BRADYPHRENIA IN PARKINSON'S DISEASE AND PSYCHOMOTOR RETARDATION IN DEPRESSIVE ILLNESS
Identifieur interne : 000B71 ( Main/Corpus ); précédent : 000B70; suivant : 000B72BRADYPHRENIA IN PARKINSON'S DISEASE AND PSYCHOMOTOR RETARDATION IN DEPRESSIVE ILLNESS
Auteurs : D. Rogers ; A. J. Lees ; Eileen Smith ; M. Trimble ; G. M. SternSource :
- Brain [ 0006-8950 ] ; 1987-06.
Abstract
Thirty newly diagnosed patients with Parkinson's disease and 30 patients with primary depressive illness showed slowing of response on a computerized digit symbol substitution test when compared with 30 matched normal control subjects. Significant slowing was related, in the parkinsonian patients, to structural brain disorder and affective impairment and, in the depressed patients, to motor impairment. A second computerized test, cognitively simpler but requiring the same motor response, was also administered to each subject. Both cognitive and motor slowing seemed to contribute to slowing of response in the digit symbol test in both parkinsonian and depressed patients. The tests were repeated after about six months in 12 subjects from each group. The parkinsonian patients, on dopaminergic treatment, showed neither significant change in motor or affective impairment, nor improvement in response time for the digit symbol test, but change in response time was related to change in depression rating. The depessed patients, on conventional treatment, showed significant improvement in both affective and motor impairment and improvement in response time for the digit symbol test, due to improvement in cognitive slowing. It is proposed that bradyphrenia in Parkinson's disease and psychomotor retardation in depressive illness are closely related, and that impairment of dopaminergic systems may be involved in both.
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DOI: 10.1093/brain/110.3.761
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Stern</name><affiliation><mods:affiliation>Department of Neurology, University College Hospital London</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Brain</title><idno type="ISSN">0006-8950</idno><idno type="eISSN">1460-2156</idno><imprint><publisher>Oxford University Press</publisher><date type="published" when="1987-06">1987-06</date><biblScope unit="volume">110</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="761">761</biblScope><biblScope unit="page" to="776">776</biblScope></imprint><idno type="ISSN">0006-8950</idno></series><idno type="istex">6474B7B36C76C0280DBB1B9EF5A70B1AB050E781</idno><idno type="DOI">10.1093/brain/110.3.761</idno><idno type="ArticleID">110.3.761</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0006-8950</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract">Thirty newly diagnosed patients with Parkinson's disease and 30 patients with primary depressive illness showed slowing of response on a computerized digit symbol substitution test when compared with 30 matched normal control subjects. Significant slowing was related, in the parkinsonian patients, to structural brain disorder and affective impairment and, in the depressed patients, to motor impairment. A second computerized test, cognitively simpler but requiring the same motor response, was also administered to each subject. Both cognitive and motor slowing seemed to contribute to slowing of response in the digit symbol test in both parkinsonian and depressed patients. The tests were repeated after about six months in 12 subjects from each group. The parkinsonian patients, on dopaminergic treatment, showed neither significant change in motor or affective impairment, nor improvement in response time for the digit symbol test, but change in response time was related to change in depression rating. The depessed patients, on conventional treatment, showed significant improvement in both affective and motor impairment and improvement in response time for the digit symbol test, due to improvement in cognitive slowing. It is proposed that bradyphrenia in Parkinson's disease and psychomotor retardation in depressive illness are closely related, and that impairment of dopaminergic systems may be involved in both.</div></front></TEI><istex><corpusName>oup</corpusName><author><json:item><name>D. ROGERS</name><affiliations><json:string>Department of Neuropsychiatry, University College Hospital London</json:string></affiliations></json:item><json:item><name>A. J. LEES</name><affiliations><json:string>Institute of Neurology ,the National Hospitals for Nervous Disease London</json:string><json:string>Correspondence to: Dr. A. J. Lees, Institute of Neurology, Queen Square, London WC1N 3BG.</json:string></affiliations></json:item><json:item><name>EILEEN SMITH</name><affiliations><json:string>Institute of Neurology ,the National Hospitals for Nervous Disease London</json:string></affiliations></json:item><json:item><name>M. TRIMBLE</name><affiliations><json:string>Department of Neuropsychiatry, University College Hospital London</json:string></affiliations></json:item><json:item><name>G. M. STERN</name><affiliations><json:string>Department of Neurology, University College Hospital London</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Articles</value></json:item></subject><language><json:string>eng</json:string></language><abstract>Thirty newly diagnosed patients with Parkinson's disease and 30 patients with primary depressive illness showed slowing of response on a computerized digit symbol substitution test when compared with 30 matched normal control subjects. Significant slowing was related, in the parkinsonian patients, to structural brain disorder and affective impairment and, in the depressed patients, to motor impairment. A second computerized test, cognitively simpler but requiring the same motor response, was also administered to each subject. Both cognitive and motor slowing seemed to contribute to slowing of response in the digit symbol test in both parkinsonian and depressed patients. The tests were repeated after about six months in 12 subjects from each group. The parkinsonian patients, on dopaminergic treatment, showed neither significant change in motor or affective impairment, nor improvement in response time for the digit symbol test, but change in response time was related to change in depression rating. The depessed patients, on conventional treatment, showed significant improvement in both affective and motor impairment and improvement in response time for the digit symbol test, due to improvement in cognitive slowing. 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J. Lees, Institute of Neurology, Queen Square, London WC1N 3BG.</note></notesStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a">BRADYPHRENIA IN PARKINSON'S DISEASE AND PSYCHOMOTOR RETARDATION IN DEPRESSIVE ILLNESS</title><title level="a" type="sub">AN EXPERIMENTAL STUDY</title><author><persName><forename type="first">D.</forename><surname>ROGERS</surname></persName><affiliation>Department of Neuropsychiatry, University College Hospital London</affiliation></author><author><persName><forename type="first">A. J.</forename><surname>LEES</surname></persName><affiliation>Institute of Neurology ,the National Hospitals for Nervous Disease London</affiliation></author><author><persName><forename type="first">EILEEN</forename><surname>SMITH</surname></persName><affiliation>Institute of Neurology ,the National Hospitals for Nervous Disease London</affiliation></author><author><persName><forename type="first">M.</forename><surname>TRIMBLE</surname></persName><affiliation>Department of Neuropsychiatry, University College Hospital London</affiliation></author><author><persName><forename type="first">G. M.</forename><surname>STERN</surname></persName><affiliation>Department of Neurology, University College Hospital London</affiliation></author></analytic><monogr><title level="j">Brain</title><idno type="pISSN">0006-8950</idno><idno type="eISSN">1460-2156</idno><imprint><publisher>Oxford University Press</publisher><date type="published" when="1987-06"></date><biblScope unit="volume">110</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="761">761</biblScope><biblScope unit="page" to="776">776</biblScope></imprint></monogr><idno type="istex">6474B7B36C76C0280DBB1B9EF5A70B1AB050E781</idno><idno type="DOI">10.1093/brain/110.3.761</idno><idno type="ArticleID">110.3.761</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1987</date></creation><langUsage><language ident="en">en</language></langUsage><abstract><p>Thirty newly diagnosed patients with Parkinson's disease and 30 patients with primary depressive illness showed slowing of response on a computerized digit symbol substitution test when compared with 30 matched normal control subjects. Significant slowing was related, in the parkinsonian patients, to structural brain disorder and affective impairment and, in the depressed patients, to motor impairment. A second computerized test, cognitively simpler but requiring the same motor response, was also administered to each subject. Both cognitive and motor slowing seemed to contribute to slowing of response in the digit symbol test in both parkinsonian and depressed patients. The tests were repeated after about six months in 12 subjects from each group. The parkinsonian patients, on dopaminergic treatment, showed neither significant change in motor or affective impairment, nor improvement in response time for the digit symbol test, but change in response time was related to change in depression rating. The depessed patients, on conventional treatment, showed significant improvement in both affective and motor impairment and improvement in response time for the digit symbol test, due to improvement in cognitive slowing. It is proposed that bradyphrenia in Parkinson's disease and psychomotor retardation in depressive illness are closely related, and that impairment of dopaminergic systems may be involved in both.</p></abstract><textClass><keywords scheme="keyword"><list><item><term>Articles</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1987-06">Published</change><change xml:id="refBibs-istex" who="#ISTEX-API" when="2016-3-14">References added</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/6474B7B36C76C0280DBB1B9EF5A70B1AB050E781/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="corpus oup" wicri:toSee="no header"><istex:docType PUBLIC="-//NLM//DTD Journal Publishing DTD v2.3 20070202//EN" URI="journalpublishing.dtd" name="istex:docType"></istex:docType><istex:document><article article-type="research-article"><front><journal-meta><journal-id journal-id-type="hwp">brain</journal-id><journal-id journal-id-type="publisher-id">brainj</journal-id><journal-id journal-id-type="pmc">brain</journal-id><journal-title>Brain</journal-title><issn pub-type="epub">1460-2156</issn><issn pub-type="ppub">0006-8950</issn><publisher><publisher-name>Oxford University Press</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="publisher-id">110.3.761</article-id><article-id pub-id-type="doi">10.1093/brain/110.3.761</article-id><article-categories><subj-group><subject>Articles</subject></subj-group></article-categories><title-group><article-title>BRADYPHRENIA IN PARKINSON'S DISEASE AND PSYCHOMOTOR RETARDATION IN DEPRESSIVE ILLNESS</article-title><subtitle>AN EXPERIMENTAL STUDY</subtitle></title-group><contrib-group><contrib contrib-type="author"><name><surname>ROGERS</surname><given-names>D.</given-names></name><xref ref-type="aff" rid="au1"><sup>1</sup></xref></contrib><contrib contrib-type="author"><name><surname>LEES</surname><given-names>A. J.</given-names></name><xref ref-type="aff" rid="au2"><sup>2</sup></xref><xref ref-type="corresp" rid="cor1"></xref></contrib><contrib contrib-type="author"><name><surname>SMITH</surname><given-names>EILEEN</given-names></name><xref ref-type="aff" rid="au2"><sup>2</sup></xref></contrib><contrib contrib-type="author"><name><surname>TRIMBLE</surname><given-names>M.</given-names></name><xref ref-type="aff" rid="au1"><sup>1</sup></xref></contrib><contrib contrib-type="author"><name><surname>STERN</surname><given-names>G. M.</given-names></name><xref ref-type="aff" rid="au3"><sup>3</sup></xref></contrib><aff id="au1"><sup>1</sup><institution>Department of Neuropsychiatry, University College Hospital</institution> <addr-line>London</addr-line></aff><aff id="au2"><sup>2</sup><institution>Institute of Neurology ,the National Hospitals for Nervous Disease</institution> <addr-line>London</addr-line></aff><aff id="au3"><sup>3</sup><institution>Department of Neurology, University College Hospital</institution> <addr-line>London</addr-line></aff></contrib-group><author-notes><corresp id="cor1">Correspondence to: Dr. A. J. Lees, Institute of Neurology, Queen Square, London WC1N 3BG.</corresp></author-notes><pub-date pub-type="ppub"><month>6</month><year>1987</year></pub-date><volume>110</volume><issue>3</issue><fpage>761</fpage><lpage>776</lpage><history><date date-type="received"><day>04</day><month>3</month><year>1986</year></date><date date-type="rev-recd"><day>13</day><month>6</month><year>1986</year></date><date date-type="accepted"><day>18</day><month>7</month><year>1986</year></date></history><copyright-statement>© Oxford University Press</copyright-statement><copyright-year>1987</copyright-year><abstract><p>Thirty newly diagnosed patients with Parkinson's disease and 30 patients with primary depressive illness showed slowing of response on a computerized digit symbol substitution test when compared with 30 matched normal control subjects. Significant slowing was related, in the parkinsonian patients, to structural brain disorder and affective impairment and, in the depressed patients, to motor impairment. A second computerized test, cognitively simpler but requiring the same motor response, was also administered to each subject. Both cognitive and motor slowing seemed to contribute to slowing of response in the digit symbol test in both parkinsonian and depressed patients. The tests were repeated after about six months in 12 subjects from each group. The parkinsonian patients, on dopaminergic treatment, showed neither significant change in motor or affective impairment, nor improvement in response time for the digit symbol test, but change in response time was related to change in depression rating. The depessed patients, on conventional treatment, showed significant improvement in both affective and motor impairment and improvement in response time for the digit symbol test, due to improvement in cognitive slowing. It is proposed that bradyphrenia in Parkinson's disease and psychomotor retardation in depressive illness are closely related, and that impairment of dopaminergic systems may be involved in both.</p></abstract></article-meta></front></article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo><title>BRADYPHRENIA IN PARKINSON'S DISEASE AND PSYCHOMOTOR RETARDATION IN DEPRESSIVE ILLNESS</title><subTitle>AN EXPERIMENTAL STUDY</subTitle></titleInfo><titleInfo type="alternative" contentType="CDATA"><title>BRADYPHRENIA IN PARKINSON'S DISEASE AND PSYCHOMOTOR RETARDATION IN DEPRESSIVE ILLNESS</title><subTitle>AN EXPERIMENTAL STUDY</subTitle></titleInfo><name type="personal"><namePart type="given">D.</namePart><namePart type="family">ROGERS</namePart><affiliation>Department of Neuropsychiatry, University College Hospital London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">A. J.</namePart><namePart type="family">LEES</namePart><affiliation>Institute of Neurology ,the National Hospitals for Nervous Disease London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">EILEEN</namePart><namePart type="family">SMITH</namePart><affiliation>Institute of Neurology ,the National Hospitals for Nervous Disease London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M.</namePart><namePart type="family">TRIMBLE</namePart><affiliation>Department of Neuropsychiatry, University College Hospital London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">G. M.</namePart><namePart type="family">STERN</namePart><affiliation>Department of Neurology, University College Hospital London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="research-article"></genre><subject><topic>Articles</topic></subject><originInfo><publisher>Oxford University Press</publisher><dateIssued encoding="w3cdtf">1987-06</dateIssued><copyrightDate encoding="w3cdtf">1987</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract>Thirty newly diagnosed patients with Parkinson's disease and 30 patients with primary depressive illness showed slowing of response on a computerized digit symbol substitution test when compared with 30 matched normal control subjects. Significant slowing was related, in the parkinsonian patients, to structural brain disorder and affective impairment and, in the depressed patients, to motor impairment. A second computerized test, cognitively simpler but requiring the same motor response, was also administered to each subject. Both cognitive and motor slowing seemed to contribute to slowing of response in the digit symbol test in both parkinsonian and depressed patients. The tests were repeated after about six months in 12 subjects from each group. The parkinsonian patients, on dopaminergic treatment, showed neither significant change in motor or affective impairment, nor improvement in response time for the digit symbol test, but change in response time was related to change in depression rating. The depessed patients, on conventional treatment, showed significant improvement in both affective and motor impairment and improvement in response time for the digit symbol test, due to improvement in cognitive slowing. It is proposed that bradyphrenia in Parkinson's disease and psychomotor retardation in depressive illness are closely related, and that impairment of dopaminergic systems may be involved in both.</abstract><note type="author-notes">Correspondence to: Dr. A. J. 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