Transesophageal pacing for prognostic evaluation of preexcitation syndrome and assessment of protective therapy
Identifieur interne : 000544 ( Main/Corpus ); précédent : 000543; suivant : 000545Transesophageal pacing for prognostic evaluation of preexcitation syndrome and assessment of protective therapy
Auteurs : Giuseppe Critelli ; Gino Grassi ; Francesco Perticone ; Fernando Coltorti ; Vittorio Monda ; Mario CondorelliSource :
- The American Journal of Cardiology [ 0002-9149 ] ; 1982.
Abstract
An esophageal lead was used to perform decremental atrial pacing and elective induction of atrial fibrillation (AF) in 5 patients with the Wolff-Parkinson-White (W-P-W) syndrome before and after amiodarone therapy. In the control state, 1:1 atrioventricular (AV) conduction over the accessory pathway ranged from 220 to 260 ms (mean 232). The shortest R-R interval during AF ranged from 190 to 210 ms (mean 198). The ventricular rate ranged from 175 to 212 beats/min (mean 196). After amiodarone therapy, the shortest cycle length with 1:1 AV conduction increased in all patients, ranging from 290 to 540 ms (mean 370); during AF, no preexcited beat was present in 2 patients, whereas the minimal preexcited R-R interval in the remaining 3 was 290, 240, and 370 ms, respectively. The ventricular response during AF decreased in all patients. Thus, esophageal pacing is a useful method for identifying patients at risk with the W-P-W syndrome and for assessing appropriate management in individual patients. Amiodarone provides protection against life-threatening arrhythmias in these patients.
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DOI: 10.1016/S0002-9149(83)80090-3
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<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Transesophageal pacing for prognostic evaluation of preexcitation syndrome and assessment of protective therapy</title><author><name sortKey="Critelli, Giuseppe" sort="Critelli, Giuseppe" uniqKey="Critelli G" first="Giuseppe" last="Critelli">Giuseppe Critelli</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Grassi, Gino" sort="Grassi, Gino" uniqKey="Grassi G" first="Gino" last="Grassi">Gino Grassi</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Perticone, Francesco" sort="Perticone, Francesco" uniqKey="Perticone F" first="Francesco" last="Perticone">Francesco Perticone</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Coltorti, Fernando" sort="Coltorti, Fernando" uniqKey="Coltorti F" first="Fernando" last="Coltorti">Fernando Coltorti</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Monda, Vittorio" sort="Monda, Vittorio" uniqKey="Monda V" first="Vittorio" last="Monda">Vittorio Monda</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Condorelli, Mario" sort="Condorelli, Mario" uniqKey="Condorelli M" first="Mario" last="Condorelli">Mario Condorelli</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. 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II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Grassi, Gino" sort="Grassi, Gino" uniqKey="Grassi G" first="Gino" last="Grassi">Gino Grassi</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Perticone, Francesco" sort="Perticone, Francesco" uniqKey="Perticone F" first="Francesco" last="Perticone">Francesco Perticone</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Coltorti, Fernando" sort="Coltorti, Fernando" uniqKey="Coltorti F" first="Fernando" last="Coltorti">Fernando Coltorti</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Monda, Vittorio" sort="Monda, Vittorio" uniqKey="Monda V" first="Vittorio" last="Monda">Vittorio Monda</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author><author><name sortKey="Condorelli, Mario" sort="Condorelli, Mario" uniqKey="Condorelli M" first="Mario" last="Condorelli">Mario Condorelli</name><affiliation><mods:affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">The American Journal of Cardiology</title><title level="j" type="abbrev">AJC</title><idno type="ISSN">0002-9149</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1982">1982</date><biblScope unit="volume">51</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="513">513</biblScope><biblScope unit="page" to="518">518</biblScope></imprint><idno type="ISSN">0002-9149</idno></series><idno type="istex">90E96612CA0B946D18E6E0EFDE48F0E421B959B6</idno><idno type="DOI">10.1016/S0002-9149(83)80090-3</idno><idno type="PII">S0002-9149(83)80090-3</idno><idno type="ArticleID">83800903</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0002-9149</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">An esophageal lead was used to perform decremental atrial pacing and elective induction of atrial fibrillation (AF) in 5 patients with the Wolff-Parkinson-White (W-P-W) syndrome before and after amiodarone therapy. In the control state, 1:1 atrioventricular (AV) conduction over the accessory pathway ranged from 220 to 260 ms (mean 232). The shortest R-R interval during AF ranged from 190 to 210 ms (mean 198). The ventricular rate ranged from 175 to 212 beats/min (mean 196). After amiodarone therapy, the shortest cycle length with 1:1 AV conduction increased in all patients, ranging from 290 to 540 ms (mean 370); during AF, no preexcited beat was present in 2 patients, whereas the minimal preexcited R-R interval in the remaining 3 was 290, 240, and 370 ms, respectively. The ventricular response during AF decreased in all patients. Thus, esophageal pacing is a useful method for identifying patients at risk with the W-P-W syndrome and for assessing appropriate management in individual patients. Amiodarone provides protection against life-threatening arrhythmias in these patients.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>Giuseppe Critelli MD</name><affiliations><json:string>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</json:string></affiliations></json:item><json:item><name>Gino Grassi PhD</name><affiliations><json:string>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</json:string></affiliations></json:item><json:item><name>Francesco Perticone MD</name><affiliations><json:string>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</json:string></affiliations></json:item><json:item><name>Fernando Coltorti MD</name><affiliations><json:string>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</json:string></affiliations></json:item><json:item><name>Vittorio Monda MD</name><affiliations><json:string>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</json:string></affiliations></json:item><json:item><name>Mario Condorelli MD</name><affiliations><json:string>From the Istituto di Patologia Medica. 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After amiodarone therapy, the shortest cycle length with 1:1 AV conduction increased in all patients, ranging from 290 to 540 ms (mean 370); during AF, no preexcited beat was present in 2 patients, whereas the minimal preexcited R-R interval in the remaining 3 was 290, 240, and 370 ms, respectively. The ventricular response during AF decreased in all patients. Thus, esophageal pacing is a useful method for identifying patients at risk with the W-P-W syndrome and for assessing appropriate management in individual patients. 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II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation></author><author><persName><forename type="first">Gino</forename><surname>Grassi</surname></persName><roleName type="degree">PhD</roleName><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation></author><author><persName><forename type="first">Francesco</forename><surname>Perticone</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation></author><author><persName><forename type="first">Fernando</forename><surname>Coltorti</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation></author><author><persName><forename type="first">Vittorio</forename><surname>Monda</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation></author><author><persName><forename type="first">Mario</forename><surname>Condorelli</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation></author></analytic><monogr><title level="j">The American Journal of Cardiology</title><title level="j" type="abbrev">AJC</title><idno type="pISSN">0002-9149</idno><idno type="PII">S0002-9149(83)X8065-9</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1982"></date><biblScope unit="volume">51</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="513">513</biblScope><biblScope unit="page" to="518">518</biblScope></imprint></monogr><idno type="istex">90E96612CA0B946D18E6E0EFDE48F0E421B959B6</idno><idno type="DOI">10.1016/S0002-9149(83)80090-3</idno><idno type="PII">S0002-9149(83)80090-3</idno><idno type="ArticleID">83800903</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1983</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>An esophageal lead was used to perform decremental atrial pacing and elective induction of atrial fibrillation (AF) in 5 patients with the Wolff-Parkinson-White (W-P-W) syndrome before and after amiodarone therapy. In the control state, 1:1 atrioventricular (AV) conduction over the accessory pathway ranged from 220 to 260 ms (mean 232). The shortest R-R interval during AF ranged from 190 to 210 ms (mean 198). The ventricular rate ranged from 175 to 212 beats/min (mean 196). After amiodarone therapy, the shortest cycle length with 1:1 AV conduction increased in all patients, ranging from 290 to 540 ms (mean 370); during AF, no preexcited beat was present in 2 patients, whereas the minimal preexcited R-R interval in the remaining 3 was 290, 240, and 370 ms, respectively. The ventricular response during AF decreased in all patients. Thus, esophageal pacing is a useful method for identifying patients at risk with the W-P-W syndrome and for assessing appropriate management in individual patients. Amiodarone provides protection against life-threatening arrhythmias in these patients.</p></abstract><textClass><keywords scheme="keyword"><list><head>Article category</head><item><term>Arrhythmias and conduction disturbance</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1982-09-29">Registration</change><change when="1982">Published</change></revisionDesc></teiHeader></istex:fulltextTEI></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier doc found" wicri:toSee="Elsevier, no converted or simple article"><istex:xmlDeclaration>version="1.0" encoding="UTF-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 5.0.1//EN//XML" URI="art501.dtd" name="istex:docType"></istex:docType><istex:document><article version="5.0" xml:lang="en" docsubtype="fla"><item-info><jid>AJC</jid><aid>83800903</aid><ce:pii>S0002-9149(83)80090-3</ce:pii><ce:doi>10.1016/S0002-9149(83)80090-3</ce:doi><ce:copyright type="unknown" year="1983"></ce:copyright><ce:doctopics><ce:doctopic><ce:text>Arrhythmias and conduction disturbance</ce:text></ce:doctopic></ce:doctopics></item-info><head><ce:article-footnote><ce:label>*</ce:label><ce:note-para>This study was supported in part by Grant 80,02493,11 from the National Research Council (CNR), Rome, Italy.</ce:note-para></ce:article-footnote><ce:title>Transesophageal pacing for prognostic evaluation of preexcitation syndrome and assessment of protective therapy</ce:title><ce:author-group><ce:author><ce:degrees>MD</ce:degrees><ce:given-name>Giuseppe</ce:given-name><ce:surname>Critelli</ce:surname><ce:cross-ref refid="cor1"><ce:sup loc="post">1</ce:sup></ce:cross-ref></ce:author><ce:author><ce:degrees>PhD</ce:degrees><ce:given-name>Gino</ce:given-name><ce:surname>Grassi</ce:surname></ce:author><ce:author><ce:degrees>MD</ce:degrees><ce:given-name>Francesco</ce:given-name><ce:surname>Perticone</ce:surname></ce:author><ce:author><ce:degrees>MD</ce:degrees><ce:given-name>Fernando</ce:given-name><ce:surname>Coltorti</ce:surname></ce:author><ce:author><ce:degrees>MD</ce:degrees><ce:given-name>Vittorio</ce:given-name><ce:surname>Monda</ce:surname></ce:author><ce:author><ce:degrees>MD</ce:degrees><ce:given-name>Mario</ce:given-name><ce:surname>Condorelli</ce:surname></ce:author><ce:affiliation id="aff1"><ce:textfn>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</ce:textfn></ce:affiliation><ce:correspondence id="cor1"><ce:label>1</ce:label><ce:text>Address for reprints: Prof. Giuseppe Critelli, Istituto di Patologia Medica, II Policlinico, Via Sergio Pansini, 5, 80131, Napoli, Italy.</ce:text></ce:correspondence></ce:author-group><ce:date-received day="14" month="7" year="1982"></ce:date-received><ce:date-accepted day="29" month="9" year="1982"></ce:date-accepted><ce:abstract id="ab1" class="author" xml:lang="en"><ce:abstract-sec><ce:simple-para view="all" id="simple-para.0010">An esophageal lead was used to perform decremental atrial pacing and elective induction of atrial fibrillation (AF) in 5 patients with the Wolff-Parkinson-White (W-P-W) syndrome before and after amiodarone therapy. In the control state, 1:1 atrioventricular (AV) conduction over the accessory pathway ranged from 220 to 260 ms (mean 232). The shortest R-R interval during AF ranged from 190 to 210 ms (mean 198). The ventricular rate ranged from 175 to 212 beats/min (mean 196). After amiodarone therapy, the shortest cycle length with 1:1 AV conduction increased in all patients, ranging from 290 to 540 ms (mean 370); during AF, no preexcited beat was present in 2 patients, whereas the minimal preexcited R-R interval in the remaining 3 was 290, 240, and 370 ms, respectively. The ventricular response during AF decreased in all patients. Thus, esophageal pacing is a useful method for identifying patients at risk with the W-P-W syndrome and for assessing appropriate management in individual patients. Amiodarone provides protection against life-threatening arrhythmias in these patients.</ce:simple-para></ce:abstract-sec></ce:abstract></head><tail view="all"><ce:bibliography view="all" id="bibliography.0010"><ce:section-title>References</ce:section-title><ce:bibliography-sec id="bibliography-sec.0010"><ce:bib-reference id="bib1"><ce:label>1.</ce:label><sb:reference><sb:contribution langtype="en"><sb:authors><sb:author><ce:surname>Okel</ce:surname><ce:given-name>BB</ce:given-name></sb:author></sb:authors><sb:title><sb:maintitle>The Wolff-Parkinson-White syndrome. Report of a case with fatal arrhythmia and autopsy findings of myocarditis. 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II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation><description>Address for reprints: Prof. Giuseppe Critelli, Istituto di Patologia Medica, II Policlinico, Via Sergio Pansini, 5, 80131, Napoli, Italy.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Gino</namePart><namePart type="family">Grassi</namePart><namePart type="termsOfAddress">PhD</namePart><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Francesco</namePart><namePart type="family">Perticone</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Fernando</namePart><namePart type="family">Coltorti</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Vittorio</namePart><namePart type="family">Monda</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Mario</namePart><namePart type="family">Condorelli</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Istituto di Patologia Medica. II Facoltà di Medicina e Chirurgia, Università di Napoli, Naples, Italy</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="Full-length article"></genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1982</dateIssued><dateValid encoding="w3cdtf">1982-09-29</dateValid><copyrightDate encoding="w3cdtf">1983</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">An esophageal lead was used to perform decremental atrial pacing and elective induction of atrial fibrillation (AF) in 5 patients with the Wolff-Parkinson-White (W-P-W) syndrome before and after amiodarone therapy. In the control state, 1:1 atrioventricular (AV) conduction over the accessory pathway ranged from 220 to 260 ms (mean 232). The shortest R-R interval during AF ranged from 190 to 210 ms (mean 198). The ventricular rate ranged from 175 to 212 beats/min (mean 196). After amiodarone therapy, the shortest cycle length with 1:1 AV conduction increased in all patients, ranging from 290 to 540 ms (mean 370); during AF, no preexcited beat was present in 2 patients, whereas the minimal preexcited R-R interval in the remaining 3 was 290, 240, and 370 ms, respectively. The ventricular response during AF decreased in all patients. Thus, esophageal pacing is a useful method for identifying patients at risk with the W-P-W syndrome and for assessing appropriate management in individual patients. Amiodarone provides protection against life-threatening arrhythmias in these patients.</abstract><note>This study was supported in part by Grant 80,02493,11 from the National Research Council (CNR), Rome, Italy.</note><subject><genre>Article category</genre><topic>Arrhythmias and conduction disturbance</topic></subject><relatedItem type="host"><titleInfo><title>The American Journal of Cardiology</title></titleInfo><titleInfo type="abbreviated"><title>AJC</title></titleInfo><genre type="Journal">journal</genre><originInfo><dateIssued encoding="w3cdtf">198302</dateIssued></originInfo><identifier type="ISSN">0002-9149</identifier><identifier type="PII">S0002-9149(83)X8065-9</identifier><part><detail type="volume"><number>51</number><caption>vol.</caption></detail><detail type="issue"><number>3</number><caption>no.</caption></detail><extent unit="issue pages"><start>353</start><end>616</end></extent><extent unit="pages"><start>513</start><end>518</end></extent></part></relatedItem><identifier type="istex">90E96612CA0B946D18E6E0EFDE48F0E421B959B6</identifier><identifier type="DOI">10.1016/S0002-9149(83)80090-3</identifier><identifier type="PII">S0002-9149(83)80090-3</identifier><identifier type="ArticleID">83800903</identifier><recordInfo><recordContentSource>ELSEVIER</recordContentSource></recordInfo></mods></metadata><enrichments><istex:catWosTEI uri="https://api.istex.fr/document/90E96612CA0B946D18E6E0EFDE48F0E421B959B6/enrichments/catWos"><teiHeader><profileDesc><textClass><classCode scheme="WOS">CARDIAC & CARDIOVASCULAR SYSTEMS</classCode></textClass></profileDesc></teiHeader></istex:catWosTEI></enrichments><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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