La maladie de Parkinson en France (serveur d'exploration)

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Emotional Processing in Parkinson's Disease: A Systematic Review

Identifieur interne : 001033 ( PascalFrancis/Curation ); précédent : 001032; suivant : 001034

Emotional Processing in Parkinson's Disease: A Systematic Review

Auteurs : Julie Peron [France, Suisse] ; Thibaut Dondaine [France] ; Florence Le Jeune [France] ; Didier Grandjean [Suisse] ; Marc Verin [France]

Source :

RBID : Pascal:12-0113822

Descripteurs français

English descriptors

Abstract

Parkinson's disease provides a useful model for studying the neural substrates of emotional processing. The striato-thalamo-cortical circuits, like the mesolimbic dopamine system that modulates their function, are thought to be involved in emotional processing. As Parkinson's disease is histopathologically characterized by the selective, progressive, and chronic degeneration of the nigrostriatal and mesocorticolimbic dopamine systems, it can therefore serve as a model for assessing the functional role of these circuits in humans. In the present review, we begin by providing a synopsis of the emotional disturbances observed in Parkinson's disease. We then discuss the functional roles of the striato-thalamo-cortical and mesolimbic circuits, ending with the conclusion that both these pathways are indeed involved in emotional processing.
pA  
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A08 01  1  ENG  @1 Emotional Processing in Parkinson's Disease: A Systematic Review
A11 01  1    @1 PERON (Julie)
A11 02  1    @1 DONDAINE (Thibaut)
A11 03  1    @1 JEUNE (Florence Le)
A11 04  1    @1 GRANDJEAN (Didier)
A11 05  1    @1 VERIN (Marc)
A14 01      @1 Behavior and Basal Ganglia Research Unit (EM 425), University of Rennes 1, Hôpital Pontchaillou, CHU de Rennes @2 Rennes @3 FRA @Z 1 aut. @Z 2 aut. @Z 3 aut. @Z 5 aut.
A14 02      @1 Neurology Unit, Hôpital Pontchaillou, CHU de Rennes @2 Rennes @3 FRA @Z 1 aut. @Z 2 aut. @Z 5 aut.
A14 03      @1 Neuroscience of Emotion and Affective Dynamics Laboratory, Department of Psychology and Swiss Center for Affective Sciences, University of Geneva @2 Geneva @3 CHE @Z 1 aut. @Z 4 aut.
A14 04      @1 Nuclear Medicine Department, Eugene Marquis AntiCancer Centre @2 Rennes @3 FRA @Z 3 aut.
A20       @1 186-199
A21       @1 2012
A23 01      @0 ENG
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A44       @0 0000 @1 © 2012 INIST-CNRS. All rights reserved.
A45       @0 112 ref.
A47 01  1    @0 12-0113822
A60       @1 P
A61       @0 A
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C01 01    ENG  @0 Parkinson's disease provides a useful model for studying the neural substrates of emotional processing. The striato-thalamo-cortical circuits, like the mesolimbic dopamine system that modulates their function, are thought to be involved in emotional processing. As Parkinson's disease is histopathologically characterized by the selective, progressive, and chronic degeneration of the nigrostriatal and mesocorticolimbic dopamine systems, it can therefore serve as a model for assessing the functional role of these circuits in humans. In the present review, we begin by providing a synopsis of the emotional disturbances observed in Parkinson's disease. We then discuss the functional roles of the striato-thalamo-cortical and mesolimbic circuits, ending with the conclusion that both these pathways are indeed involved in emotional processing.
C02 01  X    @0 002B17
C02 02  X    @0 002B17G
C03 01  X  FRE  @0 Maladie de Parkinson @2 NM @5 01
C03 01  X  ENG  @0 Parkinson disease @2 NM @5 01
C03 01  X  SPA  @0 Parkinson enfermedad @2 NM @5 01
C03 02  X  FRE  @0 Pathologie du système nerveux @5 02
C03 02  X  ENG  @0 Nervous system diseases @5 02
C03 02  X  SPA  @0 Sistema nervioso patología @5 02
C03 03  X  FRE  @0 Amygdale @5 09
C03 03  X  ENG  @0 Amygdala @5 09
C03 03  X  SPA  @0 Amígdala @5 09
C03 04  X  FRE  @0 Noyau gris central @5 10
C03 04  X  ENG  @0 Basal ganglion @5 10
C03 04  X  SPA  @0 Núcleo basal @5 10
C07 01  X  FRE  @0 Pathologie de l'encéphale @5 37
C07 01  X  ENG  @0 Cerebral disorder @5 37
C07 01  X  SPA  @0 Encéfalo patología @5 37
C07 02  X  FRE  @0 Syndrome extrapyramidal @5 38
C07 02  X  ENG  @0 Extrapyramidal syndrome @5 38
C07 02  X  SPA  @0 Extrapiramidal síndrome @5 38
C07 03  X  FRE  @0 Maladie dégénérative @5 39
C07 03  X  ENG  @0 Degenerative disease @5 39
C07 03  X  SPA  @0 Enfermedad degenerativa @5 39
C07 04  X  FRE  @0 Pathologie du système nerveux central @5 40
C07 04  X  ENG  @0 Central nervous system disease @5 40
C07 04  X  SPA  @0 Sistema nervosio central patología @5 40
C07 05  X  FRE  @0 Encéphale @5 42
C07 05  X  ENG  @0 Encephalon @5 42
C07 05  X  SPA  @0 Encéfalo @5 42
C07 06  X  FRE  @0 Système nerveux central @5 43
C07 06  X  ENG  @0 Central nervous system @5 43
C07 06  X  SPA  @0 Sistema nervioso central @5 43
N21       @1 086
N44 01      @1 OTO
N82       @1 OTO

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Pascal:12-0113822

Le document en format XML

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<div type="abstract" xml:lang="en">Parkinson's disease provides a useful model for studying the neural substrates of emotional processing. The striato-thalamo-cortical circuits, like the mesolimbic dopamine system that modulates their function, are thought to be involved in emotional processing. As Parkinson's disease is histopathologically characterized by the selective, progressive, and chronic degeneration of the nigrostriatal and mesocorticolimbic dopamine systems, it can therefore serve as a model for assessing the functional role of these circuits in humans. In the present review, we begin by providing a synopsis of the emotional disturbances observed in Parkinson's disease. We then discuss the functional roles of the striato-thalamo-cortical and mesolimbic circuits, ending with the conclusion that both these pathways are indeed involved in emotional processing.</div>
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<s0>Amygdala</s0>
<s5>09</s5>
</fC03>
<fC03 i1="03" i2="X" l="SPA">
<s0>Amígdala</s0>
<s5>09</s5>
</fC03>
<fC03 i1="04" i2="X" l="FRE">
<s0>Noyau gris central</s0>
<s5>10</s5>
</fC03>
<fC03 i1="04" i2="X" l="ENG">
<s0>Basal ganglion</s0>
<s5>10</s5>
</fC03>
<fC03 i1="04" i2="X" l="SPA">
<s0>Núcleo basal</s0>
<s5>10</s5>
</fC03>
<fC07 i1="01" i2="X" l="FRE">
<s0>Pathologie de l'encéphale</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="ENG">
<s0>Cerebral disorder</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="SPA">
<s0>Encéfalo patología</s0>
<s5>37</s5>
</fC07>
<fC07 i1="02" i2="X" l="FRE">
<s0>Syndrome extrapyramidal</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="ENG">
<s0>Extrapyramidal syndrome</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="SPA">
<s0>Extrapiramidal síndrome</s0>
<s5>38</s5>
</fC07>
<fC07 i1="03" i2="X" l="FRE">
<s0>Maladie dégénérative</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="ENG">
<s0>Degenerative disease</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="SPA">
<s0>Enfermedad degenerativa</s0>
<s5>39</s5>
</fC07>
<fC07 i1="04" i2="X" l="FRE">
<s0>Pathologie du système nerveux central</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="ENG">
<s0>Central nervous system disease</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="SPA">
<s0>Sistema nervosio central patología</s0>
<s5>40</s5>
</fC07>
<fC07 i1="05" i2="X" l="FRE">
<s0>Encéphale</s0>
<s5>42</s5>
</fC07>
<fC07 i1="05" i2="X" l="ENG">
<s0>Encephalon</s0>
<s5>42</s5>
</fC07>
<fC07 i1="05" i2="X" l="SPA">
<s0>Encéfalo</s0>
<s5>42</s5>
</fC07>
<fC07 i1="06" i2="X" l="FRE">
<s0>Système nerveux central</s0>
<s5>43</s5>
</fC07>
<fC07 i1="06" i2="X" l="ENG">
<s0>Central nervous system</s0>
<s5>43</s5>
</fC07>
<fC07 i1="06" i2="X" l="SPA">
<s0>Sistema nervioso central</s0>
<s5>43</s5>
</fC07>
<fN21>
<s1>086</s1>
</fN21>
<fN44 i1="01">
<s1>OTO</s1>
</fN44>
<fN82>
<s1>OTO</s1>
</fN82>
</pA>
</standard>
</inist>
</record>

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