Subthalamic neuronal firing in obsessive-compulsive disorder and Parkinson disease.
Identifieur interne : 000934 ( PubMed/Curation ); précédent : 000933; suivant : 000935Subthalamic neuronal firing in obsessive-compulsive disorder and Parkinson disease.
Auteurs : Brigitte Piallat [France] ; Mircea Polosan ; Valérie Fraix ; Laurent Goetz ; Olivier David ; Albert Fenoy ; Napoleon Torres ; Jean-Louis Quesada ; Eric Seigneuret ; Pierre Pollak ; Paul Krack ; Thierry Bougerol ; Alim L. Benabid ; Stéphan ChabardèsSource :
- Annals of neurology [ 1531-8249 ] ; 2011.
English descriptors
- KwdEn :
- Action Potentials (physiology), Adult, Aged, Female, Functional Laterality (physiology), Humans, Imaging, Three-Dimensional (methods), Magnetic Resonance Imaging (methods), Male, Middle Aged, Neurons (physiology), Obsessive-Compulsive Disorder (pathology), Parkinson Disease (pathology), Subthalamic Nucleus (pathology).
- MESH :
- methods : Imaging, Three-Dimensional, Magnetic Resonance Imaging.
- pathology : Obsessive-Compulsive Disorder, Parkinson Disease, Subthalamic Nucleus.
- physiology : Action Potentials, Functional Laterality, Neurons.
- Adult, Aged, Female, Humans, Male, Middle Aged.
Abstract
Although electrophysiologic dysfunction of the subthalamic nucleus is putative, deep brain stimulation of this structure has recently been reported to improve obsessions and compulsions. In Parkinson disease, sensorimotor subthalamic neurons display high-frequency burst firing, which is considered as an electrophysiologic signature of motor loop dysfunction. We addressed whether such neuronal dysfunction of the subthalamic nucleus also exists in the nonmotor loops involved in patients with obsessive-compulsive disorder.
DOI: 10.1002/ana.22222
PubMed: 21520240
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pubmed:21520240Le document en format XML
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<front><div type="abstract" xml:lang="en">Although electrophysiologic dysfunction of the subthalamic nucleus is putative, deep brain stimulation of this structure has recently been reported to improve obsessions and compulsions. In Parkinson disease, sensorimotor subthalamic neurons display high-frequency burst firing, which is considered as an electrophysiologic signature of motor loop dysfunction. We addressed whether such neuronal dysfunction of the subthalamic nucleus also exists in the nonmotor loops involved in patients with obsessive-compulsive disorder.</div>
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<ArticleTitle>Subthalamic neuronal firing in obsessive-compulsive disorder and Parkinson disease.</ArticleTitle>
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<Abstract><AbstractText Label="OBJECTIVE" NlmCategory="OBJECTIVE">Although electrophysiologic dysfunction of the subthalamic nucleus is putative, deep brain stimulation of this structure has recently been reported to improve obsessions and compulsions. In Parkinson disease, sensorimotor subthalamic neurons display high-frequency burst firing, which is considered as an electrophysiologic signature of motor loop dysfunction. We addressed whether such neuronal dysfunction of the subthalamic nucleus also exists in the nonmotor loops involved in patients with obsessive-compulsive disorder.</AbstractText>
<AbstractText Label="METHODS" NlmCategory="METHODS">We compared the neuronal activity of the subthalamic nucleus recorded in 9 patients with obsessive-compulsive disorder with that of 11 patients with Parkinson disease measured during intraoperative exploration for deep brain stimulation.</AbstractText>
<AbstractText Label="RESULTS" NlmCategory="RESULTS">The mean subthalamic neuron discharge rate was statistically lower in patients with obsessive-compulsive disorder than in patients with Parkinson disease (20.5 ± 11.0 Hz, n = 100 and 30.8 ± 15.6 Hz, n = 93, respectively, p < 0.001). The relative proportion of burst neurons did not differ significantly between the 2 diseases (75% vs 73%). Interestingly, burst neurons were predominantly left-sided in obsessive-compulsive disorder.</AbstractText>
<AbstractText Label="INTERPRETATION" NlmCategory="CONCLUSIONS">The recording of burst neurons within the nonmotor subthalamic nucleus in patients with obsessive-compulsive disorder is a novel finding that suggests the existence of deregulation of the nonmotor basal ganglia loop, possibly left-sided. Potentially, burst activity might interfere with normal processes occurring within nonmotor loops.</AbstractText>
<CopyrightInformation>Copyright © 2010 American Neurological Association.</CopyrightInformation>
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