Effect of chronic kidney disease on excessive daytime sleepiness in Parkinson disease
Identifieur interne : 000783 ( Main/Corpus ); précédent : 000782; suivant : 000784Effect of chronic kidney disease on excessive daytime sleepiness in Parkinson disease
Auteurs : Y. Baba ; M. Higuchi ; K. Fukuyama ; H. Abe ; Y. Uehara ; T. Inoue ; T. YamadaSource :
- European Journal of Neurology [ 1351-5101 ] ; 2011-11.
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- KwdEn :
Abstract
Background and purposes: Excessive daytime sleepiness (EDS) is a common sleep disorder in patients with Parkinson disease (PD). Non‐ergot dopamine agonists increase the risk of unanticipated sleep episodes. Objective: We aimed to assess the influence of renal function on EDS in patients with PD. Methods: Sixty‐two patients treated with ropinirole or pramipexole were recruited for this study. We evaluated the historical and clinical characteristics including the motor symptom rating scales, Epworth Sleepiness Scale (ESS), and estimated glomerular filtration rate (eGFR). An ESS score of 10 or greater was defined as EDS. Participants with eGFR < 60 ml/min/1.73 m2 were determined to have chronic kidney disease (CKD). Multiple logistic regression analysis was performed to determine the predictive factors of EDS. Results: Chronic kidney disease was found to be a significant predictive factor for EDS in all patients (P = 0.014). We observed a negative correlation between the severity of daytime sleepiness and renal function in patients treated with pramipexole alone (rs = −0.637, P < 0.001). Conclusions: Chronic kidney disease may be a risk factor for EDS, especially in patients treated with pramipexole, which is directly excreted in the urine.
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DOI: 10.1111/j.1468-1331.2011.03391.x
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ISTEX:96BAF938C52C3B7433289EFF95C766A8158A85C5Le document en format XML
<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Effect of chronic kidney disease on excessive daytime sleepiness in Parkinson disease</title><author><name sortKey="Baba, Y" sort="Baba, Y" uniqKey="Baba Y" first="Y." last="Baba">Y. Baba</name><affiliation><mods:affiliation>Department of Neurology, Fukuoka University School of Medicine</mods:affiliation></affiliation></author><author><name sortKey="Higuchi, M" sort="Higuchi, M" uniqKey="Higuchi M" first="M." last="Higuchi">M. Higuchi</name><affiliation><mods:affiliation>Department of Neurology, Fukuoka University School of Medicine</mods:affiliation></affiliation></author><author><name sortKey="Fukuyama, K" sort="Fukuyama, K" uniqKey="Fukuyama K" first="K." last="Fukuyama">K. Fukuyama</name><affiliation><mods:affiliation>Department of Neurosurgery, Fukuoka Wajiro Hospital</mods:affiliation></affiliation></author><author><name sortKey="Abe, H" sort="Abe, H" uniqKey="Abe H" first="H." last="Abe">H. Abe</name><affiliation><mods:affiliation>Departments of Neurosurgery</mods:affiliation></affiliation></author><author><name sortKey="Uehara, Y" sort="Uehara, Y" uniqKey="Uehara Y" first="Y." last="Uehara">Y. Uehara</name><affiliation><mods:affiliation>Cardiology, Fukuoka University School of Medicine, Fukuoka, Japan</mods:affiliation></affiliation></author><author><name sortKey="Inoue, T" sort="Inoue, T" uniqKey="Inoue T" first="T." last="Inoue">T. Inoue</name><affiliation><mods:affiliation>Departments of Neurosurgery</mods:affiliation></affiliation></author><author><name sortKey="Yamada, T" sort="Yamada, T" uniqKey="Yamada T" first="T." last="Yamada">T. Yamada</name><affiliation><mods:affiliation>Department of Neurology, Fukuoka University School of Medicine</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:96BAF938C52C3B7433289EFF95C766A8158A85C5</idno><date when="2011" year="2011">2011</date><idno type="doi">10.1111/j.1468-1331.2011.03391.x</idno><idno type="url">https://api.istex.fr/document/96BAF938C52C3B7433289EFF95C766A8158A85C5/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">000783</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Effect of chronic kidney disease on excessive daytime sleepiness in Parkinson disease</title><author><name sortKey="Baba, Y" sort="Baba, Y" uniqKey="Baba Y" first="Y." last="Baba">Y. Baba</name><affiliation><mods:affiliation>Department of Neurology, Fukuoka University School of Medicine</mods:affiliation></affiliation></author><author><name sortKey="Higuchi, M" sort="Higuchi, M" uniqKey="Higuchi M" first="M." last="Higuchi">M. Higuchi</name><affiliation><mods:affiliation>Department of Neurology, Fukuoka University School of Medicine</mods:affiliation></affiliation></author><author><name sortKey="Fukuyama, K" sort="Fukuyama, K" uniqKey="Fukuyama K" first="K." last="Fukuyama">K. Fukuyama</name><affiliation><mods:affiliation>Department of Neurosurgery, Fukuoka Wajiro Hospital</mods:affiliation></affiliation></author><author><name sortKey="Abe, H" sort="Abe, H" uniqKey="Abe H" first="H." last="Abe">H. Abe</name><affiliation><mods:affiliation>Departments of Neurosurgery</mods:affiliation></affiliation></author><author><name sortKey="Uehara, Y" sort="Uehara, Y" uniqKey="Uehara Y" first="Y." last="Uehara">Y. Uehara</name><affiliation><mods:affiliation>Cardiology, Fukuoka University School of Medicine, Fukuoka, Japan</mods:affiliation></affiliation></author><author><name sortKey="Inoue, T" sort="Inoue, T" uniqKey="Inoue T" first="T." last="Inoue">T. Inoue</name><affiliation><mods:affiliation>Departments of Neurosurgery</mods:affiliation></affiliation></author><author><name sortKey="Yamada, T" sort="Yamada, T" uniqKey="Yamada T" first="T." last="Yamada">T. Yamada</name><affiliation><mods:affiliation>Department of Neurology, Fukuoka University School of Medicine</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">European Journal of Neurology</title><idno type="ISSN">1351-5101</idno><idno type="eISSN">1468-1331</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="2011-11">2011-11</date><biblScope unit="volume">18</biblScope><biblScope unit="issue">11</biblScope><biblScope unit="page" from="1299">1299</biblScope><biblScope unit="page" to="1303">1303</biblScope></imprint><idno type="ISSN">1351-5101</idno></series><idno type="istex">96BAF938C52C3B7433289EFF95C766A8158A85C5</idno><idno type="DOI">10.1111/j.1468-1331.2011.03391.x</idno><idno type="ArticleID">ENE3391</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">1351-5101</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Parkinson disease</term><term>chronic kidney disease</term><term>excessive daytime sleepiness</term><term>non‐ergot dopamine agonist</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Background and purposes: Excessive daytime sleepiness (EDS) is a common sleep disorder in patients with Parkinson disease (PD). Non‐ergot dopamine agonists increase the risk of unanticipated sleep episodes. Objective: We aimed to assess the influence of renal function on EDS in patients with PD. Methods: Sixty‐two patients treated with ropinirole or pramipexole were recruited for this study. We evaluated the historical and clinical characteristics including the motor symptom rating scales, Epworth Sleepiness Scale (ESS), and estimated glomerular filtration rate (eGFR). An ESS score of 10 or greater was defined as EDS. Participants with eGFR < 60 ml/min/1.73 m2 were determined to have chronic kidney disease (CKD). Multiple logistic regression analysis was performed to determine the predictive factors of EDS. Results: Chronic kidney disease was found to be a significant predictive factor for EDS in all patients (P = 0.014). We observed a negative correlation between the severity of daytime sleepiness and renal function in patients treated with pramipexole alone (rs = −0.637, P < 0.001). Conclusions: Chronic kidney disease may be a risk factor for EDS, especially in patients treated with pramipexole, which is directly excreted in the urine.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Y. Baba</name><affiliations><json:string>Department of Neurology, Fukuoka University School of Medicine</json:string></affiliations></json:item><json:item><name>M.‐A. Higuchi</name><affiliations><json:string>Department of Neurology, Fukuoka University School of Medicine</json:string></affiliations></json:item><json:item><name>K. Fukuyama</name><affiliations><json:string>Department of Neurosurgery, Fukuoka Wajiro Hospital</json:string></affiliations></json:item><json:item><name>H. Abe</name><affiliations><json:string>Departments of Neurosurgery</json:string></affiliations></json:item><json:item><name>Y. Uehara</name><affiliations><json:string>Cardiology, Fukuoka University School of Medicine, Fukuoka, Japan</json:string></affiliations></json:item><json:item><name>T. Inoue</name><affiliations><json:string>Departments of Neurosurgery</json:string></affiliations></json:item><json:item><name>T. Yamada</name><affiliations><json:string>Department of Neurology, Fukuoka University School of Medicine</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>chronic kidney disease</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>excessive daytime sleepiness</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>non‐ergot dopamine agonist</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Parkinson disease</value></json:item></subject><articleId><json:string>ENE3391</json:string></articleId><language><json:string>eng</json:string></language><abstract>Background and purposes: Excessive daytime sleepiness (EDS) is a common sleep disorder in patients with Parkinson disease (PD). Non‐ergot dopamine agonists increase the risk of unanticipated sleep episodes. Objective: We aimed to assess the influence of renal function on EDS in patients with PD. Methods: Sixty‐two patients treated with ropinirole or pramipexole were recruited for this study. We evaluated the historical and clinical characteristics including the motor symptom rating scales, Epworth Sleepiness Scale (ESS), and estimated glomerular filtration rate (eGFR). An ESS score of 10 or greater was defined as EDS. Participants with eGFR > 60 ml/min/1.73 m2 were determined to have chronic kidney disease (CKD). Multiple logistic regression analysis was performed to determine the predictive factors of EDS. Results: Chronic kidney disease was found to be a significant predictive factor for EDS in all patients (P = 0.014). We observed a negative correlation between the severity of daytime sleepiness and renal function in patients treated with pramipexole alone (rs = −0.637, P > 0.001). 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Non‐ergot dopamine agonists increase the risk of unanticipated sleep episodes. Objective: We aimed to assess the influence of renal function on EDS in patients with PD. Methods: Sixty‐two patients treated with ropinirole or pramipexole were recruited for this study. We evaluated the historical and clinical characteristics including the motor symptom rating scales, Epworth Sleepiness Scale (ESS), and estimated glomerular filtration rate (eGFR). An ESS score of 10 or greater was defined as EDS. Participants with eGFR < 60 ml/min/1.73 m2 were determined to have chronic kidney disease (CKD). Multiple logistic regression analysis was performed to determine the predictive factors of EDS. Results: Chronic kidney disease was found to be a significant predictive factor for EDS in all patients (P = 0.014). We observed a negative correlation between the severity of daytime sleepiness and renal function in patients treated with pramipexole alone (rs = −0.637, P < 0.001). Conclusions: Chronic kidney disease may be a risk factor for EDS, especially in patients treated with pramipexole, which is directly excreted in the urine.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>chronic kidney disease</term></item><item><term>excessive daytime sleepiness</term></item><item><term>non‐ergot dopamine agonist</term></item><item><term>Parkinson disease</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="2011-11">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/96BAF938C52C3B7433289EFF95C766A8158A85C5/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1468-1331</doi><issn type="print">1351-5101</issn><issn type="electronic">1468-1331</issn><idGroup><id type="product" value="ENE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="EUROPEAN JOURNAL OF NEUROLOGY">European Journal of Neurology</title></titleGroup></publicationMeta><publicationMeta level="part" position="11111"><doi origin="wiley">10.1111/ene.2011.18.issue-11</doi><numberingGroup><numbering type="journalVolume" number="18">18</numbering><numbering type="journalIssue" number="11">11</numbering></numberingGroup><coverDate startDate="2011-11">November 2011</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="4" status="forIssue"><doi origin="wiley">10.1111/j.1468-1331.2011.03391.x</doi><idGroup><id type="unit" value="ENE3391"></id></idGroup><countGroup><count type="pageTotal" number="5"></count></countGroup><titleGroup><title type="tocHeading1">Original Articles</title></titleGroup><copyright>© 2011 The Author(s). European Journal of Neurology © 2011 EFNS</copyright><eventGroup><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.10 mode:FullText" date="2011-10-11"></event><event type="publishedOnlineEarlyUnpaginated" date="2011-03-24"></event><event type="firstOnline" date="2011-03-24"></event><event type="publishedOnlineFinalForm" date="2011-10-11"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-01-24"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-16"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="1299">1299</numbering><numbering type="pageLast" number="1303">1303</numbering></numberingGroup><correspondenceTo>Y. Baba, MD, Departments of Neurology, Fukuoka University School of Medicine, 7‐45‐1 Nanakuma, Jonan‐ku, Fukuoka 814‐0180, Japan (tel.: +81 92 801 1011; fax: +81 92 865 7900; e‐mail: <email normalForm="yasubaba@fukuoka-u.ac.jp">yasubaba@fukuoka‐u.ac.jp</email>).</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:ENE.ENE3391.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Received 30 November 2010 Accepted 22 February 2011</unparsedEditorialHistory><countGroup><count type="figureTotal" number="1"></count><count type="tableTotal" number="2"></count></countGroup><titleGroup><title type="main">Effect of chronic kidney disease on excessive daytime sleepiness in Parkinson disease</title><title type="shortAuthors">Y. Baba <i>et al.</i></title><title type="short">CKD and EDS in PD</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1"><personName><givenNames>Y.</givenNames><familyName>Baba</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1"><personName><givenNames>M.‐A.</givenNames><familyName>Higuchi</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a2"><personName><givenNames>K.</givenNames><familyName>Fukuyama</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a3"><personName><givenNames>H.</givenNames><familyName>Abe</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a4"><personName><givenNames>Y.</givenNames><familyName>Uehara</familyName></personName></creator><creator creatorRole="author" xml:id="cr6" affiliationRef="#a3"><personName><givenNames>T.</givenNames><familyName>Inoue</familyName></personName></creator><creator creatorRole="author" xml:id="cr7" affiliationRef="#a1"><personName><givenNames>T.</givenNames><familyName>Yamada</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1"><unparsedAffiliation>Department of Neurology, Fukuoka University School of Medicine</unparsedAffiliation></affiliation><affiliation xml:id="a2"><unparsedAffiliation>Department of Neurosurgery, Fukuoka Wajiro Hospital</unparsedAffiliation></affiliation><affiliation xml:id="a3"><unparsedAffiliation>Departments of Neurosurgery</unparsedAffiliation></affiliation><affiliation xml:id="a4" countryCode="JP"><unparsedAffiliation>Cardiology, Fukuoka University School of Medicine, Fukuoka, Japan</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">chronic kidney disease</keyword><keyword xml:id="k2">excessive daytime sleepiness</keyword><keyword xml:id="k3">non‐ergot dopamine agonist</keyword><keyword xml:id="k4">Parkinson disease</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p><b>Background and purposes: </b> Excessive daytime sleepiness (EDS) is a common sleep disorder in patients with Parkinson disease (PD). Non‐ergot dopamine agonists increase the risk of unanticipated sleep episodes.</p><p><b>Objective: </b> We aimed to assess the influence of renal function on EDS in patients with PD.</p><p><b>Methods: </b> Sixty‐two patients treated with ropinirole or pramipexole were recruited for this study. We evaluated the historical and clinical characteristics including the motor symptom rating scales, Epworth Sleepiness Scale (ESS), and estimated glomerular filtration rate (eGFR). An ESS score of 10 or greater was defined as EDS. Participants with eGFR < 60 ml/min/1.73 m<sup>2</sup> were determined to have chronic kidney disease (CKD). Multiple logistic regression analysis was performed to determine the predictive factors of EDS.</p><p><b>Results: </b> Chronic kidney disease was found to be a significant predictive factor for EDS in all patients (<i>P </i>=<i> </i>0.014). We observed a negative correlation between the severity of daytime sleepiness and renal function in patients treated with pramipexole alone (<i>r</i><sub><i>s</i></sub> = −0.637, <i>P </i><<i> </i>0.001).</p><p><b>Conclusions: </b> Chronic kidney disease may be a risk factor for EDS, especially in patients treated with pramipexole, which is directly excreted in the urine.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Effect of chronic kidney disease on excessive daytime sleepiness in Parkinson disease</title></titleInfo><titleInfo type="abbreviated"><title>CKD and EDS in PD</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Effect of chronic kidney disease on excessive daytime sleepiness in Parkinson disease</title></titleInfo><name type="personal"><namePart type="given">Y.</namePart><namePart type="family">Baba</namePart><affiliation>Department of Neurology, Fukuoka University School of Medicine</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M.‐A.</namePart><namePart type="family">Higuchi</namePart><affiliation>Department of Neurology, Fukuoka University School of Medicine</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">K.</namePart><namePart type="family">Fukuyama</namePart><affiliation>Department of Neurosurgery, Fukuoka Wajiro Hospital</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">H.</namePart><namePart type="family">Abe</namePart><affiliation>Departments of Neurosurgery</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Y.</namePart><namePart type="family">Uehara</namePart><affiliation>Cardiology, Fukuoka University School of Medicine, Fukuoka, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">T.</namePart><namePart type="family">Inoue</namePart><affiliation>Departments of Neurosurgery</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">T.</namePart><namePart type="family">Yamada</namePart><affiliation>Department of Neurology, Fukuoka University School of Medicine</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">2011-11</dateIssued><edition>Received 30 November 2010 Accepted 22 February 2011</edition><copyrightDate encoding="w3cdtf">2011</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="figures">1</extent><extent unit="tables">2</extent></physicalDescription><abstract lang="en">Background and purposes: Excessive daytime sleepiness (EDS) is a common sleep disorder in patients with Parkinson disease (PD). Non‐ergot dopamine agonists increase the risk of unanticipated sleep episodes. Objective: We aimed to assess the influence of renal function on EDS in patients with PD. Methods: Sixty‐two patients treated with ropinirole or pramipexole were recruited for this study. We evaluated the historical and clinical characteristics including the motor symptom rating scales, Epworth Sleepiness Scale (ESS), and estimated glomerular filtration rate (eGFR). An ESS score of 10 or greater was defined as EDS. Participants with eGFR < 60 ml/min/1.73 m2 were determined to have chronic kidney disease (CKD). Multiple logistic regression analysis was performed to determine the predictive factors of EDS. Results: Chronic kidney disease was found to be a significant predictive factor for EDS in all patients (P = 0.014). We observed a negative correlation between the severity of daytime sleepiness and renal function in patients treated with pramipexole alone (rs = −0.637, P < 0.001). Conclusions: Chronic kidney disease may be a risk factor for EDS, especially in patients treated with pramipexole, which is directly excreted in the urine.</abstract><subject lang="en"><genre>Keywords</genre><topic>chronic kidney disease</topic><topic>excessive daytime sleepiness</topic><topic>non‐ergot dopamine agonist</topic><topic>Parkinson disease</topic></subject><relatedItem type="host"><titleInfo><title>European Journal of Neurology</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">1351-5101</identifier><identifier type="eISSN">1468-1331</identifier><identifier type="DOI">10.1111/(ISSN)1468-1331</identifier><identifier type="PublisherID">ENE</identifier><part><date>2011</date><detail type="volume"><caption>vol.</caption><number>18</number></detail><detail type="issue"><caption>no.</caption><number>11</number></detail><extent unit="pages"><start>1299</start><end>1303</end><total>5</total></extent></part></relatedItem><identifier type="istex">96BAF938C52C3B7433289EFF95C766A8158A85C5</identifier><identifier type="DOI">10.1111/j.1468-1331.2011.03391.x</identifier><identifier type="ArticleID">ENE3391</identifier><accessCondition type="use and reproduction" contentType="copyright">© 2011 The Author(s). 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