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La maladie de Parkinson en France (serveur d'exploration)

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Alzheimer's disease revisited

Identifieur interne : 001367 ( PascalFrancis/Corpus ); précédent : 001366; suivant : 001368

Alzheimer's disease revisited

Auteurs : P. E. Newman

Source :

RBID : Pascal:00-0327319

Descripteurs français

English descriptors

Abstract

In a previous paper, it was suggested that a relative deficiency of essential fatty acids might play a role in the etiology of sporadic or non-familial Alzheimer's disease. A recent article regarding dementia in the Rotterdam Study reinforces this suggestion. It is also hypothesized that this relative deficiency could facilitate passage of aluminum into the brain, aluminum being increasingly suggested as one of the possible pathogenic factors in AD. It is further suggested that hypomethylation caused by a deficiency of S-adenosylmethionine might also play a role in the etiology of this disease and perhaps even of Parkinson's disease.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

pA  
A01 01  1    @0 0306-9877
A03   1    @0 Med. hypotheses
A05       @2 54
A06       @2 5
A08 01  1  ENG  @1 Alzheimer's disease revisited
A11 01  1    @1 NEWMAN (P. E.)
A14 01      @2 10, Anatole de la Forge, 75017 Paris @3 FRA @Z 1 aut.
A20       @1 774-776
A21       @1 2000
A23 01      @0 ENG
A43 01      @1 INIST @2 18253 @5 354000088753050210
A44       @0 0000 @1 © 2000 INIST-CNRS. All rights reserved.
A45       @0 9 ref.
A47 01  1    @0 00-0327319
A60       @1 P
A61       @0 A
A64 01  1    @0 Medical hypotheses
A66 01      @0 GBR
C01 01    ENG  @0 In a previous paper, it was suggested that a relative deficiency of essential fatty acids might play a role in the etiology of sporadic or non-familial Alzheimer's disease. A recent article regarding dementia in the Rotterdam Study reinforces this suggestion. It is also hypothesized that this relative deficiency could facilitate passage of aluminum into the brain, aluminum being increasingly suggested as one of the possible pathogenic factors in AD. It is further suggested that hypomethylation caused by a deficiency of S-adenosylmethionine might also play a role in the etiology of this disease and perhaps even of Parkinson's disease.
C02 01  X    @0 002B17G
C03 01  X  FRE  @0 Démence Alzheimer @5 01
C03 01  X  ENG  @0 Alzheimer disease @5 01
C03 01  X  SPA  @0 Demencia Alzheimer @5 01
C03 02  X  FRE  @0 Pathogénie @5 02
C03 02  X  ENG  @0 Pathogenesis @5 02
C03 02  X  SPA  @0 Patogenia @5 02
C03 03  X  FRE  @0 Déficit @5 03
C03 03  X  ENG  @0 Deficiency @5 03
C03 03  X  SPA  @0 Déficiencia @5 03
C03 04  X  FRE  @0 Acide gras @5 04
C03 04  X  ENG  @0 Fatty acids @5 04
C03 04  X  SPA  @0 Acido graso @5 04
C03 05  X  FRE  @0 Essentiel @5 05
C03 05  X  ENG  @0 Essential @5 05
C03 05  X  SPA  @0 Esencial @5 05
C03 06  X  FRE  @0 Taux concentration @5 06
C03 06  X  ENG  @0 Concentration factor @5 06
C03 06  X  SPA  @0 Indice concentración @5 06
C03 07  X  FRE  @0 Aluminium @2 NC @2 FR @2 FX @5 07
C03 07  X  ENG  @0 Aluminium @2 NC @2 FR @2 FX @5 07
C03 07  X  SPA  @0 Aluminio @2 NC @2 FR @2 FX @5 07
C03 08  X  FRE  @0 Encéphale @5 08
C03 08  X  ENG  @0 Brain (vertebrata) @5 08
C03 08  X  SPA  @0 Encéfalo @5 08
C03 09  X  FRE  @0 Prévention @5 09
C03 09  X  ENG  @0 Prevention @5 09
C03 09  X  SPA  @0 Prevención @5 09
C03 10  X  FRE  @0 Homme @5 20
C03 10  X  ENG  @0 Human @5 20
C03 10  X  SPA  @0 Hombre @5 20
C07 01  X  FRE  @0 Système nerveux pathologie @5 37
C07 01  X  ENG  @0 Nervous system diseases @5 37
C07 01  X  SPA  @0 Sistema nervioso patología @5 37
C07 02  X  FRE  @0 Système nerveux central pathologie @5 38
C07 02  X  ENG  @0 Central nervous system disease @5 38
C07 02  X  SPA  @0 Sistema nervosio central patología @5 38
C07 03  X  FRE  @0 Encéphale pathologie @5 39
C07 03  X  ENG  @0 Cerebral disorder @5 39
C07 03  X  SPA  @0 Encéfalo patología @5 39
C07 04  X  FRE  @0 Maladie dégénérative @5 40
C07 04  X  ENG  @0 Degenerative disease @5 40
C07 04  X  SPA  @0 Enfermedad degenerativa @5 40
C07 05  X  FRE  @0 Métabolisme pathologie @5 45
C07 05  X  ENG  @0 Metabolic diseases @5 45
C07 05  X  SPA  @0 Metabolismo patología @5 45
N21       @1 220

Format Inist (serveur)

NO : PASCAL 00-0327319 INIST
ET : Alzheimer's disease revisited
AU : NEWMAN (P. E.)
AF : 10, Anatole de la Forge, 75017 Paris/France (1 aut.)
DT : Publication en série; Niveau analytique
SO : Medical hypotheses; ISSN 0306-9877; Royaume-Uni; Da. 2000; Vol. 54; No. 5; Pp. 774-776; Bibl. 9 ref.
LA : Anglais
EA : In a previous paper, it was suggested that a relative deficiency of essential fatty acids might play a role in the etiology of sporadic or non-familial Alzheimer's disease. A recent article regarding dementia in the Rotterdam Study reinforces this suggestion. It is also hypothesized that this relative deficiency could facilitate passage of aluminum into the brain, aluminum being increasingly suggested as one of the possible pathogenic factors in AD. It is further suggested that hypomethylation caused by a deficiency of S-adenosylmethionine might also play a role in the etiology of this disease and perhaps even of Parkinson's disease.
CC : 002B17G
FD : Démence Alzheimer; Pathogénie; Déficit; Acide gras; Essentiel; Taux concentration; Aluminium; Encéphale; Prévention; Homme
FG : Système nerveux pathologie; Système nerveux central pathologie; Encéphale pathologie; Maladie dégénérative; Métabolisme pathologie
ED : Alzheimer disease; Pathogenesis; Deficiency; Fatty acids; Essential; Concentration factor; Aluminium; Brain (vertebrata); Prevention; Human
EG : Nervous system diseases; Central nervous system disease; Cerebral disorder; Degenerative disease; Metabolic diseases
SD : Demencia Alzheimer; Patogenia; Déficiencia; Acido graso; Esencial; Indice concentración; Aluminio; Encéfalo; Prevención; Hombre
LO : INIST-18253.354000088753050210
ID : 00-0327319

Links to Exploration step

Pascal:00-0327319

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