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La maladie de Parkinson en France (serveur d'exploration) - Checkpoint (Ncbi)

Index « Auteurs » - entrée « Jacques Mallet »
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Jacques Magnan < Jacques Mallet < Jacques Montplaisir  Facettes :

List of bibliographic references

Number of relevant bibliographic references: 6.
Ident.Authors (with country if any)Title
000069 (1999) Olga Corti [France] ; Amelia Sánchez-Capelo [France] ; Philippe Colin [France] ; Naïma Hanoun [France] ; Michel Hamon [France] ; Jacques Mallet [France]Long-term doxycycline-controlled expression of human tyrosine hydroxylase after direct adenovirus-mediated gene transfer to a rat model of Parkinson’s disease
000425 (2003) Pierre-François Pradat [France] ; Jacques MalletGene transfer into the central and peripheral nervous system: applications for the treatment of neurodegenerative diseases and peripheral neuropathies.
000541 (2006) Mara Brizard [France] ; Carole Carcenac ; Alexis-Pierre Bemelmans ; Claude Feuerstein ; Jacques Mallet ; Marc SavastaFunctional reinnervation from remaining DA terminals induced by GDNF lentivirus in a rat model of early Parkinson's disease.
000575 (2006) Martine Barkats [France] ; Philippe Horellou ; Philippe Colin ; Stéphanie Millecamps ; Nicole Faucon-Biguet ; Jacques Mallet1-methyl-4-phenylpyridinium neurotoxicity is attenuated by adenoviral gene transfer of human Cu/Zn superoxide dismutase.
000A30 (2009) Mathieu Desclaux [France] ; Marisa Teigell [France] ; Lahouari Amar [France] ; Roland Vogel [France] ; Minerva Gimenez Y Ribotta [Espagne] ; Alain Privat [France] ; Jacques Mallet [France]A Novel and Efficient Gene Transfer Strategy Reduces Glial Reactivity and Improves Neuronal Survival and Axonal Growth In Vitro
001E75 (1997) Alicia Bilang-Bleuel [France] ; Frédéric Revah [France] ; Phillippe Colin [France] ; Isabelle Locquet [France] ; Jean-Jacques Robert [France] ; Jacques Mallet [France] ; Philippe Horellou [France]Intrastriatal injection of an adenoviral vector expressing glial-cell-line-derived neurotrophic factor prevents dopaminergic neuron degeneration and behavioral impairment in a rat model of Parkinson disease

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