Heart rate variability and Parkinson's disease severity.
Identifieur interne : 000F96 ( PubMed/Checkpoint ); précédent : 000F95; suivant : 000F97Heart rate variability and Parkinson's disease severity.
Auteurs : D. Devos [France] ; M. Kroumova ; R. Bordet ; H. Vodougnon ; J D Guieu ; C. Libersa ; A. DesteeSource :
- Journal of neural transmission (Vienna, Austria : 1996) [ 0300-9564 ] ; 2003.
English descriptors
- KwdEn :
- Autonomic Nervous System Diseases (diagnosis), Autonomic Nervous System Diseases (etiology), Autonomic Nervous System Diseases (physiopathology), Cardiovascular System (physiopathology), Chronobiology Disorders (diagnosis), Chronobiology Disorders (etiology), Chronobiology Disorders (physiopathology), Disease Progression, Female, Heart Rate (drug effects), Heart Rate (physiology), Humans, Hypokinesia (complications), Hypokinesia (drug therapy), Hypokinesia (physiopathology), Levodopa (therapeutic use), Male, Middle Aged, Parkinson Disease (complications), Parkinson Disease (drug therapy), Parkinson Disease (physiopathology), Surveys and Questionnaires, Sympathetic Nervous System (physiopathology), Up-Regulation (drug effects), Up-Regulation (physiology), Vagus Nerve (physiopathology).
- MESH :
- chemical , therapeutic use : Levodopa.
- complications : Hypokinesia, Parkinson Disease.
- diagnosis : Autonomic Nervous System Diseases, Chronobiology Disorders.
- drug effects : Heart Rate, Up-Regulation.
- drug therapy : Hypokinesia, Parkinson Disease.
- etiology : Autonomic Nervous System Diseases, Chronobiology Disorders.
- physiology : Heart Rate, Up-Regulation.
- physiopathology : Autonomic Nervous System Diseases, Cardiovascular System, Chronobiology Disorders, Hypokinesia, Parkinson Disease, Sympathetic Nervous System, Vagus Nerve.
- Disease Progression, Female, Humans, Male, Middle Aged, Surveys and Questionnaires.
Abstract
Heart rate variability (HRV) decrease in Parkinson's disease (PD) could only be a consequence of reduce motor activity besides of being a marker of cardiovascular dysautonomia. Under continuously recorded and standardised motor activity, we studied thirty patients compared to controls in 3 PD stages: group I: less than 2 year-evolution, slight impaired without L-dopa; group II: mildly impaired with L-dopa; group III: advanced PD with motor complications. No difference was observed between group I and controls. The diurnal low frequency power (LF) and the ratio of LF/high frequency (HF) power decreased in groups II and III. The nocturnal vagal indicators: HF power and pNN50 were decreased in group III. Those parameters were correlated with Off-drug-motor handicap, suggesting an evolutive HRV decrease with disease severity but not with On-drug-motor activity. The low LF despite the higher motor activity in group III, due to dyskinesias, suggested a defective cardiovascular up-regulation.
DOI: 10.1007/s00702-003-0016-8
PubMed: 12928836
Affiliations:
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Links to Exploration step
pubmed:12928836Le document en format XML
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<term>Autonomic Nervous System Diseases (etiology)</term>
<term>Autonomic Nervous System Diseases (physiopathology)</term>
<term>Cardiovascular System (physiopathology)</term>
<term>Chronobiology Disorders (diagnosis)</term>
<term>Chronobiology Disorders (etiology)</term>
<term>Chronobiology Disorders (physiopathology)</term>
<term>Disease Progression</term>
<term>Female</term>
<term>Heart Rate (drug effects)</term>
<term>Heart Rate (physiology)</term>
<term>Humans</term>
<term>Hypokinesia (complications)</term>
<term>Hypokinesia (drug therapy)</term>
<term>Hypokinesia (physiopathology)</term>
<term>Levodopa (therapeutic use)</term>
<term>Male</term>
<term>Middle Aged</term>
<term>Parkinson Disease (complications)</term>
<term>Parkinson Disease (drug therapy)</term>
<term>Parkinson Disease (physiopathology)</term>
<term>Surveys and Questionnaires</term>
<term>Sympathetic Nervous System (physiopathology)</term>
<term>Up-Regulation (drug effects)</term>
<term>Up-Regulation (physiology)</term>
<term>Vagus Nerve (physiopathology)</term>
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<keywords scheme="MESH" type="chemical" qualifier="therapeutic use" xml:lang="en"><term>Levodopa</term>
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<term>Parkinson Disease</term>
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<term>Chronobiology Disorders</term>
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<term>Parkinson Disease</term>
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<term>Parkinson Disease</term>
<term>Sympathetic Nervous System</term>
<term>Vagus Nerve</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Disease Progression</term>
<term>Female</term>
<term>Humans</term>
<term>Male</term>
<term>Middle Aged</term>
<term>Surveys and Questionnaires</term>
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<front><div type="abstract" xml:lang="en">Heart rate variability (HRV) decrease in Parkinson's disease (PD) could only be a consequence of reduce motor activity besides of being a marker of cardiovascular dysautonomia. Under continuously recorded and standardised motor activity, we studied thirty patients compared to controls in 3 PD stages: group I: less than 2 year-evolution, slight impaired without L-dopa; group II: mildly impaired with L-dopa; group III: advanced PD with motor complications. No difference was observed between group I and controls. The diurnal low frequency power (LF) and the ratio of LF/high frequency (HF) power decreased in groups II and III. The nocturnal vagal indicators: HF power and pNN50 were decreased in group III. Those parameters were correlated with Off-drug-motor handicap, suggesting an evolutive HRV decrease with disease severity but not with On-drug-motor activity. The low LF despite the higher motor activity in group III, due to dyskinesias, suggested a defective cardiovascular up-regulation.</div>
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<Abstract><AbstractText>Heart rate variability (HRV) decrease in Parkinson's disease (PD) could only be a consequence of reduce motor activity besides of being a marker of cardiovascular dysautonomia. Under continuously recorded and standardised motor activity, we studied thirty patients compared to controls in 3 PD stages: group I: less than 2 year-evolution, slight impaired without L-dopa; group II: mildly impaired with L-dopa; group III: advanced PD with motor complications. No difference was observed between group I and controls. The diurnal low frequency power (LF) and the ratio of LF/high frequency (HF) power decreased in groups II and III. The nocturnal vagal indicators: HF power and pNN50 were decreased in group III. Those parameters were correlated with Off-drug-motor handicap, suggesting an evolutive HRV decrease with disease severity but not with On-drug-motor activity. The low LF despite the higher motor activity in group III, due to dyskinesias, suggested a defective cardiovascular up-regulation.</AbstractText>
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