Dopamine depletion impairs precursor cell proliferation in Parkinson disease.

Dopamine depletion impairs precursor cell proliferation in Parkinson disease.

Source :

• Nature neuroscience [ 1097-6256 ] ; 2004.

English descriptors

• KwdEn :
  • Aged, Analysis of Variance, Animals, Antiparkinson Agents (therapeutic use), Bromodeoxyuridine (metabolism), Case-Control Studies, Cell Count (methods), Cell Death (physiology), Cell Division (drug effects), Cells, Cultured, Dopamine (deficiency), Dopamine (physiology), Dopamine Antagonists (pharmacology), Dopamine Plasma Membrane Transport Proteins, Drug Interactions, Ependyma (ultrastructure), Female, Glial Fibrillary Acidic Protein (metabolism), Glycoproteins (metabolism), Humans, Immunohistochemistry (methods), In Situ Nick-End Labeling (methods), Indoles (therapeutic use), Intermediate Filament Proteins (metabolism), Levodopa (therapeutic use), Male, Membrane Glycoproteins (metabolism), Membrane Transport Proteins (metabolism), Mice, Mice, Inbred C57BL, Microscopy, Confocal (methods), Microscopy, Immunoelectron (methods), Microspheres, Middle Aged, Nerve Tissue Proteins (metabolism), Nestin, Neural Cell Adhesion Molecule L1 (metabolism), Neurons (metabolism), Neurons (pathology), Neurons (ultrastructure), Olfactory Bulb (cytology), Olfactory Bulb (metabolism), Oxidopamine (toxicity), Parkinson Disease (drug therapy), Parkinson Disease (metabolism), Parkinson Disease (pathology), Parkinsonian Disorders (metabolism), Parkinsonian Disorders (pathology), Postmortem Changes, Rats, Rats, Sprague-Dawley, Receptor, Epidermal Growth Factor, Receptor, ErbB-2, Receptors, Dopamine (metabolism), Sialic Acids (metabolism), Stem Cells (metabolism), Stem Cells (pathology), Stem Cells (ultrastructure), Time Factors, Tubulin (metabolism), Tyrosine 3-Monooxygenase (metabolism).
• MESH :
  • chemical , deficiency : Dopamine.
  • chemical , metabolism : Bromodeoxyuridine, Glial Fibrillary Acidic Protein, Glycoproteins, Intermediate Filament Proteins, Membrane Glycoproteins, Membrane Transport Proteins, Nerve Tissue Proteins, Neural Cell Adhesion Molecule L1, Receptors, Dopamine, Sialic Acids, Tubulin, Tyrosine 3-Monooxygenase.
  • chemical , pharmacology : Dopamine Antagonists.
  • chemical , physiology : Dopamine.
  • chemical , therapeutic use : Antiparkinson Agents, Indoles, Levodopa.
  • cytology : Olfactory Bulb.
  • drug effects : Cell Division.
  • drug therapy : Parkinson Disease.
  • metabolism : Neurons, Olfactory Bulb, Parkinson Disease, Parkinsonian Disorders, Stem Cells.
  • methods : Cell Count, Immunohistochemistry, In Situ Nick-End Labeling, Microscopy, Confocal, Microscopy, Immunoelectron.
  • pathology : Neurons, Parkinson Disease, Parkinsonian Disorders, Stem Cells.
  • physiology : Cell Death.
  • chemical , toxicity : Oxidopamine.
  • ultrastructure : Ependyma, Neurons, Stem Cells.
  • Aged, Analysis of Variance, Animals, Case-Control Studies, Cells, Cultured, Dopamine Plasma Membrane Transport Proteins, Drug Interactions, Female, Humans, Male, Mice, Mice, Inbred C57BL, Microspheres, Middle Aged, Nestin, Postmortem Changes, Rats, Rats, Sprague-Dawley, Receptor, Epidermal Growth Factor, Receptor, ErbB-2, Time Factors.

Abstract

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