Links to Exploration step
Le document en format XML
<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Parkinson's disease with camptocormia</title><author><name sortKey="Bloch, F" sort="Bloch, F" uniqKey="Bloch F" first="F" last="Bloch">F. Bloch</name></author><author><name sortKey="Houeto, J L" sort="Houeto, J L" uniqKey="Houeto J" first="J L" last="Houeto">J L Houeto</name></author><author><name sortKey="Du Montcel, S Tezenas" sort="Du Montcel, S Tezenas" uniqKey="Du Montcel S" first="S Tezenas" last="Du Montcel">S Tezenas Du Montcel</name></author><author><name sortKey="Bonneville, F" sort="Bonneville, F" uniqKey="Bonneville F" first="F" last="Bonneville">F. Bonneville</name></author><author><name sortKey="Etchepare, F" sort="Etchepare, F" uniqKey="Etchepare F" first="F" last="Etchepare">F. Etchepare</name></author><author><name sortKey="Welter, M L" sort="Welter, M L" uniqKey="Welter M" first="M L" last="Welter">M L Welter</name></author><author><name sortKey="Rivaud Echoux, S" sort="Rivaud Echoux, S" uniqKey="Rivaud Echoux S" first="S" last="Rivaud-Pechoux">S. Rivaud-Pechoux</name></author><author><name sortKey="Hahn Arma, V" sort="Hahn Arma, V" uniqKey="Hahn Arma V" first="V" last="Hahn-Barma">V. Hahn-Barma</name></author><author><name sortKey="Maisonobe, T" sort="Maisonobe, T" uniqKey="Maisonobe T" first="T" last="Maisonobe">T. Maisonobe</name></author><author><name sortKey="Behar, C" sort="Behar, C" uniqKey="Behar C" first="C" last="Behar">C. Behar</name></author><author><name sortKey="Lazennec, J Y" sort="Lazennec, J Y" uniqKey="Lazennec J" first="J Y" last="Lazennec">J Y Lazennec</name></author><author><name sortKey="Kurys, E" sort="Kurys, E" uniqKey="Kurys E" first="E" last="Kurys">E. Kurys</name></author><author><name sortKey="Arnulf, I" sort="Arnulf, I" uniqKey="Arnulf I" first="I" last="Arnulf">I. Arnulf</name></author><author><name sortKey="Bonnet, A M" sort="Bonnet, A M" uniqKey="Bonnet A" first="A M" last="Bonnet">A M Bonnet</name></author><author><name sortKey="Agid, Y" sort="Agid, Y" uniqKey="Agid Y" first="Y" last="Agid">Y. Agid</name></author></titleStmt><publicationStmt><idno type="wicri:source">PMC</idno><idno type="pmid">16754693</idno><idno type="pmc">2077378</idno><idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2077378</idno><idno type="RBID">PMC:2077378</idno><idno type="doi">10.1136/jnnp.2006.087908</idno><date when="2006">2006</date><idno type="wicri:Area/Pmc/Corpus">000298</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Corpus" wicri:corpus="PMC">000298</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">Parkinson's disease with camptocormia</title><author><name sortKey="Bloch, F" sort="Bloch, F" uniqKey="Bloch F" first="F" last="Bloch">F. Bloch</name></author><author><name sortKey="Houeto, J L" sort="Houeto, J L" uniqKey="Houeto J" first="J L" last="Houeto">J L Houeto</name></author><author><name sortKey="Du Montcel, S Tezenas" sort="Du Montcel, S Tezenas" uniqKey="Du Montcel S" first="S Tezenas" last="Du Montcel">S Tezenas Du Montcel</name></author><author><name sortKey="Bonneville, F" sort="Bonneville, F" uniqKey="Bonneville F" first="F" last="Bonneville">F. Bonneville</name></author><author><name sortKey="Etchepare, F" sort="Etchepare, F" uniqKey="Etchepare F" first="F" last="Etchepare">F. Etchepare</name></author><author><name sortKey="Welter, M L" sort="Welter, M L" uniqKey="Welter M" first="M L" last="Welter">M L Welter</name></author><author><name sortKey="Rivaud Echoux, S" sort="Rivaud Echoux, S" uniqKey="Rivaud Echoux S" first="S" last="Rivaud-Pechoux">S. Rivaud-Pechoux</name></author><author><name sortKey="Hahn Arma, V" sort="Hahn Arma, V" uniqKey="Hahn Arma V" first="V" last="Hahn-Barma">V. Hahn-Barma</name></author><author><name sortKey="Maisonobe, T" sort="Maisonobe, T" uniqKey="Maisonobe T" first="T" last="Maisonobe">T. Maisonobe</name></author><author><name sortKey="Behar, C" sort="Behar, C" uniqKey="Behar C" first="C" last="Behar">C. Behar</name></author><author><name sortKey="Lazennec, J Y" sort="Lazennec, J Y" uniqKey="Lazennec J" first="J Y" last="Lazennec">J Y Lazennec</name></author><author><name sortKey="Kurys, E" sort="Kurys, E" uniqKey="Kurys E" first="E" last="Kurys">E. Kurys</name></author><author><name sortKey="Arnulf, I" sort="Arnulf, I" uniqKey="Arnulf I" first="I" last="Arnulf">I. Arnulf</name></author><author><name sortKey="Bonnet, A M" sort="Bonnet, A M" uniqKey="Bonnet A" first="A M" last="Bonnet">A M Bonnet</name></author><author><name sortKey="Agid, Y" sort="Agid, Y" uniqKey="Agid Y" first="Y" last="Agid">Y. Agid</name></author></analytic><series><title level="j">Journal of Neurology, Neurosurgery, and Psychiatry</title><idno type="ISSN">0022-3050</idno><idno type="eISSN">1468-330X</idno><imprint><date when="2006">2006</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en"><sec><title>Background</title><p>Camptocormia is defined as an abnormal flexion of the trunk that appears when standing or walking and disappears in the supine position. The origin of the disorder is unknown, but it is usually attributed either to a primary or a secondary paravertebral muscle myopathy or a motor neurone disorder. Camptocormia is also observed in a minority of patients with parkinsonism.</p></sec><sec><title>Objective</title><p>To characterise the clinical and electrophysiological features of camptocormia and parkinsonian symptoms in patients with Parkinson's disease and camptocormia compared with patients with Parkinson's disease without camptocormia.</p></sec><sec><title>Methods</title><p>Patients with parkinsonism and camptocormia (excluding patients with multiple system atrophy) prospectively underwent a multidisciplinary clinical (neurological, neuropsychological, psychological, rheumatological) and neurophysiological (electromyogram, ocular movement recording) examination and were compared with age‐matched patients with Parkinson's disease without camptocormia.</p></sec><sec><title>Results</title><p>The camptocormia developed after 8.5 (SD 5.3) years of parkinsonism, responded poorly to levodopa treatment (20%) and displayed features consistent with axial dystonia. Patients with camptocormia were characterised by prominent levodopa‐unresponsive axial symptoms (ie, axial rigidity, gait disorder and postural instability), along with a tendency for greater error in the antisaccade paradigm.</p></sec><sec><title>Conclusion</title><p>We suggest that (1) the salient features of parkinsonism observed in patients with camptocormia are likely to represent a specific form of Parkinson's disease and camptocormia is an axial dystonia and (2) both camptocormia and parkinsonism in these patients might result from additional, non‐dopaminergic neuronal dysfunction in the basal ganglia.</p></sec></div></front></TEI><pmc article-type="research-article"><pmc-comment>The publisher of this article does not allow downloading of the full text in XML form.</pmc-comment>
<front><journal-meta><journal-id journal-id-type="nlm-ta">J Neurol Neurosurg Psychiatry</journal-id><journal-title>Journal of Neurology, Neurosurgery, and Psychiatry</journal-title><issn pub-type="ppub">0022-3050</issn><issn pub-type="epub">1468-330X</issn><publisher><publisher-name>BMJ Group</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="pmid">16754693</article-id><article-id pub-id-type="pmc">2077378</article-id><article-id pub-id-type="publisher-id">jn87908</article-id><article-id pub-id-type="doi">10.1136/jnnp.2006.087908</article-id><article-categories><subj-group subj-group-type="heading"><subject>Paper</subject></subj-group></article-categories><title-group><article-title>Parkinson's disease with camptocormia</article-title></title-group><contrib-group><contrib contrib-type="author"><name><surname>Bloch</surname><given-names>F</given-names></name></contrib><contrib contrib-type="author"><name><surname>Houeto</surname><given-names>J L</given-names></name></contrib><contrib contrib-type="author"><name><surname>du Montcel</surname><given-names>S Tezenas</given-names></name></contrib><contrib contrib-type="author"><name><surname>Bonneville</surname><given-names>F</given-names></name></contrib><contrib contrib-type="author"><name><surname>Etchepare</surname><given-names>F</given-names></name></contrib><contrib contrib-type="author"><name><surname>Welter</surname><given-names>M L</given-names></name></contrib><contrib contrib-type="author"><name><surname>Rivaud‐Pechoux</surname><given-names>S</given-names></name></contrib><contrib contrib-type="author"><name><surname>Hahn‐Barma</surname><given-names>V</given-names></name></contrib><contrib contrib-type="author"><name><surname>Maisonobe</surname><given-names>T</given-names></name></contrib><contrib contrib-type="author"><name><surname>Behar</surname><given-names>C</given-names></name></contrib><contrib contrib-type="author"><name><surname>Lazennec</surname><given-names>J Y</given-names></name></contrib><contrib contrib-type="author"><name><surname>Kurys</surname><given-names>E</given-names></name></contrib><contrib contrib-type="author"><name><surname>Arnulf</surname><given-names>I</given-names></name></contrib><contrib contrib-type="author"><name><surname>Bonnet</surname><given-names>A M</given-names></name></contrib><contrib contrib-type="author"><name><surname>Agid</surname><given-names>Y</given-names></name></contrib></contrib-group><aff><bold>F Bloch</bold>,<bold>J L Houeto</bold>,<bold>M L Welter</bold>,<bold>V Hahn‐Barma</bold>,<bold>C Behar</bold>,<bold>A M Bonnet</bold>,<bold>Y Agid</bold>, Centre d'Investigation Clinique‐Fédération des Maladies du Système Nerveux, Groupe‐Hospitalier Pitié‐Salpêtrière, Paris, France</aff><aff><bold>S Tezenas du Montcel</bold>, Service de Biostatistiques et Information Médicale, Groupe‐Hospitalier Pitié‐Salpêtrière</aff><aff><bold>F Bonneville</bold>,<bold>E Kurys</bold>, Fédération de Neuroradiologie, Groupe‐Hospitalier Pitié‐Salpêtrière; UPR640 CNRS LENA, Poitiers, France</aff><aff><bold>F Etchepare</bold>, Service de Rhumatologie, Groupe‐Hospitalier Pitié‐Salpêtrière</aff><aff><bold>S Rivaud‐Pechoux</bold>, INSERM U679, Groupe‐Hospitalier Pitié‐Salpêtrière; IFR 70, Paris</aff><aff><bold>V Hahn‐Barma</bold>, INSERM U610, Groupe‐Hospitalier Pitié‐Salpêtrière</aff><aff><bold>T Maisonobe</bold>, Fédération de Neurophysiologie, Groupe‐Hospitalier Pitié‐Salpêtrière</aff><aff><bold>J Y Lazennec</bold>, Service d'Orthopédie, Groupe‐Hospitalier Pitié‐Salpêtrière</aff><aff><bold>I Arnulf</bold>, Fédération des Pathologies du Sommeil, Groupe‐Hospitalier Pitié‐Salpêtrière (Assistance Publique—Hôpitaux de Paris), Paris</aff><aff><bold>J L Houeto</bold>, Service de Neurologie, CHU la Milétrie, Poitiers</aff><author-notes><corresp>Correspondence to: Y Agid<break></break>Centre d'Investigation Clinique, Hôpital de la Salpêtrière, 47 Boulevard de l'Hôpital, 75013 Paris, France; agid@ccr.jussieu.fr</corresp></author-notes><pub-date pub-type="ppub"><month>11</month><year>2006</year></pub-date><pub-date pub-type="epub"><day>5</day><month>6</month><year>2006</year></pub-date><volume>77</volume><issue>11</issue><fpage>1223</fpage><lpage>1228</lpage><history><date date-type="received"><day>10</day><month>1</month><year>2006</year></date><date date-type="rev-recd"><day>9</day><month>5</month><year>2006</year></date><date date-type="accepted"><day>26</day><month>5</month><year>2006</year></date></history><permissions><copyright-statement>Copyright © 2006 BMJ Publishing Group</copyright-statement></permissions><abstract><sec><title>Background</title><p>Camptocormia is defined as an abnormal flexion of the trunk that appears when standing or walking and disappears in the supine position. The origin of the disorder is unknown, but it is usually attributed either to a primary or a secondary paravertebral muscle myopathy or a motor neurone disorder. Camptocormia is also observed in a minority of patients with parkinsonism.</p></sec><sec><title>Objective</title><p>To characterise the clinical and electrophysiological features of camptocormia and parkinsonian symptoms in patients with Parkinson's disease and camptocormia compared with patients with Parkinson's disease without camptocormia.</p></sec><sec><title>Methods</title><p>Patients with parkinsonism and camptocormia (excluding patients with multiple system atrophy) prospectively underwent a multidisciplinary clinical (neurological, neuropsychological, psychological, rheumatological) and neurophysiological (electromyogram, ocular movement recording) examination and were compared with age‐matched patients with Parkinson's disease without camptocormia.</p></sec><sec><title>Results</title><p>The camptocormia developed after 8.5 (SD 5.3) years of parkinsonism, responded poorly to levodopa treatment (20%) and displayed features consistent with axial dystonia. Patients with camptocormia were characterised by prominent levodopa‐unresponsive axial symptoms (ie, axial rigidity, gait disorder and postural instability), along with a tendency for greater error in the antisaccade paradigm.</p></sec><sec><title>Conclusion</title><p>We suggest that (1) the salient features of parkinsonism observed in patients with camptocormia are likely to represent a specific form of Parkinson's disease and camptocormia is an axial dystonia and (2) both camptocormia and parkinsonism in these patients might result from additional, non‐dopaminergic neuronal dysfunction in the basal ganglia.</p></sec></abstract></article-meta></front></pmc></record>Pour manipuler ce document sous Unix (Dilib)
EXPLOR_STEP=$WICRI_ROOT/Wicri/Sante/explor/ParkinsonFranceV1/Data/Pmc/Corpus
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 0002980 | SxmlIndent | more
Ou
HfdSelect -h $EXPLOR_AREA/Data/Pmc/Corpus/biblio.hfd -nk 0002980 | SxmlIndent | more
Pour mettre un lien sur cette page dans le réseau Wicri
{{Explor lien
|wiki= Wicri/Sante
|area= ParkinsonFranceV1
|flux= Pmc
|étape= Corpus
|type= RBID
|clé=
|texte=
}}
| This area was generated with Dilib version V0.6.29. |  |