A myocardial syndrome
Identifieur interne : 001506 ( Main/Corpus ); précédent : 001505; suivant : 001507A myocardial syndrome
Auteurs : F. Horace Smirk ; David G. PalmerSource :
- The American Journal of Cardiology [ 0002-9149 ] ; 1960.
Abstract
Eighty cases of a well defined myocardial syndrome, an important feature of which is the interruption of T waves by premature QRS complexes, are reported. Other features of the syndrome include myocardial damage and sudden death, multiform premature ventricular complexes, aberration in the form of ventricular complexes of supraventricular origin, variation in the interval between successive ectopic complexes and the preceding sinus complex and ectopic complexes in runs of two or more. Post-extrasystolic T wave changes are sometimes observed.The R on T phenomenon is by no means uncommon when looked for, and it may be seen in a wide variety of pathologic states involving the heart, such as myocardial infarction, ischemic heart disease, hypertensive heart disease and several distinct myocardiopathies. It may also be seen during cardiac surgery and during diagnostic procedures such as cardiac catheterization, angiocardiography and ventricular puncture.The phenomenon may be suspected clinically when very premature beats are noted on auscultation, and it is frequently found in electrocardiograms recording such arrhythmias as multiform ventricular systoles, ventricular tachycardia, flutter and fibrillation, auricular paroxysmal ftachycardia and auricular fibrillation.The incomplete syndrome without T wave interruption is very common and, frequently, further observation and electrocardiography will reveal the missing features.The syndrome is frequently associated with sudden death and prophylactic administration of quinidine is recommended as a therapeutic measure.
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DOI: 10.1016/0002-9149(60)90265-4
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Palmer</name><affiliation><mods:affiliation>From the Department of Medicine, Otago University Medical School, Dunedin, New Zealand</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:F9399254528F73AB6D81B4F6FFD12662656AD91F</idno><date when="1960" year="1960">1960</date><idno type="doi">10.1016/0002-9149(60)90265-4</idno><idno type="url">https://api.istex.fr/document/F9399254528F73AB6D81B4F6FFD12662656AD91F/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">001506</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">A myocardial syndrome</title><author><name sortKey="Smirk, F Horace" sort="Smirk, F Horace" uniqKey="Smirk F" first="F. Horace" last="Smirk">F. Horace Smirk</name><affiliation><mods:affiliation>From the Department of Medicine, Otago University Medical School, Dunedin, New Zealand</mods:affiliation></affiliation></author><author><name sortKey="Palmer, David G" sort="Palmer, David G" uniqKey="Palmer D" first="David G." last="Palmer">David G. Palmer</name><affiliation><mods:affiliation>From the Department of Medicine, Otago University Medical School, Dunedin, New Zealand</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">The American Journal of Cardiology</title><title level="j" type="abbrev">AJC</title><idno type="ISSN">0002-9149</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1960">1960</date><biblScope unit="volume">6</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="620">620</biblScope><biblScope unit="page" to="629">629</biblScope></imprint><idno type="ISSN">0002-9149</idno></series><idno type="istex">F9399254528F73AB6D81B4F6FFD12662656AD91F</idno><idno type="DOI">10.1016/0002-9149(60)90265-4</idno><idno type="PII">0002-9149(60)90265-4</idno><idno type="ArticleID">60902654</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0002-9149</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Eighty cases of a well defined myocardial syndrome, an important feature of which is the interruption of T waves by premature QRS complexes, are reported. Other features of the syndrome include myocardial damage and sudden death, multiform premature ventricular complexes, aberration in the form of ventricular complexes of supraventricular origin, variation in the interval between successive ectopic complexes and the preceding sinus complex and ectopic complexes in runs of two or more. Post-extrasystolic T wave changes are sometimes observed.The R on T phenomenon is by no means uncommon when looked for, and it may be seen in a wide variety of pathologic states involving the heart, such as myocardial infarction, ischemic heart disease, hypertensive heart disease and several distinct myocardiopathies. It may also be seen during cardiac surgery and during diagnostic procedures such as cardiac catheterization, angiocardiography and ventricular puncture.The phenomenon may be suspected clinically when very premature beats are noted on auscultation, and it is frequently found in electrocardiograms recording such arrhythmias as multiform ventricular systoles, ventricular tachycardia, flutter and fibrillation, auricular paroxysmal ftachycardia and auricular fibrillation.The incomplete syndrome without T wave interruption is very common and, frequently, further observation and electrocardiography will reveal the missing features.The syndrome is frequently associated with sudden death and prophylactic administration of quinidine is recommended as a therapeutic measure.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>F.Horace Smirk K.B.E., M.D., F.R.C.P., F.R.A.C.P.</name><affiliations><json:string>From the Department of Medicine, Otago University Medical School, Dunedin, New Zealand</json:string></affiliations></json:item><json:item><name>David G. Palmer M.B., ch.B.</name><affiliations><json:string>From the Department of Medicine, Otago University Medical School, Dunedin, New Zealand</json:string></affiliations></json:item></author><articleId><json:string>60902654</json:string></articleId><language><json:string>eng</json:string></language><abstract>Eighty cases of a well defined myocardial syndrome, an important feature of which is the interruption of T waves by premature QRS complexes, are reported. Other features of the syndrome include myocardial damage and sudden death, multiform premature ventricular complexes, aberration in the form of ventricular complexes of supraventricular origin, variation in the interval between successive ectopic complexes and the preceding sinus complex and ectopic complexes in runs of two or more. Post-extrasystolic T wave changes are sometimes observed.The R on T phenomenon is by no means uncommon when looked for, and it may be seen in a wide variety of pathologic states involving the heart, such as myocardial infarction, ischemic heart disease, hypertensive heart disease and several distinct myocardiopathies. It may also be seen during cardiac surgery and during diagnostic procedures such as cardiac catheterization, angiocardiography and ventricular puncture.The phenomenon may be suspected clinically when very premature beats are noted on auscultation, and it is frequently found in electrocardiograms recording such arrhythmias as multiform ventricular systoles, ventricular tachycardia, flutter and fibrillation, auricular paroxysmal ftachycardia and auricular fibrillation.The incomplete syndrome without T wave interruption is very common and, frequently, further observation and electrocardiography will reveal the missing features.The syndrome is frequently associated with sudden death and prophylactic administration of quinidine is recommended as a therapeutic measure.</abstract><qualityIndicators><score>6.45</score><pdfVersion>1.2</pdfVersion><pdfPageSize>540 x 756 pts</pdfPageSize><refBibsNative>true</refBibsNative><keywordCount>0</keywordCount><abstractCharCount>1583</abstractCharCount><pdfWordCount>3858</pdfWordCount><pdfCharCount>31693</pdfCharCount><pdfPageCount>10</pdfPageCount><abstractWordCount>216</abstractWordCount></qualityIndicators><title>A myocardial syndrome</title><pii><json:string>0002-9149(60)90265-4</json:string></pii><genre><json:string>research-article</json:string></genre><serie><volume>37</volume><pages><first>25</first></pages><genre></genre><language><json:string>unknown</json:string></language><title>Proc. 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Other features of the syndrome include myocardial damage and sudden death, multiform premature ventricular complexes, aberration in the form of ventricular complexes of supraventricular origin, variation in the interval between successive ectopic complexes and the preceding sinus complex and ectopic complexes in runs of two or more. Post-extrasystolic T wave changes are sometimes observed.The R on T phenomenon is by no means uncommon when looked for, and it may be seen in a wide variety of pathologic states involving the heart, such as myocardial infarction, ischemic heart disease, hypertensive heart disease and several distinct myocardiopathies. It may also be seen during cardiac surgery and during diagnostic procedures such as cardiac catheterization, angiocardiography and ventricular puncture.The phenomenon may be suspected clinically when very premature beats are noted on auscultation, and it is frequently found in electrocardiograms recording such arrhythmias as multiform ventricular systoles, ventricular tachycardia, flutter and fibrillation, auricular paroxysmal ftachycardia and auricular fibrillation.The incomplete syndrome without T wave interruption is very common and, frequently, further observation and electrocardiography will reveal the missing features.The syndrome is frequently associated with sudden death and prophylactic administration of quinidine is recommended as a therapeutic measure.</p></abstract></profileDesc><revisionDesc><change when="1960">Published</change></revisionDesc></teiHeader></istex:fulltextTEI></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier, elements deleted: tail"><istex:xmlDeclaration>version="1.0" encoding="UTF-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 4.5.2//EN//XML" URI="art452.dtd" name="istex:docType"></istex:docType><istex:document><converted-article version="4.5.2" docsubtype="fla" xml:lang="en"><item-info><jid>AJC</jid><aid>60902654</aid><ce:pii>0002-9149(60)90265-4</ce:pii><ce:doi>10.1016/0002-9149(60)90265-4</ce:doi><ce:copyright type="unknown" year="1960"></ce:copyright></item-info><head><ce:dochead><ce:textfn>Clinical study</ce:textfn></ce:dochead><ce:title>A myocardial syndrome</ce:title><ce:subtitle>With particular reference to the occurrence of sudden death and of premature systoles interrupting antecedent T waves<ce:sup loc="post">∗</ce:sup></ce:subtitle><ce:author-group><ce:author><ce:given-name>F.Horace</ce:given-name><ce:surname>Smirk</ce:surname><ce:degrees>K.B.E., M.D., F.R.C.P., F.R.A.C.P.</ce:degrees><ce:cross-ref refid="AFF1"><ce:sup loc="post">∗</ce:sup></ce:cross-ref></ce:author><ce:author><ce:given-name>David G.</ce:given-name><ce:surname>Palmer</ce:surname><ce:degrees>M.B., ch.B.</ce:degrees><ce:cross-ref refid="AFF1"><ce:sup loc="post">∗</ce:sup></ce:cross-ref></ce:author><ce:affiliation id="AFF1"><ce:label>∗</ce:label><ce:textfn>From the Department of Medicine, Otago University Medical School, Dunedin, New Zealand</ce:textfn></ce:affiliation></ce:author-group><ce:abstract class="author"><ce:section-title>Abstract</ce:section-title><ce:abstract-sec><ce:simple-para view="all" id="simple-para.0010">Eighty cases of a well defined myocardial syndrome, an important feature of which is the interruption of T waves by premature QRS complexes, are reported. Other features of the syndrome include myocardial damage and sudden death, multiform premature ventricular complexes, aberration in the form of ventricular complexes of supraventricular origin, variation in the interval between successive ectopic complexes and the preceding sinus complex and ectopic complexes in runs of two or more. Post-extrasystolic T wave changes are sometimes observed.</ce:simple-para><ce:simple-para view="all" id="simple-para.0015">The R on T phenomenon is by no means uncommon when looked for, and it may be seen in a wide variety of pathologic states involving the heart, such as myocardial infarction, ischemic heart disease, hypertensive heart disease and several distinct myocardiopathies. It may also be seen during cardiac surgery and during diagnostic procedures such as cardiac catheterization, angiocardiography and ventricular puncture.</ce:simple-para><ce:simple-para view="all" id="simple-para.0020">The phenomenon may be suspected clinically when very premature beats are noted on auscultation, and it is frequently found in electrocardiograms recording such arrhythmias as multiform ventricular systoles, ventricular tachycardia, flutter and fibrillation, auricular paroxysmal ftachycardia and auricular fibrillation.</ce:simple-para><ce:simple-para view="all" id="simple-para.0025">The incomplete syndrome without T wave interruption is very common and, frequently, further observation and electrocardiography will reveal the missing features.</ce:simple-para><ce:simple-para view="all" id="simple-para.0030">The syndrome is frequently associated with sudden death and prophylactic administration of quinidine is recommended as a therapeutic measure.</ce:simple-para></ce:abstract-sec></ce:abstract></head></converted-article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>A myocardial syndrome</title><subTitle>With particular reference to the occurrence of sudden death and of premature systoles interrupting antecedent T waves∗</subTitle></titleInfo><titleInfo type="alternative" lang="en" contentType="CDATA"><title>A myocardial syndrome</title><subTitle>With particular reference to the occurrence of sudden death and of premature systoles interrupting antecedent T waves∗</subTitle></titleInfo><name type="personal"><namePart type="given">F.Horace</namePart><namePart type="family">Smirk</namePart><namePart type="termsOfAddress">K.B.E., M.D., F.R.C.P., F.R.A.C.P.</namePart><affiliation>From the Department of Medicine, Otago University Medical School, Dunedin, New Zealand</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">David G.</namePart><namePart type="family">Palmer</namePart><namePart type="termsOfAddress">M.B., ch.B.</namePart><affiliation>From the Department of Medicine, Otago University Medical School, Dunedin, New Zealand</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="Full-length article"></genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1960</dateIssued><copyrightDate encoding="w3cdtf">1960</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">Eighty cases of a well defined myocardial syndrome, an important feature of which is the interruption of T waves by premature QRS complexes, are reported. Other features of the syndrome include myocardial damage and sudden death, multiform premature ventricular complexes, aberration in the form of ventricular complexes of supraventricular origin, variation in the interval between successive ectopic complexes and the preceding sinus complex and ectopic complexes in runs of two or more. Post-extrasystolic T wave changes are sometimes observed.The R on T phenomenon is by no means uncommon when looked for, and it may be seen in a wide variety of pathologic states involving the heart, such as myocardial infarction, ischemic heart disease, hypertensive heart disease and several distinct myocardiopathies. It may also be seen during cardiac surgery and during diagnostic procedures such as cardiac catheterization, angiocardiography and ventricular puncture.The phenomenon may be suspected clinically when very premature beats are noted on auscultation, and it is frequently found in electrocardiograms recording such arrhythmias as multiform ventricular systoles, ventricular tachycardia, flutter and fibrillation, auricular paroxysmal ftachycardia and auricular fibrillation.The incomplete syndrome without T wave interruption is very common and, frequently, further observation and electrocardiography will reveal the missing features.The syndrome is frequently associated with sudden death and prophylactic administration of quinidine is recommended as a therapeutic measure.</abstract><note type="content">Section title: Clinical study</note><relatedItem type="host"><titleInfo><title>The American Journal of Cardiology</title></titleInfo><titleInfo type="abbreviated"><title>AJC</title></titleInfo><genre type="Journal">journal</genre><originInfo><dateIssued encoding="w3cdtf">196009</dateIssued></originInfo><identifier type="ISSN">0002-9149</identifier><identifier type="PII">S0002-9149(00)X0307-4</identifier><part><date>196009</date><detail type="issue"><title>Symposium on Persistent Atrioventricular Canal</title></detail><detail type="volume"><number>6</number><caption>vol.</caption></detail><detail type="issue"><number>3</number><caption>no.</caption></detail><extent unit="issue pages"><start>1</start><end>704</end></extent><extent unit="pages"><start>620</start><end>629</end></extent></part></relatedItem><identifier type="istex">F9399254528F73AB6D81B4F6FFD12662656AD91F</identifier><identifier type="DOI">10.1016/0002-9149(60)90265-4</identifier><identifier type="PII">0002-9149(60)90265-4</identifier><identifier type="ArticleID">60902654</identifier><recordInfo><recordContentSource>ELSEVIER</recordContentSource></recordInfo></mods></metadata><enrichments><istex:refBibTEI uri="https://api.istex.fr/document/F9399254528F73AB6D81B4F6FFD12662656AD91F/enrichments/refBib"><teiHeader></teiHeader><text><front></front><body></body><back><listBibl><biblStruct><analytic><title level="a" type="main">R waves interrupting T waves</title><author><persName><forename type="first">F</forename><forename type="middle">H</forename><surname>Smirk</surname></persName></author></analytic><monogr><title level="j">Brit. Heart J</title><imprint><biblScope unit="volume">11</biblScope><biblScope unit="page">23</biblScope><date type="published" when="1949"></date></imprint></monogr></biblStruct></listBibl></back></text></istex:refBibTEI></enrichments></istex></record>Pour manipuler ce document sous Unix (Dilib)
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