Berlin/Heidelberg</placeTerm></place><dateCreated encoding="w3cdtf">1984-03-01</dateCreated><dateIssued encoding="w3cdtf">1985-03-01</dateIssued><copyrightDate encoding="w3cdtf">1985</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">Summary: Multiple parameters for brain volume and mass were studied in 85 parkinsonian patients and in 149 normal controls aged 24 to 84 using CT scanning. In controls there was reduction in brain substance with advancing age. Increased brain atrophy in patients with Parkinson's disease (PD) was mainly observed in the younger age group of 24 to 49. This included parameters evaluating the size of the lateral and third ventricles (P<0.05 and P<0.01 respectively) and the size of the subarachnoid space in the frontal interhemispheric and Sylvian fissures (P<0.005 and P<0.01). With computed canonical correlation analysis a formula was obtained which expressed the tendency of the atrophic process in PD to involve the areas surrounding the third ventricle and the mesial aspect of the frontal lobes more than during normal aging.</abstract><note>Originals</note><relatedItem type="host"><titleInfo><title>Neuroradiology</title><subTitle>A Journal Devoted to Neuroimaging and Interventional Neuroradiology</subTitle></titleInfo><titleInfo type="abbreviated"><title>Neuroradiology</title></titleInfo><genre type="Journal" displayLabel="Archive Journal"></genre><originInfo><dateIssued encoding="w3cdtf">1985-03-01</dateIssued><copyrightDate encoding="w3cdtf">1985</copyrightDate></originInfo><subject><genre>Medicine & Public Health</genre><topic>Neurosciences</topic><topic>Imaging / Radiology</topic><topic>Neuroradiology</topic><topic>Neurology</topic><topic>Neurosurgery</topic></subject><identifier type="ISSN">0028-3940</identifier><identifier type="eISSN">1432-1920</identifier><identifier type="JournalID">234</identifier><identifier type="IssueArticleCount">22</identifier><identifier type="VolumeIssueCount">6</identifier><part><date>1985</date><detail type="volume"><number>27</number><caption>vol.</caption></detail><detail type="issue"><number>2</number><caption>no.</caption></detail><extent unit="pages"><start>158</start><end>160</end></extent></part><recordInfo><recordOrigin>Springer-Verlag, 1985</recordOrigin></recordInfo></relatedItem><identifier type="istex">DED868865AE128D74CBC45122BF79D38F2294D17</identifier><identifier type="DOI">10.1007/BF00343788</identifier><identifier type="ArticleID">Art11</identifier><identifier type="ArticleID">BF00343788</identifier><accessCondition type="use and reproduction" contentType="copyright">Springer-Verlag</accessCondition><recordInfo><recordContentSource>SPRINGER</recordContentSource><recordOrigin>Springer-Verlag, 1985</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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