[Camptocormia disclosing Parkinson's disease].
Identifieur interne : 000E10 ( PubMed/Corpus ); précédent : 000E09; suivant : 000E11[Camptocormia disclosing Parkinson's disease].
Auteurs : E. Bouzgarou ; A. Dupeyron ; G. Castelnovo ; V. Boudousq ; L. Collombier ; P. Labauge ; J. PélissierSource :
- Annales de readaptation et de medecine physique : revue scientifique de la Societe francaise de reeducation fonctionnelle de readaptation et de medecine physique [ 0168-6054 ] ; 2007.
English descriptors
- KwdEn :
- MESH :
- diagnosis : Parkinson Disease.
- etiology : Spinal Curvatures.
- Aged, Female, Humans, Posture.
Abstract
Camptocormia is characterised as an extreme bent-forward posture of the trunk that disappears in the recumbent position. On X-ray, trunk flexion appears without vertebral rotation as in scoliosis. The condition is a well-known complication of Parkinson's disease (PD) at the late stage. The authors present the case of a 77-year-old woman affected by severe camptocormia, which appeared and worsened in less than 6 months and hindered gait. Despite no signs of PD, neuro-imaging (DAT-Scan) showed an L-Dopa transducer decrease in putamens. A few weeks later, bradykinesia appeared and the clinical diagnosis of PD became more obvious. L-Dopa improved bradykinesia but did not change the bent-spine posture. A 1-year follow-up showed no other signs of PD other than bradykinesia, but the camptocormia was unchanged.
DOI: 10.1016/j.annrmp.2006.07.060
PubMed: 17027115
Links to Exploration step
pubmed:17027115Le document en format XML
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<author><name sortKey="Bouzgarou, E" sort="Bouzgarou, E" uniqKey="Bouzgarou E" first="E" last="Bouzgarou">E. Bouzgarou</name>
<affiliation><nlm:affiliation>Département de MPR, groupe hospitalier Carémeau, 30029 Nîmes cedex 04, France.</nlm:affiliation>
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<author><name sortKey="Dupeyron, A" sort="Dupeyron, A" uniqKey="Dupeyron A" first="A" last="Dupeyron">A. Dupeyron</name>
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<author><name sortKey="Castelnovo, G" sort="Castelnovo, G" uniqKey="Castelnovo G" first="G" last="Castelnovo">G. Castelnovo</name>
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<author><name sortKey="Boudousq, V" sort="Boudousq, V" uniqKey="Boudousq V" first="V" last="Boudousq">V. Boudousq</name>
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<author><name sortKey="Collombier, L" sort="Collombier, L" uniqKey="Collombier L" first="L" last="Collombier">L. Collombier</name>
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<author><name sortKey="Labauge, P" sort="Labauge, P" uniqKey="Labauge P" first="P" last="Labauge">P. Labauge</name>
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<author><name sortKey="Pelissier, J" sort="Pelissier, J" uniqKey="Pelissier J" first="J" last="Pélissier">J. Pélissier</name>
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<series><title level="j">Annales de readaptation et de medecine physique : revue scientifique de la Societe francaise de reeducation fonctionnelle de readaptation et de medecine physique</title>
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<front><div type="abstract" xml:lang="en">Camptocormia is characterised as an extreme bent-forward posture of the trunk that disappears in the recumbent position. On X-ray, trunk flexion appears without vertebral rotation as in scoliosis. The condition is a well-known complication of Parkinson's disease (PD) at the late stage. The authors present the case of a 77-year-old woman affected by severe camptocormia, which appeared and worsened in less than 6 months and hindered gait. Despite no signs of PD, neuro-imaging (DAT-Scan) showed an L-Dopa transducer decrease in putamens. A few weeks later, bradykinesia appeared and the clinical diagnosis of PD became more obvious. L-Dopa improved bradykinesia but did not change the bent-spine posture. A 1-year follow-up showed no other signs of PD other than bradykinesia, but the camptocormia was unchanged.</div>
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<Abstract><AbstractText>Camptocormia is characterised as an extreme bent-forward posture of the trunk that disappears in the recumbent position. On X-ray, trunk flexion appears without vertebral rotation as in scoliosis. The condition is a well-known complication of Parkinson's disease (PD) at the late stage. The authors present the case of a 77-year-old woman affected by severe camptocormia, which appeared and worsened in less than 6 months and hindered gait. Despite no signs of PD, neuro-imaging (DAT-Scan) showed an L-Dopa transducer decrease in putamens. A few weeks later, bradykinesia appeared and the clinical diagnosis of PD became more obvious. L-Dopa improved bradykinesia but did not change the bent-spine posture. A 1-year follow-up showed no other signs of PD other than bradykinesia, but the camptocormia was unchanged.</AbstractText>
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<VernacularTitle>Camptocormie révélatrice d'une maladie de Parkinson.</VernacularTitle>
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