Are explicit memory disorders of progressive supranuclear palsy related to damage to striatofrontal circuits ? : Comparison with Alzheimer's, Parkinson's, and Huntington's diseases
Identifieur interne : 001699 ( PascalFrancis/Curation ); précédent : 001698; suivant : 001700Are explicit memory disorders of progressive supranuclear palsy related to damage to striatofrontal circuits ? : Comparison with Alzheimer's, Parkinson's, and Huntington's diseases
Auteurs : B. Pillon [France] ; B. Deweer ; A. Michon ; C. Malapani ; Y. Agid ; B. DuboisSource :
- Neurology [ 0028-3878 ] ; 1994.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Homme.
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Abstract
To test the hypothesis that memory disorders of subcortico-frontal dementia result mainly from inefficiency of retrieval processes of stored information, we compared verbal learning in 15 patients with progressive supranuclear palsy, prototypical of «subcortical dementia,» in free (California Verbal Learning Test) and controlled (Grober and Buschke's Test) encoding situations, with that of 19 controls, matched for age and level of education. The progressive supranuclear palsy patients showed memory deficits characterized by impaired immediate memory span, disturbed learning and consistency of recall, and abnormal number of false alarms at recognition, which were dramatically alleviated by controlled encoding associated with cued recall, using the same semantic cues
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<front><div type="abstract" xml:lang="en">To test the hypothesis that memory disorders of subcortico-frontal dementia result mainly from inefficiency of retrieval processes of stored information, we compared verbal learning in 15 patients with progressive supranuclear palsy, prototypical of «subcortical dementia,» in free (California Verbal Learning Test) and controlled (Grober and Buschke's Test) encoding situations, with that of 19 controls, matched for age and level of education. The progressive supranuclear palsy patients showed memory deficits characterized by impaired immediate memory span, disturbed learning and consistency of recall, and abnormal number of false alarms at recognition, which were dramatically alleviated by controlled encoding associated with cued recall, using the same semantic cues</div>
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