Electrophysiology and long-term efficacy of pentisomide in patients with supraventricular tachycardia
Identifieur interne : 001223 ( Main/Corpus ); précédent : 001222; suivant : 001224Electrophysiology and long-term efficacy of pentisomide in patients with supraventricular tachycardia
Auteurs : Volker Kühlkamp ; Christian Mewis ; Ludger SeipelSource :
- International Journal of Cardiology [ 0167-5273 ] ; 1992.
Abstract
The electrophysiologic effects of pentisomide were investigated after intravenous (5 mg/kg) and oral (900–1200 mg three times a day) application in 9 patients with drug refractory atrioventricular nodal tachycardia and 6 patients with orthodromic atrioventricular re-entrant tachycardia. Pentisomide did not change sinus cycle length, effective refractory period of the right ventricle and the atrioventricular node. AH, HV interval, effective refractory period of the right atrium, QRS duration and QTc duration were (p ≤ 0.01) increased. Tachycardia cycle length was only increased after intravenous application of pentisomide, antegrade effective refractory periods of the accessory pathways and shortest fully pre-excited R-R intervals during atrial fibrillation were increased after the oral treatment phase (p = 0.054). Intravenous pentisomide prevented tachycardia in 69 patients with atrioventricular nodal tachycardia and in 26 patients with atrioventricular re-entrant tachycardia. If intravenous pentisomide did not prevent induction of the tachycardia, oral pentisomide was not effective either. During long-term follow-up 27 patients with atrioventricular nodal tachycardia and 14 patient with atrioventricular re-entrant tachycardia had a recurrence. Long-term treatment with pentisomide had to be discontinued because of possible side effects in 2 patients. It is concluded, that the electrophysiological effects of pentisomide are similar to those of flecainide and propafenone.
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DOI: 10.1016/0167-5273(92)90110-O
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Pentisomide did not change sinus cycle length, effective refractory period of the right ventricle and the atrioventricular node. AH, HV interval, effective refractory period of the right atrium, QRS duration and QTc duration were (p ≤ 0.01) increased. Tachycardia cycle length was only increased after intravenous application of pentisomide, antegrade effective refractory periods of the accessory pathways and shortest fully pre-excited R-R intervals during atrial fibrillation were increased after the oral treatment phase (p = 0.054). Intravenous pentisomide prevented tachycardia in 69 patients with atrioventricular nodal tachycardia and in 26 patients with atrioventricular re-entrant tachycardia. If intravenous pentisomide did not prevent induction of the tachycardia, oral pentisomide was not effective either. During long-term follow-up 27 patients with atrioventricular nodal tachycardia and 14 patient with atrioventricular re-entrant tachycardia had a recurrence. Long-term treatment with pentisomide had to be discontinued because of possible side effects in 2 patients. It is concluded, that the electrophysiological effects of pentisomide are similar to those of flecainide and propafenone.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>Volker Kühlkamp</name><affiliations><json:string>Medizinische Klinik Abteilung III, Tübingen, Germany</json:string></affiliations></json:item><json:item><name>Christian Mewis</name><affiliations><json:string>Medizinische Klinik Abteilung III, Tübingen, Germany</json:string></affiliations></json:item><json:item><name>Ludger Seipel</name><affiliations><json:string>Medizinische Klinik Abteilung III, Tübingen, Germany</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Electrophysiology</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Pentisomide</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Supraventricular tachycardia</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Medical treatment of arrhythmia</value></json:item></subject><language><json:string>eng</json:string></language><abstract>The electrophysiologic effects of pentisomide were investigated after intravenous (5 mg/kg) and oral (900–1200 mg three times a day) application in 9 patients with drug refractory atrioventricular nodal tachycardia and 6 patients with orthodromic atrioventricular re-entrant tachycardia. Pentisomide did not change sinus cycle length, effective refractory period of the right ventricle and the atrioventricular node. AH, HV interval, effective refractory period of the right atrium, QRS duration and QTc duration were (p ≤ 0.01) increased. Tachycardia cycle length was only increased after intravenous application of pentisomide, antegrade effective refractory periods of the accessory pathways and shortest fully pre-excited R-R intervals during atrial fibrillation were increased after the oral treatment phase (p = 0.054). Intravenous pentisomide prevented tachycardia in 69 patients with atrioventricular nodal tachycardia and in 26 patients with atrioventricular re-entrant tachycardia. If intravenous pentisomide did not prevent induction of the tachycardia, oral pentisomide was not effective either. During long-term follow-up 27 patients with atrioventricular nodal tachycardia and 14 patient with atrioventricular re-entrant tachycardia had a recurrence. Long-term treatment with pentisomide had to be discontinued because of possible side effects in 2 patients. It is concluded, that the electrophysiological effects of pentisomide are similar to those of flecainide and propafenone.</abstract><qualityIndicators><score>6.258</score><pdfVersion>1.2</pdfVersion><pdfPageSize>545 x 749 pts</pdfPageSize><refBibsNative>true</refBibsNative><keywordCount>4</keywordCount><abstractCharCount>1494</abstractCharCount><pdfWordCount>3858</pdfWordCount><pdfCharCount>29343</pdfCharCount><pdfPageCount>11</pdfPageCount><abstractWordCount>200</abstractWordCount></qualityIndicators><title>Electrophysiology and long-term efficacy of pentisomide in patients with supraventricular tachycardia</title><pii><json:string>0167-5273(92)90110-O</json:string></pii><genre><json:string>research-article</json:string></genre><serie><volume>43</volume><pages><first>3256</first></pages><genre></genre><language><json:string>unknown</json:string></language><title>Fed Proc</title></serie><host><volume>36</volume><pii><json:string>S0167-5273(00)X0177-5</json:string></pii><pages><last>79</last><first>69</first></pages><issn><json:string>0167-5273</json:string></issn><issue>1</issue><genre><json:string>Journal</json:string></genre><language><json:string>unknown</json:string></language><title>International Journal of Cardiology</title><publicationDate>1992</publicationDate></host><categories><wos><json:string>CARDIAC & CARDIOVASCULAR SYSTEMS</json:string></wos></categories><publicationDate>1992</publicationDate><copyrightDate>1992</copyrightDate><doi><json:string>10.1016/0167-5273(92)90110-O</json:string></doi><id>04D7D7F767F9F3104FAC178805B6E200C1604262</id><fulltext><json:item><original>true</original><mimetype>application/pdf</mimetype><extension>pdf</extension><uri>https://api.istex.fr/document/04D7D7F767F9F3104FAC178805B6E200C1604262/fulltext/pdf</uri></json:item><json:item><original>true</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/04D7D7F767F9F3104FAC178805B6E200C1604262/fulltext/txt</uri></json:item><json:item><original>false</original><mimetype>application/zip</mimetype><extension>zip</extension><uri>https://api.istex.fr/document/04D7D7F767F9F3104FAC178805B6E200C1604262/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/04D7D7F767F9F3104FAC178805B6E200C1604262/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a">Electrophysiology and long-term efficacy of pentisomide in patients with supraventricular tachycardia</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>ELSEVIER</publisher><availability><p>ELSEVIER</p></availability><date>1992</date></publicationStmt><notesStmt><note type="content">Section title: Original study</note></notesStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a">Electrophysiology and long-term efficacy of pentisomide in patients with supraventricular tachycardia</title><author><persName><forename type="first">Volker</forename><surname>Kühlkamp</surname></persName><note type="correspondence"><p>Correspondence to: V. Kühlkamp M.D., Medizinische Klinik Abteilung III, Otfried Müller Str. 10, D-7400 Tübingen, Germany.</p></note><affiliation>Medizinische Klinik Abteilung III, Tübingen, Germany</affiliation></author><author><persName><forename type="first">Christian</forename><surname>Mewis</surname></persName><affiliation>Medizinische Klinik Abteilung III, Tübingen, Germany</affiliation></author><author><persName><forename type="first">Ludger</forename><surname>Seipel</surname></persName><affiliation>Medizinische Klinik Abteilung III, Tübingen, Germany</affiliation></author></analytic><monogr><title level="j">International Journal of Cardiology</title><title level="j" type="abbrev">IJCA</title><idno type="pISSN">0167-5273</idno><idno type="PII">S0167-5273(00)X0177-5</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1992"></date><biblScope unit="volume">36</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="69">69</biblScope><biblScope unit="page" to="79">79</biblScope></imprint></monogr><idno type="istex">04D7D7F767F9F3104FAC178805B6E200C1604262</idno><idno type="DOI">10.1016/0167-5273(92)90110-O</idno><idno type="PII">0167-5273(92)90110-O</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1992</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>The electrophysiologic effects of pentisomide were investigated after intravenous (5 mg/kg) and oral (900–1200 mg three times a day) application in 9 patients with drug refractory atrioventricular nodal tachycardia and 6 patients with orthodromic atrioventricular re-entrant tachycardia. Pentisomide did not change sinus cycle length, effective refractory period of the right ventricle and the atrioventricular node. AH, HV interval, effective refractory period of the right atrium, QRS duration and QTc duration were (p ≤ 0.01) increased. Tachycardia cycle length was only increased after intravenous application of pentisomide, antegrade effective refractory periods of the accessory pathways and shortest fully pre-excited R-R intervals during atrial fibrillation were increased after the oral treatment phase (p = 0.054). Intravenous pentisomide prevented tachycardia in 69 patients with atrioventricular nodal tachycardia and in 26 patients with atrioventricular re-entrant tachycardia. If intravenous pentisomide did not prevent induction of the tachycardia, oral pentisomide was not effective either. During long-term follow-up 27 patients with atrioventricular nodal tachycardia and 14 patient with atrioventricular re-entrant tachycardia had a recurrence. Long-term treatment with pentisomide had to be discontinued because of possible side effects in 2 patients. It is concluded, that the electrophysiological effects of pentisomide are similar to those of flecainide and propafenone.</p></abstract><textClass><keywords scheme="keyword"><list><head>Article category</head><item><term>Electrophysiology</term></item></list></keywords></textClass><textClass><keywords scheme="keyword"><list><head>Keywords</head><item><term>Pentisomide</term></item><item><term>Supraventricular tachycardia</term></item><item><term>Medical treatment of arrhythmia</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1992-02-05">Registration</change><change when="1992">Published</change></revisionDesc></teiHeader></istex:fulltextTEI></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier, elements deleted: tail"><istex:xmlDeclaration>version="1.0" encoding="utf-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 4.5.2//EN//XML" URI="art452.dtd" name="istex:docType"></istex:docType><istex:document><converted-article version="4.5.2" docsubtype="fla"><item-info><jid>IJCA</jid><aid>9290110O</aid><ce:pii>0167-5273(92)90110-O</ce:pii><ce:doi>10.1016/0167-5273(92)90110-O</ce:doi><ce:copyright type="unknown" year="1992"></ce:copyright><ce:doctopics><ce:doctopic><ce:text>Electrophysiology</ce:text></ce:doctopic></ce:doctopics></item-info><head><ce:dochead><ce:textfn>Original study</ce:textfn></ce:dochead><ce:title>Electrophysiology and long-term efficacy of pentisomide in patients with supraventricular tachycardia</ce:title><ce:author-group><ce:author><ce:given-name>Volker</ce:given-name><ce:surname>Kühlkamp</ce:surname><ce:cross-ref refid="COR1"><ce:sup>∗</ce:sup></ce:cross-ref></ce:author><ce:author><ce:given-name>Christian</ce:given-name><ce:surname>Mewis</ce:surname></ce:author><ce:author><ce:given-name>Ludger</ce:given-name><ce:surname>Seipel</ce:surname></ce:author><ce:affiliation><ce:textfn>Medizinische Klinik Abteilung III, Tübingen, Germany</ce:textfn></ce:affiliation><ce:correspondence id="COR1"><ce:label>∗</ce:label><ce:text>Correspondence to: V. Kühlkamp M.D., Medizinische Klinik Abteilung III, Otfried Müller Str. 10, D-7400 Tübingen, Germany.</ce:text></ce:correspondence></ce:author-group><ce:date-received day="18" month="10" year="1991"></ce:date-received><ce:date-accepted day="5" month="2" year="1992"></ce:date-accepted><ce:abstract><ce:section-title>Abstract</ce:section-title><ce:abstract-sec><ce:simple-para>The electrophysiologic effects of pentisomide were investigated after intravenous (5 mg/kg) and oral (900–1200 mg three times a day) application in 9 patients with drug refractory atrioventricular nodal tachycardia and 6 patients with orthodromic atrioventricular re-entrant tachycardia. Pentisomide did not change sinus cycle length, effective refractory period of the right ventricle and the atrioventricular node. AH, HV interval, effective refractory period of the right atrium, QRS duration and QTc duration were (<ce:italic>p</ce:italic> ≤ 0.01) increased. Tachycardia cycle length was only increased after intravenous application of pentisomide, antegrade effective refractory periods of the accessory pathways and shortest fully pre-excited R-R intervals during atrial fibrillation were increased after the oral treatment phase (<ce:italic>p</ce:italic> = 0.054). Intravenous pentisomide prevented tachycardia in <math altimg="si1.gif"><fr shape="sol"><nu>6</nu><de>9</de></fr></math> patients with atrioventricular nodal tachycardia and in <math altimg="si2.gif"><fr shape="sol"><nu>2</nu><de>6</de></fr></math> patients with atrioventricular re-entrant tachycardia. If intravenous pentisomide did not prevent induction of the tachycardia, oral pentisomide was not effective either. During long-term follow-up <math altimg="si3.gif"><fr shape="sol"><nu>2</nu><de>7</de></fr></math> patients with atrioventricular nodal tachycardia and <math altimg="si4.gif"><fr shape="sol"><nu>1</nu><de>4</de></fr></math> patient with atrioventricular re-entrant tachycardia had a recurrence. Long-term treatment with pentisomide had to be discontinued because of possible side effects in 2 patients. It is concluded, that the electrophysiological effects of pentisomide are similar to those of flecainide and propafenone.</ce:simple-para></ce:abstract-sec></ce:abstract><ce:keywords><ce:section-title>Keywords</ce:section-title><ce:keyword><ce:text>Pentisomide</ce:text></ce:keyword><ce:keyword><ce:text>Supraventricular tachycardia</ce:text></ce:keyword><ce:keyword><ce:text>Medical treatment of arrhythmia</ce:text></ce:keyword></ce:keywords></head></converted-article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo><title>Electrophysiology and long-term efficacy of pentisomide in patients with supraventricular tachycardia</title></titleInfo><titleInfo type="alternative" contentType="CDATA"><title>Electrophysiology and long-term efficacy of pentisomide in patients with supraventricular tachycardia</title></titleInfo><name type="personal"><namePart type="given">Volker</namePart><namePart type="family">Kühlkamp</namePart><affiliation>Medizinische Klinik Abteilung III, Tübingen, Germany</affiliation><description>Correspondence to: V. Kühlkamp M.D., Medizinische Klinik Abteilung III, Otfried Müller Str. 10, D-7400 Tübingen, Germany.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Christian</namePart><namePart type="family">Mewis</namePart><affiliation>Medizinische Klinik Abteilung III, Tübingen, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Ludger</namePart><namePart type="family">Seipel</namePart><affiliation>Medizinische Klinik Abteilung III, Tübingen, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="Full-length article"></genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1992</dateIssued><dateValid encoding="w3cdtf">1992-02-05</dateValid><copyrightDate encoding="w3cdtf">1992</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">The electrophysiologic effects of pentisomide were investigated after intravenous (5 mg/kg) and oral (900–1200 mg three times a day) application in 9 patients with drug refractory atrioventricular nodal tachycardia and 6 patients with orthodromic atrioventricular re-entrant tachycardia. Pentisomide did not change sinus cycle length, effective refractory period of the right ventricle and the atrioventricular node. AH, HV interval, effective refractory period of the right atrium, QRS duration and QTc duration were (p ≤ 0.01) increased. Tachycardia cycle length was only increased after intravenous application of pentisomide, antegrade effective refractory periods of the accessory pathways and shortest fully pre-excited R-R intervals during atrial fibrillation were increased after the oral treatment phase (p = 0.054). Intravenous pentisomide prevented tachycardia in 69 patients with atrioventricular nodal tachycardia and in 26 patients with atrioventricular re-entrant tachycardia. If intravenous pentisomide did not prevent induction of the tachycardia, oral pentisomide was not effective either. During long-term follow-up 27 patients with atrioventricular nodal tachycardia and 14 patient with atrioventricular re-entrant tachycardia had a recurrence. Long-term treatment with pentisomide had to be discontinued because of possible side effects in 2 patients. It is concluded, that the electrophysiological effects of pentisomide are similar to those of flecainide and propafenone.</abstract><note type="content">Section title: Original study</note><subject><genre>Article category</genre><topic>Electrophysiology</topic></subject><subject><genre>Keywords</genre><topic>Pentisomide</topic><topic>Supraventricular tachycardia</topic><topic>Medical treatment of arrhythmia</topic></subject><relatedItem type="host"><titleInfo><title>International Journal of Cardiology</title></titleInfo><titleInfo type="abbreviated"><title>IJCA</title></titleInfo><genre type="Journal">journal</genre><originInfo><dateIssued encoding="w3cdtf">199207</dateIssued></originInfo><identifier type="ISSN">0167-5273</identifier><identifier type="PII">S0167-5273(00)X0177-5</identifier><part><date>199207</date><detail type="volume"><number>36</number><caption>vol.</caption></detail><detail type="issue"><number>1</number><caption>no.</caption></detail><extent unit="issue pages"><start>1</start><end>134</end></extent><extent unit="pages"><start>69</start><end>79</end></extent></part></relatedItem><identifier type="istex">04D7D7F767F9F3104FAC178805B6E200C1604262</identifier><identifier type="DOI">10.1016/0167-5273(92)90110-O</identifier><identifier type="PII">0167-5273(92)90110-O</identifier><recordInfo><recordContentSource>ELSEVIER</recordContentSource></recordInfo></mods></metadata><enrichments><istex:catWosTEI uri="https://api.istex.fr/document/04D7D7F767F9F3104FAC178805B6E200C1604262/enrichments/catWos"><teiHeader><profileDesc><textClass><classCode scheme="WOS">CARDIAC & CARDIOVASCULAR SYSTEMS</classCode></textClass></profileDesc></teiHeader></istex:catWosTEI></enrichments></istex></record>Pour manipuler ce document sous Unix (Dilib)
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