Could Wallerian degeneration contribute to "leuko-araiosis" in subjects free of any vascular disorder?
Identifieur interne : 001653 ( PubMed/Checkpoint ); précédent : 001652; suivant : 001654Could Wallerian degeneration contribute to "leuko-araiosis" in subjects free of any vascular disorder?
Auteurs : D. Leys [France] ; J P Pruvo ; M. Parent ; P. Vermersch ; G. Soetaert ; M. Steinling ; A. Delacourte ; A. Défossez ; A. Rapoport ; J. ClarisseSource :
- Journal of neurology, neurosurgery, and psychiatry [ 0022-3050 ] ; 1991.
English descriptors
- KwdEn :
- Aged, Aged, 80 and over, Alzheimer Disease (pathology), Cerebral Ventricles (pathology), Female, Humans, Male, Middle Aged, Neurofibrils (ultrastructure), Neurons (pathology), Parkinson Disease (pathology), Supranuclear Palsy, Progressive (pathology), Tomography, X-Ray Computed, Wallerian Degeneration (physiology).
- MESH :
- pathology : Alzheimer Disease, Cerebral Ventricles, Neurons, Parkinson Disease, Supranuclear Palsy, Progressive.
- physiology : Wallerian Degeneration.
- ultrastructure : Neurofibrils.
- Aged, Aged, 80 and over, Female, Humans, Male, Middle Aged, Tomography, X-Ray Computed.
Abstract
To determine the possible role of Wallerian degeneration secondary to the grey matter neuronal loss in the pathogenesis of "leuko-araiosis", computerised tomography (CT) of the brain was studied in 98 normotensive and non diabetic subjects free of cardiac diseases: 32 with Alzheimer's disease, 36 with Parkinson's disease, eight with progressive supranuclear palsy, and 22 controls. In Alzheimer's disease, leuko-araiosis scores were greater than in control subjects. Leuko-araiosis was more prominent in anterior periventricular areas in Parkinson's disease and progressive supranuclear palsy, and in posterior periventricular areas in Alzheimer's disease. In two patients with Alzheimer's disease and leuko-araiosis, necropsy revealed diffuse white matter pallor, mild fibrillary astrocytosis, and in one patient limited hyaline thickening of small white matter vessels, without any infarction or hypertensive change. Changes were more severe in white matter close to cortical areas with a great density of neurofibrillary tangles. Leuko-araiosis was more severe or more widespread in Alzheimer's disease than in Parkinson's disease, progressive supranuclear palsy and normal ageing. Differences in the location of leuko-araiosis between the four groups might be due to differences in the location of the grey matter disorder and Wallerian degeneration rather than amyloid in Alzheimer's disease, Parkinson's disease, progressive supranuclear palsy and normal ageing. Wallerian degeneration might be another cause of leuko-araiosis in neuro-degenerative disorders beside previously reported extra-cerebral predisposing factors and amyloid angiopathy.
PubMed: 2010759
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Parent</name></author><author><name sortKey="Vermersch, P" sort="Vermersch, P" uniqKey="Vermersch P" first="P" last="Vermersch">P. Vermersch</name></author><author><name sortKey="Soetaert, G" sort="Soetaert, G" uniqKey="Soetaert G" first="G" last="Soetaert">G. Soetaert</name></author><author><name sortKey="Steinling, M" sort="Steinling, M" uniqKey="Steinling M" first="M" last="Steinling">M. Steinling</name></author><author><name sortKey="Delacourte, A" sort="Delacourte, A" uniqKey="Delacourte A" first="A" last="Delacourte">A. Delacourte</name></author><author><name sortKey="Defossez, A" sort="Defossez, A" uniqKey="Defossez A" first="A" last="Défossez">A. Défossez</name></author><author><name sortKey="Rapoport, A" sort="Rapoport, A" uniqKey="Rapoport A" first="A" last="Rapoport">A. Rapoport</name></author><author><name sortKey="Clarisse, J" sort="Clarisse, J" uniqKey="Clarisse J" first="J" last="Clarisse">J. Clarisse</name></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="1991">1991</date><idno type="RBID">pubmed:2010759</idno><idno type="pmid">2010759</idno><idno type="wicri:Area/PubMed/Corpus">001808</idno><idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Corpus" wicri:corpus="PubMed">001808</idno><idno type="wicri:Area/PubMed/Curation">001767</idno><idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Curation">001767</idno><idno type="wicri:Area/PubMed/Checkpoint">001767</idno><idno type="wicri:explorRef" wicri:stream="Checkpoint" wicri:step="PubMed">001767</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">Could Wallerian degeneration contribute to "leuko-araiosis" in subjects free of any vascular disorder?</title><author><name sortKey="Leys, D" sort="Leys, D" uniqKey="Leys D" first="D" last="Leys">D. Leys</name><affiliation wicri:level="1"><nlm:affiliation>Hopital B, Lille Department of Neurology, France.</nlm:affiliation><country xml:lang="fr">France</country><wicri:regionArea>Hopital B, Lille Department of Neurology</wicri:regionArea><wicri:noRegion>Lille Department of Neurology</wicri:noRegion><wicri:noRegion>Lille Department of Neurology</wicri:noRegion></affiliation></author><author><name sortKey="Pruvo, J P" sort="Pruvo, J P" uniqKey="Pruvo J" first="J P" last="Pruvo">J P Pruvo</name></author><author><name sortKey="Parent, M" sort="Parent, M" uniqKey="Parent M" first="M" last="Parent">M. Parent</name></author><author><name sortKey="Vermersch, P" sort="Vermersch, P" uniqKey="Vermersch P" first="P" last="Vermersch">P. Vermersch</name></author><author><name sortKey="Soetaert, G" sort="Soetaert, G" uniqKey="Soetaert G" first="G" last="Soetaert">G. Soetaert</name></author><author><name sortKey="Steinling, M" sort="Steinling, M" uniqKey="Steinling M" first="M" last="Steinling">M. Steinling</name></author><author><name sortKey="Delacourte, A" sort="Delacourte, A" uniqKey="Delacourte A" first="A" last="Delacourte">A. Delacourte</name></author><author><name sortKey="Defossez, A" sort="Defossez, A" uniqKey="Defossez A" first="A" last="Défossez">A. Défossez</name></author><author><name sortKey="Rapoport, A" sort="Rapoport, A" uniqKey="Rapoport A" first="A" last="Rapoport">A. Rapoport</name></author><author><name sortKey="Clarisse, J" sort="Clarisse, J" uniqKey="Clarisse J" first="J" last="Clarisse">J. Clarisse</name></author></analytic><series><title level="j">Journal of neurology, neurosurgery, and psychiatry</title><idno type="ISSN">0022-3050</idno><imprint><date when="1991" type="published">1991</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Aged, 80 and over</term><term>Alzheimer Disease (pathology)</term><term>Cerebral Ventricles (pathology)</term><term>Female</term><term>Humans</term><term>Male</term><term>Middle Aged</term><term>Neurofibrils (ultrastructure)</term><term>Neurons (pathology)</term><term>Parkinson Disease (pathology)</term><term>Supranuclear Palsy, Progressive (pathology)</term><term>Tomography, X-Ray Computed</term><term>Wallerian Degeneration (physiology)</term></keywords><keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Alzheimer Disease</term><term>Cerebral Ventricles</term><term>Neurons</term><term>Parkinson Disease</term><term>Supranuclear Palsy, Progressive</term></keywords><keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>Wallerian Degeneration</term></keywords><keywords scheme="MESH" qualifier="ultrastructure" xml:lang="en"><term>Neurofibrils</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Aged, 80 and over</term><term>Female</term><term>Humans</term><term>Male</term><term>Middle Aged</term><term>Tomography, X-Ray Computed</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">To determine the possible role of Wallerian degeneration secondary to the grey matter neuronal loss in the pathogenesis of "leuko-araiosis", computerised tomography (CT) of the brain was studied in 98 normotensive and non diabetic subjects free of cardiac diseases: 32 with Alzheimer's disease, 36 with Parkinson's disease, eight with progressive supranuclear palsy, and 22 controls. In Alzheimer's disease, leuko-araiosis scores were greater than in control subjects. Leuko-araiosis was more prominent in anterior periventricular areas in Parkinson's disease and progressive supranuclear palsy, and in posterior periventricular areas in Alzheimer's disease. In two patients with Alzheimer's disease and leuko-araiosis, necropsy revealed diffuse white matter pallor, mild fibrillary astrocytosis, and in one patient limited hyaline thickening of small white matter vessels, without any infarction or hypertensive change. Changes were more severe in white matter close to cortical areas with a great density of neurofibrillary tangles. Leuko-araiosis was more severe or more widespread in Alzheimer's disease than in Parkinson's disease, progressive supranuclear palsy and normal ageing. Differences in the location of leuko-araiosis between the four groups might be due to differences in the location of the grey matter disorder and Wallerian degeneration rather than amyloid in Alzheimer's disease, Parkinson's disease, progressive supranuclear palsy and normal ageing. Wallerian degeneration might be another cause of leuko-araiosis in neuro-degenerative disorders beside previously reported extra-cerebral predisposing factors and amyloid angiopathy.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">2010759</PMID><DateCreated><Year>1991</Year><Month>05</Month><Day>08</Day></DateCreated><DateCompleted><Year>1991</Year><Month>05</Month><Day>08</Day></DateCompleted><DateRevised><Year>2016</Year><Month>11</Month><Day>22</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0022-3050</ISSN><JournalIssue CitedMedium="Print"><Volume>54</Volume><Issue>1</Issue><PubDate><Year>1991</Year><Month>Jan</Month></PubDate></JournalIssue><Title>Journal of neurology, neurosurgery, and psychiatry</Title><ISOAbbreviation>J. Neurol. Neurosurg. Psychiatr.</ISOAbbreviation></Journal><ArticleTitle>Could Wallerian degeneration contribute to "leuko-araiosis" in subjects free of any vascular disorder?</ArticleTitle><Pagination><MedlinePgn>46-50</MedlinePgn></Pagination><Abstract><AbstractText>To determine the possible role of Wallerian degeneration secondary to the grey matter neuronal loss in the pathogenesis of "leuko-araiosis", computerised tomography (CT) of the brain was studied in 98 normotensive and non diabetic subjects free of cardiac diseases: 32 with Alzheimer's disease, 36 with Parkinson's disease, eight with progressive supranuclear palsy, and 22 controls. In Alzheimer's disease, leuko-araiosis scores were greater than in control subjects. Leuko-araiosis was more prominent in anterior periventricular areas in Parkinson's disease and progressive supranuclear palsy, and in posterior periventricular areas in Alzheimer's disease. In two patients with Alzheimer's disease and leuko-araiosis, necropsy revealed diffuse white matter pallor, mild fibrillary astrocytosis, and in one patient limited hyaline thickening of small white matter vessels, without any infarction or hypertensive change. Changes were more severe in white matter close to cortical areas with a great density of neurofibrillary tangles. Leuko-araiosis was more severe or more widespread in Alzheimer's disease than in Parkinson's disease, progressive supranuclear palsy and normal ageing. Differences in the location of leuko-araiosis between the four groups might be due to differences in the location of the grey matter disorder and Wallerian degeneration rather than amyloid in Alzheimer's disease, Parkinson's disease, progressive supranuclear palsy and normal ageing. Wallerian degeneration might be another cause of leuko-araiosis in neuro-degenerative disorders beside previously reported extra-cerebral predisposing factors and amyloid angiopathy.</AbstractText></Abstract><AuthorList CompleteYN="N"><Author ValidYN="Y"><LastName>Leys</LastName><ForeName>D</ForeName><Initials>D</Initials><AffiliationInfo><Affiliation>Hopital B, Lille Department of Neurology, France.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Pruvo</LastName><ForeName>J P</ForeName><Initials>JP</Initials></Author><Author ValidYN="Y"><LastName>Parent</LastName><ForeName>M</ForeName><Initials>M</Initials></Author><Author ValidYN="Y"><LastName>Vermersch</LastName><ForeName>P</ForeName><Initials>P</Initials></Author><Author ValidYN="Y"><LastName>Soetaert</LastName><ForeName>G</ForeName><Initials>G</Initials></Author><Author ValidYN="Y"><LastName>Steinling</LastName><ForeName>M</ForeName><Initials>M</Initials></Author><Author ValidYN="Y"><LastName>Delacourte</LastName><ForeName>A</ForeName><Initials>A</Initials></Author><Author ValidYN="Y"><LastName>Défossez</LastName><ForeName>A</ForeName><Initials>A</Initials></Author><Author ValidYN="Y"><LastName>Rapoport</LastName><ForeName>A</ForeName><Initials>A</Initials></Author><Author ValidYN="Y"><LastName>Clarisse</LastName><ForeName>J</ForeName><Initials>J</Initials></Author></AuthorList><Language>eng</Language><GrantList CompleteYN="Y"><Grant><GrantID>GGP08119</GrantID><Agency>Telethon</Agency><Country>Italy</Country></Grant></GrantList><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>England</Country><MedlineTA>J Neurol Neurosurg Psychiatry</MedlineTA><NlmUniqueID>2985191R</NlmUniqueID><ISSNLinking>0022-3050</ISSNLinking></MedlineJournalInfo><CitationSubset>IM</CitationSubset><CommentsCorrectionsList><CommentsCorrections RefType="Cites"><RefSource>J Psychiatr Res. 1975 Nov;12(3):189-98</RefSource><PMID Version="1">1202204</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Arch Neurol. 1989 Oct;46(10):1124-8</RefSource><PMID Version="1">2803072</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Neurology. 1980 Sep;30(9):920-8</RefSource><PMID Version="1">7191536</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>J Neurol Neurosurg Psychiatry. 1981 Apr;44(4):294-304</RefSource><PMID Version="1">7241156</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Stroke. 1983 May-Jun;14(3):399-401</RefSource><PMID Version="1">6658909</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Neurology. 1984 Jul;34(7):939-44</RefSource><PMID Version="1">6610841</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Can J Neurol Sci. 1985 May;12(2):88-94</RefSource><PMID Version="1">4016596</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Arch Neurol. 1985 Oct;42(10):951-9</RefSource><PMID Version="1">4038102</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Brain. 1985 Sep;108 ( Pt 3):785-99</RefSource><PMID Version="1">3876136</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Rev Neurol (Paris). 1985;141(8-9):528-36</RefSource><PMID Version="1">4089415</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Ann Neurol. 1986 Mar;19(3):253-62</RefSource><PMID Version="1">3963770</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Brain. 1986 Oct;109 ( Pt 5):845-83</RefSource><PMID Version="1">3779372</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Arch Neurol. 1987 Jan;44(1):21-3</RefSource><PMID Version="1">3800716</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Arch Neurol. 1987 Jan;44(1):32-5</RefSource><PMID Version="1">3800719</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Arch Neurol. 1987 Jan;44(1):36-9</RefSource><PMID Version="1">3800720</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Arch Neurol. 1987 Jan;44(1):42-7</RefSource><PMID Version="1">3800721</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Neurology. 1987 Aug;37(8):1365-8</RefSource><PMID Version="1">3614659</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Arch Neurol. 1988 Jun;45(6):637-41</RefSource><PMID Version="1">3259421</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Arch Neurol. 1988 Jul;45(7):719-21</RefSource><PMID Version="1">3260480</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Brain. 1988 Dec;111 ( Pt 6):1425-39</RefSource><PMID Version="1">3208064</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Ann Neurol. 1989 Feb;25(2):131-9</RefSource><PMID Version="1">2784043</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Acta Neuropathol. 1989;78(2):131-6</RefSource><PMID Version="1">2546358</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Gastroenterology. 1980 Aug;79(2):232-41</RefSource><PMID Version="1">7399228</PMID></CommentsCorrections></CommentsCorrectionsList><MeshHeadingList><MeshHeading><DescriptorName UI="D000368" MajorTopicYN="N">Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D000369" MajorTopicYN="N">Aged, 80 and over</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D000544" MajorTopicYN="N">Alzheimer Disease</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="Y">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D002552" MajorTopicYN="N">Cerebral Ventricles</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="Y">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008875" MajorTopicYN="N">Middle Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D009454" MajorTopicYN="N">Neurofibrils</DescriptorName><QualifierName UI="Q000648" MajorTopicYN="N">ultrastructure</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D009474" MajorTopicYN="N">Neurons</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="N">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D010300" MajorTopicYN="N">Parkinson Disease</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="Y">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D013494" MajorTopicYN="N">Supranuclear Palsy, Progressive</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="Y">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D014057" MajorTopicYN="Y">Tomography, X-Ray Computed</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D014855" MajorTopicYN="N">Wallerian Degeneration</DescriptorName><QualifierName UI="Q000502" MajorTopicYN="Y">physiology</QualifierName></MeshHeading></MeshHeadingList><OtherID Source="NLM">PMC1014298</OtherID></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>1991</Year><Month>1</Month><Day>1</Day></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>1991</Year><Month>1</Month><Day>1</Day><Hour>0</Hour><Minute>1</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>1991</Year><Month>1</Month><Day>1</Day><Hour>0</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">2010759</ArticleId><ArticleId IdType="pmc">PMC1014298</ArticleId></ArticleIdList></PubmedData></pubmed><affiliations><list><country><li>France</li></country></list><tree><noCountry><name sortKey="Clarisse, J" sort="Clarisse, J" uniqKey="Clarisse J" first="J" last="Clarisse">J. Clarisse</name><name sortKey="Defossez, A" sort="Defossez, A" uniqKey="Defossez A" first="A" last="Défossez">A. Défossez</name><name sortKey="Delacourte, A" sort="Delacourte, A" uniqKey="Delacourte A" first="A" last="Delacourte">A. Delacourte</name><name sortKey="Parent, M" sort="Parent, M" uniqKey="Parent M" first="M" last="Parent">M. Parent</name><name sortKey="Pruvo, J P" sort="Pruvo, J P" uniqKey="Pruvo J" first="J P" last="Pruvo">J P Pruvo</name><name sortKey="Rapoport, A" sort="Rapoport, A" uniqKey="Rapoport A" first="A" last="Rapoport">A. Rapoport</name><name sortKey="Soetaert, G" sort="Soetaert, G" uniqKey="Soetaert G" first="G" last="Soetaert">G. Soetaert</name><name sortKey="Steinling, M" sort="Steinling, M" uniqKey="Steinling M" first="M" last="Steinling">M. Steinling</name><name sortKey="Vermersch, P" sort="Vermersch, P" uniqKey="Vermersch P" first="P" last="Vermersch">P. Vermersch</name></noCountry><country name="France"><noRegion><name sortKey="Leys, D" sort="Leys, D" uniqKey="Leys D" first="D" last="Leys">D. Leys</name></noRegion></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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